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1 g, arteriovenous shunt vasoconstriction, and shivering.
2 cing the thresholds for vasoconstriction and shivering.
3 erimental cold exposure designed to minimize shivering.
4 e comfortable in the cold, and reported less shivering.
5  and clinical protocols on how to best treat shivering.
6 nverse relationship between BAT activity and shivering.
7 are arteriovenous shunt vasoconstriction and shivering.
8  can also produce heat metabolically without shivering.
9      Nine patients experienced mild episodic shivering.
10 widely expressed uncoupling protein UCP2, or shivering.
11                                              Shivering (152 [37%] vs 59 [15%]; RR 2.54, 95% CI 1.95-3
12                                              Shivering (229 [47%] vs 82 [17%]; RR 2.80, 95% CI 2.25-3
13                                              Shivering after anesthesia or in the critical care setti
14  Brown adipose tissue (BAT) functions in non-shivering and diet-induced thermogenesis via its capacit
15 iac arrest requires deep sedation to prevent shivering and discomfort.
16    Surface CW provides beneficial control of shivering and improves the metabolic profile during TTM.
17 ratory distress syndrome, but cooling causes shivering and increases metabolism.
18 ections resulted in short-lived increases in shivering and longer decreases in locomotor activity, as
19 e of thermoregulation was caused by impaired shivering and nonshivering thermogenesis, whereas thermo
20  sham controls and iBAT-ablated mice stopped shivering and resumed routine physical activity, indicat
21  ingestion at 52 degrees C rapidly decreased shivering and sensations of cold compared to 37 degrees
22 id ingestion at 22 and 7 degrees C increased shivering and sensations of coolness to similar levels,
23 n, fluid ingestion at 52 degrees C decreased shivering and the sensation of coolness, whereas fluid i
24              With LRG ingestion, compared to shivering and thermal sensations with ingestion at 37 de
25 t study, we characterize the modification of shivering and whole-body thermal sensation during cold s
26 delayed drug metabolism, prolonged recovery, shivering, and thermal discomfort.
27                    Both vasoconstriction and shivering are associated with autonomic and hemodynamic
28 s scored on a scale of 0-3 using the Bedside Shivering Assessment Scale (BSAS).
29 s in puberty, either dystonic/dyskinetic or "shivering" attacks, triggered by stretching, motor initi
30 he time of IDC, 72% of patients had signs of shivering (BSAS >0).
31 irmed that the industrial sections; cutting, shivering/crusting, and stitching were the principal con
32  were decreases in body movements, including shivering, during NREM sleep; body temperature and heart
33 scuss complications of hypothermia including shivering, electrolyte abnormalities, hemodynamic change
34 , and brown adipose tissue temperatures; and shivering EMGs in anesthetized rats following central an
35 ) and clonidine (100 mug/kg, i.v.) inhibited shivering EMGs, BAT SNA, and BAT thermogenesis, effects
36 y control, and prevents vasoconstriction and shivering in blocked areas.
37 -blocking agents may be used to manage overt shivering in therapeutic hypothermia.
38                                     Although shivering is one alternative mechanism of thermogenesis
39                                              Shivering occurred more frequently in the Arctic Sun gro
40 n in a dose-dependent manner without causing shivering or altering physiological parameters.
41  use (p = 0.03), baseline moderate to severe shivering (p = 0.04), and lower serum magnesium levels (
42                                          The shivering response appeared to be delayed and much reduc
43  on four occasions, to induce a steady-state shivering response, at which point two aliquots of 1.5 m
44 mine whether visceral thermoreceptors modify shivering responses to cold stress.
45 nalysis using linear comparisons calculating shivering risk-reduction ratios.
46  misoprostol group more commonly experienced shivering (RR 1.91, 95% CI 1.65 to 2.21, p<0.001) and fe
47                                              Shivering severity during each phase was scored on a sca
48  thermogenesis is a key player in muscle non-shivering thermogenesis (NST) and can compensate for los
49 ults indicate a potent inhibition of BAT and shivering thermogenesis by alpha2-AR activation in the r
50 bition of rat brown adipose tissue (BAT) and shivering thermogenesis by alpha2-AR agonists.
51  in obesity, but the effect of impairing non-shivering thermogenesis in BAT on lipid storage in WAT r
52 protein-1 (UCP1), which mediate adaptive non-shivering thermogenesis in mammals.
53 nes on brown adipose tissue suggest that non-shivering thermogenesis is an arena for intragenomic con
54                                          Non-shivering thermogenesis is required for survival of rode
55 to an inhibition of brown adipose tissue and shivering thermogenesis that is mediated by neurons in t
56 nd acute cold tolerance (partly a measure of shivering thermogenesis) in s/s mice were modestly but s
57 the first day of cold, a time of intense non-shivering thermogenesis, AMPK activity remained at basal
58 s, re-routing fatty acids to BAT to fuel non-shivering thermogenesis.
59 apacity of brown adipose tissue and thus non-shivering thermogenesis.
60 ssue, and not in the acute activation of non-shivering thermogenesis.
61 rd pathway mediates not only sympathetic and shivering thermogenic responses but also metabolic and c
62                                          The shivering threshold is usually a full 1 degrees C below
63 travenous meperidine were used to reduce the shivering threshold.
64 , triggering of the ventilator and degree of shivering) to assess the degree of neuromuscular blockad
65                                              Shivering was common in the misoprostol group, and nause
66 AT thermogenesis and survive by compensatory shivering with consequent acute weight loss.

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