ライフサイエンスコーパス: shigella infection

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1 for the reduction of immune responses during Shigella infection.

2 e intestine by 24 hours post-intraperitoneal Shigella infection.

3 e; however, adult mice are resistant to oral Shigella infection.

4 protective efficacy against intraperitoneal Shigella infection.

5 p90 host-cell vinculin fragment generated by Shigella infection.

6 is essential to resistance following primary Shigella infection.

7 e interleukin-1beta is cleaved shortly after Shigella infection.

8 tic therapy for children with non-dysenteric Shigella infection.

9 y help identify patients most likely to have Shigella infection.

10 n to be altered during the course of natural Shigella infection.

11 dentified that surface loops of OmpA mediate Shigella infection.

12 lgi-associated ARF/ARL family GTPases during Shigella infection.

13 ProD was associated with Cryptosporidium and Shigella infections.

14 Ciprofloxacin is a recommended treatment for Shigella infections.

15 ella infection, 28.3 {CI, 7.2-112.2}; OR for Shigella infection, 14.5 {CI 1.5-141.0} or any recreatio

16 Abl and Arg are catalytically activated upon Shigella infection, accumulate at the site of bacterial

17 d. vaccination with IpaB and IpaD to prevent Shigella infection and support further studies in humans

18 or programmed cell death, is induced during Shigella infections and has been proposed to be a key ev

19 value of dysentery for the identification of Shigella infection, and the efficacy of antibiotics for

20 Shigella infections are a leading cause of diarrhoeal de

21 he human intestinal xenograft in response to shigella infection but failed to significantly alter tis

22 ith human intestine in vivo, we have studied shigella infection in human intestinal xenografts in sev

23 Essential events in the pathogenesis of Shigella infections include bacterial invasion of epithe

24 ction of MSD cases that were attributable to Shigella infection increased from 9.6% (n = 129) for cul

25 Western blots revealed that Shigella infection induces vinculin proteolysis in PtK2

26 sentery for identification and management of Shigella infection might miss an opportunity to reduce S

27 g infection, the susceptibility to pulmonary Shigella infection of each of various mouse strains that

28 h has been previously described for virulent Shigella infection of HMDM and is termed oncosis.

29 During Shigella infections of human macrophages, interleukin-1b

30 xhibited a 10-fold-greater susceptibility to Shigella infection than immunocompetent mice.

31 and suggest a novel approach to treatment of Shigella infections through inhibition of host cell sign

32 Shigella infection was associated with human intestinal

33 Shigella infection was associated with mortality (pooled

34 dysentery identified 1.9-85.9% of confirmed Shigella infections, with sensitivity decreasing over ti

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