ライフサイエンスコーパス: severe hepatitis

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1 ion occurs with high frequency and can cause severe hepatitis.

2 of staining in many cells, concomitant with severe hepatitis.

3 d 37% of patients with genotype 2a or 2b had severe hepatitis.

4 patitis (F <or= 2/6, NI <or= 3/18), moderate/severe hepatitis (3 <or= F < 6 or NI >or= 4/18), and cir

5 were abnormal serum potassium (32/97 [33%]), severe hepatitis (54/92 [59%]), and raised C-reactive pr

6 rwent liver biopsy, only 13% had evidence of severe hepatitis (8%) or cirrhosis (5%), despite a durat

7 Severe hepatitis A infection is an infrequent but well-r

8 deficient in inhibitory FcgammaRIIb had more severe hepatitis and accelerated mortality.

9 itonavir were reported to be associated with severe hepatitis and acute liver failure.

10 stingly, ILC2 depletion correlated with less severe hepatitis and reduced accumulation of eosinophils

11 these entities, mice experience increasingly severe hepatitis and tissue necrosis and die within a fe

12 VTTs in control, mild hepatitis, moderate or severe hepatitis, and cirrhosis groups were 38.3 seconds

13 levels of platelets, multifocal moderate to severe hepatitis, and perivascular edema.

14 patients had mild hepatitis; 18, moderate or severe hepatitis; and 10, cirrhosis.

15 post ConA injection, Wt mice presented with severe hepatitis as assessed by increased liver transfer

16 antagonist flutamide had significantly less severe hepatitis as determined by histologic activity in

17 infected with MHV-CXCL10 were protected from severe hepatitis as evidenced by reduced pathology and s

18 here have been no published reports of acute severe hepatitis associated with the use of OKT3 in non-

19 Until now, however, no cases of severe hepatitis attributed to zafirlukast have been rep

20 tation (OLTX) in patients retransplanted for severe hepatitis B virus (HBV) in the first allograft ha

21 fication of chemotherapy in patients who had severe hepatitis B virus flares.

22 opportunistic infection, graft rejection and severe hepatitis C virus recurrence).

23 9 (MHV-A59) produces meningoencephalitis and severe hepatitis during acute infection.

24 We describe here a case of acute severe hepatitis encountered during treatment of acute r

25 e liver failure, decompensated cirrhosis, or severe hepatitis flare) and in those with compensated ci

26 atic failure in a patient and, subsequently, severe hepatitis in a chimpanzee (CH1422), to analyze th

27 tomegalovirus strain v70 induces a rapid and severe hepatitis in immunocompetent mice that requires t

28 BALB/c females developed more severe hepatitis (p<0.01) and produced more pro-inflamma

29 Castrated males developed more severe hepatitis than did intact males (p<0.001) and fem

30 e infected with HhG and HhNET developed less-severe hepatitis than male A/JCr mice infected with Hh3B

31 Patient 3 developed severe hepatitis that improved after treatment with cort

32 occur-though the occurrence of accompanying severe hepatitis was rare.

33 low RT-PCR cycle threshold (<20 cycles), and severe hepatitis were independently associated with mort

34 an be stratified into those with moderate to severe hepatitis, who merit imminent therapy, and those

35 in the liver, similar to MHV-2, and induced severe hepatitis with extensive hepatocellular necrosis.

36 etransplantation (2 = chronic rejection; 1 = severe hepatitis with panacinar necrosis, resembling ori

37 somewhat lower titer and induced moderate to severe hepatitis with zonal necrosis, similar to MHV-A59

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