ライフサイエンスコーパス: severe hypertension

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1 cardial reperfusion injury in the setting of severe hypertension.

2 nd further raises blood pressure, leading to severe hypertension.

3 n of kidney dysfunction and outcome in acute severe hypertension.

4 outcome in patients hospitalized with acute severe hypertension.

5 ension are rare genetic disorders that cause severe hypertension.

6 Cyp4a14, a murine homologue of CYP4A11, have severe hypertension.

7 e rats (SHR), a widely used genetic model of severe hypertension.

8 mented sympathetic output compared with more severe hypertension.

9 reated groups progressed from less severe to severe hypertension.

10 ormone aldosterone and are a common cause of severe hypertension.

11 However, 1K1C produced more severe hypertension (164+/-2 mm Hg) and cardiac hypertro

12 mably as a consequence of abrupt, transient, severe hypertension and catecholamine release.

13 in pathogenesis in a subset of patients with severe hypertension and implicate loss of K(+) channel s

14 morbidity among patients admitted with acute severe hypertension, and AKI is a frequent form of acute

15 -protein signaling-2)-deficient mice exhibit severe hypertension, and genetic variations of RGS2 occu

16 gulator of G-protein signaling-2 (RGS2) have severe hypertension, and RGS2 genetic variations occur i

17 At baseline, patients had severe hypertension (aortic systolic pressure, 176+/-26

18 ms and provided DNA samples but did not have severe hypertension, aortic prosthesis, or significant a

19 ovariate data, from 2,044 sibling pairs with severe hypertension, collected by the British Genetics o

20 (STAT) registry enrolled patients with acute severe hypertension, defined as > or =1 blood pressure m

21 er, reported significantly more frequent and severe hypertension, diabetes, cancer, heart disease, ar

22 Dahl salt-sensitive rats develop severe hypertension during a high-sodium diet, but the b

23 on factor is required for the development of severe hypertension during chronic angiotensin II signal

24 roducing adrenal adenomas (APAs), a cause of severe hypertension, feature constitutive hormone produc

25 Severe hypertension (>/=160/110 mm Hg) developed in 40.6

26 A few forms of severe hypertension have been linked to single genes.

27 Severe hypertension in ACE2-deficient mice was associate

28 Renovascular disease is a frequent cause of severe hypertension in children and may result in signif

29 e despite having no effect on the sustained, severe hypertension induced by L-NAME.

30 entricular rhythm disorders (n = 22 [0.7%]), severe hypertension (n = 24 [0.8%]) and hypotension or l

31 with hypokalaemia, together with those with severe hypertension or a family history of hypertension

32 in undetermined, the current study rules out severe hypertension or advanced renal disease as sole ca

33 beta1 subunit during genetic borderline and severe hypertension reduced BK channel activity by decre

34 In an ASA subject with severe hypertension refractory to antihypertensive medic

35 However, so far, only patients with severe hypertension (systolic BP >/=160 mm Hg or >/=150

36 activated ion channel, in turn resulting in severe hypertension that is resistant to most forms of c

37 25 d after the induction of severe hypertension with deoxycorticosterone acetate and

38 KCNJ5 mutation in a Mendelian form of early severe hypertension with massive adrenal hyperplasia.

39 duced FBF, altered flow distribution, and/or severe hypertension with the progression toward diabetes

40 e aldosterone and cause millions of cases of severe hypertension worldwide.

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