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1 th Idd3 and Idd5) have been shown to control sialadenitis.
2 eously develop autoimmune dacryoadenitis and sialadenitis and are a model for the human disorder Sjog
5 xperienced observers, nonspecific changes of sialadenitis are frequently confused with the focal lymp
6 lpha-transgenic mice develop autoantibodies, sialadenitis, as in Sjogren's syndrome, and immune compl
9 pecimens included 61% with focal lymphocytic sialadenitis (FLS; 69% of which had focus scores of >/=1
10 development of salivary gland inflammation (sialadenitis), indicating that the effect was tissue-sel
13 "atypical" manifestations were noted: fever, sialadenitis, lymphadenopathy, erythema nodosum, leukocy
20 ary glands from 37 SS patients and 9 chronic sialadenitis patients were analyzed by immunohistochemis
21 nces between the groups in the prevalence of sialadenitis, stomatitis, xerostomia, or dysgeusia over
23 ,726 LSG specimens exhibiting any pattern of sialadenitis, we compared biopsy diagnoses against concu
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