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1 proliferation in the presence of respective serum immunoglobulin G.
2 e deficiency, characterized by low levels of serum immunoglobulin G, A, and/or M with loss of antibod
3 ly correlated with the level of prechallenge serum immunoglobulin G against the O127 lipopolysacchari
4 l aplasia, several vaccine/pathogen-specific serum immunoglobulin G and A (IgG and IgA) titers remain
9 ith the level of protection correlating with serum immunoglobulin G anti-protective antigen titers.
11 VRP resulted in high levels of DENV-specific serum immunoglobulin G antibodies and significant titers
13 ll-length block 2 antigens showed that human serum immunoglobulin G antibodies induced by infection c
14 orbent assays (ELISAs) for quantification of serum immunoglobulin G antibodies specific for N. mening
15 uced specific secretory immunoglobulin A and serum immunoglobulin G antibodies that persisted at subs
16 concentrations and neutralizing activity of serum immunoglobulin G antibodies to the RSV prefusion (
17 tion, as well as the specificity, of anti-P1 serum immunoglobulin G antibodies were demonstrated in g
19 d formation, an impaired ability to generate serum immunoglobulin G antibodies, and significant inhib
20 treated patients with LA have high levels of serum immunoglobulin G antibodies, and sometimes low lev
21 cator of inflammation), or edentulism and 2) serum immunoglobulin G antibody response to Aggregatibac
22 e also developed sustained anti-H. hepaticus serum immunoglobulin G antibody responses and elevated a
23 Less marked, but significant, anti-SREHP serum immunoglobulin G antibody responses were also indu
25 PI) owing to the presence of highly elevated serum immunoglobulin G antibody titers with a high avidi
26 cant levels of salivary immunoglobulin A and serum immunoglobulin G antibody to the respective inject
32 ity to S. typhimurium severely depressed the serum immunoglobulin G (IgG) and IgA anti-FrgC response
33 -CAT construct induced significant levels of serum immunoglobulin G (IgG) and IgA antibody to both pa
35 with Invaplex was found to enhance anti-OVA serum immunoglobulin G (IgG) and IgA responses and induc
36 oximately 2-week intervals and evaluated for serum immunoglobulin G (IgG) and IgG subclass and for sa
38 throughout the experiment and evaluated for serum immunoglobulin G (IgG) and IgM and salivary IgA an
40 or no bacteria in the liver and lungs, high serum immunoglobulin G (IgG) and IgM antibody titers, an
41 lpha stimulated strong antigen (Ag)-specific serum immunoglobulin G (IgG) and IgM responses, while MI
42 ion of mice with CVLPs induced gp41-specific serum immunoglobulin G (IgG) and intestinal secretory Ig
43 lones were found to stimulate high levels of serum immunoglobulin G (IgG) and mucosal IgA antibodies
44 luenza virus showed significant increases in serum immunoglobulin G (IgG) and mucosal IgA antibodies
45 oxin (CT) A2 and B subunits (CTA2/B) develop serum immunoglobulin G (IgG) and mucosal IgA antibody re
46 hesizing either PspA fusion protein elicited serum immunoglobulin G (IgG) and mucosal IgA responses a
49 hK63 or LThR72 exhibited high titers of both serum immunoglobulin G (IgG) and mucosal secretory IgA a
50 ed animals demonstrated significantly higher serum immunoglobulin G (IgG) and salivary IgA antibody l
54 enuation elicited higher Salmonella-specific serum immunoglobulin G (IgG) and/or mucosal secretory-Ig
55 later, significant reductions were seen for serum immunoglobulin G (IgG) antibodies at week 14 and f
57 recombinant proteins were reacted with human serum immunoglobulin G (IgG) antibodies in Western immun
60 ta provide evidence that a critical level of serum immunoglobulin G (IgG) antibodies to the surface p
61 were found to be immunogenic since elevated serum immunoglobulin G (IgG) antibodies without a specif
62 Oral and i.p. vaccines elicited O11-specific serum immunoglobulin G (IgG) antibodies, but IgA was obs
63 isms are found and the induction of specific serum immunoglobulin G (IgG) antibodies, we examined the
64 ay was used to measure the seroprevalence of serum immunoglobulin G (IgG) antibody among NHANES parti
66 ria during the study interval, and increased serum immunoglobulin G (IgG) antibody levels substantiat
67 nce of P. gingivalis in dental plaque and of serum immunoglobulin G (IgG) antibody levels to P. gingi
69 oPn produced greater titers of MoPn-specific serum immunoglobulin G (IgG) antibody than mice that rec
70 zed by enzyme-linked immunosorbent assay for serum immunoglobulin G (IgG) antibody to Aggregatibacter
73 rticular) demonstrated significantly reduced serum immunoglobulin G (IgG) antibody, salivary IgA anti
74 train RB51 (SRB51) produced small amounts of serum immunoglobulin G (IgG) but no IgM antibody to smoo
75 Ribi had the highest levels of PorB-specific serum immunoglobulin G (IgG) by enzyme-linked immunosorb
77 mean titer (GMTs) and 4-fold rise of anti-Vi serum immunoglobulin G (IgG) enzyme-linked immunosorbent
78 ntigenic MDI-albumin products, as defined by serum immunoglobulin G (IgG) from MDI-exposed workers, w
80 immunosorbent assay to measure beta-specific serum immunoglobulin G (IgG) levels and used it to compa
83 haride (PS) conjugate vaccines may prime for serum immunoglobulin G (IgG) memory responses to meningo
84 transplantations indicate that low levels of serum immunoglobulin G (IgG) negatively impact outcomes.
85 Overall, 23% of patients developed either a serum immunoglobulin G (IgG) response (9%) or sputum IgA
86 . denticola induced a significant (400-fold) serum immunoglobulin G (IgG) response compared to that i
88 ized with replication-defective HSV, durable serum immunoglobulin G (IgG) responses were elicited.
89 e the long-term course of H. pylori-specific serum immunoglobulin G (IgG) responses with respect to s
90 were effective in eliciting antigen-specific serum immunoglobulin G (IgG) responses, and these respon
92 fold increase over the prechallenge anti-SMV serum immunoglobulin G (IgG) titer, mostly subclass IgG1
93 were consistent with significantly declined serum immunoglobulin G (IgG) titers for HHV-6 of MS pati
94 ckerboard analyses of eight plaque bacteria, serum immunoglobulin G (IgG) titers to 17 bacteria, and
96 B) toxoid in saline elicited higher anti-SEB serum immunoglobulin G (IgG) titers when the toxoid was
98 colonization, 9 of 40 patients developed new serum immunoglobulin G (IgG) to OMP CD, as measured by e
99 ollowing infection and who had developed new serum immunoglobulin G (IgG) to their infecting strain w
100 eleased from bovine fetuin, polyclonal human serum immunoglobulin G (IgG), and human alpha1-acid glyc
101 he most effective means of immunization: the serum immunoglobulin G (IgG), IgA, and IgM anti-fragment
104 s resulted in the induction of FimA-specific serum (immunoglobulin G [IgG] and IgA) and salivary (IgA
105 adermal comparative tuberculin skin test and serum immunoglobulin G [IgG] responses) against a range
106 vated during primary infections (acute-phase serum immunoglobulin G [IgG] titers, <25), compared with
107 nses (HI, antibody-secreting cell [ASC], and serum immunoglobulin G [IgG]) in 15 LRs and 25 control H
108 minent specific anti-CtxB responses in vivo (serum immunoglobulin G [IgG], P </= 0.05; serum IgA, P <
109 ortal vein diameter, splenomegaly, increased serum immunoglobulin G level, and increasing number of a
114 , with a 44% reduction in intracardiomyocyte serum immunoglobulin G localization in het-treated-8 mic
115 cination geometric mean titre of LT-specific serum immunoglobulin G of 3400.29, compared with 315.41
116 asma could not be duplicated by pooled human serum, immunoglobulin G, or fibrinogen, whether used sep
119 the naturally infected SCID/NCr mice had no serum immunoglobulin G response against H. hepaticus.
120 vaccine doses elicited a strong PA-specific serum immunoglobulin G response with a geometric mean ti
122 ufficient to decrease P. gingivalis-specific serum immunoglobulin G responses, but lower antibody tit
123 osted 4 weeks later induced higher levels of serum immunoglobulin G specific for PspA and for outer m
126 he vhs(-)/ICP8(-) mutant showed prechallenge serum immunoglobulin G titers comparable to those immuni
127 a clinical oral-health examination, and had serum immunoglobulin G titers measured against 19 period
128 of variants with ChoP correlates with higher serum immunoglobulin G titers to LPS containing this str
132 ut adjuvant stimulated induction of specific serum immunoglobulin G, whereas antigen alone or admixed
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