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1 ed anti-rubella virus immunoglobulin G (IgG) seroprotection.
2 dults aged >/= 80 years), among whom 80% had seroprotection.
3 TIV), with or without adjuvant, may increase seroprotection.
4 years, half of teens and adults showed H3N2v seroprotection.
5 lations of TIV do not substantially increase seroprotection.
6 A similar trend was identified for seroprotection.
7 with regard to an absolute titer indicating seroprotection.
8 ization opportunity window and could improve seroprotection.
9 d in a meta-analysis to provide estimates of seroprotection 2 and 5 years after the last vaccine admi
11 ificantly lower in PHIV children for measles seroprotection (57% [95% confidence interval {CI}, 52%-6
12 primary efficacy parameter was the degree of seroprotection 6 or 7 months (26 +/- 2 weeks) after begi
13 52%-62%] vs 99% [95% CI, 96%-100%]), rubella seroprotection (65% [95% CI, 60%-70%] vs 98% [95% CI, 95
15 -microg regimen elicited the highest rate of seroprotection (96.2%), with a geometric mean titer of a
17 a "complete response" (both seroresponse and seroprotection) after first vaccination was associated w
20 inst influenza A viruses (P < .001), greater seroprotection against influenza A/H1N1 (P = .01), and g
21 day 28 and percentages of seroconversion and seroprotection, all determined by haemagglutination inhi
22 Knowledge of the age-specific prevalence of seroprotection and incidence of seroconversion infection
23 Measles revaccination induced high rates of seroprotection and memory in children receiving HAART.
24 e reduction neutralization assay and rubella seroprotection and mumps seropositivity by enzyme immuno
26 S aureus colonization experienced (1) lower seroprotection and seroconversion rates and lower hemagg
33 red age-related cross-sectional estimates of seroprotection before the pandemic (during 2009) and aft
34 logic specimen was used to determine measles seroprotection by plaque reduction neutralization assay
35 n the 10(11) VP cohort (89%; 67-99) achieved seroprotection compared with four of 22 placebo recipien
37 ded in HIV-infected patients to estimate how seroprotection decreases over time in those who initiall
40 ith baseline titers had significantly higher seroprotection for the 2009-H1N1 strain (100% vs. 73%, r
42 Children in all groups had evidence of seroprotection (>10 mIU/mL) at 1 month after the second
44 aforementioned age groups, respectively, and seroprotection (HAI titers >/= 40) was shown in 79.6%, 8
45 in the proportion of participants achieving seroprotection (hemagglutination-inhibition antibody tit
47 ted persons reach higher levels of influenza seroprotection if vaccinated with the high-dose trivalen
51 Of those lacking seroresponse (n = 43) or seroprotection (n = 37) after the first vaccination, 46.
58 mune responses in subjects aged 18-64 years (seroprotection rate [SPR], 97.2%; seroconversion rate [S
59 the 98.3% confidence interval for the day 42 seroprotection rate was >/=70%, thus fulfilling the US a
60 lations, seroconversion rates were >/=85.7%, seroprotection rates >/=91.1%, and geometric mean titers
75 study vaccination was associated with higher seroprotection rates, greater antibody concentrations, a
77 on, 46.5% and 40.5% achieved seroresponse or seroprotection, respectively, after the second vaccinati
78 suggest that a two-dose regimen can achieve seroprotection similar to that of the three-dose regimen
80 eipt of 2 doses, 61.17% of subjects retained seroprotection titers at 24 months, and immunogenicity c
82 In 212 evaluable patients (105 IM, 107 ID), seroprotection to H1N1, H3N2 and B strains was 70.5%, 63
83 trieved from the literature, the decrease of seroprotection was modeled with a log binomial generaliz
85 Our analyses confirmed that the duration of seroprotection was shorter in HIV-infected patients and
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