ライフサイエンスコーパス: severe acute pancreatitis

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1 t and activin may be a therapeutic target in severe acute pancreatitis.

2 exocrine lineage-specific regeneration after severe acute pancreatitis.

3 elf-migrating jejunal tubes in patients with severe acute pancreatitis.

4 titis and enteral feeding and antibiotics in severe acute pancreatitis.

5 ral feeding has been strongly recommended in severe acute pancreatitis.

6 he usefulness of prophylactic antibiotics in severe acute pancreatitis.

7 leads to improved survival in patients with severe acute pancreatitis.

8 s concerning the management of patients with severe acute pancreatitis.

9 ctomy is performed sooner than 3 weeks after severe acute pancreatitis.

10 ocrine regeneration after the induction of a severe acute pancreatitis.

11 ortion of the morbidity and mortality during severe acute pancreatitis.

12 tted to the surgical ICU with a diagnosis of severe acute pancreatitis.

13 relationship of necrosis to organ failure in severe acute pancreatitis.

14 ined from rats 18 hrs after the induction of severe, acute pancreatitis.

15 Animal models of both severe acute pancreatitis and chronic pancreatitis have

16 ections, common in patients with moderate to severe acute pancreatitis, are additionally problematic.

17 ponse genes in initiation and development of severe acute pancreatitis as a model of acute inflammati

18 BP was significantly raised in patients with severe acute pancreatitis but would seem to be of limite

19 ticular risk are those with septic shock and severe acute pancreatitis, but the adverse effects of IA

20 gative predictive values of the test to show severe acute pancreatitis compared with mild acute pancr

21 ular fluid status during the early course of severe acute pancreatitis, compared with a treatment str

22 luid resuscitation was started 2 hours after severe acute pancreatitis induction and continued for 6

23 ues of stroke volume index assessed prior to severe acute pancreatitis induction as therapeutic hemod

24 diated vasodilation before and 6 hours after severe acute pancreatitis induction, revealed less impai

25 In severe acute pancreatitis, intensive medical support and

26 R in 4 (2.8%) and CTRC in 2 (1.4%) patients; severe acute pancreatitis, mutation of CFTR and CTRC in

27 ) genetic, (4) autoimmune, (5) recurrent and severe acute pancreatitis, or (6) obstructive (TIGAR-O s

28 nificant decreased risk associated with both severe acute pancreatitis, (OR 0 .

29 2) Should patients with severe acute pancreatitis receive prophylactic antibioti

30 The outcome of severe acute pancreatitis relates to its pulmonary and s

31 sceral fat necrosis has been associated with severe acute pancreatitis (SAP) for over 100 years; howe

32 Initial management of severe acute pancreatitis (SAP) is conservative.

33 sepsis, ALI is also a major complication in severe acute pancreatitis (SAP).

34 hospital volume and outcome in patients with severe acute pancreatitis (SAP).

35 an effort to salvage patients afflicted with severe acute pancreatitis (SAP).

36 anagement of the critically ill patient with severe acute pancreatitis (SAP).

37 group, stroke volume index assessed prior to severe acute pancreatitis served as primary hemodynamic

38 pancreatitis, cftr (-/-) mice developed more severe acute pancreatitis than wild-type, as indicated b

39 In severe acute pancreatitis, the administration of fluids

40 Within the first 6 hours of severe acute pancreatitis, the study group received a to

41 feeding tube is often used in patients with severe acute pancreatitis to prevent gut-derived infecti

42 The optimal threshold for predicting severe acute pancreatitis was 100 mL.

43 By comparison, severe acute pancreatitis was associated with significan

44 Moderate to severe acute pancreatitis was defined as > 5 Ranson prog

45 sensitize cftr (-/-) mice to developing more severe acute pancreatitis with an exuberant pancreatic i

46 f delaying cholecystectomy after moderate to severe acute pancreatitis with early cholecystectomy.

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