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1 egrins or selectin carbohydrate ligands (eg, sialyl-Lewis(x)).
2 provide functional oligosaccharides such as sialyl Lewis(X).
3 receptors that present carbohydrates such as sialyl Lewis(x).
4 -detectable glyconanoparticles conjugated to sialyl Lewis(X).
5 essed normal levels of CD18, L-selectin, and sialyl-Lewis(x).
6 ctable sulfation of GlcNAc in the context of sialyl Lewis x.
7 that comprise structural motifs derived from sialyl Lewis x.
8 lanoma and human lung tumor cells expressing sialyl Lewis X.
9 humans through the generation of functional sialyl Lewis X.
10 ent groups based on the expression levels of sialyl Lewis X.
11 lung nodules as B16-FTIII.N cells which lack sialyl Lewis X.
12 various functional oligosaccharides, such as sialyl Lewis X.
13 lfate and GlcNAc-6-sulfate in the context of sialyl Lewis x.
14 d N-acetylglucosamine [GlcNAc]-6-sulfate) of sialyl Lewis x.
15 y 4-fold 3'-sulfo Lewis x, as compared to 3'-sialyl Lewis x.
16 Several N-acylglucosamine derivatives of sialyl Lewis X (1-3) were prepared using a combined chem
20 omplex with its prime glycan target 6'-sulfo sialyl Lewis(x) A canonical motif for sialic acid recogn
21 rate structural analysis showed that 6-sulfo sialyl Lewis X, a dominant ligand for L-selectin, was al
23 ro and in vivo because of markedly decreased sialyl-Lewis X/A carbohydrate ligand-binding epitopes on
24 pport the cooperative role of E-cadherin and sialyl-Lewis X/A-deficient MUC1 in the passive dissemina
25 ell binding is because of markedly decreased sialyl-Lewis(x/a) (sLe(x/a)) carbohydrate ligand-binding
26 tment resulted in dose-dependent ablation of sialyl Lewis X and CHO-131 antigen expression on PSGL-1,
29 ere inhibited, we analyzed the expression of sialyl Lewis X and sialyl-fucosylated core 2 O-glycan (C
31 nhibitors of selectin binding to immobilized sialyl Lewis(X) and of cell adhesion to immobilized sele
32 introduced glyco-patterns range from common sialyl Lewis(x) and sialyl lacto-chains to chemically fu
33 detect binding of the SabA adhesin domain to sialyl-Lewis(X) and Lewis(X) but not to Lewis(A), Lewis(
35 oll" (via interactions between selectins and sialyl-Lewis-x), and then firmly adhere to the vascular
36 cytometry, bimodal patterns of expression of sialyl-Lewis X- and sialyl-dimeric-Lewis X were observed
37 re colon cancer HT29 cells by using multiple Sialyl Lewis X antibodies (aSlex)-conjugated PAMAM dendr
38 at occur as two major species containing the sialyl Lewis x antigen; one species is a disialylated, m
40 relevant branched oligosaccharides, such as sialyl Lewis(x), as well as sulfated glycosaminoglycan-l
41 ugh it has been shown to more effective than sialyl Lewis x at blocking the L-selectin-GlyCAM-1 inter
42 nd CD11b are all required for PMN binding to sialyl Lewis(x)-bearing LS174T cells at high shear (800
44 ts, we propose possible pathways for 6-sulfo sialyl Lewis x biosynthesis and suggest that sulfation m
45 and fucosylated alpha2-3 glycans (including sialyl Lewis x), both of which may be important receptor
46 monosialylated, core 2-based O-glycan with a sialyl Lewis x (C2-O-sLe(x)) motif at a specific Thr res
47 pating in the synthesis of core 2-associated sialyl Lewis x (C2-O-sLe(x)), a ligand involved in selec
50 correlated with their reduced expression of sialyl Lewis x (CD15s), a putative cellular receptor com
52 e developed a cell-free adhesion assay using sialyl Lewis(x)-coated microspheres and E-selection-IgG
54 m the neutrophil surface during rolling on a sialyl Lewis x-coated planar surface at physiological sh
55 -selectin on neutrophils during rolling on a sialyl Lewis x-coated surface that involves mechanical f
56 on both E- and P-selectin and suggest that a sialyl Lewis x-containing glycan at Threonine-16 is crit
58 n the lung vasculature plays a major role in sialyl Lewis X-dependent cancer cells targeting to the l
59 ational analysis with NMR indicated that the sialyl Lewis x domain of 1 retained the conformation of
61 and glycosphingolipid structures displaying sialyl Lewis X epitopes as potential E-selectin ligands
62 n E/P-selectin doubly deficient mutant mice, sialyl Lewis X-expressing B16 melanoma cells colonized t
63 carbohydrate-dependent lung colonization of sialyl Lewis X-expressing B16-FTIII-M cells in E/P-selec
64 ranose) action by contrasting the effects on sialyl Lewis X expression displayed by P-selectin glycop
65 inhibitor to reduce O-linked glycosylation, sialyl Lewis-X formation, and leukocyte adhesion via the
66 e selectins were blocked with an analogue of sialyl-Lewis(x) given as an I.V. bolus of 10 mg/kg follo
67 of soluble complement receptor-1 (sCR1) was sialyl Lewis x glycosylated (sCR1sLex) to inhibit comple
71 nsfected cells carried comparable amounts of sialyl Lewis x in extended core 1 and core 2 branched O-
75 critical function of N-glycan-linked 6-sulfo sialyl Lewis X in L-selectin-dependent lymphocyte homing
76 FTIII.M cells expressing moderate amounts of sialyl Lewis X in poly-N-acetyllactosamines produced lar
77 III.H cells expressing the highest amount of sialyl Lewis X in shorter N-glycans died in lung blood v
78 esults indicate that excessive expression of sialyl Lewis X in tumor cells leads to rejection by NK c
81 ession of cell surface carbohydrates such as sialyl Lewis X is associated with tumor formation and me
83 ied a class of N-glycans bearing the 6-sulfo sialyl Lewis X L-selectin ligand in high endothelial ven
84 egulation of endothelial selectins that bind sialyl Lewis(x) ligands and activation of beta(2)-integr
88 at adding fucose to human Tregs, forming the Sialyl Lewis X moiety on P-selectin glycoprotein ligand-
89 eu5Acalpha2-3Galbeta1-4GlcNAc, including the sialyl-Lewis(x) motif and structures containing 6-sulfog
90 de 1) diminishes the formation of the glycan sialyl Lewis X (Neu5Acalpha2-3Galbeta1-4(Fucalpha1-3) Gl
91 ied by the emergence of the CSLEX-1 epitope, sialyl Lewis x (NeuAcalpha2,3Galbeta1,4(Fucalpha1,3)GlcN
92 t adhesion on SEC apoptosis was tested using sialyl Lewis-X oligosaccharide (sLe(x)), a natural ligan
93 d SIGNR7 binds preferentially to the 6-sulfo-sialyl Lewis(x) oligosaccharide, whereas SIGNR2 binds al
95 to recognize both nonsulfated and 6-sulfated sialyl Lewis X on core 2 branched O-glycans, and MECA-79
96 AcT) enabled the construction of the 6-sulfo sialyl Lewis x on extended core1 O-glycans, recapitulati
99 Here we show that binding of E-selectin to sialyl Lewis(x) on L-selectin and PSGL-1 drives their co
100 T, that directs the synthesis of the 6-sulfo sialyl Lewis(x) on L-selectin counterreceptors CD34, Gly
101 sulfated carbohydrate structures (6-sulfated sialyl Lewis x or 6-sulfo-sLex) as a recognition determi
102 g the same sequence inhibited the binding of sialyl Lewis X or sialyl Lewis A oligosaccharides to E-s
103 E-selectin glycoprotein ligand(s); distinct sialyl Lewis X (or HECA-452 antigen)-bearing membrane pr
104 intracellular signals elicited by binding of sialyl Lewis(X) present on salival mucins to l-selectin
105 test the hypothesis that plasma C1INH bears sialyl Lewis(x)-related moieties and therefore binds to
106 demonstrated that plasma C1INH does express sialyl Lewis(x)-related moieties on its N-glycan as dete
107 bind tightly to sugar moieties Lewis(B) and sialyl-Lewis(X), respectively, on the surface of epithel
108 ass spectrometric analyses revealed that the sialyl-Lewis(x) sequence [NeuAcalpha2-3Galbeta1-4(Fucalp
110 inhibited by glycoconjugates terminated with sialyl-Lewis(x) sequences or by antibodies directed agai
111 ual sulfated tetrasaccharide epitope 6-sulfo sialyl Lewis x (Siaalpha2-->3Galbeta1-->4[Fucalpha1-->3]
112 ell line, HT-29LMM, expressed high levels of sialyl Lewis x, sialyl Lewis a, alpha(1,3/1,4)fucosyltra
113 the canonical E-selectin binding determinant sialyl Lewis X (sLe(X)) and display markedly greater adh
115 s based on the monomeric carbohydrate ligand sialyl Lewis X (SLe(X)) have low affinities and are not
117 ligand sulfotransferase (LSST) forms 6-sulfo sialyl Lewis x (sLe(x)) on both core 2 branch and MECA-7
120 lpha1-3-fucosylated selectin ligands such as sialyl Lewis x (sLe(x)), although monoclonal antibodies
121 ty by ablating N-acetyllactosamine (LacNAc), sialyl Lewis X (sLe(X)), and related lectin ligands on e
122 hesion proteins P- and L-selectin binding to sialyl Lewis X (sLe(X))-containing ligands, and the myos
123 an intragranulocytic bacterium that utilizes sialyl Lewis x (sLe(x))-modified P-selectin glycoprotein
125 binding of simple oligosaccharides based on sialyl Lewis-X (sLe(X)) and complex molecules with the c
126 amine structure (Galbeta1,4GlcNAc) to create sialyl Lewis-X (sLe(X)) and related sialofucosylated gly
127 nalysis demonstrate the presence of both the sialyl Lewis-X (sLe(X)) and the di-sialylated T-antigen
128 rks that participate in the formation of the sialyl Lewis-X (sLe(X)) epitope on O-glycans linked to a
129 r conventional Hbonds in the pentasaccharide sialyl Lewis-X (sLe(X)-5) between 5 and 37 degrees C in
132 -sialylated- and alpha1,3-fucosylated-moiety sialyl-Lewis x (sLe(x)), which modifies the PSGL-1 N ter
133 ctin, is capped with sulfated derivatives of sialyl Lewis x [sLe(x): Sia alpha 2-->3Gal beta 1-->4(Fu
137 have reported that microspheres coated with sialyl Lewis(x) (sLe(x)) interact specifically and roll
138 -terminus of PSGL-1 through recognition of a sialyl Lewis(x) (SLe(x)) moiety linked to a properly pos
139 Binding of the P-, L-, and E-selectins to sialyl Lewis(x) (sLe(x)) retards circulating leukocytes,
140 nanometer-scale polymer construct containing sialyl Lewis(x) (sLe(x)) that is found on the surface of
141 f L-selectin and the minimal selectin ligand sialyl Lewis(x) (sLe(x)) to interact with postcapillary
142 res functionalized with the selectin ligand, sialyl Lewis(X) (sLe(X)), and an antibody against ICAM-1
143 find that beads coated with the saccharides sialyl Lewis(x) (sLe(x)), sialyl Lewis(a) (sLe(a)), and
144 ds are glycosylated with the tetrasaccharide sialyl Lewis(x) (sLe(x)), which contributes to bond affi
145 However, the only millimolar affinity of sialyl Lewis(x) (sLe(x)), which is the common tetrasacch
146 ted in a cell-free system; it was shown that sialyl Lewis(x) (sLe(x))-coated microspheres roll over E
147 ed for the ability to inhibit the binding of sialyl Lewis(x) (sLe(x), 2) bearing HL-60 cells to E-, P
149 (AFM) with cantilevers biofunctionalized by sialyl-Lewis(x) (sLe(x)) were employed to investigate Ab
150 s and displays copies of the tetrasaccharide sialyl-Lewis(x) (sLe(X)), as well as a cluster of three
151 the sialylated, fucosylated tetrasaccharide sialyl Lewis X (sLex) is an important component of leuko
154 igand-1 (PSGL-1), which displays appropriate sialyl Lewis x (sLex)-like carbohydrate determinants for
158 -selectin glycoprotein ligand-1 [PSGL-1] and Sialyl-Lewis(x) [SLeX]) to rapidly target inflamed tissu
162 -60 cells and serum albumin derivatized with sialyl-Lewis X tetrasaccharide, thus mimicking the prope
164 n of HL-60 and B16 melanoma cells expressing sialyl Lewis X to E-selectin was also inhibited by the p
168 -long glass fibers, and the selectin ligand, sialyl Lewis(x), was coupled to latex microspheres.
169 kingly, bonds between L-selectin and 6-sulfo-sialyl Lewis X were impervious to ramp rate changes.
170 ion to unique carbohydrate ligands, sulfated sialyl Lewis(x), which are expressed on high endothelial
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