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1 for human diphtheria, its use often induces serum sickness.
2 None of the patients had evidence for serum sickness.
4 five renal transplant patients who developed serum sickness after polyclonal antibody treatment with
8 gic vasculitis, leukocytoclastic vasculitis, serum sickness, and others, which are often used interch
11 strongly immunogenic and rabbit ATG induces serum sickness disease in almost all patients without ad
16 lement, which led to spontaneous and chronic serum sickness-induced proliferative glomerulonephritis.
19 der untoward autoimmune reactions, including serum sickness-like disease, drug-induced lupus, and aut
20 of infrequent but serious events, including serum sickness-like reaction, opportunistic infection an
21 -inflammatory drug-associated angioedema and serum sickness-like reactions, are more frequent among y
22 ephritis, which was accelerated in a chronic serum sickness model by active immunization with heterol
24 discontinuation of treatment, 1 developed a serum sickness reaction after completion of therapy, and
27 oup examining the models of acute or chronic serum sickness were major for our understanding of renal
28 group had both cytokine release syndrome and serum sickness, which was associated with a transient ri
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