ライフサイエンスコーパス: simian virus 5

1 ulatory proteins of other viruses, including simian virus 5 and hepatitis B.

2 In this study, we report that simian virus 5 and Sendai virus heterologous HN proteins

3 ssing of Hendra virus F but had no effect on simian virus 5 F processing.

4 rainfluenza virus induces STAT2 degradation, simian virus 5 induces STAT1 degradation.

5 ER virus, a paramyxovirus closely related to simian virus 5, induces no syncytium formation.

6 ainfluenza virus 5 (PIV5), formerly known as simian virus 5, is a prototypical paramyxovirus and enco

7 Recombinant simian virus 5 (rSV5) mutants containing substitutions i

8 HeLa cells were infected with a recombinant simian virus 5 (rSV5) that expressed GFP.

9 HPIV3 HN and heterologous HN/H proteins from simian virus 5, Sendai virus, and measles virus in coexp

10 etely conserved residues of CBF1 in both the simian virus 5 (SV5) and Hendra virus F proteins.

11 conserved positions were created in both the simian virus 5 (SV5) and Hendra virus F proteins.

12 The paramyxoviruses simian virus 5 (SV5) and human parainfluenza virus 2 (HP

13 usion reaction promoted by the paramyxovirus simian virus 5 (SV5) and human parainfluenza virus type

14 usion (F) glycoproteins of the paramyxovirus simian virus 5 (SV5) are expressed on the surface of vir

15 irus is closely related to the paramyxovirus simian virus 5 (SV5) but is defective in syncytium forma

16 2), and type 3 (PI3), Sendai virus (SN), and simian virus 5 (SV5) by expression of their glycoprotein

17 In some cell types the paramyxovirus simian virus 5 (SV5) causes little cytopathic effect (CP

18 asal administration, the model paramyxovirus simian virus 5 (SV5) establishes an infection in the res

19 The paramyxovirus simian virus 5 (SV5) establishes highly productive persi

20 In contrast to proteolytic cleavage of the simian virus 5 (SV5) F protein by the Ca(2+)-dependent p

21 idues of the N-terminal heptad repeat in the simian virus 5 (SV5) F protein on protein folding, trans

22 The role of the simian virus 5 (SV5) fusion (F) protein 20 residue COOH-

23 nction was used to determine the role of the simian virus 5 (SV5) intergenic regions in transcription

24 Simian virus 5 (SV5) is a member of the paramyxovirus fa

25 The paramyxovirus simian virus 5 (SV5) is a poor activator of human dendri

26 The parainfluenza virus simian virus 5 (SV5) is a poor inducer of innate immune

27 The V protein of the paramyxovirus simian virus 5 (SV5) is responsible for targeted degrada

28 A replication promoter for the paramyxovirus simian virus 5 (SV5) requires two essential and disconti

29 cells infected with the parainfluenza virus simian virus 5 (SV5) show minimal activation of host cel

30 Simian virus 5 (SV5) targets STAT1, human parainfluenza

31 have previously shown for the paramyxovirus simian virus 5 (SV5) that a functional promoter for RNA

32 sly shown that a mutant of the paramyxovirus simian virus 5 (SV5) that harbors mutations in the P/V g

33 spiratory tract with the model paramyxovirus simian virus 5 (SV5) to study CD8+ T-cell responses in t

34 ve shown that treatment of the paramyxovirus simian virus 5 (SV5) with human serum results in deposit

35 ion from human lung A549 cells infected with simian virus 5 (SV5) with other members of the Rubulavir

36 Simian virus 5 (SV5), a closely related rubulavirus, enc

37 The V protein of simian virus 5 (SV5), a prototype of the paramyxoviruses

38 , a paramyxovirus that is closely related to simian virus 5 (SV5), is unusual in that it fails to ind

39 equirements for budding of the paramyxovirus simian virus 5 (SV5), its M protein was expressed in mam

40 For the paramyxovirus simian virus 5 (SV5), the cytoplasmic tail of the hemagg

41 tein convert the noncytopathic paramyxovirus simian virus 5 (SV5), which is a poor inducer of host ce

42 , BALB/c and C57BL/6, with the paramyxovirus simian virus 5 (SV5).

43 d following infection with the paramyxovirus simian virus 5 (SV5).

44 nsegmented negative-strand RNA paramyxovirus simian virus 5 (SV5).

45 We exploited the ability of V protein of simian virus 5 to degrade Stat1 and prevent interferon s

46 ctures of DDB1 alone and in complex with the simian virus 5 V protein.