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1 ocking properties of fluoxetine, a selective serotonin reuptake inhibitor.
2 lack of remission with an initial selective serotonin reuptake inhibitor.
3 mpus following administration of a selective serotonin reuptake inhibitor.
4 itor, the other from paroxetine, a selective serotonin-reuptake inhibitor.
5 Depressive patients medicated with Selective Serotonin Reuptake Inhibitors.
6 tracyclic antidepressants and norepinephrine-serotonin reuptake inhibitors.
7 itivity, and response to long-term selective serotonin reuptake inhibitors.
8 in patients that do not respond to selective serotonin reuptake inhibitors.
9 he absence of prenatal exposure to selective serotonin reuptake inhibitors.
10 ly active medications such as tricyclics and serotonin reuptake inhibitors.
11 tive to SERT inhibitors, including selective serotonin reuptake inhibitors.
12 ared with TCAs and strong compared with weak serotonin reuptake inhibitors.
13 disorder, GAD, or use of benzodiazepines or serotonin reuptake inhibitors.
14 a, often respond to treatment with selective serotonin reuptake inhibitors.
15 ame FDA "black box" warning as the selective serotonin reuptake inhibitors.
16 ars to lack drug interactions with selective serotonin reuptake inhibitors.
17 ting the antidepressant actions of selective serotonin reuptake inhibitors.
21 inhibitors, patients taking higher doses of serotonin reuptake inhibitors and who had a longer durat
22 cological treatments, particularly selective serotonin-reuptake inhibitors and serotonin-noradrenalin
23 iazepines (-0.96, -1.56 to -0.36), selective serotonin-reuptake inhibitors and serotonin-norepinephri
24 to empirical treatments for the disorder-ie, serotonin-reuptake inhibitors and various forms of cogni
25 lthy volunteers with citalopram (a selective serotonin reuptake inhibitor) and contrasted its effects
26 cts differential response to escitalopram (a serotonin reuptake inhibitor) and nortriptyline (a norep
27 benzodiazapines, and withdrawal of selective serotonin-reuptake inhibitor) and received cyproheptadin
28 erational effects of sertraline, a selective serotonin reuptake inhibitor, and venlafaxine, a seroton
29 cluding tricyclic antidepressants, selective serotonin reuptake inhibitors, and norepinephrine reupta
30 as atypical antipsychotic agents, selective serotonin reuptake inhibitors, and selective serotonin-n
31 -related issues: suicide risk with selective serotonin reuptake inhibitors, and the safety of antidep
32 and that the neurogenic effects of selective serotonin reuptake inhibitor antidepressants can success
39 ce continues to support the use of selective serotonin reuptake inhibitors as first-line pharmacologi
40 vidence supporting the efficacy of selective serotonin reuptake inhibitors, augmentation strategies a
41 hift in prescribing toward the non-selective serotonin reuptake inhibitor bupropion hydrochloride, ev
42 ASD associated with treatment with selective serotonin reuptake inhibitors by the mother during the y
43 year risk of MOF by 36% for use of selective serotonin reuptake inhibitors, by 63% for use of mood st
44 of hip fracture by 57% for use of selective serotonin reuptake inhibitors, by 98% for use of mood st
45 double-blind design to receive the selective serotonin reuptake inhibitor citalopram (20 mg) or place
47 We therefore examined whether the selective serotonin reuptake inhibitor citalopram improves respons
50 agonists (BI-11A7 and BI-2A7); the selective serotonin reuptake inhibitor citalopram was used as a po
52 hallenge with an acute dose of the selective serotonin reuptake inhibitor, citalopram, revealed a gen
55 pre-HAART or HAART era (P = .23), and use of serotonin reuptake inhibitors did not alter the risk of
56 ether fluoxetine, the prototypical selective serotonin reuptake inhibitor, differentially modulates n
57 entin and pregabalin) and the norepinephrine/serotonin reuptake inhibitors (duloxetine and milnacipra
59 c deletion or brief treatment with selective serotonin reuptake inhibitors during development, have a
61 of exposure to fluoxetine (FLX), a selective serotonin reuptake inhibitor, during adolescence on beha
62 e of antidepressants, specifically selective serotonin reuptake inhibitors, during the second and/or
63 get for antidepressants, including selective serotonin reuptake inhibitors (e.g. (S)-citalopram) and
66 with subsequent remission with the selective serotonin reuptake inhibitor escitalopram; this is consi
67 8 weeks after randomization to the selective serotonin reuptake inhibitors escitalopram and sertralin
68 ponded significantly better to the selective serotonin reuptake inhibitors escitalopram and sertralin
69 oma were randomized to either placebo or the serotonin reuptake inhibitor, escitalopram (ESC) 10-20 m
70 ether a 3-week administration of a selective serotonin reuptake inhibitor, expected to increase brain
71 the antidepressant efficacy of the selective-serotonin reuptake inhibitor fluoxetine (FLX) in a chron
78 ort and binding of cocaine and the selective serotonin reuptake inhibitors fluoxetine and escitalopra
79 droxyphenylserine at 5 mg/kg) or a selective serotonin reuptake inhibitor (fluoxetine at 20 mg/kg) at
80 ally exposed to acute doses of the selective serotonin reuptake inhibitor, fluoxetine (5 or 25 mg/L),
82 sterone blocked the ability of the selective serotonin reuptake inhibitor, fluvoxamine, to inhibit se
83 gether with the demonstrated efficacy of the serotonin reuptake inhibitors for childhood-onset obsess
84 egan demonstrating the efficacy of selective serotonin reuptake inhibitors for these conditions in th
85 -current stimulation (tDCS) with a selective serotonin-reuptake inhibitor for the treatment of depres
86 -CCKR-2 tg mice with fluoxetine, a selective serotonin reuptake inhibitor, for a period of 4 wk signi
90 h cognitive behavioral therapy and selective serotonin-reuptake inhibitors have shown efficacy in tre
91 cortex are powerfully modulated by selective serotonin reuptake inhibitors, however, direct measureme
93 er a 12-week treatment course with selective serotonin reuptake inhibitors in late-life depression.
94 f cognitive behavioral therapy and selective serotonin reuptake inhibitors in the treatment of pediat
95 The efficacy and adverse events of selective serotonin reuptake inhibitors in these patients are unkn
96 Long-term efficacy and safety of selective serotonin reuptake inhibitors in these patients are unkn
97 of children exposed prenatally to selective serotonin reuptake inhibitors in this population was low
99 iological effects of fluoxetine, a selective serotonin reuptake inhibitor, in filter and deposit feed
105 substances, such as increasingly prescribed serotonin reuptake inhibitors, may produce an independen
106 rols); unmedicated-MDD; and MDD treated with serotonin reuptake inhibitors (MDD*SSRI) or tricyclics (
107 treated, n = 12), MDD treated with selective serotonin reuptake inhibitors (MDD*SSRI, n = 6) or tricy
108 e-enhanced behavior therapy) and medication (serotonin reuptake inhibitors, N-acetylcysteine, or nalt
109 second-generation antidepressants (selective serotonin reuptake inhibitors, nefazodone, venlafaxine,
111 ession classified as responsive to selective serotonin reuptake inhibitors (odds ratio=1.55, 95% CI=1
112 s into the effects of OLZ, but not selective serotonin reuptake inhibitors, on core features of AN.
113 antidepressant medication therapy (selective serotonin reuptake inhibitor or serotonin and norepineph
114 ndomized to receive either another selective serotonin reuptake inhibitor or venlafaxine, with or wit
115 0-week open-label treatment with a selective serotonin reuptake inhibitor or, in some cases, another
116 setron) either in combination with selective serotonin reuptake inhibitors or as monotherapy in the t
117 treatment of depression by use of selective serotonin reuptake inhibitors or psychological approache
118 lly untreated maternal depression, selective serotonin reuptake inhibitors or serotonin and norepinep
119 wnregulated by the acute action of selective serotonin reuptake inhibitors or serotonin at the site o
120 preterm birth are high for women who used a serotonin reuptake inhibitor (OR, 1.55 [95% CI, 1.02-2.3
121 rval [CI] = 1.10-15.14) and use of selective serotonin reuptake inhibitors (OR = 2.66; 95% CI = 1.01-
122 edicted response to antidepressants overall, serotonin reuptake inhibitors, or noradrenaline reuptake
123 ncludes tricyclic antidepressants, selective serotonin reuptake inhibitors, or psychosocial intervent
127 Compared with patients who were not taking serotonin reuptake inhibitors, patients taking higher do
128 primary care settings, the effectiveness of serotonin reuptake inhibitor prescription by pediatric p
129 Although current antidepressants, such as serotonin-reuptake inhibitors, produce subtle changes th
131 reatment of Hoxb8 mutants with fluoxetine, a serotonin reuptake inhibitor, reduces excessive grooming
133 behavioral therapy with or without selective serotonin reuptake inhibitors remains a preferred initia
135 in women with MDD or anxiety, with selective serotonin reuptake inhibitors reported as the most commo
136 f classic antidepressants, such as selective serotonin reuptake inhibitors, require a month or longer
137 e itch triggered by serotonin or a selective serotonin reuptake inhibitor required both HTR7 and TRPA
138 nt exposure to fluoxetine (FLX), a selective serotonin reuptake inhibitor, results in changes to the
139 case report suggested benefit from selective serotonin reuptake inhibitor/serotonin-norepinephrine re
140 excess morbidity in critically ill selective serotonin reuptake inhibitor/serotonin-norepinephrine re
141 as generally unclear if outpatient selective serotonin reuptake inhibitor/serotonin-norepinephrine re
143 dies of ICU patients with recorded selective serotonin reuptake inhibitor/serotonin-norepinephrine re
144 exposed and control groups; use of selective serotonin reuptake inhibitor/serotonin-norepinephrine re
145 help clinicians decide when to use selective serotonin reuptake inhibitor/serotonin-norepinephrine re
146 antidepressant treatment with the selective serotonin reuptake inhibitor sertraline hydrochloride.
153 der (OCD) indicates that partial response to serotonin reuptake inhibitors (SRIs) is the norm and tha
158 ndicated that the combination of a selective serotonin reuptake inhibitor (SSRI) and a statin may hav
159 ollowing administration of several selective serotonin reuptake inhibitor (SSRI) antidepressant drugs
160 splays distinct sensitivity to the selective serotonin reuptake inhibitor (SSRI) antidepressant fluox
162 ychotic medications; fluoxetine, a selective serotonin reuptake inhibitor (SSRI) antidepressant; and
163 sant medication or specifically to selective serotonin reuptake inhibitor (SSRI) antidepressants, all
164 , either untreated or treated with selective serotonin reuptake inhibitor (SSRI) antidepressants, and
166 ian subjects were administered the selective serotonin reuptake inhibitor (SSRI) citalopram (10 mg) i
168 his study investigated whether the selective serotonin reuptake inhibitor (SSRI) citalopram downregul
169 (TCA) desipramine (10 mg/kg), the selective serotonin reuptake inhibitor (SSRI) fluoxetine (10 mg/kg
171 e early anxiogenic response to the selective serotonin reuptake inhibitor (SSRI) fluoxetine and had i
172 onstrated that chronic administration of the serotonin reuptake inhibitor (SSRI) fluoxetine augments
173 hat 6 weeks' administration of the selective serotonin reuptake inhibitor (SSRI) fluoxetine would amp
175 aled that chronic treatment with a selective serotonin reuptake inhibitor (SSRI) impairs the acquisit
176 A subset of patients started on a selective serotonin reuptake inhibitor (SSRI) initially experience
177 cally supported psychotherapy with selective serotonin reuptake inhibitor (SSRI) pharmacotherapy as t
178 or to beginning treatment with the selective serotonin reuptake inhibitor (SSRI) sertraline or cognit
181 was synthesized by linking a novel selective serotonin reuptake inhibitor (SSRI) to a PDE4 inhibitor.
182 the relationship between prenatal selective serotonin reuptake inhibitor (SSRI) treatment and pregna
186 ERT) were synthesized based on the selective serotonin reuptake inhibitor (SSRI), (S)-citalopram (1).
187 fects of duloxetine hydrochloride, selective serotonin reuptake inhibitor (SSRI), and covariates on t
189 In the presence of fluvoxamine, a selective serotonin reuptake inhibitor (SSRI), concentrations of 5
191 ted the effects of escitalopram, a selective serotonin reuptake inhibitor (SSRI), on brain activation
192 es demonstrated that fluoxetine, a selective serotonin reuptake inhibitor (SSRI), provided the most s
193 dolescents (ages 12-18 years) with selective serotonin reuptake inhibitor (SSRI)-resistant depression
198 we show that when the effects of a selective serotonin reuptake inhibitor (SSRI, citalopram) are stud
202 rated that acute administration of selective serotonin reuptake inhibitors (SSRI) can affect P-gp fun
203 studied the effect of fluoxetine (a specific serotonin reuptake inhibitor--SSRI) on active avoidance
204 sms explaining hormetic effects of selective serotonin reuptake inhibitors (SSRIs) and 4-nonylphenol
205 strongest evidence supports use of selective serotonin reuptake inhibitors (SSRIs) and cognitive-beha
206 ors, both of which are reversed by selective serotonin reuptake inhibitors (SSRIs) and the tricyclic
224 an association between the use of selective serotonin reuptake inhibitors (SSRIs) during pregnancy a
226 It is unknown whether exposure to selective serotonin reuptake inhibitors (SSRIs) during pregnancy i
227 ongoing controversy as to whether selective serotonin reuptake inhibitors (SSRIs) exhibit the same a
228 ality in pediatric patients taking selective serotonin reuptake inhibitors (SSRIs) for depression.
233 dulation of synaptic plasticity by selective serotonin reuptake inhibitors (SSRIs) in hippocampal bra
234 ted that the treatment response to selective serotonin reuptake inhibitors (SSRIs) in major depressiv
235 blockers potentiate the effects of selective serotonin reuptake inhibitors (SSRIs) in some treatment-
236 gated whether in utero exposure to selective serotonin reuptake inhibitors (SSRIs) increases the risk
237 ating serotonin (5-HT) levels with selective serotonin reuptake inhibitors (SSRIs) is the most widely
239 g animals with MAO-A inhibitors or selective serotonin reuptake inhibitors (SSRIs) normalized anxiety
240 sess the effects of treatment with selective serotonin reuptake inhibitors (SSRIs) on the risks of ge
241 SM) advised against treatment with selective serotonin reuptake inhibitors (SSRIs) other than fluoxet
244 D, and clinical data indicate that selective serotonin reuptake inhibitors (SSRIs) such as fluoxetine
245 ion accounting for unsatisfactory effects of serotonin reuptake inhibitors (SSRIs) such as insufficie
246 utomer, escitalopram (S-(+)-1) are selective serotonin reuptake inhibitors (SSRIs) that are used clin
247 m concomitant use of nsNSAIDs with selective serotonin reuptake inhibitors (SSRIs) was 1.6, whereas t
248 udies linking in utero exposure to selective serotonin reuptake inhibitors (SSRIs) with persistent pu
249 orepinephrine reuptake inhibitor), selective serotonin reuptake inhibitors (SSRIs), and maternal depr
250 respond to initial treatment with selective serotonin reuptake inhibitors (SSRIs), and this is assoc
251 rinatal exposure to drugs, such as selective serotonin reuptake inhibitors (SSRIs), impacts cortical
253 compared for patients who received selective serotonin reuptake inhibitors (SSRIs), new-generation no
254 ge of therapeutic agents including selective serotonin reuptake inhibitors (SSRIs), serotonin-noradre
255 he relative efficacy and safety of selective serotonin reuptake inhibitors (SSRIs), serotonin-norepin
257 ich dampen limbic irritability, or selective serotonin reuptake inhibitors (SSRIs), which may enhance
258 of fluoxetine and paroxetine, two selective serotonin reuptake inhibitors (SSRIs), with the human (h
270 ation and is commonly treated with selective serotonin reuptake inhibitors (SSRIs; e.g., Prozac).
272 eductase inhibitors (statins), and selective serotonin-reuptake inhibitors (SSRIs) and serotonin-nore
274 efects after antenatal exposure to selective serotonin-reuptake inhibitors (SSRIs) remains controvers
275 nuates the therapeutic activity of selective serotonin-reuptake inhibitors (SSRIs), whereas their fun
277 ine to comparators (venlafaxine or selective serotonin reuptake inhibitors [SSRIs], and individuals w
278 d declines in case finding and non-selective serotonin reuptake inhibitor substitute treatment were s
279 ith subchronic administration of a selective serotonin reuptake inhibitor, suggesting the predictive
281 oral changes were rescued by acute selective serotonin reuptake inhibitor treatment, supporting the h
286 iazepine hypnotics, beta-blockers, selective serotonin reuptake inhibitors, tricyclic antidepressants
287 to whether patients were receiving selective serotonin reuptake inhibitors, tricyclic antidepressants
288 ute odds ratios for differences in selective serotonin reuptake inhibitor use between cases and contr
290 and adolescents, the risk benefit ratio for serotonin reuptake inhibitor use in pediatric anxiety di
294 ion was shortened by 3.6 days; with maternal serotonin reuptake inhibitor use, gestation was shortene
296 of 6024 residents, 1024 (17%) were selective serotonin reuptake inhibitor users compared to 788 (13%)
297 For SERT analysis, patients on selective serotonin reuptake inhibitor were excluded (n = 48 remai
298 s who were partial responders to a selective serotonin reuptake inhibitor were randomized to receive
300 used to examine the effect of treatment with serotonin reuptake inhibitors, with or without antipsych
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