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   2 a provide evidence for the importance of the sex-limited chromosome in a female heterogametic species
     3 ation, gene expression levels decline on the sex-limited chromosome, and this can lead to selection f
  
  
  
  
     8  neurodevelopmental disorders, and suggest a sex-limited effect on the penetrance of the pathological
  
    10 h male lethality is probably responsible for sex-limited expression of these disorders, as affected w
  
  
    13 ance, which include sex-linked transmission, sex-limited fertility reduction, and X-autosome epistasi
  
  
    16 rehensive genomic sampling of biparental and sex-limited genetic variation to identify and control fo
    17  collection of D. pulex clones suggests that sex-limited meiosis suppression in D. pulex has spread w
    18 st striking examples of sexual dimorphism is sex-limited mimicry in butterflies, a phenomenon in whic
  
  
  
    22 0-base pair enhancer, derived from the mouse sex-limited protein (Slp) gene, is activated solely by t
  
  
    25 erwise male-specific liver proteins, such as sex-limited protein (Slp), major urinary proteins (MUPs)
    26 gene expression, most dramatically for mouse sex-limited protein (Slp), which provides an in vivo rep
  
    28 ns requires the N/C interaction, whereas the sex-limited protein gene and mouse mammary tumor virus l
  
  
    31 a large number of families and is frequently sex limited: Sex linkage has not yet been demonstrated, 
    32 wever, because the majority of genes are not sex-limited, the potential for substantial conflict may 
    33 design because the spermatozoal phenotype is sex-limited (to males) while the genotype is sex-linked 
  
  
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