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1 ster rate of decline of serum hematocrit and serum bicarbonate.
2 ce [CrCl] 15 to 30 ml/min per 1.73 m(2)) and serum bicarbonate 16 to 20 mmol/L to either supplementat
3 mbrane depolarization correlated with raised serum bicarbonate and base excess, indicating compensate
10 albuminuria, serum calcium, serum phosphate, serum bicarbonate, and serum albumin (C statistic, 0.917
11 2 mm Hg; P = .25), although daily changes of serum bicarbonate (between-group difference, -0.8 mEq/L;
13 of the patients initially had acidosis (mean serum bicarbonate concentration, 12.9 mmol per liter).
15 higher prevalence of vasopressor use, lower serum bicarbonate concentrations, and a higher prevalenc
16 tinoculation percent weight change per h and serum bicarbonate concentrations, the virulence of the S
17 entilation in multivariate analysis included serum bicarbonates less than 20 mM (odds ratio, 4.9 [95%
18 ted multinomial logistic regression model, a serum bicarbonate level less than 10 mEq/L (compared wit
19 pH was greater than 7.45, and .4 days where serum bicarbonate level was greater than 28 mmol/L, duri
20 ssociated with the number of days with a low serum bicarbonate level, but was not associated with inc
23 4.5; 95% confidence interval [CI], 2.7-7.6), serum bicarbonate of <20 mmol/L (OR, 2.9; 95% CI, 1.6-5.
25 aps earlier acid-base indicator of risk than serum bicarbonate, particularly in patients without acid
27 imens at any point over the study period for serum bicarbonate, serum potassium, or urine chloride en
28 asing proportion of days with elevated pH or serum bicarbonate was not associated with increased mort
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