ライフサイエンスコーパス: shock state

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1 some of the pathophysiologic derangements in shock states.

2 organ preservation and transplantation, and shock states.

3 nd estimation of the severity of circulatory shock states.

4 ponse quantitator of severity of circulatory shock states.

5 ncorporates serum lactate as a biomarker for shock states.

6 ough it is released in response to stress or shock states, a relative deficiency of vasopressin has b

7 bserve weak correlations between plagioclase shock state and some REE+Y systematics (e.g., La/Y and S

8 d, includes serum lactate as a biomarker for shock states, and incorporates all vasopressors used in

9 proach to the study of acute inflammation in shock states, and suggest hypotheses with regard to the

10 forms of PSGL-1 are able to ameliorate acute shock states by suppressing selectin-mediated leukocyte-

11 ynamic measurements in severe cases showed a shock state characterized by a low cardiac index (range

12 Due to the wide array of shock states dealt with by intensivists, an integrated a

13 eakly shocked (<15 GPa) and examples of high shock states (>30 GPa; maskelynite) are uncommon.

14 elates to mechanical ventilation and various shock states including hypovolemic, cardiogenic, obstruc

15 Shock states induce the expression of inducible nitric o

16 Intestinal ischemia-reperfusion after severe shock states is often associated with bacterial transloc

17 During sepsis and shock states, mitochondrial dysfunction occurs.

18 f cardiac output during low-flow circulatory shock states of diverse causes.

19 In particular, we demonstrate how these shock states perturb venous return/right heart interacti

20 cardiopulmonary bypass, and the treatment of shock states possibly associated with adrenocortical ins

21 ution have studied a variety of vasodilatory shock states that are characterized by vasopressin defic

22 As a result of the wide array of shock states with which intensivists contend, an approac

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