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2 The Kaplan-Meier time-to-event analyses showed that OCT detected progression earlier than VF in both
8 iferase reporter assays, spanning all major TE classes, and showed that 95.6% of tested TEs can function as either transc
9 genic mouse under the regulation of the Nkx2.5 enhancer and showed that neonatal Nkx2.5+ cardiomyoblasts mature into card
10 is increased in B-lineage cells from Gli3-deficient FL and showed that these cells expressed reduced levels of B-lineage
12 lular imaging using a phospho-specific p-T153/Y155 antibody showed that phosphorylated TDP-43 was specifically recruited
13 CSK9 hypomethylation and a translational mouse model of AUD showed that alcohol exposure leads to PCSK9 downregulation.
14 ore, a transgenic mouse line specifically labeling SL cells showed that they send profuse axonal projections to olfactory
15 Confocal laser scanning microscopy (CLSM) showed that the number of fungal cells attached to the modifi
16 odels (BS-REL, BUSTED and RELAX)), our results consistently showed that ancestral primates were subjected to enhanced pos
17 results obtained from simulated and real genotype datasets showed that the ELS algorithm was able to accurately estimate
19 t-mean-squared error) of load estimates a modeling exercise showed that passive samplers were a viable alternative to gra
20 ted against in all populations, our competition experiments showed that antibiotics significantly increased the advantage
27 evidence of "silencing", intracellular free calcium imaging showed that the cells were still viable.
33 results from previous studies on other viruses, our results showed that filtration and nuclease treatment did not discern
37 enetic analysis of SNAP genes from 22 diverse plant species showed that SNAPs were distributed in six monophyletic clades
39 thought to be the underlying mechanism, and recent studies showed that sKlotho interacts with alpha2-3-sialyllactose-con
40 gy activity of the Kalpha2-helix peptide, the present study showed that treatment with the Kalpha2 peptide fused with the
44 portantly, through in vitro binding and activity assays, we showed that CML36 interacts directly with the regulative N te
45 isable nonredundant subunits of mTORC1, mTORC2, or both, we showed that mice lacking mTORC1 or mTORC1/mTORC2 but not mTOR
47 immunofluorescent and transmission electron microscopy, we showed that S. pneumoniae rapidly adhered to and invaded card
50 ng fluorescence microscopy and cryo-electron tomography, we showed that Pseudomonas chlororaphis phage 201phi2-1 assemble
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