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1 acterized by deep refractoriness to seizure (silent period).
2  did not affect the duration of the cortical silent period.
3 de with voluntary facilitation and a shorter silent period.
4 physiology occurring during the seizure-free silent period.
5  status) and 4.3+/-0.7 late (14 days) in the silent period.
6 terval intracortical inhibition and cortical silent period.
7 xplained by a decrease in the density of the silent periods.
8 t bursts of RNA synthesis followed by longer silent periods.
9 nerve response (16 x baseline) followed by a silent period (1-2 s) during which another stimulus evok
10  = 0.01) and the post-motor evoked potential silent period (101 ms; SEM +/- 10) was significantly sho
11 fore conversion, P<.01); (2) an electrically silent period (267+/-45 ms); (3) "organized atrial fibri
12 lices when depolarized during their normally silent period and (2) bursting when depolarized in nonrh
13 t zolpidem reduced hyperexcitability in both silent period and chronically epileptic cells, but was m
14 rtical inhibition was measured with cortical silent period and intracortical inhibition paradigms.
15 al intracortical inhibition and the cortical silent period) and GABAA (short-interval intracortical i
16  thresholds, input/output curves or cortical silent period between patients with secondary and primar
17 between respiratory and syringeal control of silent periods between sound units and wing movement cyc
18 eurons that alternate bursting activity with silent periods, but the mechanism underlying this vital
19 ed by gamma-aminobutyric acid-A receptors in silent period cells differed markedly from controls.
20 or-evoked potentials (MEPs) and the cortical silent period (CSP) evoked by a single-pulse TMS, short-
21 cortical facilitation, and the contralateral silent period (CSP).
22 sting motor threshold (P < .05) and cortical silent period duration (P < .001).
23 shold, central motor conduction time (CMCT), silent period duration and the amplitude of compound mus
24                                              Silent period duration was shorter than normal early in
25 s (APs) by 21 mV without affecting burst- or silent-period durations.
26 d of two bursts per respiratory cycle with a silent period during inspiration.
27 racortical inhibition and prolonged cortical silent period during voluntary activity of an intrinsic
28 onal coinactivation: the occurrence of brief silent periods during which all neurons in the local net
29 igms, such as trace conditioning, in which a silent period elapses between the offset of the conditio
30                      The early shortening of silent period, however, probably represents a shift in t
31 rformed a duration discrimination task for a silent period in a rhythmic auditory sequence.
32 -evaluation of the concept of post-traumatic silent period in both animals and humans.
33             The burst rate and the pre-burst silent period in nDF firing of organic dystonia were con
34 ements of their display with atypically long silent periods in their song, potentially avoiding adver
35        An additional analysis of ipsilateral silent periods indicated that interhemispheric inhibitio
36 ion of the reflex occurred within a cortical silent period induced by transcranial magnetic stimulati
37 spheric inhibition (IHI) and the ipsilateral silent period (iSP), whilst excitability of CTS pathways
38 s seizures develop following an asymptomatic silent period lasting several weeks.
39 ffective at unmasking the underlying IPSP in silent period neurons.
40             VPd neurons only fire during the silent period of DP-PCN neurons, suggesting that they pr
41 vements of the display are synchronized with silent periods of song, but it is unknown how this coord
42 iated IPSPs diminished more gradually in the silent period, reaching a minimum at day 14.
43 tracortical facilitation (ICF), the cortical silent period (SP) and spinal reciprocal inhibition (RI)
44 eter muscles of 12 subjects and the cortical silent period (SP) in nine subjects.
45 rhythmic CS discharges were interleaved with silent periods, suggesting that apamin- and CTX-sensitiv
46 d spontaneous transitions between active and silent periods termed cell cycling.
47 tials and decreased duration of the cortical silent period (the latter only in the conditioned group)
48                                 The cortical silent period, the startle response and the second and t
49            The distribution of the pre-burst silent period was bimodal with a longer mode of approxim
50                                              Silent period was estimated during a small background co
51                                     Cortical silent periods were shortened in leg muscles.
52  asynchronous state) and (2) "filling-in" of silent periods with low-frequency (2-4 Hz) activity (beg

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