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1 e BALB/cJ mice showed a fivefold increase in serum corticosterone.
2 icular nucleus with a concurrent increase in serum corticosterone.
4 ose of corticosterone, which did not elevate serum corticosterone, acted synergistically with sodium
5 nduced brain levels of 3alpha,5alpha-THP and serum corticosterone, although gender, gonadectomy and h
6 trophin-1) also potentiated the elevation of serum corticosterone and IL-6 levels induced by IL-1.
7 o stresses were observed including increased serum corticosterone and increased dopamine metabolism i
8 l stress-predictive cues exhibited increased serum corticosterone and significantly greater reinstate
9 dator of the rat, on fear-induced behaviors, serum corticosterone, and central dopamine metabolism.
10 Also, they displayed higher levels of blood serum corticosterone, as well as decreased body weight.
11 one in situ showed a significant increase in serum corticosterone associated with visceral hyperalges
14 rleukin-6 (IL-6) potentiate the elevation of serum corticosterone induced by suboptimal doses of inte
16 raint stress and a slower descending rate in serum corticosterone level during a 30-min post-stress p
17 sults suggest that the chronically increased serum corticosterone levels contribute to the diabetes o
18 While AED did not antagonize the elevated serum corticosterone levels following acute infection, A
22 w that acute cold stress leading to elevated serum corticosterone levels neither induces LTP-like inc
23 citation across an extended trial range, and serum corticosterone levels were evaluated in response t
29 social deficits are associated with elevated serum corticosterone levels; however, in the F2 and F3 g
33 fact, in IL-6 deficient mice, IL-1 increased serum corticosterone to a level comparable to that obser
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