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1 ediate global urgency, such as influenza and severe acute respiratory syndrome.
2 patic infections such as influenza virus and severe acute respiratory syndrome.
3 ric diseases in animals and humans including severe acute respiratory syndrome.
4 ory disease in humans and animals, including severe acute respiratory syndrome.
5 with MERS closely matches that of those with severe acute respiratory syndrome.
6 emergence of human disease epidemics such as severe acute respiratory syndrome, AIDS, dengue, and inf
7 ding HIV and the coronavirus associated with severe acute respiratory syndrome, and is also exploited
9 ting and characterizing new pathogens (e.g., severe acute respiratory syndrome-associated coronavirus
11 a two-segmented virus expressing GFP and the severe acute respiratory syndrome-associated coronavirus
15 r coronaviruses, the membrane (M) protein of severe acute respiratory syndrome-associated coronavirus
16 animal species that can be infected with the severe acute respiratory syndrome-associated coronavirus
20 gens, including the recently emerged viruses severe acute respiratory syndrome-associated coronavirus
21 read of infectious diseases, including SARS (severe acute respiratory syndrome), avian influenza, and
22 ections, including varicella, influenza, and severe acute respiratory syndrome, can be associated wit
23 and biological evaluation of peptidomimetic severe acute respiratory syndrome chymotrypsin-like prot
24 nase) is required for the replication of the severe acute respiratory syndrome coronavirus (SARS CoV)
27 of several dangerous pathogens (e.g. HIV and severe acute respiratory syndrome coronavirus (SARS)) vi
30 a number of higher-affinity variants of the severe acute respiratory syndrome coronavirus (SARS-CoV)
32 ne-protective and immunopathogenic events in severe acute respiratory syndrome coronavirus (SARS-CoV)
33 failure was caused by a newly emerged virus, severe acute respiratory syndrome coronavirus (SARS-CoV)
38 The most prominent of these viruses is the severe acute respiratory syndrome coronavirus (SARS-CoV)
45 haracterized the cellular immune response to severe acute respiratory syndrome coronavirus (SARS-CoV)
46 mechanisms used by different viruses such as severe acute respiratory syndrome coronavirus (SARS-CoV)
49 atory viruses, including influenza virus and severe acute respiratory syndrome coronavirus (SARS-CoV)
50 onverting enzyme 2 (hACE2), the receptor for severe acute respiratory syndrome coronavirus (SARS-CoV)
60 observed when gene expression profiles from severe acute respiratory syndrome coronavirus (SARS-CoV)
62 immunodeficiency virus (HIV), influenza, and severe acute respiratory syndrome coronavirus (SARS-CoV)
64 It is believed that a novel coronavirus, severe acute respiratory syndrome coronavirus (SARS-CoV)
67 ation of nsp3a, the N-terminal domain of the severe acute respiratory syndrome coronavirus (SARS-CoV)
70 re nonstructural protein-15 (nsp15) from the severe acute respiratory syndrome coronavirus (SARS-CoV)
73 e characterized the structures of N-NTD from severe acute respiratory syndrome coronavirus (SARS-CoV)
77 e glycoprotein, termed the spike protein, of severe acute respiratory syndrome coronavirus (SARS-CoV)
79 lphacoronavirus and Betacoronavirus, such as severe acute respiratory syndrome coronavirus (SARS-CoV)
83 phylogenetically divergent counterpart from severe acute respiratory syndrome coronavirus (SARS-CoV)
85 ted residues in pFPs of two other beta-CoVs, severe acute respiratory syndrome coronavirus (SARS-CoV)
87 n-like protease (SARS-3CLpro) inhibitors for severe acute respiratory syndrome coronavirus (SARS-CoV)
88 (IgG1), a human monoclonal antibody against severe acute respiratory syndrome coronavirus (SARS-CoV)
89 we report the three-dimensional structure of severe acute respiratory syndrome coronavirus (SARS-CoV)
91 localization in Vero cells infected with the severe acute respiratory syndrome coronavirus (SARS-CoV)
98 RS was identified to be a novel coronavirus, severe acute respiratory syndrome coronavirus (SARS-CoV)
99 bat coronavirus which is much closer to the severe acute respiratory syndrome coronavirus (SARS-CoV)
105 omain (RBD) of the spike (S) glycoprotein of severe acute respiratory syndrome coronavirus (SARS-CoV)
106 ced by its counterpart from the N protein of severe acute respiratory syndrome coronavirus (SARS-CoV)
108 against the papain-like protease (PLpro) of severe acute respiratory syndrome coronavirus (SARS-CoV)
112 of the first highly pathogenic coronavirus, severe acute respiratory syndrome coronavirus (SARS-CoV)
113 pro were determined and compared to those of severe acute respiratory syndrome coronavirus (SARS-CoV)
114 coronaviruses (CoVs), including ancestors of severe acute respiratory syndrome coronavirus (SARS-CoV)
115 requirement slightly different from that of severe acute respiratory syndrome coronavirus (SARS-CoV)
117 LXCXE/D) in the majority of the Nsp15 of the severe acute respiratory syndrome coronavirus (SARS-CoV)
119 (EC(50) 39 microM) against cowpox virus and severe acute respiratory syndrome coronavirus (SARSCoV)
122 he present study showed the association of a severe acute respiratory syndrome coronavirus (SCoV) acc
124 5'UTRs) from mouse hepatitis virus (MHV) and severe acute respiratory syndrome coronavirus (SCoV) wer
125 lines and primary cells examined, productive severe acute respiratory syndrome coronavirus (Urbani st
127 s which carry a macro domain: Coronaviridae (severe acute respiratory syndrome coronavirus and human
128 to the entry pathway recently described for severe acute respiratory syndrome coronavirus and indica
129 t are important agents of disease, including severe acute respiratory syndrome coronavirus and Middle
131 es of the S, M, and N structural proteins of severe acute respiratory syndrome coronavirus and two ot
133 nslated region (3' UTR) of the genome of the severe acute respiratory syndrome coronavirus can functi
137 It was recently shown that the 7a protein of severe acute respiratory syndrome coronavirus induces bi
138 icantly influence disease outcomes following severe acute respiratory syndrome coronavirus infection
139 sin RTD-1 protects mice from an experimental severe acute respiratory syndrome coronavirus infection,
140 The replication/transcription complex of severe acute respiratory syndrome coronavirus is compose
141 aring an insertion of the SR region from the severe acute respiratory syndrome coronavirus N protein
143 notated nucleic acid-binding region (NAB) of severe acute respiratory syndrome coronavirus nonstructu
144 sidues of mNsp15 are superimposable with its severe acute respiratory syndrome coronavirus ortholog.
145 use they had no effect on the ability of the severe acute respiratory syndrome coronavirus papainlike
146 o predicted heptad repeat regions within the severe acute respiratory syndrome coronavirus S protein.
148 DPUP has close structural similarity to the severe acute respiratory syndrome coronavirus unique dom
149 ar viral entry mechanisms (such as HIV-1 and severe acute respiratory syndrome coronavirus) can be in
151 uses, including henipaviruses, lyssaviruses, severe acute respiratory syndrome coronavirus, and filov
152 ad antiviral spectrum includes HIV-1, HSV-1, severe acute respiratory syndrome coronavirus, and influ
153 ith highly pathogenic avian influenza virus, severe acute respiratory syndrome coronavirus, and Middl
154 engue virus, Ebola virus, influenza A virus, severe acute respiratory syndrome coronavirus, and West
155 ratory disease were found, including (i) the severe acute respiratory syndrome coronavirus, associate
156 tick-borne encephalitis virus, rabies virus, severe acute respiratory syndrome coronavirus, human imm
164 s to assess the interactions between MBL and severe acute respiratory syndrome-coronavirus (SARS-CoV)
165 rburg, Nipah, Hendra, and rabies viruses and severe acute respiratory syndrome-coronavirus (SARS-CoV)
167 of the Middle East respiratory syndrome and severe acute respiratory syndrome coronaviruses for cell
168 hyl glutaminyl fluoromethyl ketones (fmk) as severe acute respiratory syndrome coronovirus (SARS-CoV)
170 thogenic human pathogens of zoonotic origin: severe acute respiratory syndrome CoV (SARS-CoV) and Mid
171 including DNA proofreading enzymes, and the severe acute respiratory syndrome CoV (SARS-CoV) nsp14 h
172 piratory syndrome coronavirus (MERS-CoV) and severe acute respiratory syndrome CoV (SARS-CoV) represe
173 ond cleavage site has been identified in the severe acute respiratory syndrome CoV (SARS-CoV) S2 doma
174 bind to its receptor, whereas the beta-CoVs severe acute respiratory syndrome CoV in group B and Mid
175 Ds from infectious bronchitis virus and from severe acute respiratory syndrome CoV revealed that, alt
176 es (CoVs) revealed an adjacent Ubl domain in severe acute respiratory syndrome CoV, Middle East respi
180 cluding Middle East respiratory syndrome and severe acute respiratory syndrome, generating significan
181 aviruses, including the etiological cause of severe acute respiratory syndrome, has significantly inc
182 ance by using data from the 2003 epidemic of severe acute respiratory syndrome in Hong Kong, People's
184 d re-emerging infectious diseases, including severe acute respiratory syndrome-like coronaviruses, he
185 ory Syndrome coronavirus (MERS-CoV) causes a Severe Acute Respiratory Syndrome-like disease with appr
191 es utilize host cell cyclophilins, including severe acute respiratory syndrome (SARS) and human immun
192 that cause two emerging diseases of humans, severe acute respiratory syndrome (SARS) and Middle East
193 been discovered, including those that cause severe acute respiratory syndrome (SARS) and Middle East
194 us diseases in humans and animals, including severe acute respiratory syndrome (SARS) and Middle East
195 rus (CoV), including the causative agents of severe acute respiratory syndrome (SARS) and Middle East
196 ts that have spread internationally, such as severe acute respiratory syndrome (SARS) and the 2009 pa
197 s potentially lethal zoonotic agents causing severe acute respiratory syndrome (SARS) and the recentl
198 The pathogenesis and optimal treatments for severe acute respiratory syndrome (SARS) are unclear, al
199 s highlighted during the global emergence of severe acute respiratory syndrome (SARS) by numerous 'su
200 -term protection against future outbreaks of severe acute respiratory syndrome (SARS) caused by a nov
202 a prophylactic antiviral in a mouse model of severe acute respiratory syndrome (SARS) coronavirus (Co
203 inoviruses (RhVs), enteroviruses (EnVs), and severe acute respiratory syndrome (SARS) coronavirus (Co
205 an unusual three-stemmed pseudoknot from the severe acute respiratory syndrome (SARS) coronavirus (Co
209 coronavirus (BCoV) and 339-nt 3' UTR in the severe acute respiratory syndrome (SARS) coronavirus (SC
211 L (EC 3.4.22.15) and as an entry blocker of severe acute respiratory syndrome (SARS) coronavirus and
212 y, the predicted stem-loop IV homolog in the severe acute respiratory syndrome (SARS) coronavirus app
217 tenuated vaccine consisting of a recombinant severe acute respiratory syndrome (SARS) coronavirus lac
218 '-proximal open reading frames (ORFs) of the severe acute respiratory syndrome (SARS) coronavirus may
222 he PLP domains of human coronavirus NL63 and severe acute respiratory syndrome (SARS) coronavirus to
224 olecule inhibitor that blocks replication of severe acute respiratory syndrome (SARS) CoV and murine
225 ting enzymes (DUBs), we confirmed that, like severe acute respiratory syndrome (SARS) CoV PLpro, HCoV
232 ng that occurred during the 2003 outbreak of severe acute respiratory syndrome (SARS) in Hong Kong, C
247 imization of a lead dipeptide-like series of severe acute respiratory syndrome (SARS) main protease (
249 icture was remarkably similar to that of the severe acute respiratory syndrome (SARS) outbreak in 200
251 inflammatory glucocorticoids in the lungs of severe acute respiratory syndrome (SARS) patients are un
255 nal experiments revealed, unexpectedly, that severe acute respiratory syndrome (SARS) S-mediated entr
259 ic health emergencies involving anthrax, the severe acute respiratory syndrome (SARS), and shortages
260 coronaviruses, including the one that caused severe acute respiratory syndrome (SARS), cause signific
261 o be critical factors in the pathogenesis of severe acute respiratory syndrome (SARS), caused by a co
264 ing human pathogens hepatitis C virus (HCV), Severe acute respiratory syndrome (SARS), coxsackie viru
265 ses (CoVs), including the causative agent of severe acute respiratory syndrome (SARS), encode a nucle
267 eginning of the 2003 Singaporean outbreak of Severe Acute Respiratory Syndrome (SARS), providing new
273 pathologic features observed in the lungs of severe acute respiratory syndrome (SARS)-coronavirus-inf
276 enic human coronaviruses (CoVs), such as the severe acute respiratory syndrome (SARS)-CoV and the Mid
278 d that HCoV-NL63 and the genetically distant severe acute respiratory syndrome (SARS)-CoV employ the
279 demonstrate that engineered inactivation of severe acute respiratory syndrome (SARS)-CoV ExoN activi
281 des analogous to the aromatic domains of the severe acute respiratory syndrome (SARS)-CoV, mouse hepa
284 nthetic replicating life form, a 29.7-kb bat severe acute respiratory syndrome (SARS)-like coronaviru
286 as received considerable attention since the severe acute respiratory syndrome (SARS)-like CoV was id
287 e coronavirus (MERS-CoV) caused outbreaks of severe acute respiratory syndrome (SARS)-like illness wi
288 HV-A59 background were able to reproduce the severe acute respiratory syndrome (SARS)-like pathology
290 central segment of the previously annotated severe acute respiratory syndrome (SARS)-unique domain (
296 cellular receptor for the causative agent of severe-acute respiratory syndrome (SARS), SARS-CoV (coro
297 infections with epidemic potential, such as severe acute respiratory syndrome, swine-origin influenz
298 Herein, we show that the E protein in the severe acute respiratory syndrome virus has only one TM
299 the mutations onto the crystal structure of severe acute respiratory syndrome virus nsp10 identified
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