コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 adycardia, consistent with AC9 expression in sinoatrial node.
2 nscriptional regulator within the developing sinoatrial node.
3 otein was not expressed in the centre of the sinoatrial node.
4 lowering agent that acts specifically on the sinoatrial node.
5 of ionic currents in pacemaker cells of the sinoatrial node.
6 ial node cells as well as that of the intact sinoatrial node.
7 It also is present in areas within the sinoatrial node.
8 properties of pacemaker myocytes in the aged sinoatrial node.
9 sistently Islet-1 mRNA was detected in human sinoatrial node.
10 unction, with initial, larger effects on the sinoatrial node.
11 er-law behavior comparable to those of human sinoatrial node.
12 The AVJ is a primary backup pacemaker to the sinoatrial node.
13 may mask intrinsic fractal behaviour of the sinoatrial node.
14 and peripheral cells that make up the native sinoatrial node.
15 right atrial foci, especially those near the sinoatrial node.
16 e wall, corresponding to the location of the sinoatrial node.
17 ine in situ detection of connexins in rabbit sinoatrial node (a tissue that is particularly rich in f
18 dly rectifying K+ (GIRK or KACh) channels of sinoatrial node and atria play a major role in beat-to-b
19 shown at birth to be present throughout the sinoatrial node and atrial muscle, however, at one month
21 ty of cell types and distribution within the sinoatrial node and cell-cell interactions add complexit
22 ntify Islet-1 as a novel marker of the adult sinoatrial node and do not provide evidence for Islet-1(
23 izes the leading pacemaker region within the sinoatrial node and hence is crucial for stable heart ra
24 the HCN1 protein is highly expressed in the sinoatrial node and is colocalized with HCN4, the main s
26 els of MiRP1 and HCN subunits in the cardiac sinoatrial node and the contribution of pacemaker channe
29 ) hearts the focus ultimately shifted to the sinoatrial node at a very prolonged cycle length (initia
31 within regions of the heart that become the sinoatrial node, atrioventricular node, and bundle of Hi
32 t density contributes to the acceleration of sinoatrial node automaticity and explains, in part, the
36 r evaluated in rabbit isolated patch-clamped sinoatrial node cells (n = 21), where we found that 5 mu
38 neath the cell plasma membrane (subspace) of sinoatrial node cells (SANCs) occurring during diastolic
39 ct in concert with ion channels to confer on sinoatrial node cells (SANCs) their status of dominance
41 caffeine (2-4 mM) to isolated single rabbit sinoatrial node cells acutely reduces their spontaneous
42 geneity of the electrical activity of single sinoatrial node cells as well as that of the intact sino
48 (as measured by cell capacitance) of rabbit sinoatrial node cells was investigated using the whole-c
49 n of pacemaker mechanisms in single isolated sinoatrial node cells, explanted beating sinoatrial node
54 ysis that Cx43 protein expression within the sinoatrial node decreased with age; however, the express
56 r conduction block and arrhythmias caused by sinoatrial node dysfunction are clinically important and
59 hmias: sinus pauses and bradycardia indicate sinoatrial node dysfunction, whereas preexcitation and a
61 ession and network analysis identified novel sinoatrial node-enriched genes and predicted that the tr
62 P maps during normal atrial excitation (i.e. sinoatrial node excitation) were compared to those obser
63 h-frequency ratio [P=0.03]) and less erratic sinoatrial node firing (eg, lower Poincare ratio [P=0.02
64 opagation of the action potential across the sinoatrial node, from the initiation point to the crista
69 [Ca(2+)]i release and Ca(2+) handling in the sinoatrial node, impaired pacemaker activity and symptom
71 beta-adrenergic receptor stimulation of the sinoatrial node in intact dogs is markedly blunted when
75 ectrical signal is known to originate in the sinoatrial node myocyte, but exactly what role Ca plays
76 ow that depressed excitability of individual sinoatrial node myocytes (SAMs) contributes to reduction
80 ted sinoatrial node cells, explanted beating sinoatrial node preparation, telemetric in vivo electroc
81 nitiates early in development, represses the sinoatrial node program and pacemaker activity on the le
82 d indices of sympathovagal modulation of the sinoatrial node (ratio of low-frequency to high-frequenc
83 by bradycardia, sinus dysrhythmia, prolonged sinoatrial node recovery time, increased sinoatrial cond
84 tructive heart surgery include injury of the sinoatrial node (SAN) and atrioventricular node (AVN), r
85 highly expressed in embryonic myocardium and sinoatrial node (SAN) and is required for cardiac automa
89 te the activation within the human or canine sinoatrial node (SAN) because they are intramural struct
90 rucial regulatory role in the development of sinoatrial node (SAN) by repressing the expression of Nk
93 t these sites and how this relates to normal sinoatrial node (SAN) development remain uncharacterized
95 polymorphic ventricular tachycardia manifest sinoatrial node (SAN) dysfunction, the mechanisms of whi
96 primary pacemaker area of the intact rabbit sinoatrial node (SAN) exhibits robust positive labeling
97 d by Ras-related small G proteins, regulates sinoatrial node (SAN) ion channel activity through a mec
98 ght to confirm our hypothesis that the human sinoatrial node (SAN) is functionally insulated from the
100 her their presence nor their contribution to sinoatrial node (SAN) pacemaker activity has been invest
101 ated in controlling automaticity in isolated sinoatrial node (SAN) pacemaker cells, but the potential
103 rough K(+) channels are essential for proper sinoatrial node (SAN) pacemaking, but the influence of i
105 ces in cardiomyocyte automaticity permit the sinoatrial node (SAN) to function as the leading cardiac
106 Dysfunction of pacemaker activity in the sinoatrial node (SAN) underlies "sick sinus" syndrome (S
108 pecialized cardiomyocytes located within the sinoatrial node (SAN), and is responsible for originatin
112 situ studies indicated that Pitx2 suppresses sinoatrial node (SAN)-specific gene expression, includin
118 of this is the use of optical mapping in the sinoatrial node (SAN): when microelectrode and optical r
119 We developed a computational model of the sinoatrial node that showed that a loss of SAN cells bel
124 pecifically inhibits the I(f) current in the sinoatrial node to lower heart rate, without affecting o
125 en by an electrical impulse generated in the sinoatrial node to propagate from atria to ventricles.
127 ional computerized numerical modeling of the sinoatrial node was conducted to validate the theoretica
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。