ライフサイエンスコーパス: seta

1 onuclear infiltrates predominated in SeA and SeTA.

2 se to predicting the tip size of Tokay gecko seta.

3 thesis loosening (septic total arthroplasty [SeTA]; 9 specimens), rheumatoid arthritis (RA; 25 specim

4 In co-immunoprecipitation experiments, SETA and AIP1 interacted and could form a complex with a

5 Endogenous SETA and AIP1 proteins showed similar patterns of staini

6 erence in the interaction between endogenous SETA and AIP1 sensitizes astrocytes to apoptosis in resp

7 in DeltaEGFR resulted in the dissociation of SETA and Cbl proteins and a concomitant attenuation of r

8 We show that both SetA and SetB can transport lactose and glucose.

9 ric enterotoxin called ShET1, encoded by the setA and setB genes.

10 sgrS genes are located directly upstream of setA, and this gene organization is conserved in numerou

11 In addition, strains that hyperexpress SetA are resistant to the growth inhibitory sugar analog

12 Each seta branches into hundreds of 200-nm spatulae that make

13 Despite previous in vitro evidence that SetA can promote efflux of the stress-causing glucose an

14 Ruk/CIN85/SETA/CD2BP3 and CD2AP/CMS/METS-1 comprise a new family o

15 However, SETA/CIN85 and Alix were capable of mutually promoting t

16 attenuating the interaction between Cbl and SETA/CIN85 and by inhibiting Cbl-mediated ubiquitination

17 vel of Alix weakened the interaction between SETA/CIN85 and Cbl and reduced the tyrosine phosphorylat

18 This antagonism of the Cbl-SETA/CIN85 complex by Alix was reflected in its diminuti

19 It has been suggested that SETA/CIN85 promotes receptor internalization by recruiti

20 Cbl and the level of ubiquitination of EGFR, SETA/CIN85, and Cbls.

21 educed its interaction with binding partners SETA/CIN85, epidermal growth factor receptor, and Pyk2.

22 The assembly of the Cbl-SETA/CIN85-endophilin complex at the C terminus of the e

23 We found that the SETA/CIN85-interacting protein Alix/AIP1, which also bin

24 ith this, Alix interaction did not occur via SETA/CIN85.

25 s at low intensity and does not bind Cbls or SETA/CIN85.

26 by the presence of the Alix binding partner SETA/CIN85.

27 in of 85 kDa/regulator of ubiquitous kinase (SETA/CIN85/Ruk).

28 teric species, prompting the hypothesis that SetA contributes to the glucose-phosphate stress respons

29 mplemented by setA in trans, suggesting that SetA contributes to the optimal cellular recovery from s

30 SETA encodes a variety of adapter proteins containing ei

31 on that the lack of interaction with the Cbl-SETA-endophilin complex is because of differences in sig

32 This study demonstrates that setA expression is induced in response to glucose-phosph

33 taining expressed in tumorigenic astrocytes (SETA) gene is associated with the tumorigenic state in a

34 uced its polyubiquitination, suggesting that SETA has a regulatory function in the formation of the E

35 In addition, SetA has broad substrate specificity, with preferences f

36 calized with the PtdIns(3)P markers FYVE and SetA in cotransfected cells.

37 tress conditions that can be complemented by setA in trans, suggesting that SetA contributes to the o

38 d by the chemical probe, S-ethylacetimidate (SETA), in order to evaluate conformational changes as a

39 acroscopic orientation and preloading of the seta increased attachment force 600-fold above that of f

40 experiments showed that the SH3-N domain of SETA interacted with the proline-rich C terminus of AIP1

41 cells grown at high density, high levels of SETA interfered in the recruitment of Casitas B-lineage

42 SetA is a proton motive force-driven efflux pump capable

43 Under stress conditions, setA is cotranscribed with sgrS from the sgrS promoter.

44 de, in vivo data in this study indicate that SetA is not the major efflux pump responsible for remova

45 Each 30-130 microm long seta is only one-tenth the diameter of a human hair and

46 Measurements revealed that a seta is ten times more effective at adhesion than predic

47 kinase (Ruk) protein, also known as CIN85 or SETA, is an adaptor-type protein belonging to the CD2AP/

48 The SETA labeling/UVPD-MS methodology was used to monitor th

49 1.5 kb upstream of the setB structural gene; setA may be transcribed via readthrough of the setB tran

50 A setA mutant exhibits a growth defect under stress condit

51 he vascularized areas of the SeTA specimens (SeTA:normal ratio 1.9:1).

52 alues support the hypothesis that individual seta operate by van der Waals forces.

53 ate specificities, strains that hyperexpress SetA or SetB are desensitized to lactose analogues as me

54 w that DeltaEGFR did not interact with Cbls, SETA, or endophilin A1, providing a mechanistic explanat

55 Misexpression of a variety of SETA protein isoforms in these astrocytes revealed that

56 Furthermore, SETA proteins containing the SH3-N domain were able to s

57 Additionally, those lysines for which the SETA reactivity increased under denaturing conditions we

58 in terms of sequence coverage, allowing the SETA reactivity of greater number of lysine amines to be

59 ypical recognition consensus shared by CIN85/SETA/Ruk SH3 domains, PX(P/A)XXR.

60 rties of the three Escherichia coli members (SetA, SetB, and SetC) of the family are characterized.

61 ser extent, in the vascularized areas of the SeTA specimens (SeTA:normal ratio 1.9:1).

62 Gram stains were positive in all SeA and SeTA specimens.

63 supernumerary sporangia attached to a single seta, suggesting that it negatively regulates branching

64 ydrogen bonding, providing evidence that the SETA/UVPD-MS approach offers a versatile means to probe

65 milar to SeA but contained fewer CD3+ cells (SeTA versus SeA 0.26:1; P = 0.009) and a tendency toward

66 Adhesion of a single isolated gecko seta was equally effective on the hydrophobic and hydrop

67 The inflammatory infiltrate of SeTA was similar to SeA but contained fewer CD3+ cells (