ライフサイエンスコーパス: severe aplastic anemia

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1 mbopag in previously untreated patients with severe aplastic anemia.

2 l responses in some patients with refractory severe aplastic anemia.

3 ophosphamide is highly effective therapy for severe aplastic anemia.

4 produces durable treatment-free remission in severe aplastic anemia.

5 ar survival in patients who received HCT for severe aplastic anemia.

6 Severe aplastic anemia (AA) is a bone marrow (BM) failur

7 induces durable treatment-free remissions in severe aplastic anemia and a variety of other autoimmune

8 ion of the skin in an 11-year-old child with severe aplastic anemia and prolonged neutropenia is repo

9 whereas MECOM patients presented early-onset severe aplastic anemia, and ERCC6L2 patients, mild pancy

10 preferred for children and young adults with severe aplastic anemia, and immunosuppressive therapy is

11 d unrelated donor bone marrow transplant for severe aplastic anemia as a manifestation of Schwachman-

12 ferior to horse ATG as a first treatment for severe aplastic anemia, as indicated by hematologic resp

13 rbent assays in 18 consecutive patients with severe aplastic anemia before and after treatment with h

14 ons in telomere length have been reported in severe aplastic anemia but their clinical significance i

15 ge-adjusted telomere length of patients with severe aplastic anemia consecutively enrolled in immunos

16 d 1993, 40 patients received transplants for severe aplastic anemia from related donors other than HL

17 o a distinct cell surface receptor and cause severe aplastic anemia in vivo and erythroblast destruct

18 Severe aplastic anemia is a life-threatening bone marrow

19 In a cohort of patients with severe aplastic anemia receiving immunosuppressive thera

20 nic immune thrombocytopenia, Evans syndrome, severe aplastic anemia/refractory cytopenia, and others.

21 About a quarter of patients with severe aplastic anemia remain pancytopenic despite immun

22 Severe aplastic anemia (SAA) appears to be an immunologi

23 Severe aplastic anemia (SAA) can be successfully treated

24 d donor bone marrow (BM) transplantation for severe aplastic anemia (SAA) has improved, with survival

25 Survival in severe aplastic anemia (SAA) has markedly improved in th

26 Severe aplastic anemia (SAA) is a life-threatening bone

27 Severe aplastic anemia (SAA) is a life-threatening bone

28 Acquired severe aplastic anemia (SAA) is a rare hematologic disea

29 First-line therapy of severe aplastic anemia (SAA) with high-dose cyclophospha

30 In contrast to severe aplastic anemia (sAA), the appropriate management

31 e is effective in restoring hematopoiesis in severe aplastic anemia (SAA).

32 ed conditioning regimen in HSCT for acquired severe aplastic anemia (SAA).

33 geneic bone marrow transplantation (BMT) for severe aplastic anemia (SAA).

34 y) has been promoted as curative therapy for severe aplastic anemia (SAA).

35 results were noted in analyses stratified on severe aplastic anemia subtype, recipient age, HLA match

36 of hematologic response among patients with severe aplastic anemia than in a historical cohort.

37 in the great majority of young patients with severe aplastic anemia; the major challenges are extendi

38 dy, we analyzed results in 700 patients with severe aplastic anemia treated with allogeneic marrow tr

39 Approximately half of patients with severe aplastic anemia treated with antithymocyte globul

40 gkin Lymphoma, 1 Chronic Myeloid Leukemia, 2 Severe Aplastic Anemia) undergoing allo-HSCT.

41 Between 1970 and 1996, 333 patients with severe aplastic anemia underwent HLA-matched related mar

42 Bone marrow transplants for severe aplastic anemia were first performed in the 1970s

43 Severe aplastic anemia, which is characterized by immune

44 oach deserves further study in patients with severe aplastic anemia who are not suitable candidates f

45 le institution analysis of 183 patients with severe aplastic anemia who were treated in sequential pr

46 son Cancer Research Center for patients with severe aplastic anemia whose donors were HLA-nonidentica

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