ライフサイエンスコーパス: serum amyloid A

1 ein, 25% for C-reactive protein, and 32% for serum amyloid A).

2 f IgM, IgE, IgG2b, IgG3, anti-dsDNA Abs, and serum amyloid A.

3 is a receptor for the amyloidogenic form of serum amyloid A.

4 um-derived factor, surfactant protein B, and serum amyloid A.

5 ivity was monitored by serial measurement of serum amyloid A.

6 s the activating and proinflammatory protein serum amyloid A.

7 rleukin-1beta, interleukin-6, fibrinogen, or serum amyloid A.

8 roteins such as C-reactive protein (CRP) and serum amyloid A].

9 We now show that systemic serum amyloid A 1 (SAA-1) controls the plasticity of neu

10 lated the IL-1beta-induced expression of the serum amyloid A 1 (SAA1) and SAA2 genes.

11 RNA inhibited cytokine induction of the APP serum amyloid A-1, demonstrating that both transcription

12 ple assayed for C-reactive protein (CRP) and serum amyloid A (30 months after diagnosis) and complete

13 The human acute phase serum amyloid A (A-SAA) genes, SAA1 and SAA2, have a hig

14 The acute-phase protein serum amyloid A (A-SAA) was significantly increased by 2

15 Serum amyloid A (A-SAA/Saa3) was shown before to affect

16 e to the generation of lipid-poor apoA-I and serum amyloid A acceptors for cholesterol efflux.

17 ter with a zinc finger transcription factor, serum amyloid A activating factor (SAF)-1, was demonstra

18 l that overexpresses a transcription factor, serum amyloid A activating factor-1 (SAF-1), leading to

19 t promoter constructs of MMP-9, we show that serum amyloid A-activating factor (SAF)-1, a novel trans

20 The role of serum amyloid A-activating factor 1 (SAF-1) in MMP-1 exp

21 nflammation-responsive transcription factor, serum amyloid A-activating factor 1 (SAF-1), has been sh

22 The transcription factor serum amyloid A-activating factor-1 (SAF-1) has been ide

23 Serum amyloid A-activating factor-1 (SAF-1) is a zinc fi

24 report, IL-6 failed to induce activation of serum amyloid A-activating factor-1/c-Myc-associated zin

25 Serum amyloid A-activating transcription factor-1 (SAF-1

26 tokine-mediated transcriptional induction of serum amyloid A, an acute-phase plasma protein that is a

27 Serum amyloid A: an ozone-induced circulating factor wit

28 motactic peptide fMet-Leu-Phe, lipoxin A(4), serum amyloid A and beta-amyloid peptides.

29 ation and acute phase proteins, particularly serum amyloid A and group IIa secretory phospholipase A2

30 LVS-infected IL-6 KO mice produced much less serum amyloid A and haptoglobin (two acute-phase protein

31 culating levels of the acute phase proteins, serum amyloid A and IL-6, and the neutrophil-selective C

32 therosclerosis, serum levels of CD40 ligand, serum amyloid A and monocyte chemoattractant protein-1,

33 inflammation markers C-reactive protein and serum amyloid A and quality-of-life scores were signific

34 model for the acute phase response in which serum amyloid A and sPLA2-IIa, present at sites of infla

35 d a decrease in serum C-reactive protein and serum amyloid-A and an increase in serum retinol-binding

36 inflammation markers (C-reactive protein and serum amyloid A), and quality-of-life assessments (Derma

37 itive C-reactive protein, interleukin 6, HDL serum amyloid A, and adiponectin concentrations were mea

38 oid-beta(1-40), alpha-synuclein, ABri, ADan, serum amyloid A, and amylin undergo supramolecular confo

39 is-suppressing signals from myeloperoxidase, serum amyloid A, and bacterial DNA, shifting the balance

40 ave high circulating concentrations of IL-6, serum amyloid A, and C-reactive protein, each of which d

41 ol was required for elevation of circulating serum amyloid A, and cholate was required for accumulati

42 s apolipoprotein J, fibrinogen, haptoglobin, serum amyloid A, and complement factors (B, C3, and C9).

43 ric oxide, oxidized low-density lipoprotein, serum amyloid A, and lipid peroxidation, were significan

44 igh levels of apolipoproteins A-II and B and serum amyloid A, and low levels of haptoglobin dimers an

45 estingly, some of the serum proteins such as Serum amyloid A, Apolipoprotein A1, C-reactive protein,

46 r, C-reactive protein (CRP), fibrinogen, and serum amyloid A are associated independently with functi

47 ficant elevation of transcripts for the APPs serum amyloid A, complement C3, pentraxin 3, and alpha2-

48 Amyloid A deposits regress upon reduction of serum amyloid A concentration, indicating that the amylo

49 reased apoA-I content and markedly increased serum amyloid A content in HDL during the acute phase re

50 c lipase, phospholipid transfer protein, and serum amyloid A) could decrease the ability of HDL to pr

51 These effects were associated with reduced serum amyloid A expression in ileum and synovial tissue.

52 e inflammation, including three genes of the serum amyloid A family, three major histocompatibility c

53 o 2.5 +/- 0.5 mg/L (P < 0.01), decreased HDL serum amyloid A from 10.3 +/- 1.8 to 5.7 +/- 1.3 mg/L (P

54 Diary scores improved (P<0.001) and serum amyloid A (from a median of 174 mg to 8 mg per lit

55 ha1-antichymotrypsin, C-reactive protein, or serum amyloid A) from 15 studies of apparently healthy i

56 Serum amyloid A functions efficiently in a lipid-free or

57 n shown to regulate several genes, including serum amyloid A, gamma-fibrinogen, and matrix metallopro

58 t is characterized by enhanced expression of serum amyloid A, haptoglobin and tissue inhibitor for me

59 les were enriched with acute-phase proteins (serum amyloid A, haptoglobin, and hemopexin) and deplete

60 acute-phase proteins (C-reactive protein and serum amyloid A), however, has been found to be associat

61 Blood C-reactive protein, serum amyloid A, IL-6, IL-1ra, G-CSF, but not TNF-alpha

62 C-reactive protein, alpha-1-antitrypsin, and serum amyloid A), immune response (high IgA), leakage of

63 onal to that of either C-reactive protein or serum amyloid A in both HD and PD patients.

64 Serum amyloid A increases the ability of acute phase HDL

65 igh-sensitivity C-reactive protein (hs-CRP), serum amyloid A, interleukin-6, and soluble intercellula

66 Serum amyloid A is an acute phase protein that is carrie

67 as no significant difference in steady-state serum amyloid A level in the serum of aged non-Tg and Fa

68 ad increased IL-1beta, IL-6, IL-23, C3a, and serum amyloid A levels in BAL fluid, and these correlate

69 Serum amyloid A levels were significantly lower in ApoE(

70 and matrix metalloproteinase-9, and systemic serum amyloid A levels.

71 ammation as assessed by circulating IL-6 and serum amyloid A levels.

72 oles of high-sensitivity C-reactive protein, serum amyloid-A, lipoprotein(a), and homocysteine were a

73 lteration in the induction of APP, including serum amyloid A, LPS-binding protein, fibrinogen, or cer

74 The displacement of paraoxonase by serum amyloid A may explain in part the proinflammatory

75 Serum amyloid A measurement and a rapid cTnT assay were

76 ding amyloid-beta, tau, alpha-synuclein, and serum amyloid A, misfold into distinct conformers linked

77 , tyrosine phosphorylation, and IL-6-induced serum amyloid A mRNA expression.

78 ke Alzheimer beta-amyloid peptide (Abeta) or serum amyloid A, must undergo significant structural tra

79 sedimentation rates, and C-reactive protein, serum amyloid A, myeloid-related protein 8/14, and S100A

80 By comparison, serum amyloid A non-genomic responses were reliant on ex

81 lted in significantly higher serum levels of serum amyloid A on day 2 and IL-6 on days 1 and 2 and a

82 Peak interleukin-6 and serum amyloid A plasma levels were observed at 2 and 7 d

83 In mice, it induces serum amyloid A, potentiates the induction by IL-1 of co

84 etin, p53, superoxide dismutase 1, lysozyme, serum amyloid A, prions, vasopressin receptor 2, and alp

85 n in mice, while not affecting IL-22-induced serum amyloid A production or EPO-induced reticulocytosi

86 alamus-pituitary-adrenal axis, hypoglycemia, serum amyloid A production, and anorexia.

87 journal to induce an acute phase response of serum amyloid A protein (SAA) and of CRP itself, and to

88 that develops when proteolytic fragments of serum amyloid A protein (SAA) are deposited in tissues a

89 inflammatory protein 1beta (MIP-1beta), and serum amyloid A protein (SAA) during acute SIVmac251 inf

90 Serum amyloid A protein (SAA) is an acute-phase reactant

91 ed from the circulating acute-phase reactant serum amyloid A protein (SAA), but the relation between

92 is of levels of C-reactive protein (CRP) and serum amyloid A protein (SAA).

93 From these peaks, two peptides (serum amyloid A protein and transthyretin) were identifi

94 ry assessments, including measurement of the serum amyloid A protein concentration.

95 -1 activity was measured by the induction of serum amyloid A protein in cultured chondrocytes.

96 ation, including immunoglobulin light chain, serum amyloid A protein, and transthyretin.

97 cute-phase reactants, C-reactive protein and serum amyloid A protein, were measured by immunonephelom

98 egates derived from the acute-phase reactant serum amyloid A protein.

99 Acute phase serum amyloid A proteins (A-SAAs) are multifunctional ap

100 rect contact with the epithelium and induces serum amyloid A proteins 1 and 2 (SAA1/2), which promote

101 During inflammation, serum amyloid A proteins transport retinol to infected t

102 colleagues shows that hepatic and intestinal serum amyloid A proteins, which are induced in response

103 t congophilic fibrillar material composed of serum amyloid A-related protein that acted as a potent A

104 with the risk of cardiovascular events were serum amyloid A (relative risk for the highest as compar

105 The fibrillation of Serum Amyloid A (SAA) - a major acute phase protein - is

106 Serum amyloid A (SAA) 1 and 2 are produced predominantly

107 found that the acute-phase response proteins serum amyloid A (SAA) 1 and SAA3 are transcriptional tar

108 -terminal peptide of the acute phase protein serum amyloid A (SAA) 1.

109 Serum amyloid A (SAA) activating factor-1 (SAF-1) is an

110 Inflammatory markers serum amyloid A (SAA) and C-reactive protein (CRP) are p

111 Therefore, serum amyloid A (SAA) and C-reactive protein (CRP) were

112 lpha), circulatory C-reactive protein (CRP), serum amyloid A (SAA) and haptoglobin (Hpt) were analyse

113 Systemic C-reactive protein (CRP) and serum amyloid A (SAA) are measures of low-grade chronic

114 the inflammation-associated genes Fizz1 and serum amyloid A (SAA) are significantly up-regulated in

115 Here, we identify serum amyloid A (SAA) as a candidate mediator of GC refr

116 h reduced expression of TNF-alpha, IL-6, and serum amyloid A (SAA) at all time points compared with l

117 ly interleukin 6, leads to overproduction of serum amyloid A (SAA) by the liver.

118 Since serum amyloid A (SAA) concentrations in HDL increase wit

119 Because AngII increases hepatic serum amyloid A (SAA) expression in an IL-6-dependent ma

120 The serum amyloid A (SAA) family comprises a number of diffe

121 Serum amyloid A (SAA) has been linked to atherosclerosis

122 The fibrillar deposition of serum amyloid A (SAA) has been linked to the disease amy

123 The acute phase protein serum amyloid A (SAA) has been well characterized as an

124 ta demonstrating the multifunctional role of serum amyloid A (SAA) in the pathogenesis of amyloidosis

125 Serum amyloid A (SAA) is a family of acute-phase protein

126 Serum amyloid A (SAA) is a highly conserved acute phase

127 Induced secretion of acute-phase serum amyloid A (SAA) is a host response to danger signa

128 Serum amyloid A (SAA) is a major acute phase protein inv

129 Serum amyloid A (SAA) is a major acute-phase protein syn

130 Serum amyloid A (SAA) is a plasma protein that is associ

131 Serum amyloid A (SAA) is a plasma protein which has been

132 iously reported that the acute phase protein serum amyloid A (SAA) is a potent chemoattractant for hu

133 Serum amyloid A (SAA) is a small apolipoprotein that bin

134 Serum amyloid A (SAA) is an acute phase protein whose ex

135 Serum amyloid A (SAA) is an acute-phase plasma protein t

136 Serum amyloid A (SAA) is an acute-phase protein induced

137 Serum amyloid A (SAA) is an apolipoprotein primarily pro

138 Serum amyloid A (SAA) is best known for being the main c

139 The acute-phase protein serum amyloid A (SAA) is commonly considered a marker fo

140 Serum amyloid A (SAA) is expressed locally in chronic in

141 The acute-phase protein serum amyloid A (SAA) is highly induced during inflammat

142 Serum amyloid A (SAA) is one such marker but its functio

143 Elevated serum amyloid A (SAA) levels are associated with increas

144 ignificantly reduced histological damage and serum amyloid A (SAA) levels in IL-10(-/-) colitis mice,

145 used to compare C-reactive protein (CRP) and serum amyloid A (SAA) levels in prerandomization blood s

146 Serum amyloid A (SAA) promotes neutrophilic inflammation

147 kines to increase the synthesis of precursor serum amyloid A (SAA) protein and the transitory nature

148 The serum amyloid A (SAA) protein has been implicated in the

149 The serum amyloid A (SAA) protein has been implicated in the

150 Serum amyloid A (SAA) protein has recently been linked t

151 Abundantly expressed serum amyloid A (SAA) protein under chronic inflammatory

152 Serum amyloid A (SAA) proteins are a family of inflammat

153 The serum amyloid A (SAA) proteins are a polymorphic family

154 Serum amyloid A (SAA) proteins are strongly induced in t

155 Serum amyloid A (SAA) proteins were originally identifie

156 Serum amyloid A (SAA) represents an evolutionarily conse

157 Serum amyloid A (SAA) upregulation was subsequently conf

158 C-reactive protein (CRP) and serum amyloid A (SAA) were measured by latex-enhanced ne

159 Neither the normal role of serum amyloid A (SAA), a major acute phase response prot

160 Serum amyloid A (SAA), a plasma protein inducible in res

161 We evaluated the ability of serum amyloid A (SAA), alone and in combination with a r

162 ctant protein B (SP-B), apolipoprotein C-II, serum amyloid A (SAA), and alpha-1-microglobulin/bikunin

163 We measured C-reactive protein (CRP), serum amyloid A (SAA), and interleukin 6 (IL-6) in 2402

164 C-reactive protein (CRP), serum amyloid A (SAA), and lipoprotein levels were compa

165 markers including C-reactive protein (CRP), serum amyloid A (SAA), and S100 calcium-binding protein

166 acute-phase proteins, C-reactive protein and serum amyloid A (SAA), are biomarkers of infection and i

167 pocyte-derived factors, e.g., hyaluronan and serum amyloid A (SAA), can facilitate monocyte adhesion

168 ior day stressors, C-reactive protein (CRP), serum amyloid A (SAA), intercellular adhesion molecule-1

169 igh-sensitivity C-reactive protein (hs-CRP), serum amyloid A (SAA), interleukin-6 (IL-6), leukocyte,

170 Concentrations of HDL-bound serum amyloid A (SAA), lipopolysaccharide binding protei

171 ucose-stimulated production by adipocytes of serum amyloid A (SAA), monocyte chemoattractant protein

172 f high-sensitivity C-reactive protein (CRP), serum amyloid A (SAA), plasminogen activator inhibitor-1

173 SFB cause an increase in serum amyloid A (SAA), suggesting that SAA might mediate

174 e expression of C-reactive protein (CRP) and serum amyloid A (SAA), the prototype acute-phase respons

175 The amino-terminal region of serum amyloid A (SAA), the subunit precursor protein in

176 The transcription factor serum amyloid A (SAA)-activating factor (SAF), a family

177 in the blood of acute-phase proteins such as serum amyloid A (SAA).

178 histological injury score (HIS), and plasma serum amyloid A (SAA).

179 temic deposition of the acute-phase reactant serum amyloid A (SAA).

180 increased levels of the acute-phase protein, serum amyloid A (SAA).

181 daily activities, and C-reactive protein and serum amyloid A [SAA] levels).

182 high-sensitivity C-reactive protein [hsCRP], serum amyloid A [SAA], and IL-6) were determined at 3, 6

183 -6], tumor necrosis factor alpha [TNFalpha], serum amyloid A [SAA], vascular endothelial growth facto

184 served significantly increased expression of serum amyloid A (Saa3) and serine protease inhibitor 3n

185 ncluding induction of fibrinogen, CXCL1, and serum amyloid A that likely contribute to the reported c

186 all OxLDL biomarkers and C-reactive protein, serum amyloid A, tissue plasminogen activator, interleuk

187 ced IFN-gamma production and IL-22-dependent serum amyloid A to similar extents, indicating that, in

188 y was found in the case of those formed from serum amyloid A, transthyretin, and islet amyloid polype

189 amyloidogenic precursor proteins, including serum amyloid A, transthyretin, islet amyloid polypeptid

190 inflammation, including C-reactive protein, serum amyloid A, tumor necrosis factor-alpha, and IL-6.

191 Serum amyloid A was higher in patients who died compared

192 r levels of D-dimer, C-reactive protein, and serum amyloid A were associated with higher all-cause mo

193 The levels of hs-CRP and serum amyloid A were significant predictors of risk even

194 ines studied induced the acute-phase protein serum amyloid A when administered alone.