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1 ct axons, respectively (P < 0.05 compared to sham treatment).
2 nd PVD with less anti-VEGF use compared with sham treatment.
3 pacing at baseline and after 1 h of LLTS or sham treatment.
4 e subjected to full-thickness burn injury or sham treatment.
5 D at month 12 with ocriplasmin compared with sham treatment.
6 ght temporoparietal area was not superior to sham treatment.
7 th the 12- and 24-month visits compared with sham treatment.
8 affected eyes through 6 months compared with sham treatment.
9 oclonal antibody, antibody plus ramipril, or sham treatment.
10 and after ONC in animals receiving rtACS or sham treatment.
11 ding increased tumor size when compared with sham treatment.
12 K), compared with uptake in mice receiving a sham treatment.
13 0% total body surface area burn or a control sham treatment.
14 s -2 and -1) compared with prior episodes of sham treatment.
15 ment compared with 1 of 17 (5.9%) courses of sham treatment.
16 nar nuclei (ILN) and midline nuclei (MLN) or sham treatment.
17 stimulation significantly improved mood over sham treatment.
18 20 Hz at 80% motor threshold) and 2 weeks of sham treatment.
19 tion, or microvascular density compared with sham treatment.
20 e mice received 10 Gy cranial irradiation or sham-treatment.
21 P=0.015) and 16 weeks (P=0.04) compared with sham treatments.
22 SCs, mesenchymal stem cells, native ECs, and sham treatments.
24 ing at 60% peak aerobic power (VO2,peak)) or sham treatment (60 min seated rest) in nine healthy subj
27 mulation continues to display superiority to sham treatment and benefits similar to antimuscarinic th
28 ated) tubulins were increased, compared with sham treatment, and only Paxceed ameliorated motor impai
29 F-alpha gene transcription 4- to 5-fold over sham treatment, and TNF-alpha gene transcription increas
31 were exposed to an activating dose of IR or sham treatment as control, and nuclear extracts were ana
34 ntion, 10 participants (71.4%) randomized to sham treatment believed they had received WBH compared w
39 bserved following LFMS treatment relative to sham treatment for both diagnostic subgroups for our pri
42 n patients (37%; 95% CI, 25.9%-48.1%) in the sham treatment group vs 32 (23%; 95% CI, 15.8%-29.6%) in
43 9) units, whereas the baseline score for the sham treatment group was 32.4 (8.4) units and the week-1
46 changes were observed for the apheresis- and sham-treatment groups for endoscopic remission and respo
47 ranulocyte/monocyte apheresis (n = 112)- and sham-treatment groups, respectively (n = 56; P = .361).
48 DAPP mice at 2, 5, and 8 months after TBI or sham treatment (i.e., at 6, 9, and 12 months of age).
49 and safety of this regimen as compared with sham treatment in 807 infants in need of respiratory sup
50 and tolerability of such PDL treatment with sham treatment in patients with facial inflammatory acne
51 shown that sphincterotomy is no better than sham treatment in patients with post-cholecystectomy pai
56 r and that ethanol may modify the effects of sham treatment on gene expression, as well as inducing s
60 Anaesthetized pigs were subjected to either sham treatment, or an abrupt increase in cardiac workloa
63 mprovement in pain or function compared with sham treatment, raising questions about its value for th
65 Resuscitation with hydroxyethyl starch and sham treatment significantly decreased FIBTEM maximum cl
66 hypoglycemia (days -2 and -1) compared with sham treatment significantly enhanced baseline adrenal S
68 uncture is significantly more effective than sham treatment (standardized mean difference, 0.54 [95%
69 cale scores decreased 5 points, while during sham treatment the scores increased or worsened by 3 poi
71 s were given a course of eight spaced ECS or sham treatments under either halothane or ketamine anaes
72 nted sites were randomized to sonotherapy or sham treatment using a custom-built, 8-French catheter i
74 trials of 9 nonpharmacologic options versus sham treatment, wait list, or usual care, or of 1 nonpha
76 with MSC-VSVG treatment versus MSC alone or sham treatment was associated with decreased MSC retenti
77 enocarcinoma tumors were allocated to RFA or sham treatment with or without a STAT3 inhibitor (S3I-20
78 xt, animals were allocated to hepatic RFA or sham treatment with or without STAT3 (signal transducer
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