ライフサイエンスコーパス: siderosis

1 with environmental risk factors for hepatic siderosis.

2 ation p.Arg59* presented with severe hepatic siderosis.

3 gh burden of cardiac mortality in myocardial siderosis.

4 confirmed clinical diagnosis of superficial siderosis.

5 infarcts, pyknotic neurons, and progressive siderosis.

6 d by a photosensitive dermatosis and hepatic siderosis.

7 da is a skin disease associated with hepatic siderosis.

8 (POD); successive biopsies showed increasing siderosis.

9 review possible factors contributing to the siderosis.

10 he presence of cSAH and cortical superficial siderosis.

11 especially disseminated cortical superficial siderosis (49% vs 19%; p<0.0001).

12 ene (Hfe) have been used to generate hepatic siderosis, a nearly uniform finding in PCT.

13 Conclusion In the presence of siderosis, a transmetallation mechanism may be set off b

14 between acute cSAH and cortical superficial siderosis-a new CAA haemorrhagic imaging signature and (

15 Thus, in TM patients with cardiac siderosis, amlodipine combined with chelation therapy re

16 The cause of the hepatic siderosis and iron overload that is common in porphyria

17 Eight patients had siderosis and underwent chelation therapy.

18 Cerebral microbleeds, cortical superficial siderosis, and white matter hyperintensity volume were a

19 her studies of the safety of GBCAs in severe siderosis are needed.

20 These data indicate that the hepatic siderosis associated with PCT likely results from dysreg

21 Autopsy showed siderosis at many sites, including the implanted liver.

22 le logistic regression, cortical superficial siderosis burden (OR 5.53; 95% CI 2.82 to 10.8, p<0.0001

23 relating to treatment of chronic myocardial siderosis but none relating to treatment of acute heart

24 r cerebral microbleeds, cortical superficial siderosis, centrum semiovale perivascular spaces, and wh

25 r cerebral microbleeds, cortical superficial siderosis, centrum semiovale perivascular spaces, and wh

26 microbleeds (CMBs) and cortical superficial siderosis (CSS) are the characteristic markers of cerebr

27 Cortical superficial siderosis describes a distinct pattern of blood-breakdow

28 e CAA were analysed for cortical superficial siderosis (focal, </=3 sulci; disseminated, >/=4 sulci),

29 Although cortical superficial siderosis has many possible causes, it is emerging as a

30 hemochromatosis (hh) gene (HFE) explain the siderosis in approximately 20% patients, suggesting that

31 tandards for evaluating cortical superficial siderosis in research studies.

32 heart failure and arrhythmia from myocardial siderosis in thalassemia major and is superior to serum

33 These results suggest that superficial siderosis is associated with a distinct pattern of cogni

34 oid angiopathy cohorts, cortical superficial siderosis is associated with characteristic clinical sym

35 Hepatic siderosis is common in patients with porphyria cutanea t

36 Thus, cortical superficial siderosis is of relevance to neurologists working in neu

37 express a porphyric phenotype unless hepatic siderosis is present.

38 al nervous system infratentorial superficial siderosis (iSS) is increasingly detected by blood-sensit

39 dial T2* from 5 to 20 ms (indicating cardiac siderosis), left ventricular ejection fraction (LVEF) of

40 l lesions (microbleeds, cortical superficial siderosis, microinfarcts) and large-scale alterations (w

41 atients with severe transfusional myocardial siderosis (myocardial [m] T2* 5-<10 ms; left ventricular

42 g assessment of myocardial edema, myocardial siderosis, myocardial perfusion, and diffuse myocardial

43 Siderosis of interstitial trophoblasts is a novel pathol

44 tive effects are associated with superficial siderosis of the central nervous system caused by chroni

45 Superficial siderosis of the CNS is a rare condition, caused by depo

46 microscopy (n = 3) were conducted to verify siderosis of trophoblasts.

47 episodes compared with cortical superficial siderosis-positive, but cSAH-negative subjects with CAA

48 Cortical superficial siderosis prevalence (but no other CAA severity markers)

49 Disseminated superficial siderosis (subhazard ratio [SHR] 7.45, 95% CI 4.27-12.99

50 a-thalassemia major patients with myocardial siderosis (T2* 6-20 milliseconds) and no signs of cardia

51 We describe superficial siderosis that developed more than a decade following tr

52 d clinical, imaging and cortical superficial siderosis topographical mapping data between subjects wi

53 center, the presence of cortical superficial siderosis was an independent variable associated with la

54 The presence of cortical superficial siderosis was an independent variable associated with la

55 linical significance of cortical superficial siderosis, with a particular emphasis on cerebral amyloi