ライフサイエンスコーパス: serum calcium

1 centrifuge sediment increased along with the serum calcium.

2 e group had at least one elevated measure of serum calcium.

3 us, low hemoglobin level, and high corrected serum calcium.

4 ers are responsible for abnormalities in the serum calcium.

5 Henle to sense and respond to alterations in serum calcium.

6 TH play a critical role in the regulation of serum calcium.

7 GS was required because of her low levels of serum calcium.

8 tification factors haemoglobin and corrected serum calcium.

9 ns occur in response to prolonged changes in serum calcium.

10 analysis of the ePTH patients revealed that serum calcium 1-week after surgery was predictive of rec

11 changes in intestinal calcium absorption and serum calcium, 1alpha,25-dihydroxyvitamin D also repress

12 longitudinal study of the effects of GBP on serum calcium, 25-hydroxy-vitamin-D (vitamin D), and par

13 Serum calcium and 1,25-dihydroxyvitamin D(3) levels were

14 administration was associated with increased serum calcium and a lack of increase in body weight in a

15 calcitriol (12.5 microg/d) which normalized serum calcium and improved his rickets.

16 his was accompanied by significantly reduced serum calcium and increased PTH levels in patients with

17 pic and phosphaturic hormones, and urine and serum calcium and inorganic phosphorus in mice in which

18 As anticipated, parathyroidectomy reduced serum calcium and intact parathyroid hormone levels.

19 Higher serum calcium and lower PTH levels were demonstrated in

20 mice exhibited hypercalciuria and had lower serum calcium and markedly increased serum PTH levels.

21 -heritability (rg = 0.191, P = 0.03) between serum calcium and migraine headache, indicating that the

22 High levels of serum calcium and OPG are significantly associated with

23 ive factors for hypocalcemia, measurement of serum calcium and parathyroid hormone (PTH) levels, and

24 Sost KO and WT mice had similar serum calcium and parathyroid hormone concentrations.

25 r can cause an abnormal relationship between serum calcium and parathyroid hormone response, as is ty

26 ther markers of mineral metabolism including serum calcium and phosphate showed no significant associ

27 nd meat protein intakes, height, weight, and serum calcium and phosphorus concentrations all independ

28 condary hyperparathyroidism by correction of serum calcium and phosphorus concentrations and the admi

29 no significant between-group differences in serum calcium and phosphorus concentrations or in urine

30 sphocalcic product (the product of the total serum calcium and phosphorus concentrations).

31 with the patients' clinical characteristics, serum calcium and phosphorus concentrations, and other b

32 Serum calcium and phosphorus levels were not statistical

33 nalogue, appears to lessen the elevations in serum calcium and phosphorus levels, as compared with ca

34 ndex (LVMI), serum C-reactive protein (CRP), serum calcium and phosphorus, and erythropoietin resista

35 mean ratio of urinary calcium to creatinine, serum calcium and phosphorus, or change in iron status (

36 t parathyroid hormone and elevated levels of serum calcium and phosphorus, situations in which inject

37 abolism, particularly high concentrations of serum calcium and phosphorus, were associated with incre

38 the low potency of this analog in increasing serum calcium and phosphorus.

39 in darker-skinned children), and, inversely, serum calcium and phosphorus.

40 calcet for achieving long-term reductions in serum calcium and PTH concentrations in primary hyperpar

41 idectomy results in greater normalization of serum calcium and PTH levels and significantly improves

42 hyroid tumour development, and elevations in serum calcium and PTH, were similar in males and females

43 notably, a shift in the relationship between serum calcium and PTH.

44 ice exhibit a much wider range of values for serum calcium and renal excretion of calcium than we obs

45 1, 25 dihydroxy vitamin D3, FGF23, serum calcium and urinary phosphorus were significant de

46 in Red and von Kossa stains, by depletion of serum calcium, and by uptake of calcium and phosphate by

47 arction was weighted by its association with serum calcium, and estimates were combined using an inve

48 hosphorus, absence of diabetes, younger age, serum calcium, and female gender.

49 roid hormone, increased vitamin D, increased serum calcium, and normalization of bone markers such as

50 adjusting for age, gender, diabetic status, serum calcium, and phosphorus (P < 0.0001).

51 order, the magnitude of the deviation of the serum calcium, and the severity of symptoms.

52 genase, low serum hemoglobin, high corrected serum calcium, and time from initial RCC diagnosis to st

53 in parathyroid hormone secretory control by serum calcium, as had been hypothesized.

54 that the analogue 4 significantly increases serum calcium at dose levels similar to 1alpha,25-(OH)2D

55 ms (SNPs)) that independently contributed to serum calcium at genome-wide significance which we appli

56 CaSR gene SNP rs1801725 was associated with serum calcium but not with risk of diabetes.

57 efined as the multiplication of preoperative serum calcium by preoperative parathyroid hormone (PTH).

58 in EDTA injection alone, such as decrease in serum calcium (Ca), increase in urine Ca, or toxicity to

59 d compression), hypercalcemia (symptoms or a serum calcium concentration > or = 12 mg per deciliter [

60 2.31, 2.05-2.60; p<0.0001), and raised total serum calcium concentration (1.43, 1.21-1.69; p<0.0001),

61 pinal cord compression, and hypercalcemia (a serum calcium concentration above 12 mg per deciliter [3

62 ne as evidenced by a greater increase of the serum calcium concentration and urine deoxypyridinoline

63 r, and safety measures of renal function and serum calcium concentration assessed every 3 months.

64 either unable or only slightly able to raise serum calcium concentration but are nevertheless able to

65 ication of variation in CASR that influences serum calcium concentration confirms the results of earl

66 ium and active vitamin D while maintaining a serum calcium concentration greater than or the same as

67 f this study was to test the hypothesis that serum calcium concentration is positively and independen

68 Uncorrected serum calcium concentration is the first mineral metabol

69 rathyroidism has been described in which the serum calcium concentration is within normal range but p

70 Abnormalities in serum calcium concentration may have profound effects on

71 At base line, the mean (+/-SE) serum calcium concentration was 10.7+/-0.2 mg per decili

72 have assessed the association of uncorrected serum calcium concentration with clinical outcomes.

73 who did not undergo surgery had no change in serum calcium concentration, urinary calcium excretion,

74 ctions of the native hormone without raising serum calcium concentration.

75 nitiates an endocrine cascade that regulates serum calcium concentration.

76 Serum calcium concentrations also decreased by 5 to 10%

77 or without symptoms led to normalization of serum calcium concentrations and a mean (+/-SE) increase

78 um-based binders led to significantly higher serum calcium concentrations and an increased incidence

79 go parathyroid surgery include monitoring of serum calcium concentrations and bone density.

80 th the Dunnett procedure was used to compare serum calcium concentrations at different time points af

81 Sevelamer, however, maintained serum calcium concentrations closer to the lower end of

82 and Cdc73(L/L)/PTH-Cre mice had higher mean serum calcium concentrations than wild-type littermates,

83 GWAS of serum calcium concentrations was performed in 20 611 ind

84 Higher adjusted serum calcium concentrations were also associated with a

85 Total serum calcium concentrations, which had ranged from 3.5

86 ese mutations had hypercalciuria even at low serum calcium concentrations.

87 ] and accounted for 0.54% of the variance in serum calcium concentrations.

88 hundred ninety-six inpatients with available serum calcium data obtained before and after gadodiamide

89 serum PTH levels, regardless of high or low serum calcium, demonstrated that PTH/PTH1R signaling exe

90 was then maintained for another 18 wk unless serum calcium exceeded 11.5 mg/dL or Ca x P product exce

91 s (recurrence < 12 months after nephrectomy, serum calcium > 10 mg/dL, hemoglobin < lower limit of no

92 (< lower limit of normal), high "corrected" serum calcium (> 10 mg/dL), and absence of prior nephrec

93 Elevated serum calcium has been associated with a variety of meta

94 Serum calcium has been associated with cardiovascular di

95 ong-term black carbon [BC] and PM2.5 levels, serum calcium homeostasis biomarkers (parathyroid hormon

96 ic bone disease, as a consequence of reduced serum calcium, hypoproteinuria, and hypoglycemia leading

97 diverged thereafter with significantly lower serum calcium in the 19-norD(2)-treated rats by 5 d.

98 nce-daily treatment with PTH 1-34 maintained serum calcium in the normal range with decreased urine c

99 In contrast, serum calcium in the VD(3) group was significantly eleva

100 substitute for vitamin D in the elevation of serum calcium in vitamin D-deficient rats.

101 Serum calcium indexes were taken throughout the study, a

102 Outcomes measures included serum calcium, intact parathyroid hormone (iPTH), and th

103 Maintenance of the normal serum calcium is a result of tightly regulated ion trans

104 ntrol of serum phosphorus without increasing serum calcium is an important goal for patients with ESR

105 f PTH secretion in response to variations in serum calcium is mediated by G-protein coupled, calcium-

106 thyroidism of renal failure, where a rise in serum calcium is undesirable.

107 weeks after treatment, PTX resulted in lower serum calcium level (9.28 mg/dL) compared with CIN (10.2

108 vs 23 [79.3%] female; P = .78), preoperative serum calcium level (mean [SD], 11.1 [0.9] vs 10.8 [0.8]

109 Associations between serum calcium level and baseline hematoma volume and bet

110 vel and baseline hematoma volume and between serum calcium level and ICH expansion were investigated

111 PTX led to a greater reduction in serum calcium level and lower chance of persistent hyper

112 identified initial AVA, current smoking, and serum calcium level as the independent predictors of amo

113 ecreased by 52% from 92 to 44 pg/mL, and the serum calcium level increased from 7.8 to 8.5 mg/dL.

114 Objective: To investigate whether a low serum calcium level is associated with an increase in th

115 15 mg/dL (normal level, <20 mg/dL), a total serum calcium level of 2.46 mmol/L (reference range, 2.1

116 admission, and hypocalcemia was defined as a serum calcium level of less than 8.4 mg/dL.

117 In this subgroup, a higher serum calcium level was associated with reduced risk of

118 A decreased serum calcium level was the only risk factor found to di

119 Serum calcium level, parathyroid hormone (PTH) level, an

120 valuating tumor growth, CD44 expression, and serum calcium level.

121 ,285 cases, 95,425 controls) and circulating serum calcium levels (39,400 subjects).

122 p and 10 in the placebo group), and elevated serum calcium levels (6 in the vitamin D3 + calcium grou

123 m a genome-wide association meta-analysis of serum calcium levels (N = up to 61079 individuals) and f

124 This lead SNP was associated with higher serum calcium levels [0.06 mg/dl (0.015 mmol/l) per copy

125 Serum calcium levels and change in serum creatinine leve

126 CaR is necessary for the fine regulation of serum calcium levels and renal calcium excretion indepen

127 between genetic variants related to elevated serum calcium levels and risk of coronary artery disease

128 -Fc > or =100 microg suppressed elevation of serum calcium levels and suppressed the bone turnover ma

129 Serum calcium levels and the Ca x P ion product increase

130 and 123504 noncases), the 6 SNPs related to serum calcium levels and without pleiotropic association

131 Serum calcium levels are tightly controlled by an integr

132 Serum calcium levels are tightly regulated.

133 hormone (iPTH) > or = 400 pg/ml, normalized serum calcium levels between 8.0 and 10.0 mg/dl, and cal

134 Finally, we found that elevation of serum calcium levels by 1 mg/dl resulting from our genet

135 with lifelong genetic exposure to increased serum calcium levels can be translated to a risk associa

136 The maximum serum calcium levels did not change (P = 0.15).

137 FBP lowered serum calcium levels during the first 24 h after the ins

138 Furthermore, raising serum calcium levels in Cyp27b1-depleted mice directly i

139 Serum calcium levels in the analogue group were not elev

140 The introduction of routine measurement of serum calcium levels led to a sharp increase in the inci

141 The early rise in serum calcium levels observed with treatment may have co

142 We found that the rate of change in serum calcium levels varied as a function of transmural

143 A genetic predisposition to higher serum calcium levels was associated with increased risk

144 crease (about 1 SD) in genetically predicted serum calcium levels were 1.25 (95% CI, 1.08-1.45; P = .

145 ization (MR) design to determine if elevated serum calcium levels were associated with risk of migrai

146 Serum calcium levels were increased only with vitamin D3

147 Serum calcium levels were measured with inductively coup

148 Postoperative mean serum calcium levels were similar (8.78 mg/dL, NIH group

149 aused a transient artifact in measurement of serum calcium levels with an OCP assay but not with an a

150 pt mean ages, 55 versus 65, and preoperative serum calcium levels, 11.5 mg/dL versus 11.0 mg/dL (NIH

151 ion of gadoversetamide caused no decrease in serum calcium levels, as measured with inductively coupl

152 Serum calcium levels, however, were unaffected by nephre

153 D3 and parathyroid hormone levels, decreased serum calcium levels, hyperplasia of the parathyroid, an

154 s that calcium supplementation, which raises serum calcium levels, may increase the risk of cardiovas

155 their ability to act in vivo without raising serum calcium levels, they may be of considerable intere

156 parathyroid hormone levels and low-to-normal serum calcium levels, were younger, and were receiving a

157 ould be achieved without adversely affecting serum calcium levels.

158 mon genetic variations associated with total serum calcium levels.

159 type, were able to reproduce, and had normal serum calcium levels.

160 hanisms that maintain skeletal integrity and serum calcium levels.

161 l in the blood did not affect measurement of serum calcium levels.

162 ased on genetic variants related to elevated serum calcium levels.

163 wk of treatment, concomitant with a rise in serum calcium levels.

164 ed to explain about 0.8% of the variation in serum calcium levels.

165 e was 100%, as evidenced by normalization of serum calcium levels.

166 tment for potential confounders, uncorrected serum calcium <8.5 and >/=10.2 mg/dl were associated wit

167 th the experimental evidence, suggest higher serum calcium may increase the risk of CAD.

168 Changes in serum calcium measurements following gadodiamide adminis

169 lowing 42 gadodiamide-enhanced examinations, serum calcium measurements spuriously decreased by more

170 hypercalcemic that became undetectable when serum calcium normalized.

171 At the multivariate analysis, serum calcium, OPG, and estimated glomerular filtration

172 patient age, renal function, or preoperative serum calcium or parathyroid hormone levels.

173 che diagnoses, and that genetically elevated serum calcium over lifetime appears to increase risk for

174 in D intakes were positively associated with serum calcium (P < 0.005) and calcidiol (P < 0.01) conce

175 ntributed significantly to the prediction of serum calcium (P < 0.009) and calcidiol (P < 0.0001), th

176 D concentration was the primary outcome, and serum calcium, parathyroid hormone (PTH), 1,25-dihydroxy

177 Patients were followed up for serum calcium, parathyroid hormone levels, and symptomat

178 In contrast, serum calcium, parathyroid hormone, and 25-hydroxyvitami

179 sted to achieve consistent albumin-corrected serum calcium, patients were randomly assigned (2:1) via

180 pletely rescued the hypercalciuric and lower serum calcium phenotype in Ksp-cre;Pth1r(fl/fl) mice, em

181 Laboratory evaluations included serum calcium, phosphate, alkaline phosphatase, 25-hydro

182 Serum calcium, phosphate, and vitamin D levels were norm

183 Runx2 deletion did not affect serum calcium, phosphate, fibroblast growth factor-23, o

184 There were no significant changes in serum calcium, phosphate, or intact parathyroid hormone

185 Serum calcium, phosphate, urea nitrogen, and creatinine

186 us; higher body mass index; higher levels of serum calcium, phosphorous, and parathyroid hormone; and

187 In sensitivity analyses, the addition of serum calcium, phosphorus, 25-hydroxyvitamin D, intact p

188 gender, age, diabetic status, and levels of serum calcium, phosphorus, alkaline phosphatase, and alu

189 Serum calcium, phosphorus, alkaline phosphatase, and par

190 ion to the more widely recognized changes in serum calcium, phosphorus, and 1alpha,25-dihydroxyvitami

191 Cinacalcet also significantly reduced serum calcium, phosphorus, and Ca x P levels compared wi

192 l excretion of calcium; and no difference in serum calcium, phosphorus, and PTH levels.

193 rly, the hematocrit, white blood cell count, serum calcium, phosphorus, cholesterol, triglycerides, t

194 No change occurred in serum calcium, phosphorus, intact parathyroid hormone, o

195 Baseline serum calcium, phosphorus, magnesium, and PTH levels wer

196 Serum calcium, phosphorus, osteocalcin, and alkaline pho

197 The serum calcium-phosphorus product declined by 15 percent

198 FHH3 probands had significantly greater serum calcium (sCa) and magnesium (sMg) concentrations w

199 cluded age, sex, estimated GFR, albuminuria, serum calcium, serum phosphate, serum bicarbonate, and s

200 Changes in plasma PTH, serum calcium, serum phosphorus, and calcium x phosphoru

201 mmortalizes human keratinocytes and inhibits serum/calcium-stimulated differentiation.

202 in patients with an asymptomatic increase in serum calcium to >12.0 mg/dl persisting for >1 year afte

203 ys showed a significant (P <.05) decrease in serum calcium values after administration of gadoverseta

204 The decrease in serum calcium values peaked immediately after injection,

205 lues peaked immediately after injection, and serum calcium values quickly returned to baseline.

206 adoteridol produced no significant change in serum calcium values, regardless of analytic method.

207 Serum-calcium values were similar (2.1 +/- 0.3 vs 2.1 +/

208 a were compiled to determine how GBP affects serum calcium, vitamin D, and PTH.

209 Serum calcium was corrected for serum albumin.

210 ent with 3 previous cohort studies, elevated serum calcium was found to be associated with a greater

211 and U/HP mice versus sham controls, whereas serum calcium was increased in the U/HP group, and no di

212 Serum calcium was measured at baseline and corrected for

213 19-norD(2) or 1,25(OH)(2)D(3), increases in serum calcium were identical 24 h after the first inject

214 ar between groups except that hemoglobin and serum calcium were lower and serum phosphorus was higher

215 ignificant (P <.05) decrease in the value of serum calcium when analyzed with the OCP technique but n

216 ase in dietary calcium for 10 days increased serum calcium, with an associated increase in FGF23, dec