ライフサイエンスコーパス: sickness

1 rican trypanosomiasis (HAT; African sleeping sickness).

2 st mortality (=death or delisting because of sickness).

3 extracellular parasite that causes sleeping sickness.

4 ain the weight loss associated with sleeping sickness.

5 ntribute to loss of appetite observed during sickness.

6 an African trypanosomiasis (HAT) or sleeping sickness.

7 called African trypanosomiasis, or sleeping sickness.

8 t causes the deadly disease African sleeping sickness.

9 ncluding malaria, dengue fever, and sleeping sickness.

10 23andMe database who were surveyed about car sickness.

11 share underlying genetic factors with motion sickness.

12 esiense, the cause of acute African sleeping sickness.

13 somiasis (HAT), also called African sleeping sickness.

14 brucei, the causal agent of African sleeping sickness.

15 develop new treatments for African sleeping sickness.

16 tsetse fly and that causes African sleeping sickness.

17 re therapeutic treatment of African sleeping sickness.

18 result of the common side effect of visceral sickness.

19 n trypanosomiasis (HAT), or African sleeping sickness.

20 igate the social environment during times of sickness.

21 eukemia, autoimmune disorders, and radiation sickness.

22 miasis (HAT), also known as African sleeping sickness.

23 ng and leaving curves had induced the motion sickness.

24 w to maintain speed while eliminating motion sickness.

25 go faster, but passengers complain of motion sickness.

26 sitic protozoan that causes African sleeping sickness.

27 yaw and roll, and a survey evaluated motion sickness.

28 cei, the causative agent of African sleeping sickness.

29 insights into the mechanism of sleep during sickness.

30 rs who might provide support and care during sickness.

31 ral neuroendocrine mechanism of sleep during sickness.

32 in other organisms to regulate sleep during sickness.

33 ate/inefficient RH alleles exhibit synthetic sickness.

34 ore to assess the severity of acute mountain sickness.

35 d to suicide rates but others, such as staff sickness (-0.12, 0.37) and patient satisfaction (-0.06,

36 .0 emotional (+8.1; 95% CI, 1.8 to 14.4) and Sickness (-8.2; 95% CI, -14.2 to -2.2) scores.

37 Human African Trypanosomiasis (HAT, sleeping sickness), a disease that has often been considered to b

38 i is the causative agent of African sleeping sickness, a devastating disease endemic to sub-Saharan A

39 Trypanosoma brucei causes African sleeping sickness, a disease for which existing chemotherapies ar

40 t to develop new treatments against sleeping sickness, a fatal disease caused by this protozoan paras

41 thern Africa are foci for Rhodesian sleeping sickness, a fatal zoonotic disease caused by trypanosome

42 rypanosomiases, including Rhodesian sleeping sickness, a zoonosis associated with wilderness areas of

43 ported by an individual and the odds of paid sickness absence (1 adversity: odds ratio (OR) = 1.26, 9

44 The outcome variables were paid sickness absence (yes/no) and number of days of paid sic

45 The higher prevalence of sickness absence after breast cancer was mainly a result

46 The primary outcome measure was change in sickness absence among those supervised by each of the m

47 were associated with higher odds ratios for sickness absence and disability pension (odds ratio rang

48 ted and sociodemographic factors with future sickness absence and disability pension among women with

49 Purpose To explore future diagnosis-specific sickness absence and disability pension among women with

50 Information about relatively low future sickness absence and disability pension levels can be us

51 the annual prevalence of diagnosis-specific sickness absence and disability pension over 5 years.

52 absence hours divided by the sum of hours of sickness absence and hours of attendance.

53 Advanced cancer at diagnosis, > 90 days sickness absence before diagnosis, low education, and be

54 r study because of fatigue was determined by sickness absence certified by a doctor.

55 en in the first year, most women were not on sickness absence for a substantial time, and even in hig

56 Long-term sickness absence from studies and work was noted in all

57 Health care service use and sickness absence from work during the year before diagno

58 Most research on long-term sickness absence has focussed on exposure to occupationa

59 leave seperately, with rate being defined as sickness absence hours divided by the sum of hours of si

60 ondents reported a greater number of days of sickness absence in response to worsening psychosocial j

61 absence (yes/no) and number of days of paid sickness absence in the past year (2005-2012).

62 Conclusion The level of sickness absence increased substantially in women with b

63 Sickness absence is associated with adverse health, orga

64 d even in high-risk groups, many were not on sickness absence or disability pension in the following

65 e included in the study via their anonymised sickness absence records.

66 Long-term sickness absence was defined as receipt of Incapacity Be

67 breast cancer, the proportion of women with sickness absence was high but decreased continuously fro

68 iary care, use of other medical services, or sickness absence, but the consumption of nutritional sup

69 most rapidly increasing causes of long-term sickness absence, despite improved rates of detection an

70 d to a significant reduction in work-related sickness absence, with an associated return on investmen

71 ll psychosocial job quality and variation in sickness absence.

72 health problems, and its effect on employee sickness absence.

73 mprove the quality of work could help reduce sickness absence.

74 All medically certified sickness absences for depression recorded between Januar

75 redictors: age, self-rated health, number of sickness absences in previous year, socioeconomic positi

76 soma brucei, the causative agent of sleeping sickness across sub-Saharan Africa, depends on a remarka

77 ses human African trypanosomiasis ("sleeping sickness") across sub-Saharan Africa and is a model syst

78 Chagas disease, leishmaniasis and sleeping sickness affect 20 million people worldwide and lead to

79 ts association with energy-saving behaviors, sickness allows increased incentive motivation when the

80 Acute mountain sickness (AMS) affects more than 25% of individuals asce

81 to high altitude, the CBF in acute mountain sickness (AMS) subjects was higher (P < 0.05), while the

82 with symptoms consistent with acute mountain sickness (AMS).

83 The studies in serum sickness and anti-GBM nephritis led to an understanding

84 Detection of subtle cues indicating sickness and avoidance of sick conspecifics would theref

85 d public health measures drastically reduced sickness and death from infections in children and young

86 esses, enhanced inflammation and exaggerated sickness and depressive-like behaviors.

87 ents or highlanders include chronic mountain sickness and high-altitude pulmonary hypertension.

88 provocative of and protective against motion sickness and how vestibular disease may sensitize to mot

89 th yaw velocity on curves will reduce motion sickness and improve passenger comfort on tilting trains

90 i is the causative agent of African sleeping sickness and is known for its unique RNA processing mech

91 rasitic protozoa that cause African sleeping sickness and malaria.

92 and observing new associations with altitude sickness and many gastrointestinal conditions.

93 causative pathogen of human African sleeping sickness and nagana in domestic animals, myo-inositol is

94 n mechanisms and loci associated with motion sickness and nausea per se.

95 sea and vomiting (PONV), vertigo and morning sickness and observing new associations with altitude si

96 ould benefit both victims of acute radiation sickness and patients undergoing hematopoietic cell tran

97 e importance of the nervous system in motion sickness and suggest a role for glucose levels in motion

98 individuals is highly susceptible to motion sickness and yet the underlying causes of this condition

99 adverse event, including rash, fever, serum sickness, and anaphylaxis.

100 sthenia, sensory neuropathy, anorexia, serum sickness, and hypertensive encephalopathy.

101 cei, the causative agent of African sleeping sickness; and Plasmodium spp., the causative agents of m

102 the development of diagnostics for sleeping sickness are considered and progress towards a much-need

103 causative agent of the fatal human sleeping sickness, are essential for the parasite.

104 This effect was not due to sickness as icv TTR did not cause a conditioned taste av

105 There is new research on acute mountain sickness associated with inflammation with relevance for

106 morphisms (SNPs) were associated with motion sickness at a genome-wide-significant level (P < 5 x 10(

107 ore, which quantifies the overall feeling of sickness at altitude (VAS[O]; various thresholds), Acute

108 sual analog scale for the overall feeling of sickness at altitude, Acute Mountain Sickness-Cerebral,

109 ei rhodesiense causes human African sleeping sickness because it has evolved an inhibitor of TLF-1, s

110 Exaggerated sickness behavior (lethargy, piloerection, ptosis) in th

111 Sickness behavior and cognitive dysfunction occur freque

112 that probiotic (VSL#3) treatment attenuates sickness behavior development in mice with liver inflamm

113 Attenuation of sickness behavior development was associated with reduct

114 ication pathway, thereby reducing subsequent sickness behavior development.

115 croglial activation and subsequently driving sickness behavior development.

116 n, decreased central neural excitability and sickness behavior development.

117 n the initiation of cognitive impairment and sickness behavior during viral infection through inducti

118 monocytes and platelets, and development of sickness behavior following bile-duct ligation.

119 Sickness behavior in humans is characterized by low mood

120 ligation and perforation-induced symptoms of sickness behavior in rats.

121 immunoglobulin M alleviated the symptoms of sickness behavior in the septic rats, with the animals b

122 Despite how common sickness behavior is, its molecular basis is not well un

123 ddition, inflammatory cytokines in serum and sickness behavior profiles were measured before and afte

124 These animals developed sickness behavior symptoms, including anorexia, hypoacti

125 Features of the complex behavioral sickness behavior syndrome were characterized by homecag

126 ered peripherally to adult zebra finches and sickness behavior was recorded 2 or 24 hours later.

127 lls as natural gatekeepers for virus-induced sickness behavior, demonstrated tissue specific IFNAR en

128 We found that during sickness behavior, single-stranded RNA viruses, double-s

129 Sickness behavior, temperature, and cytokine profiles of

130 eriphery in response to brain injury induced sickness behavior, which could be abrogated by inhibitio

131 ate microglial activation and development of sickness behavior.

132 lls to activate microglial cells and promote sickness behavior.

133 of the microglia, as well as development of sickness behavior.

134 The resulting sickness behaviors (e.g., pain, disturbed sleep), depres

135 Sickness behaviors and metabolic responses to invading p

136 Although these so-called sickness behaviors are the most common and familiar symp

137 rious pathophysiologic mechanisms leading to sickness behaviors have been proposed.

138 tures of circulating monocytes and promoting sickness behaviors in mice with cholestatic liver injury

139 microbiome to treat inflammation-associated sickness behaviors in patients with systemic inflammator

140 ate recovery suggests that norms relevant to sickness behaviors may also vary as a function of region

141 mation in specific regions may contribute to sickness behaviors that overlap with the symptoms of MDE

142 mals, activation of the immune system causes sickness behaviors that present during a major depressiv

143 important role of TLR2 in the development of sickness behaviors via stimulation of hypothalamic micro

144 Sickness behaviors were quantified based on time spent b

145 ommonly develop debilitating symptoms (i.e., sickness behaviors) that arise from changes in brain fun

146 Sickness behaviors, such as fatigue, mood alterations, a

147 mmonly develop debilitating symptoms, called sickness behaviors, which arise via changes in brain fun

148 tion of a constellation of symptoms, termed 'sickness behaviour'.

149 nd are sufficient to generate the associated sickness behaviours, and are the first demonstration tha

150 location of metabolic resources during acute sickness but might also account for maladaptive, motivat

151 omide was used by pregnant women for morning sickness but was removed from the market because it caus

152 ferent levels of protection against sleeping sickness, but this comes with an increased risk of devel

153 may undermine efforts to eliminate sleeping sickness by 2020.

154 Decompression sickness can involve the musculoskeletal system, skin, i

155 that the risk for trypanosomiasis (sleeping sickness), carried by tsetse flies in bushy environments

156 African sleeping sickness, caused by the protozoan parasite Trypanosoma b

157 (VAS[O]; various thresholds), Acute Mountain Sickness-Cerebral score (AMS-C; >/=0.7 indicates AMS), a

158 ling of sickness at altitude, Acute Mountain Sickness-Cerebral, and clinical functional score perform

159 arterial gas embolism, severe decompression sickness, clostridial gas gangrene, necrotizing fasciiti

160 We observed patients with chronic mountain sickness (CMS) in our clinic who developed progressive n

161 , with a condition known as chronic mountain sickness (CMS) or Monge disease.

162 ome high-altitude dwellers (chronic mountain sickness [CMS] or Monge's disease) but not others living

163 searched for comorbid phenotypes with motion sickness, confirming associations with known comorbiditi

164 rucei, a causative agent of African Sleeping Sickness, constantly changes its dense variant surface g

165 ve effect of olfactory-visual integration of sickness cues was found in the intraparietal sulcus, whi

166 pathological responses such as decompression sickness (DCS).

167 cei, the etiologic agent of African Sleeping Sickness, deploys an RNA pol II that contains a non-cano

168 cei, the causative agent of African sleeping sickness, differs from its human host in several fundame

169 uce immune complex diseases, including serum sickness disease (SSD).

170 gly immunogenic and rabbit ATG induces serum sickness disease in almost all patients without addition

171 Sickness during pregnancy is associated with an increase

172 g a poor sleeper) that correlate with motion sickness, findings that could help identify risk factors

173 Trypanosoma brucei causes African sleeping sickness for which no vaccine exists and available treat

174 In 1993, the freedom to choose one's sickness fund was formally introduced, and reforms that

175 extended in 1913 to cover relations between sickness funds and doctors, which in turn led to the rig

176 initially applied mainly to the payers (the sickness funds) but was extended in 1913 to cover relati

177 w vestibular disease may sensitize to motion sickness has increased.

178 rucei, the causative agent of human sleeping sickness, has an intrinsic circadian clock that regulate

179 Human African sleeping sickness (HAT) is caused by the parasitic protozoan Tryp

180 owever, the polysymptomatic nature of motion sickness, high interindividual variability, and the exte

181 oing to high altitude include acute mountain sickness, high-altitude pulmonary edema, and high-altitu

182 soma brucei, the causative agent of sleeping sickness (Human African Trypanosomiasis, HAT), contains

183 of surveillance, for the control of sleeping sickness (human African trypanosomiasis, HAT).

184 vements in the psychosocial dimension of the Sickness Impact Profile compared with the placebo group

185 nitive abilities, quality of life (using the Sickness Impact Profile), serum levels of ammonia, level

186 elephone Interview for Cognitive Status, and Sickness Impact Profile.

187 irst genome-wide association study on motion sickness in 80 494 individuals from the 23andMe database

188 humans perceive and integrate early cues of sickness in conspecifics sampled just hours after the in

189 ed by biting midges and causes African horse sickness in equids, with mortality reaching up to 95% in

190 s African trypanosomiasis, known as sleeping sickness in humans and nagana in domestic animals.

191 ses caused by African trypanosomes: sleeping sickness in humans and Nagana in livestock.

192 African trypanosomes cause sleeping sickness in humans, a disease that is typically fatal wi

193 tozoan parasite that causes African sleeping sickness in humans.

194 infect mammals, they cause African sleeping sickness in humans.

195 has traditionally been used to treat motion sickness in humans.

196 re is a hallmark feature of chronic mountain sickness in maladapted populations living at high altitu

197 Sickness in mammals can lead to cognition deficits, alth

198 llular stress is similar to sleep induced by sickness in other animals.

199 indirect centrality is also correlated with sickness in the Agta, suggesting a trade-off.

200 mor flare") (in 11%), anemia (in 11%), serum sickness (in 8%), and fatigue (in 8%).

201 nhibitor cycloheximide and yields synthetic "sickness" in cells with limiting amounts of translation

202 The prevalence of acute mountain sickness increases with higher altitudes.

203 Now, Wang et al. identify that sickness-induced anorexia differentially shapes the meta

204 Sickness-induced anorexia is a conserved behavior induce

205 microbes have evolved mechanisms to modulate sickness-induced behaviors to promote health of their ho

206 ine article, Garre et al. (2017) report that sickness-induced cortical dendritic spine loss and impai

207 Study 2 examined the performance of the Sickness Insight in Coping Questionnaire among 100 ICU p

208 The Sickness Insight in Coping Questionnaire had good intern

209 Overall, the Sickness Insight in Coping Questionnaire has good psycho

210 This study showed that the Sickness Insight in Coping Questionnaire has good struct

211 ICU clinicians can use the Sickness Insight in Coping Questionnaire to gain insight

212 n = 103 hospitalized patients) addressed the Sickness Insight in Coping Questionnaire's performance r

213 res in testing the construct validity of the Sickness Insight in Coping Questionnaire-subscales (figh

214 t-proxy agreement of a novel instrument, the Sickness Insight in Coping Questionnaire.

215 Loss of appetite during sickness is a common and often debilitating phenomenon.

216 Human African trypanosomiasis or sleeping sickness is a deadly disease endemic in sub-Saharan Afri

217 Motion sickness is an emerging hazard in information technologi

218 Inflammation-induced sickness is associated with a large set of behavioral al

219 an African trypanosomiasis (HAT) or sleeping sickness is caused by two subspecies of Trypanosoma bruc

220 Sickness is triggered by exposure to environments such a

221 n African trypanosomiasis (HAT), or sleeping sickness, is a major threat to human health throughout s

222 Human African trypanosomiasis, or sleeping sickness, is caused by infection with parasites of the g

223 cei, the causative agent of African sleeping sickness, is transmitted to its mammalian host by the ts

224 ent fatigue, lowered functional capacity and sickness leave or delayed education after a large commun

225 xploited therapeutically to induce synthetic sickness/lethality in hematological malignancies, and po

226 Two hours post LPS, sickness-like behaviors increased, the transcription of

227 mmatory drug-associated angioedema and serum sickness-like reactions, are more frequent among young p

228 ma brucei, the etiological agent of sleeping sickness, localized its replication origins to the bound

229 tic trypanosomes that cause African sleeping sickness, mating occurs during transmission by the tsets

230 ogy and brain regions associated with motion sickness may provide for more effective medication in th

231 -like behavior, without altering LPS-induced sickness measured by body weight loss, decreased motor a

232 is, which was accelerated in a chronic serum sickness model by active immunization with heterologous

233 ity due to heart failure in chronic mountain sickness most likely reduces fitness.

234 Depending on the kind of sickness MRI and CT may fulfill basic or subsidiary role

235 Severe acute mountain sickness occurred in 314 (23.7%), high-altitude pulmonar

236 s the subcellular localization and synthetic sickness of Heh2DeltaNLS mutants.

237 compared to what would be expected from the sickness of the component single knockouts.

238 s are a cardinal feature of chronic mountain sickness offering one plausible mechanism for selection.

239 exual behaviour, and (3) reporting recurring sickness or chronic illness, suggesting infected adolesc

240 African sleeping sickness or human African trypanosomiasis, caused by Try

241 es, including those associated with satiety, sickness or unpalatable food.

242 tolerate high doses of the container without sickness or weight loss.

243 r human African trypanosomiasis (or sleeping sickness), orally dosed fexinidazole stands poised to re

244 Trypanosomatids, such as the sleeping sickness parasite Trypanosoma brucei, contain a approxim

245 Motility of the sleeping sickness parasite, Trypanosoma brucei, impacts disease t

246 The strategy that sleeping sickness parasites use to evade the mammalian immune sys

247 rare cell types in blood (including sleeping sickness parasites), and has the potential to enable sin

248 hlighting the need for better assessments of sickness, particularly in women.

249 Protozoan parasites such as the sleeping sickness pathogen Trypanosoma brucei adapt to different

250 i is the causative agent of African sleeping sickness, putting at risk up to 50 million people in sub

251 eric Core Scales (PedsQL4.0), and an overall Sickness question.

252 Motion sickness remains bothersome in conventional transport an

253 This fundamental aspect of the sickness response reduces the quality of life for people

254 blocks the protracted neuroinflammatory and sickness response to peripheral Escherichia coli (E. col

255 These sickness responses are characterized by an acute reorien

256 nged infection-induced neuroinflammatory and sickness responses in aged rats.

257 ne stimulation, and that are associated with sickness responses such as fever, anorexia, and stress h

258 inst Trypanosoma that cause African sleeping sickness, resulting in positive selection of these allel

259 s should be sensitive to the unique roles of sickness, risk perception, and isolation in the ED/UC di

260 ificantly affect average MSAS, PedsQL4.0, or Sickness score trends.

261 AS, PedsQL4.0 total and subscale scores, and Sickness scores during 20 weeks of follow-up, along with

262 pite greater morbidity (e.g. weight loss and sickness scores) during the acute infection in the 18-mo

263 ad evidence of association between increased sickness sensitivity and reduced glucocorticoid sensitiv

264 P < .03), with significant interaction with sickness severity status.

265 more, in some patients with higher levels of sickness severity, reduced mortality from an optimized a

266 Importantly, mechanisms regulating sickness sleep are partially distinct from those regulat

267 aptive mechanisms employed in the control of sickness sleep may play a role in correcting cellular ho

268 During sickness, sleep is regulated by cytokines released from

269 in these instances (e.g., report of morning sickness, start month of acetaminophen use and ibuprofen

270 Sickness status presented by odor and facial photograph

271 of some anti-mAChR drugs in treating motion sickness suggest that we may, in fact, already be target

272 that may be related to inflammation-induced sickness symptom as well as other functions, such as blo

273 Cytokines and sickness symptoms were assessed hourly till 8 h after LP

274 terleukin-6 and tumor necrosis factor-alpha, sickness symptoms, body temperature and self-reported fa

275 lammatory cytokines, vital sign changes, and sickness symptoms, well-established consequences of LPS

276 Much of the sickness syndrome is mediated by prostaglandins acting o

277 suite of brain-mediated responses called the sickness syndrome occurs, which includes fever, anorexia

278 During infection, vertebrates develop "sickness syndrome," characterized by fever, anorexia, be

279 d specific neural circuits that underlie the sickness syndrome.

280 nosoma brucei, which causes African sleeping sickness, TbISWI down-regulates RNA polymerase I (Pol I)

281 To decrease the possibility of serum sickness, the equine antisera was digested with pepsin t

282 y example of cross-resistance among sleeping sickness therapies.

283 Other SNPs may affect motion sickness through nearby genes with roles in the nervous

284 ia occurs in disorders ranging from altitude sickness to airway obstruction, apnea, and atelectasis.

285 d by the causative agent of African sleeping sickness, Trypanosoma brucei In mitochondria of this pat

286 etabolism in the causative agent of sleeping sickness, Trypanosoma brucei, with that of human erythro

287 nosomes, parasites that cause human sleeping sickness, undergo a density-dependent differentiation in

288 changes accompanying visually induced motion sickness, using a motion video, hypothesizing that diffe

289 African horse sickness virus (AHSV) is a lethal arbovirus of equids th

290 African horse sickness virus (AHSV), an orbivirus in the Reoviridae fa

291 African horse sickness virus is transmitted by biting midges and cause

292 Concurrently, motion sickness was greater (P<0.001) in the reactive mode.

293 No motion sickness was induced in the untilted mode, but the train

294 amining the models of acute or chronic serum sickness were major for our understanding of renal and v

295 t Barcroft was suffering from acute mountain sickness when he made it!

296 the arterial circulation, and decompression sickness, which is caused by in-situ bubble formation fr

297 had both cytokine release syndrome and serum sickness, which was associated with a transient rise in

298 ilar sorting defects and displayed synthetic sickness with cof1-8.

299 ase, high altitude involves chronic mountain sickness with new knowledge of associated cardiovascular

300 t of Human African Trypanosomiasis (sleeping sickness), yet little is known about which PKs are essen