ライフサイエンスコーパス: severe pancreatitis

コーパス検索結果 (１語後でソート)

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1 sk of bias) included patients with predicted severe pancreatitis.

2 ce in mediating the progression from mild to severe pancreatitis.

3 extravascular (migrated) neutrophils only in severe pancreatitis.

4 ncreatitis, but lung fMLFK only increased in severe pancreatitis.

5 the time of drainage were considered to have severe pancreatitis.

6 ain strains of mice, Coxsackievirus causes a severe pancreatitis.

7 n patients had mild pancreatitis, and 23 had severe pancreatitis.

8 e radicals, which potentiate organ injury in severe pancreatitis.

9 as-ligand double-deficient mice die early of severe pancreatitis.

10 in those patients having CT scan findings of severe pancreatitis.

11 t, and 1 patient died because of concomitant severe pancreatitis.

12 of benefit in the treatment of patients with severe pancreatitis.

13 Coxsackievirus infection causes severe pancreatitis and myocarditis in humans, often lea

14 itical steps in the progression from mild to severe pancreatitis and responsible for many of its syst

15 went biliopancreatic duct ligation to induce severe pancreatitis, and rPAF-AH administration was begu

16 replicated to high titers in vivo and caused severe pancreatitis but minimal myocarditis.

17 all significantly increased in both mild and severe pancreatitis, but lung fMLFK only increased in se

18 n of oxidative stress to the pathogenesis of severe pancreatitis by examining the prevalence of funct

19 caused by caerulein and more prominently in severe pancreatitis caused by feeding a choline-deficien

20 Moderate-to-severe pancreatitis developed in 13 patients (4.4%) in t

21 diet administered by nasogastric tube during severe pancreatitis does not worsen outcome compared wit

22 IL-33R, T1/ST2, significantly developed more severe pancreatitis, had greater weight loss, and contai

23 Earlier animal studies of moderate and severe pancreatitis have shown that blockade of this pow

24 t admission predicted the development of the severe pancreatitis in different groups of the patients.

25 ECLS is useful in treating severe pancreatitis-induced ARDS.

26 During severe pancreatitis, interleukin (IL)-1beta and tumor ne

27 Among patients with severe pancreatitis, limited evidence revealed no statis

28 Mild and severe pancreatitis models in the rat.

29 evalence of PPCF was higher in patients with severe pancreatitis (n = 16 [70%]) than in those with mi

30 ateral exocytosis in part explained the less severe pancreatitis observed in Munc18c(+/-) mice after

31 Severe pancreatitis occurs in 20%-30% of all patients wi

32 given ethanol or fatty acids developed more severe pancreatitis than mice not given ethanol or fatty

33 Moderate and severe pancreatitis were characterized by much greater l

34 Infection with CVB4-V results in an early, severe pancreatitis, which can lead to mortality or prog

35 IER3 expression was induced by both mild and severe pancreatitis, which promoted PanIN formation and

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