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1 riod, blood samples were taken for 8:00 A.M. serum cortisol.
2 letion of a corticotrophin stimulation test, serum cortisol and 11beta-deoxycortisol concentrations w
6 ealed that ozone exposure markedly increased serum cortisol and corticosterone together with increase
7 , metabolic responses (serial blood glucose, serum cortisol and insulin, plasma epinephrine, plasma n
8 xia nervosa subtype, waist-to-hip ratio, and serum cortisol and leptin levels on treatment outcome, o
10 d plasma aldosterone, plasma renin activity, serum cortisol, and estimation of trans tubular potassiu
11 y and levels of adrenocorticotropic hormone, serum cortisol, and saliva cortisol among all patients a
13 eroid hormones, and accurate measurements of serum cortisol are necessary for proper diagnosis of adr
14 vents, coping by means of denial, and higher serum cortisol as well as with lower cumulative average
15 The 91 patients with sepsis began with a serum cortisol at 29.3 +/- 2.5, and it increased to 40.1
21 in infusion raised plasma growth hormone and serum cortisol concentrations significantly (P < 0.001 f
28 epinephrine levels, morning plasma ACTH and serum cortisol, fasting glucose and insulin, and lipid p
35 addition, there was a sustained elevation of serum cortisol levels, reduced circulating melatonin lev
39 tigraphy assessment, and 24-hour analyses of serum cortisol, melatonin, and peripheral clock gene exp
40 y ill patients is best made by a delta total serum cortisol of < 9 microg/dL after adrenocorticotroph
43 (37%) were steroid responsive; the baseline serum cortisol was 14.1 +/- 5.2 microg/dL in the steroid
46 thalamic-pituitary-thyroid axis, we measured serum cortisol, which also has a circadian secretory pat
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