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1  while the peak time of the rate of response shifted to the left.
2 elicited cAMP accumulation in HeLa cells was shifted to the left.
3      Stimulus intensity-response curves were shifted to the left and displayed larger maxima in the m
4 f K(m), as the substrate dependence curve is shifted to the left and K(m) is reduced 10-fold.
5 gh the task required attention to be equally shifted to the left and to the right, eight of 10 subjec
6 to far field potentials when stimulation was shifted to the left atrium.
7    The GABA concentration-response curve was shifted to the left by low pH and non-competitively inhi
8 on relation in hypertrophied cardiocytes was shifted to the left compared with normal cells.
9   Conversely, KW-6002 produced a significant shift to the left, consistent with potentiation of the r
10 cluster from the third intracellular loop is shifted to the left due to three missing residues.
11 the normal distribution of contact areas was shifted to the left, due in part to a decreased number o
12 se curve of CBF in response to veratrine was shifted to the left; eg, 5 micrograms/kg of veratrine in
13 ally, the end-diastolic pressure-area curves shifted to the left in all patients.
14 se to lateral perforant path stimulation was shifted to the left in hippocampal slices from pregnant
15 d rats, the morphine dose-response curve was shifted to the left in sensitized as compared to non-sen
16                 The force frequency curve is shifted to the left in the FKBP12-deficient diaphragm mu
17 , a K(ATP) channel opener, was significantly shifted to the left in the inflamed smooth-muscle cells.
18 tion 623 (E623Q) displayed a titration curve shifted to the left relative to wild type channels and t
19 -state voltage dependence of Na channels was shifted to the left with almost 50% of channels unavaila
20 ve for agonist-induced cAMP accumulation was shifted to the left with increasing expression levels of

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