コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 kidney capsules of SCID mice (ie, mice with severe combined immunodeficiency).
2 benefits with gene therapy for ADA-deficient severe combined immunodeficiency.
3 ng Tgfb1-null mutant mice on a background of severe combined immunodeficiency.
4 in Matrigel, and formed tumors in mice with severe combined immunodeficiency.
5 ratoma assays in nonobese diabetic mice with severe combined immunodeficiency.
6 c), which is mutated in humans with X-linked severe combined immunodeficiency.
7 ion and subsequently received a diagnosis of severe combined immunodeficiency.
8 logical process defective in human and mouse severe combined immunodeficiency.
9 Amish infant hospitalized in Minnesota with severe combined immunodeficiency.
10 reased sensitivity to ionizing radiation and severe combined immunodeficiency.
11 ression is defective in humans with X-linked severe combined immunodeficiency.
12 ntified in patients with NHEJ deficiency and severe combined immunodeficiency.
13 ngs with T cell, B cell, natural killer cell severe combined immunodeficiency.
14 d kinase, Jak3, are the major cause of human severe combined immunodeficiency.
15 ients display radiosensitivity combined with severe combined immunodeficiency.
16 )), which is mutated in humans with X-linked severe combined immunodeficiency.
17 in humans and null mutations in mice lead to severe combined immunodeficiency.
18 ly, such protection occurred without causing severe combined immunodeficiency.
19 ally attenuated MM tumor growth in mice with severe combined immunodeficiency.
20 inactivated in human radiosensitive T(-)B(-) severe combined immunodeficiency, a syndrome characteriz
21 affected with adenosine deaminase-deficient severe combined immunodeficiency (ADA-SCID) and set out
22 c contrast, 39 adenosine deaminase-deficient severe combined immunodeficiency (ADA-SCID) patients hav
23 'difficult' conditions [adenosine deaminase-severe combined immunodeficiency (ADA-SCID), major histo
24 ites that destroy the immune system, causing severe combined immunodeficiency (ADA-SCID), often refer
25 clude adenosine deaminase deficiency-related severe combined immunodeficiency (ADA-SCID), Shwachman-B
27 clinical case of an RSV-infected infant with severe combined immunodeficiency and effectively no adap
29 g its goal of defining the true incidence of severe combined immunodeficiency and providing early tre
30 ells injected intraperitoneally (ie, in CB17 severe combined immunodeficiencies) and significantly in
31 , diagnosis of adenosine deaminase-deficient severe combined immunodeficiency, and consanguinity were
33 g in California established the incidence of severe combined immunodeficiency at 1 in 66,250 live bir
35 ) were given intranasally or i.p. to newborn severe combined immunodeficiency-beige mice exposed to 9
37 yptosporidium parasites from 6 of 7 infected severe combined immunodeficiency-beige mice, and the par
38 raftment is a common finding in infants with severe combined immunodeficiency but is not typically ob
39 ions in Janus kinase 3 (JAK3) are a cause of severe combined immunodeficiency, but hypomorphic JAK3 d
40 ete recovery occurred in VLP-dosed mice with severe combined immunodeficiency, but not in wild-type m
41 correct T-cell deficiencies in patients with severe combined immunodeficiency by replacing resident t
42 delivered via osmotic pump in an intratibial severe combined immunodeficiency CAG myeloma model or in
43 eficiency CAG myeloma model or in a systemic severe combined immunodeficiency CAG-heparanase model of
45 ells has emerged as a convincing therapy for severe combined immunodeficiency caused by ILR2G mutatio
46 chain cytokine receptor subunit give rise to severe combined immunodeficiency characterized by lack o
48 orrection of disease phenotypes for X-linked severe combined immunodeficiency, chronic granulomatous
50 was unequivocal evidence that children with severe combined immunodeficiency could be cured by gene
54 n of EBV-CTLs both in vitro and in vivo in a severe combined immunodeficiency disease (SCID) mouse mo
57 s, whereas transfers into BALB/c x Rag-/- or severe combined immunodeficiency disease mice (H-2d) did
58 ligoclonal lines into either C3H x Rag-/- or severe combined immunodeficiency disease mice (H-2k) als
61 12 was found to be mutated in patients with severe combined immunodeficiency disease, and expression
63 d adults with specific forms of PID, such as severe combined immunodeficiency, for over 10 years.
64 ications of anti-CD3epsilon mAb treatment in severe combined immunodeficiency forms characterized by
65 r from healthy donors were injected into NOD-severe combined immunodeficiency gammac(-/-) mice, follo
67 y CLL cells proliferate in nonobese diabetes/severe combined immunodeficiency/gammac(null) mice under
68 and patients with atypical presentations of severe combined immunodeficiency gene mutations presents
69 treatment advances and newborn screening for severe combined immunodeficiency, has resulted in improv
70 patients with adenosine deaminase deficient severe combined immunodeficiency have identified neutrop
71 HTLV-1-infected humanized nonobese diabetic severe combined immunodeficiency (HU-NOD/SCID) mice were
74 gnificantly inhibited C4-2B cell growth in a severe combined immunodeficiency-human model of experime
75 homing, and engraftment in nonobese diabetic/severe combined immunodeficiency IL-2gamma(null) (NSG) m
76 HEJ factor ARTEMIS cause radiation-sensitive severe combined immunodeficiency in humans and may incre
77 geneic hosts as well as in nonobese diabetic/severe combined immunodeficiency/interleukin 2Rgamma(nul
78 nstitute immune-deficient (nonobese diabetic/severe combined immunodeficiency/interleukin-2 gammac re
79 Therefore, we created a nonobese diabetic/severe combined immunodeficiency/interleukin-2 receptor-
80 stitution by transplanting nonobese diabetic/severe combined immunodeficiency/interleukin-2 receptor-
81 Spontaneously diabetic NOD mice received NOD severe combined immunodeficiency islet transplants and w
82 gene replacement for Hemophilia B, X-linked Severe Combined Immunodeficiency, Leber's Congenital Ama
83 oss of T cells in a spectrum including leaky severe combined immunodeficiency (LS) and Omenn syndrome
84 ndent growth in vitro, and tumorigenicity in severe combined immunodeficiency mice (all P < 0.05).
85 lial cells, leading to increased survival of severe combined immunodeficiency mice after transplantat
86 (+) fetal liver cells into nonobese diabetic/severe combined immunodeficiency mice allows for the lon
87 n vivo after subcutaneous transplantation in severe combined immunodeficiency mice and differentiated
88 umab and copper-DOTA-conatumumab was done in severe combined immunodeficiency mice bearing Colo205 xe
90 ession by PET and ex vivo biodistribution in severe combined immunodeficiency mice bearing H2009 tumo
91 in vivo experiments, 6- to 12-wk-old female severe combined immunodeficiency mice bearing M21 xenogr
92 ol Tyr(4)-BBN: for in vivo GRPR blockade) in severe combined immunodeficiency mice bearing PC-3 xenog
93 d not reduce infectivity or pathogenicity in severe combined immunodeficiency mice but resulted in cl
94 A33 antibodies greatly delayed the deaths of severe combined immunodeficiency mice challenged with va
95 of human erythrocytes into nonobese diabetic/severe combined immunodeficiency mice extends blood circ
96 LL cells transplanted onto nonobese diabetic/severe combined immunodeficiency mice faithfully recapit
98 omib significantly prolonged the survival of severe combined immunodeficiency mice inoculated with LC
99 in vivo and pathogenesis are not available, severe combined immunodeficiency mice into which human t
100 myofibers in the injured skeletal muscle of severe combined immunodeficiency mice more effectively t
102 tumor transplantation in Non-obese diabetic/severe combined immunodeficiency mice that the HAGE knoc
103 plantation in xenotolerant nonobese diabetic/severe combined immunodeficiency mice through intrasplen
104 ansplanted nonobese diabetic background with severe combined immunodeficiency mice to assess suppress
105 s (PBMCs) were injected into the footpads of severe combined immunodeficiency mice to measure human d
108 Humanized urokinase plasminogen activator/severe combined immunodeficiency mice were used to estab
109 transplanted with MSCs in nonobese diabetic severe combined immunodeficiency mice with a significant
110 antly improved survival of nonobese diabetic/severe combined immunodeficiency mice with HL-60 leukemi
111 ease severity during persistent infection of severe combined immunodeficiency mice with isogenic sero
112 nd decreased tumour volumes and mortality of severe combined immunodeficiency mice xenografted with P
113 EB-silenced MM cells were then injected into severe combined immunodeficiency mice, and tumor growth
115 f human coronary artery were transplanted to severe combined immunodeficiency mice, followed by recon
116 MSCs was not observed in non-obese diabetic/severe combined immunodeficiency mice, indicating the im
118 esis in immunocompromised non-obese diabetic/severe combined immunodeficiency mice, supporting an onc
119 o a different genogroup, are inoculated into severe combined immunodeficiency mice, the order of seve
120 E. chaffeensis strains were inoculated into severe combined immunodeficiency mice, the order of the
121 ng MDA-MB-231 cell-derived mammary tumors in severe combined immunodeficiency mice, we show here for
142 tudies were performed in Mec-1-bearing SCID (severe combined immunodeficiency) mice, a new animal mod
147 nd virulence in human skin xenografts in the severe combined immunodeficiency mouse model in vivo.
148 tensively in infected skin xenografts in the severe combined immunodeficiency mouse model of VZV path
150 bound to BM using both in vitro and in vivo severe combined immunodeficiency mouse models of human M
151 assayable human cells, the nonobese diabetic/severe combined immunodeficiency mouse repopulating cell
152 d engraftment in NOD-SCID (nonobese diabetic-severe combined immunodeficiency) mouse myocardium incre
153 tem with experimental systemic infections of severe combined immunodeficiency Mus musculus with the b
155 under the renal capsule of nonobese diabetic severe combined immunodeficiency (NOD SCID) mice with MS
156 l CD34(+) progenitors into nonobese diabetic/severe combined immunodeficiency (NOD-SCID) mice resulte
157 itt lymphoma xenografts in nonobese diabetic/severe combined immunodeficiency (NOD/scid) animals comp
159 ition, we demonstrate that nonobese diabetic/severe combined immunodeficiency (NOD/SCID) leukemia-ini
160 aclizumab was evaluated in nonobese diabetic/severe combined immunodeficiency (NOD/SCID) mice given i
161 cells into immunodeficient nonobese diabetic severe combined immunodeficiency (NOD/SCID) mice leads t
162 th allogeneic T cells into nonobese diabetic-severe combined immunodeficiency (NOD/SCID) mice resulte
163 A-overexpressing tumors in nonobese diabetic/severe combined immunodeficiency (NOD/SCID) mice with pa
164 lation assays performed on nonobese diabetic/severe combined immunodeficiency (NOD/SCID) mice, and in
167 and after engraftment in a nonobese diabetic/severe combined immunodeficiency (NOD/SCID) mouse xenogr
168 hat when transplanted into nonobese diabetic/severe combined immunodeficiency (NOD/SCID) recipient mi
169 ere divided into 3 main categories: T(-)B(-) severe combined immunodeficiency, Omenn syndrome, and co
170 ency causes T(-)B(+) natural killer-positive severe combined immunodeficiency or T-cell lymphopenia w
171 mmune system since it did not occur in nude, severe combined immunodeficiency, or T-cell depleted mic
172 ce represent a model for radiation-sensitive severe combined immunodeficiency, our findings suggest t
173 emis nuclease is defective in radiosensitive severe combined immunodeficiency patients and is require
174 h overexpression of Bcl2 does not rescue the severe combined immunodeficiency phenotype in Ku70-defic
177 ons are responsible for the development of a severe combined immunodeficiency [radiation-sensitive (R
178 rchy contains a rare CD34(-) population with severe combined immunodeficiency-repopulating capacity.
179 telangiectasia and a class of Radiosensitive-Severe Combined Immunodeficiency (RS-SCID), respectively
181 - and B-lymphocyte-independent protection of severe combined immunodeficiency SCID mice from dissemin
183 fection, wild-type (WT) and C3H animals with severe combined immunodeficiency (SCID animals) were ino
184 success in select diseases such as X-linked severe combined immunodeficiency (SCID) and ADA deficien
185 function of adenosine deaminase (ADA) cause severe combined immunodeficiency (SCID) and affect many
186 n the brain, brain stem, and spinal cords of severe combined immunodeficiency (SCID) and C57BL/6 (wil
187 with ARTEMIS deficiency usually present with severe combined immunodeficiency (SCID) and cellular rad
188 alues and severely impaired dissemination in severe combined immunodeficiency (SCID) and immunocompet
189 oietic-cell transplantation in children with severe combined immunodeficiency (SCID) and other primar
194 sgenic for hHGF/SF (designated hHGF-Tg) on a severe combined immunodeficiency (SCID) background.
197 tivating gene 1 (RAG1) deficiency results in severe combined immunodeficiency (SCID) caused by a comp
200 to the pathology of Omenn syndrome and leaky severe combined immunodeficiency (SCID) has not been pre
209 s of galectin-1 were higher in tumor-bearing severe combined immunodeficiency (SCID) mice breathing 1
210 L-6)-dependent human MM cell line INA-6 into severe combined immunodeficiency (SCID) mice previously
211 tumor antigen-specific TCR, we reconstituted severe combined immunodeficiency (SCID) mice with bone m
222 udies using a CD4+CD45Rbhigh T-cell transfer severe combined immunodeficiency (SCID) mouse inflammato
223 ignificant prolonged survival in a xenograft severe combined immunodeficiency (SCID) mouse model of d
224 ed autophagy in human skin xenografts in the severe combined immunodeficiency (SCID) mouse model of V
225 wth of RARalpha2-overexpressing MM tumors in severe combined immunodeficiency (SCID) mouse model.
226 me for at least a month in both nude rat and severe combined immunodeficiency (SCID) mouse xenograft
227 se of nonobese diabetic (NOD) mice, NOD with severe combined immunodeficiency (scid) mutation (SCID)
228 fetal liver cells in nonobese diabetic (NOD)/severe combined immunodeficiency (SCID) or NOD/SCID/gamm
229 ic and nonimmunologic outcomes in cohorts of severe combined immunodeficiency (SCID) patients with ei
231 Cl 4 fibrosis were adoptively transferred to severe combined immunodeficiency (SCID) recipients, whic
232 asmid patterns, were infectious in mice with severe combined immunodeficiency (SCID) regardless of th
234 Although gene therapy can cure patients with severe combined immunodeficiency (SCID) syndromes, the c
235 onsortium (PIDTC) is enrolling children with severe combined immunodeficiency (SCID) to a prospective
237 We report here a patient with T(-)B(+)NK(+) severe combined immunodeficiency (SCID) who was homozygo
239 ations that impair Rag2 function can lead to severe combined immunodeficiency (SCID), a condition cha
240 the 91 position found in some patients with severe combined immunodeficiency (SCID), and the double
241 d T-cell reconstitution in many infants with severe combined immunodeficiency (SCID), but correction
242 have been identified, often associated with severe combined immunodeficiency (SCID), consistent with
243 used into young horses (foals) affected with severe combined immunodeficiency (SCID), followed by cha
244 of typical profound T-cell dysfunction (TD), severe combined immunodeficiency (SCID), has been carefu
245 the cervical spinal cords of adult mice with severe combined immunodeficiency (SCID), human pluripote
246 cell transplantation (HCT) for patients with severe combined immunodeficiency (SCID), including survi
247 hly relevant per se because in patients with severe combined immunodeficiency (SCID), infections caus
248 minase (ADA) deficiency, a cause of X-linked severe combined immunodeficiency (SCID), is a case in po
249 t profound primary immunodeficiency disease, severe combined immunodeficiency (SCID), is fatal in inf
251 the natural history of patients treated for severe combined immunodeficiency (SCID), Wiskott-Aldrich
252 abrogate lymphocyte development and lead to severe combined immunodeficiency (SCID), XLF mutations c
254 suppressed the growth of MM in vivo using a severe combined immunodeficiency (SCID)-hu murine model.
255 tations in ORAI1 or STIM1 genes present with severe combined immunodeficiency (SCID)-like disease.
272 sies were injected intravenously into C.B-17 severe combined immunodeficiency (SCID)/beige (bg) mice.
273 cured immunodeficiencies including X-linked severe combined immunodeficiency (SCID-X1) and adenine d
276 ical trials involving children with X-linked severe combined immunodeficiency (SCID-X1), a Moloney mu
277 fective vectors for gene therapy of X-linked severe combined immunodeficiency (SCID-X1), we have eval
282 sseminating infections in wild-type (WT) and severe-combined-immunodeficiency (SCID) mice was analyze
283 growth in vivo in hairless outbred mice with severe combined immunodeficiency (SHO-Prkdc(scid)Hr(hr))
284 ell lymphopenia, patients with ADA-deficient severe combined immunodeficiency showed a partial block
286 ations that inactivate Artemis cause a human severe combined immunodeficiency syndrome associated wit
288 established tumors (ie, in nonobese diabetic-severe combined immunodeficiencies) that were derived fr
291 nts with adenosine deaminase (ADA)-deficient severe combined immunodeficiency using 2 slightly differ
292 root ganglion (DRG) xenografts in mice with severe combined immunodeficiency using multiscale correl
293 is with neutrophil infiltration in mice with severe combined immunodeficiency, which is accompanied b
294 natural killer cell-sufficient patient with severe combined immunodeficiency, whom we found had muta
295 r insertion in a clinical trial for X-linked severe combined immunodeficiency (X-SCID) has prompted s
296 of LMO2 following gene therapy for X-linked severe combined immunodeficiency (X-SCID) have led to a
299 an restore immunity to infants with X-linked severe combined immunodeficiency (XSCID) caused by mutat
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。