ライフサイエンスコーパス: sexual transmission

1 ecent studies have shown an increase through sexual transmission.

2 e ingredient of a microbicide to prevent HIV sexual transmission.

3 eports regarding the evidence for or against sexual transmission.

4 preciated role in decreasing the rate of HIV sexual transmission.

5 irus propagation and dissemination following sexual transmission.

6 of microbicides as a method to prevent HIV-1 sexual transmission.

7 s HIV trans-infection and may play a role in sexual transmission.

8 nt as a vaginal microbicide to prevent HIV-1 sexual transmission.

9 er human immunodeficiency virus (HIV) during sexual transmission.

10 bility to human immunodeficiency virus (HIV) sexual transmission.

11 nea pigs received approximately 10(2) IFU by sexual transmission.

12 ts of a topical microbicide to prevent HIV-1 sexual transmission.

13 mains essential to understanding the risk of sexual transmission.

14 es should focus on pathogenesis and modes of sexual transmission.

15 the male reproductive system poses a risk of sexual transmission.

16 both rapid disease progression and enhanced sexual transmission.

17 ce that HIV-1 selection indeed occurs during sexual transmission.

18 d to investigate potential mechanisms of HIV sexual transmission.

19 on from recovered patients have been through sexual transmission.

20 st immune cells that HIV-1 encounters during sexual transmission.

21 t immune cells to interact with HIV-1 during sexual transmission.

22 anogenital stratified squamous mucosa during sexual transmission.

23 ils that are exploited by HIV to promote its sexual transmission.

24 the first cell types to encounter HIV during sexual transmission.

25 evention strategies to protect against HIV-1 sexual transmission.

26 immunodeficiency virus (HIV) at the point of sexual transmission.

27 1 entry to prevent early dissemination after sexual transmission.

28 IV-1 infection may be a determinant of HIV-1 sexual transmission.

29 onhospital healthcare (clinic) settings; and sexual transmission among HIV-infected persons.

30 infection in the male reproductive tract and sexual transmission, an ability to cross the placenta du

31 ons may help explain the high rates of HIV-1 sexual transmission and accelerated HIV-1 disease progre

32 very different in the anatomical context of sexual transmission and begin to explain the lack of sti

33 ptimize treatment, and the relative roles of sexual transmission and endogenous infection in BV epide

34 ion in cerebrospinal fluid (CSF) and between sexual transmission and human immunodeficiency virus typ

35 in HIV-negative patients are consistent with sexual transmission and local immunity, whereas in HIV-p

36 The likelihood of sexual transmission and persistence of DRM was assessed

37 are the first cells to encounter HIV during sexual transmission and their interaction with HIV may d

38 to expanded prevention programs targeted at sexual transmission and transmission among injecting dru

39 ates due to injection drug use and secondary sexual transmission and with an apparent West Coast focu

40 ation with abnormal fetal brain development, sexual transmission, and lack of a preventive vaccine ha

41 ave not as yet resulted in either disease or sexual transmission, and may represent benign endpoint i

42 us (EBOV) RNA persistence in semen, reported sexual transmission, and sporadic clusters at the end of

43 Since viruses isolated after sexual transmission are mainly R5 viruses, this suggests

44 d mosquito-borne flavivirus, have identified sexual transmission as a new route of disease spread, wh

45 The cluster likely resulted from sexual transmission between men having sex with men livi

46 k factors account for many cases of apparent sexual transmission between sexual partners; the prevale

47 njecting heroin users (NIUs) are a potential sexual transmission bridge to "lower risk" partners, 180

48 possibly, HCV-infected NIUs, are a potential sexual transmission bridge.

49 type 1 (HIV-1) load and reduces the risk of sexual transmission, but little is known about the effic

50 t with HHV-8 being primarily associated with sexual transmission, but the HHV-8 seropositivity rate i

51 al data strongly support the hypothesis that sexual transmission by acutely infected individuals has

52 ine the approximate infection dose in actual sexual transmission by comparing the kinetics of infecti

53 -1 is suggested to be a major contributor to sexual transmission by mucosal routes.

54 Following sexual transmission, Chlamydia trachomatis specifically

55 These findings suggest that sexual transmission constrains viral diversity at the po

56 ance mutations (DRM) may be a consequence of sexual transmission, de novo mutations, or technical err

57 These data show that sexual transmission does not select for enhanced macroph

58 Here we examine whether sexual transmission during multiple matings of queens is

59 A suspected case of sexual transmission from a male survivor of Ebola virus

60 ter transmission were the consequence of (i) sexual transmission from the source, (ii) de novo emerge

61 ut can persist in human semen and sperm, and sexual transmission has been documented.

62 alent in sub-Saharan Africa, but the role of sexual transmission has not been well characterized.

63 Anecdotal cases of sexual transmission have been reported, and HCV nucleoti

64 complications such as ocular involvement and sexual transmission have been reported.

65 Among the women with a low risk of sexual transmission, HHV-8 seroprevalence was 0 percent

66 In addition to blood transfusion and sexual transmission, HTLV-1 is transmitted mainly throug

67 These data support a role for sexual transmission in HTLV-II infection.

68 raditionally at low risk for shigellosis via sexual transmission in MSM.

69 tted among injection drug users, the role of sexual transmission in the spread of HCV remains controv

70 which certain selfish genetic elements skew sexual transmission in their own favor.

71 results suggest an approach to prevent HIV-1 sexual transmission in which a virus-sensitizing microbi

72 results suggest an approach to prevent HIV-1 sexual transmission in which a virus-sensitizing microbi

73 majority of new HIV infections occur through sexual transmission, in which HIV is transferred from th

74 rate and mortality rate of mosquitoes while sexual transmission increases the risk of infection and

75 00), in which the percentage contribution of sexual transmission is 3.044% (95% CI: 0.123-45.73).

76 However, acute HIV-1 infection via sexual transmission is a low-probability event in which

77 ealth Service has estimated that the risk of sexual transmission is approximately 5%, well below the

78 Moreover, the fraction of cases due to sexual transmission is estimated below 4% of the cumulat

79 tment might relate to evidence that suggests sexual transmission is integral to the pathogenesis of b

80 xual men, but determining specific routes of sexual transmission is methodologically challenging, and

81 Sexual transmission is negligible in sex-partner studies

82 The risk of sexual transmission is probably lower than for Chlamydia

83 Sexual transmission is the main transmission route in AG

84 Sexual transmission is the most frequent (86%) route of

85 Increasing the rate of horizontal sexual transmission means that vector control rate or in

86 This provides a sexual-transmission model of human AIDS that can be used

87 conceptual framework for the male to female sexual transmission observed during ZIKV infection.

88 From January 2003 to December 2013, sexual transmission of 2 clades of Campylobacter jejuni

89 V(+)) youth, and the attendant potential for sexual transmission of antiretroviral (ARV) drug-resista

90 reviewed trials of interventions to prevent sexual transmission of any STI, employing a multilevel p

91 Fortunately, sexual transmission of chlamydiae has been described for

92 based combination microbicide to prevent the sexual transmission of diverse HIV-1 variants.

93 ve response to the threat of persistence and sexual transmission of Ebola is required and should buil

94 Sexual transmission of Ebola virus in Liberia has now be

95 a, the genomic analysis provides evidence of sexual transmission of EBOV and evidence of the persiste

96 ne transfer (HGT) in bdelloids precludes the sexual transmission of entire haplotypes, for which we h

97 se (EVD) in Western Africa may contribute to sexual transmission of EVD and generate new clusters of

98 study did not evaluate directly the risk of sexual transmission of EVD.

99 r model suggests that BV is initiated by the sexual transmission of Gardnerella vaginalis, which has

100 Epidemiologic studies suggest that most sexual transmission of genital herpes occurs when person

101 idence is that there is no increased risk of sexual transmission of HCV among heterosexual couples in

102 e providers need to pay special attention to sexual transmission of HCV among HIV-infected individual

103 sed injection drugs; however, more recently, sexual transmission of HCV has been recognized among HIV

104 Other observations, however, weigh against sexual transmission of HCV infection.

105 voir of HCV-infected individuals is sizable, sexual transmission of HCV likely contributes to the tot

106 d seminal HCV RNA levels could contribute to sexual transmission of HCV, but other factors, including

107 te HCV infection given its potential role in sexual transmission of HCV.

108 is approximately 5%, well below the risk of sexual transmission of hepatitis B or human immunodefici

109 ations have been cited to support a role for sexual transmission of hepatitis C.

110 Sexual transmission of herpes simplex virus type 2 (HSV-

111 ve therapy with valacyclovir reduces risk of sexual transmission of herpes simplex virus type 2 (HSV-

112 be more important than semen or stool in the sexual transmission of HHV-8.

113 There is strong epidemiologic evidence for sexual transmission of high-risk genital human papilloma

114 tential candidate microbicide to prevent the sexual transmission of HIV and AIDS.

115 of the data on the use of ART to prevent the sexual transmission of HIV and identify challenges to im

116 ignificance, for example, as a ligand in the sexual transmission of HIV and stimulator of natural kil

117 on (VMMC) is capable of reducing the risk of sexual transmission of HIV from females to males by appr

118 tes in HIV-infected semen, may contribute to sexual transmission of HIV from men to their partners.

119 s could be effective tools in preventing the sexual transmission of HIV in humans.

120 During sexual transmission of HIV in women, the first cells lik

121 Strategies to prevent the sexual transmission of HIV include vaccines that elicit

122 lopment of topical microbicides that prevent sexual transmission of HIV is an active area of investig

123 ne responses required for protection against sexual transmission of HIV is essential for the developm

124 These results suggest that protection from sexual transmission of HIV may be possible by effectivel

125 Sexual transmission of HIV selects for highly fit drug-r

126 We also investigated the sexual transmission of HIV to and from these participant

127 nistered antiretroviral drugs to prevent the sexual transmission of HIV to receptive partners.

128 Only 1 intervention showed efficacy against sexual transmission of HIV, but 22 (53.7%) showed effect

129 nfectivity and may play an important role in sexual transmission of HIV, making them a potential micr

130 During sexual transmission of HIV, virus crosses mucosal epithe

131 n of antiretroviral therapy reduced rates of sexual transmission of HIV-1 and clinical events, indica

132 ve important implications for the biology of sexual transmission of HIV-1 and its potential reduction

133 Initial biologic events that underlie sexual transmission of HIV-1 are poorly understood.

134 4 cell count may be associated with enhanced sexual transmission of HIV-1 because of increased number

135 esent a novel strategy to reduce the rate of sexual transmission of HIV-1 in humans.

136 Sexual transmission of HIV-1 is an inefficient process,

137 tiviral therapy on HIV-1 in semen and on the sexual transmission of HIV-1 require further study.

138 Sexual transmission of HIV-1 requires that small amounts

139 viral entry inhibitors can help prevent the sexual transmission of HIV-1 to humans.

140 onsidered independent methods to prevent the sexual transmission of HIV-1 to women.

141 es in the earliest phases of infection after sexual transmission of HIV-1, a process that is ineffici

142 icrobicide that could be used to prevent the sexual transmission of HIV-1.

143 ecretions have important roles in preventing sexual transmission of HIV-1.

144 logic association of trichomoniasis with the sexual transmission of HIV-1.

145 rent genetic bottlenecks associated with the sexual transmission of HIV-1.

146 ecretions will improve studies that evaluate sexual transmission of HIV-1.

147 ical microbicides for their ability to block sexual transmission of HIV-1.

148 vated immune cells and have implications for sexual transmission of HIV-1.

149 ons for the selection of resistant virus and sexual transmission of HIV-1.

150 odel for testing of agents designed to block sexual transmission of HIV-1.

151 ogesterone affects mechanisms underlying the sexual transmission of HIV-1.

152 ction likely increases the efficiency of the sexual transmission of HIV-1.

153 e that such migration may be involved in the sexual transmission of HIV-1.

154 pears to protect homozygous individuals from sexual transmission of HIV-1.

155 related morbidity and mortality and prevents sexual transmission of HIV-1.

156 or HIV target cell during the early stage of sexual transmission of HIV.

157 tinually present, has the potential to block sexual transmission of HIV.

158 be useful as a topical microbicide to block sexual transmission of HIV.

159 ls may be a novel strategy to interfere with sexual transmission of HIV.

160 an immunodeficiency virus (HIV) may decrease sexual transmission of HIV.

161 g, protecting, and transferring virus during sexual transmission of HIV.

162 alis may be valuable in reducing the risk of sexual transmission of HIV.

163 vaginal flora could lead to a higher rate of sexual transmission of HIV.

164 this protein as a microbicide to prevent the sexual transmission of HIV.

165 ory role in the efficiency of male-to-female sexual transmission of HIV.

166 women and may play an important role in the sexual transmission of HIV.

167 h less advanced immunodeficiency and reduces sexual transmission of HIV.

168 with a 10- to 20-fold reduction in risk for sexual transmission of HIV.

169 they could present a natural barrier to the sexual transmission of HPV and could serve as the basis

170 accine's target disease cervical cancer, the sexual transmission of HPV was minimized, the threat of

171 A microbicide to prevent sexual transmission of HSV-2 would contribute substantia

172 d partners and condom use reduce the risk of sexual transmission of HSV-2.

173 on genital mucosal surfaces and may prevent sexual transmission of HSV.

174 The incidence of sexual transmission of HTLV-II may be similar to that of

175 Both intrauterine and sexual transmission of human herpesvirus (HHV)-6 and HHV

176 iretroviral resistance and the prevention of sexual transmission of human immunodeficiency virus (HIV

177 In the acute stage of infection following sexual transmission of human immunodeficiency virus (HIV

178 Sexual transmission of human immunodeficiency virus (HIV

179 a (SP) may reduce virus burden and influence sexual transmission of human immunodeficiency virus (HIV

180 as been associated with an increased rate of sexual transmission of human immunodeficiency virus (HIV

181 ntified recently as a cofactor that promotes sexual transmission of human immunodeficiency virus (HIV

182 (BV) is associated with an increased rate of sexual transmission of human immunodeficiency virus (HIV

183 Recent studies indicate that sexual transmission of human immunodeficiency virus type

184 Recent findings suggest that most sexual transmission of human immunodeficiency virus type

185 the body fluid most commonly associated with sexual transmission of human immunodeficiency virus type

186 ovaginal mucosa is a relative barrier to the sexual transmission of human immunodeficiency virus type

187 To explore the mechanism of sexual transmission of human immunodeficiency virus type

188 macaque model to study events that underlie sexual transmission of human immunodeficiency virus type

189 Sexual transmission of human immunodeficiency virus type

190 ve role against invading pathogens; however, sexual transmission of human immunodeficiency virus type

191 Sexual transmission of human immunodeficiency virus type

192 In the primate model of sexual transmission of human immunodeficiency virus, tre

193 V) 1% vaginal gel has been found to decrease sexual transmission of human immunodeficiency virus.

194 -alpha response is not sufficient to prevent sexual transmission of human immunodeficiency virus.

195 inhibitor of an amyloid fibril that enhances sexual transmission of human immunodeficiency virus.

196 Sexual transmission of human papilloma virus is a leadin

197 Cross-sectional studies support sexual transmission of human T lymphotropic virus (HTLV)

198 tissue, supporting a possible mechanism for sexual transmission of KSHV.

199 nd semen, suggesting that the possibility of sexual transmission of Lassa virus should be considered.

200 nalysis revealed no substantial evidence for sexual transmission of minority DRM (BF = 0.02).

201 We found no clear evidence to support the sexual transmission of minority resistant variants, and

202 infection in rhesus macaques is a model for sexual transmission of primate retroviruses.

203 In TB, lack of sexual transmission of rearranged chromosomes associates

204 In sexual transmission of simian immunodeficiency virus, an

205 nder development as a microbicide to prevent sexual transmission of the human immunodeficiency virus

206 ntial prophylactic for the prevention of the sexual transmission of the human immunodeficiency virus

207 xposure to RNA viruses is required to combat sexual transmission of this class of pathogens.

208 ay increase cervical HTLV-I shedding and the sexual transmission of this virus.

209 sterone, a sex steroid hormone, enhances the sexual transmission of various pathogens, including SIV.

210 epresent an attractive strategy for blocking sexual transmission of virus.

211 cells to come into contact with HIV-1 during sexual transmission of virus.

212 We report on 9 cases of male-to-female sexual transmission of Zika virus in the United States o

213 Also unknown is the relative importance of sexual transmission of ZIKV and asymptomatic ZIKV infect

214 the type I interferon receptor, we examined sexual transmission of ZIKV.

215 he highest viral loads, which is relevant to sexual transmission of ZIKV.

216 investigate the impact of mosquito-borne and sexual transmission on the spread and control of ZIKV an

217 Yet, the role of sexual transmission on the spread and control of ZIKV in

218 We used data from the Antiretrovirals, Sexual Transmission Risk and Attitudes (ASTRA) questionn

219 KV should target both the mosquito-borne and sexual transmission routes.

220 observation that the guinea pigs infected by sexual transmission shed organisms for a significantly s

221 Gametocytes, the sexual transmission stages, form normally and respond in

222 In the current study, we studied both sexual transmission (STx) and vertical transmission (VTx

223 Compared with other risk groups for HIV sexual transmission, such as men who have sex with men,

224 Apart from sexual transmission, the virus can also be transmitted f

225 se of needle-exchange programmes, as well as sexual transmission to injection drug users (IDUs) and s

226 transmission, postexposure prophylaxis, and sexual transmission (topical microbicide).

227 The likelihood of sexual transmission was based on viral load, knowledge o

228 A) loads were stable during observation, and sexual transmission was not attributable to an upward tr

229 Limitation: Sexual transmission was not explicitly accounted for in

230 Sexual transmission was not observed following mating of

231 Sexual transmission was the sole risk factor for infecti

232 lective pressures on Nef functions following sexual transmission, we analyzed genetic and functional

233 ne how SEVI might function in the context of sexual transmission, we applied HIV-1 and SEVI to intact

234 have specific biological properties favoring sexual transmission, we inoculated human cervical tissue

235 ority of HIV infections are acquired through sexual transmission, we investigated whether antiviral C

236 rus-cell interactions in this barrier during sexual transmission, we studied the uptake and infection

237 used a mathematical model of HPV 6/11/16/18 sexual transmission within an MSM population in England,