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1 otein coding sequence using the technique of single-strand conformational polymorphism.
2 tation and allelic loss by the method of PLR-single-strand conformational polymorphism.
3 nce and mutational analysis was performed by single-strand conformational polymorphism.
4 ns within the coding exons of FHIT using PCR-single-strand conformational polymorphism.
5 nal diversity by CDR3 size spectratyping and single-strand conformational polymorphism.
6 e of the remaining TDG allele as analyzed by single-strand conformational polymorphism.
7 mbines heteroduplex analysis and analysis of single-stranded conformational polymorphisms.
9 alteration in exon 1 of KRAS was detected by single strand conformational polymorphism analysis of DN
10 malignancies upon polymerase chain reaction-single stranded conformational polymorphism analysis of
11 orphisms were detected using heteroduplex or single-strand conformational polymorphism analysis after
12 from brain tissue and serum were compared by single-strand conformational polymorphism analysis and d
14 or alterations in the PTEN/MMAC1 gene, using single-strand conformational polymorphism analysis and d
15 rinting, a technique combining components of single-strand conformational polymorphism analysis and d
16 utations in exons 4-8 of the p53 gene, using single-strand conformational polymorphism analysis and m
17 ction fragment length polymorphism analysis, single-strand conformational polymorphism analysis and/o
23 Restriction fragment length polymorphism and single-strand conformational polymorphism analysis of ec
31 Using reverse transcription-PCR and "Cold" single-strand conformational polymorphism analysis, 6 of
32 this locus, by polymerase chain reaction and single-strand conformational polymorphism analysis, and
33 from 146 bladder tumors by PCR, screened by single-strand conformational polymorphism analysis, and
34 al individuals were also characterized using single-strand conformational polymorphism analysis, DNA
39 3p deletion and none of 19 tumors studied by single-stranded conformational polymorphism analysis dis
41 NA from 75 patients with SLE was screened by single-stranded conformational polymorphism analysis for
42 tumor and nontumorous tissue was analyzed by single-stranded conformational polymorphism analysis for
43 erse transcriptase-polymerase chain reaction/single-stranded conformational polymorphism analysis in
44 n the micro heavy chain (IGHM) gene, we used single-stranded conformational polymorphism analysis to
47 ublecortin open reading frame as assessed by single-stranded conformational polymorphism analysis.
48 fragments were screened for mutations using single-stranded conformational polymorphism analysis; an
51 class I and class II genes was determined by single-strand conformational polymorphism and allele-spe
52 gene allelic loss, p53 gene mutations using single-strand conformational polymorphism and direct seq
53 amplified with the PCR and screened by using single-strand conformational polymorphism and direct seq
54 BCL10 mutation by polymerase chain reaction-single-strand conformational polymorphism and DNA sequen
55 LOH were screened for mutations in PTC using single-strand conformational polymorphism and heterodupl
56 ns and 120 African Americans was examined by single-strand conformational polymorphism and restrictio
58 to a single cycle of replication, employing single-stranded conformational polymorphism and DNA sequ
59 Analysis of survival difference by p53 by single-stranded conformational polymorphism and IHC, int
61 by immunohistochemistry and DNA mutations by single-stranded conformational polymorphism and sequenci
62 the P53 gene using polymerase chain reaction/single strand conformational polymorphism, and subsequen
63 uthern blot, comparative multiplex PCR, PCR -single-strand conformational polymorphism, and DNA seque
64 ty was estimated by the number of bands in a single-strand conformational polymorphism assay, whereas
65 NAs were examined by a combined heteroduplex-single-stranded conformational polymorphism assay to ass
66 31 controls) were amplified and analysed for single-strand conformational polymorphism by capillary e
67 mas and 9 breast cancer cell lines using PCR-single-strand conformational polymorphism, direct DNA se
68 N-ras mutations by polymerase chain reaction-single-strand conformational polymorphism followed by DN
69 alysis of exon-3 of the beta-catenin gene by single-strand conformational polymorphism followed by DN
71 n microsatellite repeat were isolated by PCR-single-strand conformational polymorphism from populatio
73 by heteroduplex mobility assay combined with single-stranded conformational polymorphism (HDA+SSCP) a
74 PCR products were screened for mutations by single-strand conformational polymorphism-heteroduplex a
75 genes and expressed sequence tags by scoring single-strand conformational polymorphisms in a panel of
76 med a mutation screen of the TLR2 gene using single-stranded conformational polymorphism in 110 norma
78 tients using a polymerase chain reaction and single-strand conformational polymorphism method followe
81 t diagnosis, using polymerase chain reaction-single-strand conformational polymorphism (PCR-SSCP) ana
82 rearrangements by polymerase chain reaction single-strand conformational polymorphism (PCR-SSCP) in
83 This study uses polymerase chain reaction/single-strand conformational polymorphism (PCR/SSCP) ana
84 extracted with proteinase K and analyzed by single-strand conformational polymorphism-PCR for p53 an
85 de sequencing of mobility shifts detected by single-strand conformational polymorphism revealed somat
86 +3A>T mutation by restriction-enzyme digest, single-strand conformational polymorphism screening, or
90 a hybrid technique which combines aspects of single strand conformational polymorphism (SSCP) and did
91 1B gene was inactivated in ovarian cancer by single strand conformational polymorphism (SSCP) and het
94 sed variant-specific hybridization (VSH) and single-strand conformational polymorphism (SSCP) analyse
95 ions in GCAP1 exons using DNA sequencing and single-strand conformational polymorphism (SSCP) analysi
97 techniques for identifying point mutations (single-strand conformational polymorphism (SSCP) and did
100 cipients, HCV sequences were analyzed by the single-strand conformational polymorphism (SSCP) assay a
102 on of the element, were identified using the single-strand conformational polymorphism (SSCP) method.
104 ely) and Kenya (n=1106) for polymorphisms by single-stranded conformational polymorphism (SSCP) analy
105 We did not detect germ-line mutations by single-stranded conformational polymorphism (SSCP) analy
106 J1S6 sequence, and could also be detected by single-stranded conformational polymorphism (SSCP) as th
107 lyzed by polymerase chain reaction (PCR) and single-stranded conformational polymorphism (SSCP) for p
108 analysis for C34T mutation was performed by single-stranded conformational polymorphism (SSCP) in 22
109 t combined heteroduplex analysis (HDA) and a single-stranded conformational polymorphism (SSCP) metho
110 c proband was examined for base exchanges by single-stranded conformational polymorphism (SSCP).
111 nd expressed sequence tags (ESTs) by scoring single-stranded conformational polymorphisms (SSCPs) in
112 besity for single nucleotide changes using a single-strand conformational polymorphism strategy.
114 ons 2 through 11 of p53 were screened by the single-strand conformational polymorphism technique.
115 mical analysis and polymerase chain reaction/single strand conformational polymorphism to examine whe
116 r its potential role in bladder cancer using single-strand conformational polymorphism to screen for
119 RAPD markers were cloned and sequenced, and single-strand conformational polymorphisms were identifi
120 estriction fragment length polymorphisms and single-strand conformational polymorphisms were used to
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