ライフサイエンスコーパス: simplex

1 ow transplantation for epidermolysis bullosa simplex.

2 dicated dominant (43%), recessive (10%), and simplex (47%) disease.

3 Until now, both simplex and familial UHS-affected case subjects with aut

4 question, we recently published an original simplex approach helping to develop predictive models of

5 PMMs potentially contribute risk to 3%-4% of simplex ASD case subjects and future studies of PMMs in

6 of 208 genomes from 53 families affected by simplex autism.

7 ements might help explain additional risk of simplex autism.

8 vy muscular area of the ductus ejaculatorius simplex before and after mating, and these differences c

9 (c.1434_1435insC [p.Glu479Argfs( *)18]) in a simplex case subject.

10 e sequencing in a multiplex family and three simplex case subjects with an atypical association of LC

11 gnificant male predominance, particularly in simplex cases without a genetic diagnosis.

12 A simplex-centroid design comprising three solvents (water

13 strate that in the human skin disease lichen simplex chronicus, WNT5a and KRT9 are robustly activated

14 s of published genotype data from the Simons Simplex Collection (SSC) and the Autism Genetic Resource

15 this study was to extend work in the Simons Simplex Collection by comparing the phenotypic profiles

16 a large family-based ASD cohort, the Simons Simplex Collection, we systematically evaluated the pote

17 cruited, 8.6% with junctional EB, 34.3% with simplex EB, 34.3% with autosomal recessive dystrophic EB

18 ents affected by dystrophic, junctional, and simplex EB.

19 Importance: Epidermolysis bullosa simplex (EBS) is a group of clinically and genetically d

20 Herpes simplex encephalitis (HSE) is the most common form of ac

21 The herpes simplex encephalitis (HSE) model was used for induction

22 views; these include the relevance of herpes simplex encephalitis and of epilepsy to AD, the action o

23 Although herpes simplex encephalitis has been extensively studied, HSV-1

24 ssociation between TRIF mutations and herpes simplex encephalitis in patients.

25 vention is crucial to the survival of herpes simplex encephalitis patients; however, many survivors s

26 titis, highly debilitating and lethal herpes simplex encephalitis, and generalized infections that ca

27 ing necrotizing stromal keratitis and herpes simplex encephalitis.

28 e in multiplex families differs from that in simplex families and is complex, warranting more complet

29 ed in families with only one affected child (simplex families), the contribution of both de novo and

30 novo events was significantly lower than in simplex families.

31 ue T150 in cytoplasmic epidermolysis bullosa simplex granules containing R125C K14 mutants.

32 ome sequencing and Sanger sequencing in five simplex GS-affected families, we found homozygous or com

33 Many viruses, including herpes simplex (HSV), are recruited to their host cells via int

34 oma (BK) viremia (8.2% vs 11.1%), and herpes simplex infections (5.5% vs 4.0%).

35 kin blistering disease epidermolysis bullosa simplex is keratin filament (KF) network collapse caused

36 ients with a unilateral and relapsing herpes simplex keratitis (HSK group) that was quiescent during

37 life (QoL) in patients with quiescent herpes simplex keratitis compared with control patients without

38 ents were diagnosed sequentially with herpes simplex keratitis, then Acanthamoeba keratitis before re

39 ndritiform keratopathy, such as prior herpes simplex keratitis, varicella-zoster viral keratitis, the

40 th antiviral medications for presumed herpes simplex keratitis; 4 patients underwent diagnostic testi

41 The experiment was designed according to simplex-lattice method and different types of gluten-fre

42 The optimization was carried out using a simplex method.

43 nterior uveitis (AU), owing to either herpes simplex or varicella zoster virus, by using the Standard

44 ith a possible role in epidermolysis bullosa simplex pathogenesis.

45 fully recapitulate the epidermolysis bullosa simplex phenotype, is advisable before commencing clinic

46 as increased over time, standard-curve based simplex quantitative polymerase chain reaction (qPCR) an

47 of toxocariasis seropositivity and Anisakis simplex sensitization when compared to healthy controls.

48 had seropositivity of fasciolosis, Anisakis simplex sensitization, and the presence of Blastocystis

49 Exact simplex solvers based on rational arithmetic require a n

50 y, the profiles were regarded as data in the Simplex space and subjected to its specific tools or tra

51 of a safe and effective asymptomatic herpes simplex vaccine that is selectively based on CD8(+) T-ce

52 of a safe and effective T-cell-based herpes simplex vaccine.

53 ase chain reaction (PCR) analysis for Herpes simplex, varicella zoster, cytomegalovirus, Epstein-Barr

54 sessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficiency viru

55 Herpes simplex virus (HSV) 1 stimulates type I IFN expression t

56 eurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people worldwide

57 hat US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses, such a

58 Herpes simplex virus (HSV) anterograde transport in neuronal ax

59 d antimicrobial factor that restricts herpes simplex virus (HSV) by activating type I interferon and

60 The herpes simplex virus (HSV) capsid is released into the cytoplas

61 dy employed a novel strategy in which herpes simplex virus (HSV) carrying a small interfering RNA (si

62 Herpes simplex virus (HSV) causes acute and relapsing symptoms

63 CSF) commonly predicts the absence of herpes simplex virus (HSV) central nervous system (CNS) infecti

64 After entry into the nucleus, herpes simplex virus (HSV) DNA is coated with repressive protei

65 During lytic infection, herpes simplex virus (HSV) DNA is replicated by a mechanism inv

66 Herpes simplex virus (HSV) entry into a subset of cells require

67 rus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions with a vi

68 Herpes simplex virus (HSV) establishes a latent reservoir in ne

69 Herpes simplex virus (HSV) establishes latency and reactivates

70 on of 'pore dead' GluA1 subunits (via herpes simplex virus (HSV) GluA1-Q582E) in the lateral core or

71 Pritelivir is a well-tolerated novel herpes simplex virus (HSV) helicase-primase inhibitor that redu

72 n as a transcriptional coactivator of herpes simplex virus (HSV) immediate early (IE) transcription,

73 The herpes simplex virus (HSV) infected cell culture polypeptide 27

74 -virus interactions that occur during herpes simplex virus (HSV) infection are not fully understood.

75 Importance: Genital herpes simplex virus (HSV) infection is a prevalent sexually tr

76 Herpes simplex virus (HSV) infection is restricted to epithelia

77 Herpes simplex virus (HSV) infection is widespread in the human

78 Studies of herpes simplex virus (HSV) infections of humans are limited by

79 Herpes simplex virus (HSV) infections of the central nervous sy

80 During lytic herpes simplex virus (HSV) infections, the virion host shutoff

81 The terminase of herpes simplex virus (HSV) is composed of three subunits encode

82 Herpes simplex virus (HSV) is investigated not only as a human

83 ic penetrating keratoplasty (PPK) for herpes simplex virus (HSV) keratitis.

84 r HCF-1 is required for initiation of herpes simplex virus (HSV) lytic infection and for reactivation

85 us or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 7

86 isualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.

87 Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerve destr

88 The rate of oral herpes simplex virus (HSV) shedding was lower overall, and chil

89 Chronic infections with herpes simplex virus (HSV) type 1 are highly prevalent in popul

90 The herpes simplex virus (HSV) type I alkaline nuclease, UL12, has

91 Samples were tested for herpes simplex virus (HSV) types 1 and 2, Epstein-Barr virus (E

92 croinjecting before training a single herpes simplex virus (HSV) vector expressing either CRISPR/Cas9

93 Presence of CMV, EBV, and herpes simplex virus (HSV) were independent predictors of genit

94 Caused by the herpes simplex virus (HSV), herpes is a viral infection that is

95 ection is caused by 2 subtypes of the herpes simplex virus (HSV), HSV-1 and HSV-2.

96 nomolar irreversible activity against herpes simplex virus (HSV), human papilloma virus, respiratory

97 ntry and subsequent lateral spread of herpes simplex virus (HSV), requires the four envelope glycopro

98 Herpes simplex virus (HSV), which triggers the STING signaling

99 ignificant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated divergen

100 before test HIV counselling, HIV and herpes simplex virus (HSV)-2 testing, and post-test counselling

101 In herpes simplex virus (HSV)-infected cells, ND10 bodies assemble

102 ) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).

103 l brain inflammation, commonly due to herpes simplex virus (HSV).

104 ron (IFN) is important for control of herpes simplex virus (HSV-1) in the central nervous system (CNS

105 ered to the same sensory neurons that herpes simplex virus (HSV-1) infects when applied peripherally

106 mediates recognition and clearance of herpes simplex virus (HSV1)-infected cells.

107 on on tga20/CD11b-thymidine kinase of Herpes simplex virus (HSVTK) cerebellar organotypic cultured sl

108 Oncolytic herpes simplex virus (oHSV) selectively replicates in cancer ce

109 ed the fate and efficacy of oncolytic herpes simplex virus (oHSV)-armed mesenchymal stem cells (MSCs)

110 reign DNA-sensing pathways.IMPORTANCE Herpes simplex virus 1 (HSV-1) afflicts 80% of the population w

111 Herpes simplex virus 1 (HSV-1) and HSV-2 are large, double-stra

112 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (PRV), ha

113 cts of intrinsic immunity to restrict herpes simplex virus 1 (HSV-1) as well as other viruses.

114 The herpes simplex virus 1 (HSV-1) capsid is a huge assembly, appro

115 ase in virus-infected mice.IMPORTANCE Herpes simplex virus 1 (HSV-1) causes cold sores and neonatal h

116 Herpes simplex virus 1 (HSV-1) encodes the multifunctional neur

117 Herpes simplex virus 1 (HSV-1) enters mice via olfactory epithe

118 of cell membranes and is required for herpes simplex virus 1 (HSV-1) entry (1-3).

119 The dissection of the herpes simplex virus 1 (HSV-1) entry mechanism is complicated b

120 ether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 from the

121 Herpes simplex virus 1 (HSV-1) establishes latency within the s

122 Herpes simplex virus 1 (HSV-1) establishes lifelong infection i

123 us presence of a helper virus such as herpes simplex virus 1 (HSV-1) for productive replication.

124 on greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by still unkno

125 construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so that the

126 The herpes simplex virus 1 (HSV-1) ICP0 protein is an E3 ubiquitin

127 lear bodies (PML NBs), is a target of herpes simplex virus 1 (HSV-1) ICP0-mediated degradation.

128 For example, herpes simplex virus 1 (HSV-1) immediate early (IE) protein ICP

129 CF1 is best known for its function in herpes simplex virus 1 (HSV-1) immediate early gene transcripti

130 Herpes simplex virus 1 (HSV-1) infection is an incurable viral

131 Herpes simplex virus 1 (HSV-1) infection is widespread among hu

132 ntrinsic antiviral immune response to herpes simplex virus 1 (HSV-1) infection.

133 in IFN responses can result in lethal herpes simplex virus 1 (HSV-1) infections, usually from encepha

134 Herpes simplex virus 1 (HSV-1) infects humans through stratifie

135 Herpes simplex virus 1 (HSV-1) infects most people and can caus

136 The immediate early protein ICP0 of herpes simplex virus 1 (HSV-1) interacts with CIN85, an adaptor

137 We report here that UL37 of herpes simplex virus 1 (HSV-1) is a protein deamidase that targ

138 Human herpes simplex virus 1 (HSV-1) is a widespread pathogen, with 8

139 Infected cell protein 0 (ICP0) of herpes simplex virus 1 (HSV-1) is an alpha gene product require

140 The UL16 tegument protein of herpes simplex virus 1 (HSV-1) is conserved among all herpesvir

141 Herpes simplex virus 1 (HSV-1) is one of the eight herpesviruse

142 DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of severa

143 S) sequences previously identified in herpes simplex virus 1 (HSV-1) large terminase and human cytome

144 Herpes simplex virus 1 (HSV-1) latency entails the repression o

145 a variant to target the mRNA encoding herpes simplex virus 1 (HSV-1) major transcription regulator IC

146 Herpes simplex virus 1 (HSV-1) most commonly causes recrudescen

147 iously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclear local

148 Herpes simplex virus 1 (HSV-1) remodels nuclear membranes durin

149 reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.

150 Infection of mature HD10.6 neurons by herpes simplex virus 1 (HSV-1) results in a delayed but product

151 Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory reactio

152 ncephalomyocarditis virus (EMCV), and herpes simplex virus 1 (HSV-1) show impaired production of anti

153 4.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN signali

154 Humans occasionally transmit herpes simplex virus 1 (HSV-1) to captive primates, who recipro

155 ty of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axonal term

156 Herpes simplex virus 1 (HSV-1) typically causes recurrent cold

157 Herpes simplex virus 1 (HSV-1) UL20 plays a crucial role in the

158 The herpes simplex virus 1 (HSV-1) UL37 protein functions in virion

159 otection requisite for an efficacious herpes simplex virus 1 (HSV-1) vaccine remain unclear with resp

160 n, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which has be

161 wing infection of epithelial tissues, herpes simplex virus 1 (HSV-1) virions travel via axonal transp

162 Herpes simplex virus 1 (HSV-1), a leading cause of genital herp

163 TRIM proteins in autophagy induced by herpes simplex virus 1 (HSV-1), encephalomyocarditis virus (EMC

164 Persistent pathogens, such as herpes simplex virus 1 (HSV-1), have evolved a variety of immun

165 ive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian pseudor

166 of the more than 80 mRNAs harbored by herpes simplex virus 1 (HSV-1), only 5 are spliced.

167 Following infection with herpes simplex virus 1 (HSV-1), PIAS1 is recruited to nuclear s

168 e focus on two tegument proteins from herpes simplex virus 1 (HSV-1), pUL7 and pUL51, which have homo

169 lthough many herpesviruses, including herpes simplex virus 1 (HSV-1), stimulate mTORC1, how HSV-1-inf

170 equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inhibitors

171 replication of a related herpesvirus, herpes simplex virus 1 (HSV-1), was not impacted.

172 For herpes simplex virus 1 (HSV-1), we and others have previously p

173 Herpes simplex virus 1 (HSV-1), which causes a variety of disea

174 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed the vi

175 to restrict viral pathogens, such as herpes simplex virus 1 (HSV-1).

176 a (TG) of mice latently infected with herpes simplex virus 1 (HSV-1).

177 bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

178 n AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).

179 that have been ocularly infected with herpes simplex virus 1 (HSV-1).

180 meningitis virus (LCMV) infection, or herpes simplex virus 1 (HSV1) infection was profoundly decrease

181 [KSHV], Epstein-Barr virus [EBV], and herpes simplex virus 1 [HSV-1]) genomes and induces the inflamm

182 ulin M/immunoglobulin G antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-Barr vir

183 actiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human parech

184 y to be infected by the herpesviruses herpes simplex virus 1 and murine herpesvirus 68 and the parvov

185 ependent of varicella-zoster virus or herpes-simplex virus 1 coinfection.

186 we have characterized the full-length herpes simplex virus 1 gB in a native membrane by displaying it

187 is method to rapidly deliver a 152 kb herpes simplex virus 1 genome cloned in yeast into mammalian ce

188 uctive infection of cultured cells by herpes simplex virus 1 has made it a paradigm for this mode of

189 olecular organization of chromatin in herpes simplex virus 1 infection and its effect on the transpor

190 d survival during influenza virus and herpes simplex virus 1 infections.

191 ication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.

192 focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesviruses:

193 mains, as well as a truncated form of herpes simplex virus 1 thymidine kinase (HSV-TK).

194 dence of acute herpesvirus infection; herpes simplex virus 1 was found most often.

195 he structural, material properties of herpes simplex virus 1 were investigated.

196 8%; HHV8, 6%; Epstein-Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%; HHV7, 2

197 obulin G seropositivity for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were studied

198 eas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a strong ant

199 ion of antagomir-155 nanoparticles to herpes simplex virus 1-infected mice led to diminished SK lesio

200 Random walk modelling of herpes simplex virus 1-sized particles in a three-dimensional s

201 established that cells infected with herpes simplex virus 2 (HSV-2) are disrupted in their ability t

202 veral prophylactic vaccines targeting herpes simplex virus 2 (HSV-2) have failed in the clinic to dem

203 irion host shutoff protein (vhs) as a herpes simplex virus 2 (HSV-2) protein capable of disrupting SG

204 of people worldwide are infected with herpes simplex virus 2 (HSV-2), and to date, an efficacious pro

205 cella zoster virus, 3%; HHV7, 2%; and herpes simplex virus 2, 1%.

206 Alpha herpesviruses, such as herpes simplex virus and pseudorabies virus (PRV), are neuroinv

207 e structural stability of icosahedral herpes simplex virus capsids.

208 ecimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 months apar

209 The herpes simplex virus for example evades immune surveillance by

210 The transcription factor ICP4 from herpes simplex virus has a central role in regulating the gene

211 CP47, a small protein produced by the herpes simplex virus I.

212 its HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potential of r

213 rimase inhibitor for the treatment of herpes simplex virus infections, was prepared.

214 opment of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although nucleosi

215 The capsid of herpes simplex virus is built up of a variety of proteins, and

216 m entering the endoplasmic reticulum, herpes simplex virus is hidden from cytotoxic T lymphocytes, wh

217 creased the recurrence of any type of herpes simplex virus keratitis by approximately half.

218 ns of the superficial cornea, such as herpes simplex virus keratitis or Acanthamoeba keratitis.

219 titis is significantly different from herpes simplex virus keratitis, and further studies using this

220 Further, in the murine model of herpes simplex virus keratitis, corneal pathology and lymphangi

221 significantly different from that of herpes simplex virus keratitis.

222 ophagy/xenophagy results with mutated herpes simplex virus lacking its ICP34.5 neurovirulence gene (H

223 ation of nonintegrin receptors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associated he

224 cally diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated be

225 activation, exhibiting parallels with herpes simplex virus reactivation.

226 lipid raft-dwelling US9 protein from Herpes Simplex Virus strikingly overlaps with that of the amylo

227 nic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by the pr

228 particles served to deliver the human Herpes simplex virus thymidine kinase type-1 (HSVtk) gene, whic

229 Infectious titers of EBOV or herpes simplex virus type 1 (HSV-1) in detergents-treated cell

230 Lytic infection with herpes simplex virus type 1 (HSV-1) induces profound modificati

231 f virus reactivation following ocular herpes simplex virus type 1 (HSV-1) infection.

232 Herpes simplex virus type 1 (HSV-1) is a leading cause of neuro

233 Herpes simplex virus type 1 (HSV-1) is a ubiquitous virus and a

234 Glycoprotein D (gD) of herpes simplex virus type 1 (HSV-1) is one of four glycoprotein

235 urs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) genital i

236 l stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies upon nu

237 rted a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were functiona

238 nal approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large DNA geno

239 Knockout of TRIM14 impairs herpes simplex virus type 1 (HSV-1)-triggered antiviral respons

240 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the brains

241 Herpes simplex virus type 1 and Alzheimer's disease: possible m

242 are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).

243 und wide distribution of O-glycans on herpes simplex virus type 1 glycoproteins and demonstrated that

244 ncoding the prodrug-converting enzyme herpes simplex virus type 1 thymidine kinase (HSV1-tk) into the

245 CMV, herpes simplex virus type 1, and human herpesvirus 6 infection

246 iation of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, and Epst

247 ens were detected by mNGS (4 cases of herpes simplex virus type 1, including 1 case of coinfection wi

248 for mapping O-glycosylation sites on herpes simplex virus type 1.

249 e (TDF) has in vitro activity against herpes simplex virus type 2 (HSV-2) and reduced HSV-2 acquisiti

250 We focused on herpes simplex virus type 2 (HSV-2) because prior published dat

251 or protection against transmission of herpes simplex virus type 2 (HSV-2) has been examined in a vari

252 Despite the high prevalence of herpes simplex virus type 2 (HSV-2) in sub-Saharan Africa, the

253 Genital herpes simplex virus type 2 (HSV-2) infection causes recurrent

254 ystem's protective effect against one herpes simplex virus type 2 (HSV-2) infection protects against

255 ell function in response to a mucosal herpes simplex virus type 2 (HSV-2) infection.

256 HIV and herpes simplex virus type 2 (HSV-2) infections cause a substant

257 clinical development for treatment of herpes simplex virus type 2 (HSV-2) infections.

258 Herpes simplex virus type 2 (HSV-2) reactivation is accompanied

259 s been incompletely characterized for herpes simplex virus type 2 (HSV-2).

260 Herpes simplex virus type 2 (HSV-2; herpes) exacerbates human i

261 cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.

262 3408 persons coinfected with HIV and herpes simplex virus type 2.

263 al cord in response to infection with herpes simplex virus type 2.

264 rachomatis, Neisseria gonorrhoeae, or herpes simplex virus type 2.

265 ctions affected by lytic infection of Herpes Simplex Virus type I in Human primary fibroblasts.

266 tic neuralgia using a murine model of Herpes Simplex Virus Type-1 (HSV-1) infection.

267 en, including bacterial vaginosis and herpes simplex virus type-2 (HSV-2) infection.

268 ct of antiretroviral therapy (ART) on herpes simplex virus type-2 (HSV-2) replication is unclear.

269 trols for sociodemographic variables, herpes simplex virus type-2 status, and recreational drug use.

270 s (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and varicel

271 1 weeks) to Epstein Barr virus (EBV), herpes simplex virus types 1 and 2 (HSV-1 and HSV-2), and cytom

272 Seroprevalence of EBV, CMV, herpes simplex virus types 1 and 2, and human herpesvirus 8 was

273 r virus (EBV), cytomegalovirus (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8) using

274 irus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluation of

275 mucosal immunity and then compare the herpes simplex virus, human immunodeficiency virus, and influen

276 oma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus, and Eps

277 stantial impairment: cytomegalovirus, herpes simplex virus, rubella virus, Toxoplasma gondii, and Zik

278 oplasmosis, rubella, cytomegalovirus, herpes simplex virus, syphilis, and human immunodeficiency viru

279 V range, 52%-95%; NPV range, 79%-80%; herpes simplex virus, vulvar ulcerations: sensitivity, 20%; spe

280 Herpes simplex virus-1 (HSV-1) causes life-long morbidities in

281 Herpes Simplex Virus-1 (HSV-1) is a ubiquitous human pathogen,

282 Herein we demonstrate that oncolytic herpes simplex virus-1 (HSV-1) potently activates human periphe

283 rferon-alpha secretion in response to herpes simplex virus-1 (HSV-1), whereas granzyme-B induction up

284 HC I low) tumors respond to oncolytic herpes simplex virus-1 (oHSV-1) and PD-1 blockade combination t

285 ary generated using the high-capacity herpes simplex virus-1 amplicon technology to deliver bacterial

286 the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced production of

287 e address this deficit, focusing on a herpes simplex virus-1 protein, ICP27.

288 neralized infections that can lead to herpes simplex virus-induced acute liver failure.

289 Furthermore, an unrelated herpes simplex virus-thymidine kinase (HSV-TK) promoter was str

290 parvovirus B19), cytomegalovirus, and herpes simplex virus.

291 n also proved to be well tolerated in herpes simplex virus.

292 Herpes simplex viruses (HSV) are human pathogens that switch be

293 icant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or genita

294 us isolates to use entry receptors of herpes simplex viruses (HSV).

295 Herpes simplex viruses (HSVs) are human pathogens that can caus

296 Herpes simplex viruses (HSVs) are unusual in that unlike most e

297 Herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) infect and est

298 mps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, enterovirus

299 from the Drosophila fushi tarazu and Herpes simplex VP16 was created.

300 plied to patients with epidermolysis bullosa simplex with intraepidermal blistering.