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1 lated with unilineage dysplasia without ring sideroblasts.
2 r a diagnosis of refractory anemia with ring sideroblasts.
3 scovered in a distinct form of MDS with ring sideroblasts.
4 f myelodysplastic syndrome (MDS) with ringed sideroblasts.
5 O) therapy for refractory anemia with ringed sideroblasts.
6 d dysplasia and 15% or more bone marrow ring sideroblasts.
7 ts with myeloid neoplasm and 1% or more ring sideroblasts.
8 ry anemia (7), refractory anemia with ringed sideroblasts (5), refractory anemia with excess blasts (
9 s blasts (35), refractory anemia with ringed sideroblasts (9), and refractory anemia with excess blas
14 eloid leukemia, MDS/MPN-Unclassifiable, ring sideroblasts associated with marked thrombocytosis, and
15 n(s) in our cohort of MDS patients with ring sideroblasts can arise from CD34(+)CD38(-)CD45RA(-)CD90(
16 e of RARS/RARS-T is the presence of abnormal sideroblasts characterized by iron overload in the mitoc
17 risk (refractory anemia [RA]/RA with ringed sideroblasts/chronic myelomonocytic leukemia with < 5% b
19 eloid precursors, excess iron stores, ringed sideroblasts, iron incorporation in plasma cells, and va
22 quent in myelodysplastic syndromes with ring sideroblasts (MDS-RS; approximately 75% incidence) and S
23 efractory anemia/refractory anemia with ring sideroblasts [RA/RARS]) have low levels of NF-kappaB act
24 ed in 60%-80% of refractory anemia with ring sideroblasts (RARS) and RARS associated with thrombocyto
25 cantly between refractory anemia with ringed sideroblasts (RARS) and refractory anemia with multiline
27 refractory anemia (RA), 3 had RA with ringed sideroblasts (RARS), 5 had RA with excess blasts (RAEB),
28 penia with multilineage dysplasia and ringed sideroblasts (RCMD-RS) and UPD4q24, and five patients (r
30 nemia with multilineage dysplasia and ringed sideroblasts (RCMD/RS) with regard to therapeutic respon
31 actory anemia, refractory anemia with ringed sideroblasts, refractory anemia with excess blasts, refr
32 he presence of >/= 15% bone marrow (BM) ring sideroblasts (RS) and < 5% blasts is required for a diag
34 atients with the refractory anemia with ring sideroblasts subtype of myelodysplastic syndrome (MDS) h
35 B1 mutation with refractory anemia with ring sideroblasts, TET2/SRSF2 comutation with chronic myelomo
38 ions are prevalent in low-risk MDS with ring sideroblasts, whereas U2AF1 and SRSF2 mutations are freq
39 uency among conditions characterized by ring sideroblasts, which is consistent with a causal relation
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