ライフサイエンスコーパス: severely burned patient

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1 ar target to improve the clinical outcome of severely burned patients.

2 nas aeruginosa is a major cause of sepsis in severely burned patients.

3 PP_PA14 (PA14) was grown in whole blood from severely burned patients.

4 n a more favorable muscle protein balance in severely burned patients.

5 articipating in exercise programs, and seven severely burned patients.

6 ipid metabolism across the splanchnic bed in severely burned patients.

7 creased rate of muscle protein catabolism in severely burned patients.

8 have examined possible clinical benefits in severely burned patients.

9 re appears to be a harbinger of mortality in severely burned patients.

10 cose, fatty acids, and protein metabolism in severely burned patients.

11 ltidisciplinary approach to the treatment of severely burned patients.

12 nit resulted in improved fluid management of severely burned patients.

13 was detected in serum specimens derived from severely burned patients.

14 monocytes isolated from peripheral blood of severely burned patients (19 of 19 patients) were demons

15 PBMC from 41 of 45 severely burned patients (91%) failed to produce macroph

16 se kinetics and muscle protein metabolism in severely burned patients and assess any potential benefi

17 Severely burned patients are susceptible to infections w

18 substitutes can facilitate wound closure in severely burned patients, but deficiencies limit their o

19 ave been developed to guide resuscitation in severely burned patients during the initial 48 hrs after

20 All severely burned patients exhibited burn-induced insulin

21 Cardiopulmonary resuscitation (CPR) in severely burned patients experiencing cardiac arrest (CA

22 This system of care for severely burned patients facilitates the transfer of the

23 and further supports the use of metformin in severely burned patients for postburn control of hypergl

24 Severely burned patients had increased muscle protein de

25 The severely burned patients increased the workload of the b

26 Muscle protein catabolism was accelerated in severely burned patients, leading to a progressive loss

27 es) were demonstrated in peripheral blood of severely burned patients (M2a, 2 of 19 patients; M2c, 5

28 Fatty liver occurs in severely burned patients, often resulting in a twofold i

29 we tried to induce Th17 cells in cultures of severely burned patient PBMC by stimulation with the C.

30 lted in decreased morbidity and mortality in severely burned patients returning from war zones.

31 ociated with hypoglycemia we suggest that in severely burned patient's blood glucose of 130 mg/dL sho

32 ia and increases muscle protein synthesis in severely burned patients, thereby indicating a metabolic

33 The high susceptibility of severely burned patients to C. albicans infection might

34 Severely burned patients were shown to be carriers of M2

35 cytes are predominant in peripheral blood of severely burned patients who are carriers of CCL2 that f

36 Lipid kinetics were studied in six severely burned patients who were treated with a high do

37 he ability of insulin to improve outcomes of severely burned patients with infected burn wounds is no

38 expression on Mo, are not fully explored in severely burned patients with sepsis.

39 studies, primarily in adults, suggested that severely burned patients with tracheostomies have a high

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