1 na, including motivational conflict, that it
sets out to.
2 In this study, we
set out to 1) investigate striatal development in autism
3 We
set out to address the fundamental question of how gut m
4 Here, we
set out to address the motor network activity and synchr
5 We
set out to address this issue by studying how various mR
6 Here we
set out to address this problem and uncover a unique cat
7 We
set out to address this problem by generating multiple g
8 We
set out to address whether neglect presentation could be
9 This study
set out to analyze the brains of selected arthropods wit
10 We
set out to analyze the development of this connectivity
11 Here we
set out to answer if the dark-matter of the genome encom
12 We therefore
set out to apply the Oxford Nanopore Technologies sequen
13 In this cohort study, we
set out to assess compliance.
14 We
set out to assess the dynamics of retinal injury after a
15 Therefore, we
set out to assess the effect of introduction of ten-vale
16 Therefore, we
set out to assess the effects and possible components of
17 We
set out to assess the performance of the MYH7-specific A
18 Leveraging a collaborative RPPA model, we
set out to assess the variability between three differen
19 Here we
set out to assess this hypothesis, and find an unexpecte
20 We therefore
set out to assess whether children diagnosed after the a
21 ecognize microbial or endogenous signals, we
set out to assess whether their functions are locally in
22 Here we
set out to better understand how miR-122 inhibition infl
23 We
set out to bridge this gap.
24 We therefore
set out to characterise CD4(+)CD25(high) T cells isolate
25 We
set out to characterise the changes in macroscopic muscl
26 Therefore, we
set out to characterize alterations in miRNA expression
27 We thus
set out to characterize BAP1 in CM and discovered an une
28 Using fMRI we
set out to characterize both the task and stimulus depen
29 In this study, we
set out to characterize changes in DNA methylation and g
30 Herein we
set out to characterize the carbohydrate minimal binding
31 We therefore
set out to characterize the cellular and molecular immun
32 We
set out to characterize the human CD4(+) T cell response
33 The results of this nation-wide study
set out to characterize the impact of bariatric surgery
34 Here, we
set out to characterize the physical interaction between
35 LK in early stages of T cell development, we
set out to characterize the role NLK plays in T cell dev
36 In this investigation, we
set out to characterize the roles of the WDR5 subunit in
37 Here we
set out to characterize transcriptional heterogeneity in
38 erstand its apparent biological activity, we
set out to chemically synthesize d-allo-ShK and determin
39 We
set out to clarify these relationships using polarized h
40 We
set out to compare several measures of community viral l
41 We
set out to compare the incidence of bowel repair and/or
42 In wild-type and CC16(-/-) mice, we
set out to comprehensively examine pulmonary physiology,
43 rities in regeneration among vertebrates, we
set out to define at high resolution the changes in gene
44 Here we
set out to define genes associated with motile cilia in
45 We
set out to define precisely the morphological events of
46 e T-PLL International Study group (TPLL-ISG)
set out to define standardized criteria for diagnosis, t
47 Here, we
set out to define the biochemical and functional diversi
48 The present study
set out to define the functional activities of bovine gr
49 We
set out to define the role of 1alpha,25-dihydroxy vitami
50 We
set out to define the role of UL31 in CMV replication.
51 We
set out to define the roles of cGAS, IRF3, IRF7, the typ
52 Here, we
set out to delineate the protein degradation mechanism o
53 a first step in testing this hypothesis, we
set out to demonstrate that the tricuspid valve maladapt
54 We therefore
set out to describe cellularization in the beetle Tribol
55 Here we
set out to describe fecal metabolite composition in heal
56 We
set out to describe the natural history of keratoconus.
57 accreditation to international standards, we
set out to design and assess an accreditation procedure
58 We therefore
set out to design and synthesize an inhibitor of AasS an
59 In this work, we
set out to design novel granulopoietic agents using a re
60 We
set out to design, synthesize and optimize a DNA-minimal
61 garding giant virus infection mechanisms, we
set out to determine biomolecular conditions that promot
62 Hence, we
set out to determine CCLs that tend to be overly sensiti
63 The present study
set out to determine consistent, specific regional brain
64 We
set out to determine if adhesive ligand tether length is
65 In this study, we
set out to determine if chronically activated microglia
66 Here, we
set out to determine if K17 is as accurate as molecular
67 In this study, we
set out to determine if the same is true for B cells usi
68 genes in cases of uncomplicated malaria, we
set out to determine if there was any evidence of a sele
69 Here, we
set out to determine the consequences of alcohol-depende
70 Our studies
set out to determine the contribution of SP-A to the res
71 Here, we
set out to determine the disease mechanism of 7 de novo
72 ing a new model of mild contusion injury, we
set out to determine the effect of contusion on iGAS bac
73 We
set out to determine the effect of PCSK9 (proprotein con
74 Hence we
set out to determine the effects of TIGAR expression on
75 Here, we
set out to determine the extent to which dynamic changes
76 Here, we
set out to determine the function of human family with s
77 e factors and effective vaccine antigens, we
set out to determine the genetic determinants of K. king
78 We
set out to determine the independent association of diff
79 Therefore, we
set out to determine the intracellular signalling involv
80 Here we
set out to determine the mechanisms underpinning variant
81 We
set out to determine the minimum number of cells that ca
82 We
set out to determine the molecular mechanisms underlying
83 their numbers can be limiting; therefore we
set out to determine the parameters for robust ultra-low
84 Here, we
set out to determine the pathophysiological role of PC1
85 We
set out to determine the physiological relevance and cel
86 We therefore
set out to determine the precise contributions of Esco1
87 We
set out to determine the role of CD73 in GB pathogenesis
88 We
set out to determine the specific AMPAR subunit required
89 We
set out to determine the structural properties of membra
90 In this report, we
set out to determine what viral components are responsib
91 We
set out to determine whether a single protocol, combinin
92 On the basis of these results, we next
set out to determine whether AMG655 possibly interferes
93 We
set out to determine whether attenuated total reflection
94 Here, we
set out to determine whether CADM2 contributes to mechan
95 Here, we
set out to determine whether changes in rate constants r
96 ion genetic analysis of wild populations, we
set out to determine whether evidence supports a role fo
97 To address this challenge, we
set out to determine whether follow up for surgical site
98 We
set out to determine whether gradient-like molecular dif
99 rtant to glomerular permselectivity, we next
set out to determine whether IP receptor agonism similar
100 We
set out to determine whether movements during REM sleep
101 We therefore
set out to determine whether NS3 from the replicatively
102 Here we
set out to determine whether oscillatory activity in the
103 ses iron absorption in infants is unclear.We
set out to determine whether prebiotic consumption affec
104 or threonine, my group at the Salk Institute
set out to determine whether the polyomavirus middle T-t
105 We
set out to determine whether this important mechanical c
106 lular protein kinases, and in this study, we
set out to determine whether this modification is requir
107 fter establishing this reward modulation, we
set out to determine whether we could correctly classify
108 We
set out to determine whether, similarly, common cancer s
109 We
set out to determine which characteristics and outcomes
110 We
set out to determine, in checkpoint inhibitor resistant
111 Therefore, we
set out to develop 2 novel KOR agonist radiotracers, (11
112 Therefore, we
set out to develop a combinatorial approach toward the n
113 Here, we
set out to develop a computational framework for estimat
114 disease, glaucoma, and 10 years ago, my lab
set out to develop a deeper understanding of myocilin in
115 We
set out to develop a method to overcome this obstacle by
116 This work
set out to develop a motion-correction approach aided by
117 rate the cardiac drug discovery pipeline, we
set out to develop a platform that would be capable of q
118 We
set out to develop an algorithm that can mine differenti
119 We
set out to develop an epilepsy gene therapy vector optim
120 Here, we
set out to develop an expanded toolkit of autophagy repo
121 Guided by this rationale, we
set out to develop an immunization protocol aimed at max
122 We
set out to develop an in vitro model of tumor cell migra
123 We
set out to develop an inhibitor compound targeting the b
124 We
set out to develop and validate a prognostic model, usin
125 Therefore, we
set out to develop dCK inhibitors with favorable pharmac
126 s of affecting disease progression, our team
set out to develop LRRK2 inhibitors to test this hypothe
127 Therefore, we
set out to develop the first, to our knowledge, robust a
128 and functions in the dendritic endosome, we
set out to discover how Nsg1 and Nsg2 localization to en
129 This article
sets out to document the dawning of the age of genomics
130 The hidden talents program
sets out to document these abilities, their development,
131 Here, we
set out to elucidate the feasibility of supplementation
132 This study
set out to elucidate the functional consequences of OPN
133 Next, we
set out to elucidate the genetic network that results in
134 We
set out to elucidate the role of Hh signaling in CP-CML
135 We therefore
set out to employ a battery of behavior tests, including
136 We
set out to establish a novel model for RPGR disease to t
137 present a global Porifera microbiome survey,
set out to establish the ecological and evolutionary dri
138 We
set out to estimate the cost of the dieback of ash, Frax
139 We
set out to estimate what proportion of adolescents were
140 Thus, we
set out to evaluate the association between tumor respon
141 Thus we
set out to evaluate the best tools that can be used loca
142 Therefore, we
set out to evaluate whether NONO could be involved in th
143 Here we
set out to examine changes in airborne fungi collected o
144 To gain further insight, we
set out to examine concurrent changes in pretransplant p
145 This study
set out to examine how nurses and care workers work, the
146 Here, we
set out to examine how song-generating circuits may be i
147 We
set out to examine PG production in human ILC2s and the
148 This study
set out to examine the repeatability of OCTA quantificat
149 lly diverse and widely used CB2 agonists, we
set out to examine whether CB2 modulates primary murine
150 As both are heritable, we
set out to examine whether there is a genetic correlatio
151 This paper
sets out to examine key issues associated with biorefini
152 In this study we
set out to explain the differing effects of parabiosis w
153 Here, we
set out to explore dynamic aspects of twister RNA foldin
154 vity of peptides frequently overlap, we here
set out to explore possible antibacterial effects of int
155 We
set out to explore the prevalence and determinants of no
156 We
set out to explore the role of IL-17 in the host respons
157 Here, we
set out to explore what role FFA2 may play in regulation
158 ealed evidence of covert DWV-A infection, we
set out to explore whether this effect is due to DWV-A m
159 Here, we first
set out to express and purify the full length Reelin pro
160 Herein, we
set out to functionally characterize a novel HCM-associa
161 We then
set out to further explore and validate our hypothesis i
162 We
set out to further investigate this by studying patients
163 In this study, we
set out to further understand CD27 as an immunomodulator
164 ever been experimentally mapped, and we here
set out to gain atomistic level insights into how TETS i
165 Here, we
set out to gain further insights into the function of SM
166 antibody used has pleiotropic effects, so we
set out to gain insight into the underlying mechanism by
167 Here, we
set out to generate a model in which to decipher signali
168 n a review of reviews undertaken in 2014, we
set out to give an overview of the major approaches to a
169 This study
set out to highlight the in vitro and in vivo antifungal
170 This synopsis
sets out to highlight recent advances in the field of am
171 We
set out to identify and phenotype allergen-responsive ce
172 e melanocortin and mesolimbic DA systems, we
set out to identify both the efferent projection pattern
173 Here, we
set out to identify Ca(2+)-triggering mechanisms for dop
174 We
set out to identify disease-relevant protein changes in
175 We
set out to identify factors that bind these amyloids and
176 We
set out to identify factors that promote arterial endoth
177 We
set out to identify GCC inhibitors that may be of benefi
178 tory processes that link these responses, we
set out to identify genes that govern early responses to
179 We
set out to identify genetic alterations that underlie re
180 In this study, we
set out to identify genetic modifiers of collagen deposi
181 We
set out to identify geographical characteristics that co
182 To investigate this, we
set out to identify large chromosomal domains of epigene
183 Here, we
set out to identify mechanisms of MEK inhibitor resistan
184 Here we
set out to identify mechanisms of specificity in protein
185 We
set out to identify microbial strains with GLP-1 stimula
186 on of up to several hundred target genes, we
set out to identify microRNAs that target genes in all p
187 We
set out to identify muscles whose stimulation produced a
188 Using this knowledge, we
set out to identify new candidates for context genes whi
189 We
set out to identify novel and functionally important end
190 pounds currently in clinical development, we
set out to identify novel effective treatments for T-PLL
191 In individuals of African ancestry, we
set out to identify null and damaging missense variants,
192 human tumor development and progression, we
set out to identify PMN-MDSCs and M-MDSCs in clinical ca
193 Prospective Evaluation and Risk Assessment)
set out to identify risk factors for development of pain
194 We
set out to identify small molecules that enhance UCP1 le
195 We therefore
set out to identify specific target antigens in PAH lung
196 Here we
set out to identify the biological mechanism affected by
197 Thus, we
set out to identify the core promoter region and the tra
198 Here, we
set out to identify the determinants for the extended ne
199 We
set out to identify the genes responsible for this exclu
200 te significantly from those in the brain, we
set out to identify the optimal algorithm for PUFA in br
201 o the regulation of Glut4 palmitoylation, we
set out to identify the palmitoyl acyltransferase (PAT)
202 f a single cyclopean image (cyclopotopy), we
set out to identify the stage in visual processing at wh
203 We
set out to identify transgender women and their partners
204 We
set out to identify whether maternal copy number variant
205 Here, we
set out to identify which cell(s) contribute to colonic
206 ucidate the regulatory mechanism of Wor1, we
set out to identify Wor1-interacting proteins using a ye
207 of cGAS-dependent cytosolic DNA sensing, we
set out to investigate a role for TRIM proteins in regul
208 Here we
set out to investigate adaptation to loss of BRCA2 focus
209 We
set out to investigate DMS as a means for separating ato
210 Henceforth, we
set out to investigate effects of 3 weeks' exposure to c
211 Here, we
set out to investigate effects of UV-A and solar-simulat
212 We
set out to investigate how PGE2 regulates human ILC2 fun
213 We
set out to investigate if the failure of cultured mesenc
214 We therefore
set out to investigate mechanisms of excitability within
215 In the current study, we
set out to investigate NAbs in the recovered subjects di
216 The present study
set out to investigate non-social MSI stimuli and their
217 We
set out to investigate spatial hospital accessibility fo
218 We here
set out to investigate the biochemical function of its y
219 Nearly 50 years ago, I
set out to investigate the clinical problem of hypoglyce
220 Therefore, we
set out to investigate the effects of sexing on bovine s
221 We
set out to investigate the emerging intratumoral heterog
222 benefit and ultimately survival outcomes, we
set out to investigate the fate of MDSCs after transfer
223 Here we
set out to investigate the genome-wide localization patt
224 In this study, we
set out to investigate the long-term repeatilibity and r
225 To this end, we
set out to investigate the metabolic effects of NO in cu
226 We
set out to investigate the possibility of reducing gastr
227 We
set out to investigate the possible role of microRNAs in
228 In this study, we
set out to investigate the relationship between patholog
229 We
set out to investigate the relationship between prevalen
230 Here we
set out to investigate the role of MBL in the immune res
231 We therefore
set out to investigate the role of RIPK1 in the developm
232 We
set out to investigate the role that cytokine signaling
233 2020;189(7):660-670)
set out to investigate this relationship within 2 of the
234 d later inflammatory profiles, Huang et al.,
set out to investigate this relationship within one of t
235 Therefore, we
set out to investigate whether DJ-1 plays a role in HD.
236 Here, we
set out to investigate whether elicitation of mucosal an
237 Here, we
set out to investigate whether Galphai-GIV is a druggabl
238 e affinity of FcgammaR for the Fc domain, we
set out to investigate whether radioimmunoconjugates wit
239 Here we
set out to investigate whether such type of patterned ne
240 Here, we
set out to investigate whether the main histone acetyltr
241 This study
set out to investigate whether these chemokines could se
242 are often generated in immune responses, we
set out to investigate whether these factors are relevan
243 Here we
set out to learn more about the underlying mechanism by
244 Therefore, we
set out to look for impaired use of distal cues for spat
245 We
set out to map electrical patterns of disorganization an
246 Therefore, we
set out to map H2-IA(b)-restricted epitopes along the PV
247 This study
set out to map the "S-sulfhydrome" (ie, the spectrum of
248 important determinants of viral tropism, we
set out to map the CV-A24 receptor repertoire and establ
249 Herein, we
set out to maximize the discriminating power of HRM + SV
250 We
set out to molecularly characterize LELC-B through RNA e
251 Here, we
set out to prevent clustering of extra centrosomes.
252 We
set out to probe the role of PurAH in macroamidine forma
253 ductive and transforming viral infection, we
set out to provide the first robust estimate of the prev
254 standardize the (13)C-MFA modeling work, we
set out to publish a user-friendly and programming-free
255 We
set out to quantify and understand how WGS compares with
256 Here, we
set out to quantify Mcp5 organization and identify the b
257 In the current study, we
set out to quantify the ability of these sorting methods
258 Therefore, we
set out to resolve this controversy.
259 We
set out to reveal the mechanisms by which embryos are pr
260 Given these unexpected findings, we
set out to revisit the role of Hif-1alpha in cell-autono
261 ative and Gavi, the Vaccine Alliance, UNICEF
set out to secure access to IPV supply for around 100 co
262 In this work, we
set out to specifically test this hypothesis in the plan
263 Here, we
set out to study how specific subtypes of cerebellar nuc
264 Here we
set out to study systematically the neuroadaptive change
265 We
set out to study the contribution of common genetic vari
266 -infected intravenous drug users (IVDUs), we
set out to study the effects of smoke exposure, chronic
267 Here, we
set out to study the functional interaction of 18 relate
268 We
set out to study the influence of previously described M
269 nd E2F3b have divergent regulatory roles, we
set out to study the putative transcriptional role of th
270 We
set out to study the role of MOF in general hematopoiesi
271 important roles in cell fate transitions, we
set out to study their function during the glial progeni
272 sis for studies of antimalarial efficacy, we
set out to survey parasite carriage in 3 communities in
273 either of these variants acts alone, we have
set out to systematically analyze in a large cohort of h
274 To resolve this issue, we
set out to systematically characterize and compare the c
275 We
set out to systematically identify these subsets in huma
276 This review
set out to systematically investigate whether sex ratios
277 We
set out to take a different approach and mimic the synth
278 In the current study, we
set out to test the effectiveness of targeted chemothera
279 conventional immunohistology observation, we
set out to test the hypothesis that morphology of tumor-
280 We
set out to test the hypothesis that OGs are generated in
281 Therefore, we
set out to test this hypothesis, by manipulating SRSF6 l
282 Here we
set out to test this vaccination strategy in the ferret
283 We
set out to test whether maternal voluntary exercise duri
284 Here, we
set out to test whether RNAi triggers targeting ATXN1 co
285 We
set out to test whether these complex group behaviours c
286 We
set out to test whether these two accounts of learning p
287 We
set out to uncover additional targeting proteins using u
288 We
set out to understand how aggregated proteins are manage
289 equisite for ExPEC-mediated pathogenesis, we
set out to understand how ExPEC colonizes this niche.
290 We
set out to understand the effect of these mutations.
291 Here, we
set out to understand the extent to which shifts in geno
292 We
set out to understand the functions of the ATXN1-CIC com
293 Here, we
set out to understand the heterogeneity of HbF activatio
294 County Department of Public Health (LAC DPH)
set out to understand the impact of COVID-19 on healthca
295 The current study
set out to understand the structure-activity relationshi
296 We
set out to understand the underlying mechanisms of these
297 This review
sets out to understand the reactivity of diradicals and
298 ssue of the Journal, Isanaka et al.:861-869)
set out to update an incidence correction factor used fo
299 ents diagnosed between 1985 and 2005, and we
set out to update it by incorporating more recently repo
300 molecular structure and bulk properties, we
set out to vary the electron affinity of the molecular b