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1  proliferation in the presence of respective serum immunoglobulin G.
2 e deficiency, characterized by low levels of serum immunoglobulin G, A, and/or M with loss of antibod
3 ly correlated with the level of prechallenge serum immunoglobulin G against the O127 lipopolysacchari
4 l aplasia, several vaccine/pathogen-specific serum immunoglobulin G and A (IgG and IgA) titers remain
5             Neutralizing antibody responses, serum immunoglobulin G and A, and nasal wash specimen im
6             Antral gastritis, anti-H. pylori serum immunoglobulin G, and atrophy all increased, but w
7 or, CVD 915(pTETlpp) elicited high titers of serum immunoglobulin G anti-fragment C.
8      Only conjugates with O acetyls elicited serum immunoglobulin G anti-LPS with bactericidal activi
9 ith the level of protection correlating with serum immunoglobulin G anti-protective antigen titers.
10 l immunoglobulin A antiamebic antibodies and serum immunoglobulin G antiamebic antibodies.
11 VRP resulted in high levels of DENV-specific serum immunoglobulin G antibodies and significant titers
12                                     Although serum immunoglobulin G antibodies are significantly high
13 ll-length block 2 antigens showed that human serum immunoglobulin G antibodies induced by infection c
14 orbent assays (ELISAs) for quantification of serum immunoglobulin G antibodies specific for N. mening
15 uced specific secretory immunoglobulin A and serum immunoglobulin G antibodies that persisted at subs
16  concentrations and neutralizing activity of serum immunoglobulin G antibodies to the RSV prefusion (
17 tion, as well as the specificity, of anti-P1 serum immunoglobulin G antibodies were demonstrated in g
18                            It was shown that serum immunoglobulin G antibodies were produced against
19 d formation, an impaired ability to generate serum immunoglobulin G antibodies, and significant inhib
20 treated patients with LA have high levels of serum immunoglobulin G antibodies, and sometimes low lev
21 cator of inflammation), or edentulism and 2) serum immunoglobulin G antibody response to Aggregatibac
22 e also developed sustained anti-H. hepaticus serum immunoglobulin G antibody responses and elevated a
23     Less marked, but significant, anti-SREHP serum immunoglobulin G antibody responses were also indu
24 d animalls developed sustained anti-H. felis serum immunoglobulin G antibody responses.
25 PI) owing to the presence of highly elevated serum immunoglobulin G antibody titers with a high avidi
26 cant levels of salivary immunoglobulin A and serum immunoglobulin G antibody to the respective inject
27                                            A serum immunoglobulin G autoantibody (NMO-IgG) serves as
28       All conjugates elicited high levels of serum immunoglobulin G both to the polysaccharides and t
29               There was a slight increase in serum immunoglobulin G (IgG) against the MS11 strain and
30                    Serologic markers such as serum immunoglobulin G (IgG) and antinuclear antibody le
31 although FspA1 induced the highest levels of serum immunoglobulin G (IgG) and fecal IgA.
32 ity to S. typhimurium severely depressed the serum immunoglobulin G (IgG) and IgA anti-FrgC response
33 -CAT construct induced significant levels of serum immunoglobulin G (IgG) and IgA antibody to both pa
34                Recall responses were seen in serum immunoglobulin G (IgG) and IgA following boosting
35  with Invaplex was found to enhance anti-OVA serum immunoglobulin G (IgG) and IgA responses and induc
36 oximately 2-week intervals and evaluated for serum immunoglobulin G (IgG) and IgG subclass and for sa
37                        Despite production of serum immunoglobulin G (IgG) and IgM (median titers of 1
38  throughout the experiment and evaluated for serum immunoglobulin G (IgG) and IgM and salivary IgA an
39         In mice i.n. challenged with H10407, serum immunoglobulin G (IgG) and IgM antibodies were mea
40  or no bacteria in the liver and lungs, high serum immunoglobulin G (IgG) and IgM antibody titers, an
41 lpha stimulated strong antigen (Ag)-specific serum immunoglobulin G (IgG) and IgM responses, while MI
42 ion of mice with CVLPs induced gp41-specific serum immunoglobulin G (IgG) and intestinal secretory Ig
43 lones were found to stimulate high levels of serum immunoglobulin G (IgG) and mucosal IgA antibodies
44 luenza virus showed significant increases in serum immunoglobulin G (IgG) and mucosal IgA antibodies
45 oxin (CT) A2 and B subunits (CTA2/B) develop serum immunoglobulin G (IgG) and mucosal IgA antibody re
46 hesizing either PspA fusion protein elicited serum immunoglobulin G (IgG) and mucosal IgA responses a
47               The levels of antigen-specific serum immunoglobulin G (IgG) and mucosal IgA were higher
48 cific gamma interferon-producing T cells and serum immunoglobulin G (IgG) and mucosal IgA.
49 hK63 or LThR72 exhibited high titers of both serum immunoglobulin G (IgG) and mucosal secretory IgA a
50 ed animals demonstrated significantly higher serum immunoglobulin G (IgG) and salivary IgA antibody l
51                Both peptides readily induced serum immunoglobulin G (IgG) and salivary IgA antipeptid
52 astric tissue, histology, and measurement of serum immunoglobulin G (IgG) and salivary IgA.
53                   The specificity of anti-P1 serum immunoglobulin G (IgG) and secretory IgA antibodie
54 enuation elicited higher Salmonella-specific serum immunoglobulin G (IgG) and/or mucosal secretory-Ig
55  later, significant reductions were seen for serum immunoglobulin G (IgG) antibodies at week 14 and f
56                                              Serum immunoglobulin G (IgG) antibodies have been implic
57 recombinant proteins were reacted with human serum immunoglobulin G (IgG) antibodies in Western immun
58               We investigated the ability of serum immunoglobulin G (IgG) antibodies to periodontal b
59                       Low maternally derived serum immunoglobulin G (IgG) antibodies to Streptococcus
60 ta provide evidence that a critical level of serum immunoglobulin G (IgG) antibodies to the surface p
61  were found to be immunogenic since elevated serum immunoglobulin G (IgG) antibodies without a specif
62 Oral and i.p. vaccines elicited O11-specific serum immunoglobulin G (IgG) antibodies, but IgA was obs
63 isms are found and the induction of specific serum immunoglobulin G (IgG) antibodies, we examined the
64 ay was used to measure the seroprevalence of serum immunoglobulin G (IgG) antibody among NHANES parti
65                   We determined pneumococcal serum immunoglobulin G (IgG) antibody concentrations aga
66 ria during the study interval, and increased serum immunoglobulin G (IgG) antibody levels substantiat
67 nce of P. gingivalis in dental plaque and of serum immunoglobulin G (IgG) antibody levels to P. gingi
68                               GFPuv-specific serum immunoglobulin G (IgG) antibody responses were ana
69 oPn produced greater titers of MoPn-specific serum immunoglobulin G (IgG) antibody than mice that rec
70 zed by enzyme-linked immunosorbent assay for serum immunoglobulin G (IgG) antibody to Aggregatibacter
71                                    Levels of serum immunoglobulin G (IgG) antibody to GTF or CAT in t
72                                              Serum immunoglobulin G (IgG) antibody, induced by subcut
73 rticular) demonstrated significantly reduced serum immunoglobulin G (IgG) antibody, salivary IgA anti
74 train RB51 (SRB51) produced small amounts of serum immunoglobulin G (IgG) but no IgM antibody to smoo
75 Ribi had the highest levels of PorB-specific serum immunoglobulin G (IgG) by enzyme-linked immunosorb
76                                          New serum immunoglobulin G (IgG) detected by whole-cell enzy
77 mean titer (GMTs) and 4-fold rise of anti-Vi serum immunoglobulin G (IgG) enzyme-linked immunosorbent
78 ntigenic MDI-albumin products, as defined by serum immunoglobulin G (IgG) from MDI-exposed workers, w
79                                     Aberrant serum immunoglobulin G (IgG) glycosylation and its immun
80 immunosorbent assay to measure beta-specific serum immunoglobulin G (IgG) levels and used it to compa
81                         Significant anti-GLU serum immunoglobulin G (IgG) levels were seen in groups
82              Smoking is also known to reduce serum immunoglobulin G (IgG) levels.
83 haride (PS) conjugate vaccines may prime for serum immunoglobulin G (IgG) memory responses to meningo
84 transplantations indicate that low levels of serum immunoglobulin G (IgG) negatively impact outcomes.
85  Overall, 23% of patients developed either a serum immunoglobulin G (IgG) response (9%) or sputum IgA
86 . denticola induced a significant (400-fold) serum immunoglobulin G (IgG) response compared to that i
87                          Anamnestic anti-SEB serum immunoglobulin G (IgG) responses were elicited in
88 ized with replication-defective HSV, durable serum immunoglobulin G (IgG) responses were elicited.
89 e the long-term course of H. pylori-specific serum immunoglobulin G (IgG) responses with respect to s
90 were effective in eliciting antigen-specific serum immunoglobulin G (IgG) responses, and these respon
91 s evokes large increases in antigen-specific serum immunoglobulin G (IgG) responses.
92 fold increase over the prechallenge anti-SMV serum immunoglobulin G (IgG) titer, mostly subclass IgG1
93  were consistent with significantly declined serum immunoglobulin G (IgG) titers for HHV-6 of MS pati
94 ckerboard analyses of eight plaque bacteria, serum immunoglobulin G (IgG) titers to 17 bacteria, and
95                                              Serum immunoglobulin G (IgG) titers to 28 pneumococcal p
96 B) toxoid in saline elicited higher anti-SEB serum immunoglobulin G (IgG) titers when the toxoid was
97                  Binding activity of patient serum immunoglobulin G (IgG) to HupB did not correlate w
98 colonization, 9 of 40 patients developed new serum immunoglobulin G (IgG) to OMP CD, as measured by e
99 ollowing infection and who had developed new serum immunoglobulin G (IgG) to their infecting strain w
100 eleased from bovine fetuin, polyclonal human serum immunoglobulin G (IgG), and human alpha1-acid glyc
101 he most effective means of immunization: the serum immunoglobulin G (IgG), IgA, and IgM anti-fragment
102                                  We measured serum immunoglobulin G (IgG), IgM, and IgA antibodies to
103  increased class switched memory B cells and serum immunoglobulin G (IgG).
104 s resulted in the induction of FimA-specific serum (immunoglobulin G [IgG] and IgA) and salivary (IgA
105 adermal comparative tuberculin skin test and serum immunoglobulin G [IgG] responses) against a range
106 vated during primary infections (acute-phase serum immunoglobulin G [IgG] titers, <25), compared with
107 nses (HI, antibody-secreting cell [ASC], and serum immunoglobulin G [IgG]) in 15 LRs and 25 control H
108 minent specific anti-CtxB responses in vivo (serum immunoglobulin G [IgG], P </= 0.05; serum IgA, P <
109 ortal vein diameter, splenomegaly, increased serum immunoglobulin G level, and increasing number of a
110 tly improved liver histology (P = 0.005) and serum immunoglobulin G levels (P = 0.0002).
111                                  We compared serum immunoglobulin G levels and toxin-neutralizing ant
112                                          The serum immunoglobulin G levels correlated with protection
113                       P. gingivalis-specific serum immunoglobulin G levels were significantly elevate
114 , with a 44% reduction in intracardiomyocyte serum immunoglobulin G localization in het-treated-8 mic
115 cination geometric mean titre of LT-specific serum immunoglobulin G of 3400.29, compared with 315.41
116 asma could not be duplicated by pooled human serum, immunoglobulin G, or fibrinogen, whether used sep
117                                              Serum immunoglobulin G profiling by proteome microarray
118                                              Serum immunoglobulin G reactivity to Hsp10 and Hsp60 ant
119  the naturally infected SCID/NCr mice had no serum immunoglobulin G response against H. hepaticus.
120  vaccine doses elicited a strong PA-specific serum immunoglobulin G response with a geometric mean ti
121                            Only LAM-reactive serum immunoglobulin G responses were significantly incr
122 ufficient to decrease P. gingivalis-specific serum immunoglobulin G responses, but lower antibody tit
123 osted 4 weeks later induced higher levels of serum immunoglobulin G specific for PspA and for outer m
124                                              Serum immunoglobulin G subclass levels and antibody tite
125                                          The serum immunoglobulin G subclass profiles were indicative
126 he vhs(-)/ICP8(-) mutant showed prechallenge serum immunoglobulin G titers comparable to those immuni
127  a clinical oral-health examination, and had serum immunoglobulin G titers measured against 19 period
128 of variants with ChoP correlates with higher serum immunoglobulin G titers to LPS containing this str
129                                Vi stimulates serum immunoglobulin G Vi antibodies, whereas Ty21a, whi
130                                        A low serum immunoglobulin G was associated with number of inf
131       Subgingival P. endodontalis levels and serum immunoglobulin G were associated with a higher EL
132 ut adjuvant stimulated induction of specific serum immunoglobulin G, whereas antigen alone or admixed

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