ライフサイエンスコーパス: sinus

1 epicardial ablation from within the coronary sinus.

2 onal epicardial ablation within the coronary sinus.

3 larged and fail to merge with the urogenital sinus.

4 between the carotid system and the cavernous sinus.

5 LN parenchyma from lymph in the subcapsular sinus.

6 as the most effective way to treat pilonidal sinus.

7 ntional (69 patients) treatment of pilonidal sinus.

8 ith acute upper abdomen pain and discharging sinus.

9 kages that develops into a chronic presacral sinus.

10 als (sympetally) and modulation of growth at sinuses.

11 he drainage and ventilation of the paranasal sinuses.

12 eventing the drainage and ventilation of the sinuses.

13 ing clearance during passage through splenic sinuses.

14 R1(-/-) effector T cells failed to enter the sinuses.

15 of orbital fat and clot in the confluence of sinuses.

16 fected the asymmetry in volumes of maxillary sinuses.

17 ed by reduction in plaque necrosis in aortic sinus (35.8%) and in brachiocephalic artery (26%), with

18 CT angiography (-48.6%), CT of the paranasal sinus (-39.6%), cerebral or carotid CT angiography (-36.

19 antly less ablation from within the coronary sinus (7.0% versus 71.9%; P<0.01).

20 ass and/or allograft placed in the maxillary sinus after the osteotome technique underwent remodeling

21 old male presented with multiple discharging sinuses along the penis.

22 from lower margin to the floor of maxillary sinus and alveolar crest in the 1(st) molar and 2(nd) mo

23 or alveolar artery to the floor of maxillary sinus and alveolar crest.

24 To date, only a few genes for familial sinus and atrioventricular conduction dysfunction are kn

25 In contrast, renal sinus and femoral intermuscular fats were not differenti

26 is located on the lateral wall of maxillary sinus and may become injured during such surgical proced

27 numbers of regulatory T cells both in aortic sinus and spleen with higher mRNA expression of CTLA4 (3

28 herosclerotic plaque size in both the aortic sinus and the thoracoabdominal aorta, and were less infl

29 tions between anatomic variants of paranasal sinuses and chronic or recurrent sinusitis.

30 ions, but these infections lack the draining sinuses and fungal grains characteristic of eumycetoma.

31 ed with the use CT windows dedicated for the sinuses and head.

32 bullosa is connected with the development of sinuses and the incidence of inflammation within them.

33 nization of the urethra, a single urogenital sinus, and clitoral hypertrophy or ambiguous external ge

34 ncement of the vein of Labbe, sphenoparietal sinus, and superficial middle cerebral vein was graded b

35 The nose, paranasal sinuses, and associated lymphoid tissues play important

36 es, though are concentrated around the dural sinuses, and have a unique transcriptional profile.

37 endophytic, nearness to collecting system or sinus, anterior or posterior, and location relative to p

38 cement of bioglass and/or allograft into the sinus area using an osteotome technique in 37 patients w

39 e arrhythmia included sinoatrial (SA) block, sinus arrest, 2 degrees and 3 degrees atrioventricular (

40 syncope, advanced atrioventricular block or sinus arrest, and no structural heart disease.

41 Expiration-triggered sinus arrhythmia (ETA) is a potent and independent post-

42 Respiratory sinus arrhythmia (RSA), a measure of cardiac vagal modul

43 related to expiration (expiration-triggered sinus arrhythmia [ETA]) from short-term recordings of el

44 nent in HRV similar to mammalian respiratory sinus arrhythmia in an amphibian, the toad Rhinella schn

45 A determination by quantifying the amount of sinus arrhythmia related to expiration (expiration-trigg

46 ch of it arises independently of respiratory sinus arrhythmia.

47 ardiac vagal tone and pronounced respiratory sinus arrhythmia.

48 cardiac vagal tone give rise to respiratory sinus arryhthmia (RSA), with maximum tone in the post-in

49 choroid plexus, pituitary gland, and venous sinuses as expected from the pharmacology of dihydroergo

50 evaluate the efficacy and safety of coronary sinus aspiration (CSA) procedure to reduce the volume of

51 ecords of 551 patients who underwent lateral sinus augmentation at Tufts University School of Dental

52 lants placed in sites treated with maxillary sinus augmentation using anorganic bovine bone (ABB), an

53 SMT is, on average, 1 mm in patients seeking sinus augmentation.

54 may jeopardize the final clinical outcome of sinus augmentation.

55 ne perforation is examined in lateral window sinus augmentation.

56 rly, in HL-1 cardiomyocytes, the drug slowed sinus automaticity, reduced phase 0 upstroke slope, and

57 reover, Slc26a6(-/)(-) mice show evidence of sinus bradycardia and fragmented QRS complex, supporting

58 ent, If, underlies exercise training-induced sinus bradycardia in rodents.

59 One such disorder, familial sinus bradycardia, is caused by the S672R mutation in HC

60 ion induces electrical changes, resulting in sinus bradycardia, sinus pauses, and a susceptibility to

61 Rare episodes of sinus bradycardia, spontaneous seizure, and sudden death

62 er, pericardial, muscle, pancreas, and renal sinus) by magnetic resonance imaging.

63 d significantly lower residence times in the sinus cavity (AgNP concentrations of 3.76ppm after 3h co

64 t, we evaluated silver residence time in the sinus cavity after intranasal delivery of AgNPs and AgNO

65 ications, as concentrations of silver in the sinus cavity drop below the minimum bactericidal concent

66 eatment based on the complete removal of the sinus cavity with a minimal surgical wound.

67 Depletion of subcapsular sinus CD169-positive macrophages by clodronate-containin

68 tic endothelial cells lining the subcapsular sinus ceiling stabilizes interfollicular CCL21 gradients

69 nalysed 214 patients who underwent paranasal sinus computed tomography.

70 imally invasive treatment surgery, pilonidal sinus could become a disease treated with an endoscopic

71 ue to an inability or impediment to coronary sinus (CS) lead implantation.

72 c variants and landmarks on the preoperative sinus CT with a focus on those that predispose patients

73 t viscera sign, collar sign and other signs (sinus cut-off sign, hump sign, band sign).

74 ted with hemorrhage in patients with lateral sinus DAVFs than does CVR, and thus may offer guidance i

75 From 1995 to 2016, 163 cases of lateral sinus DAVFs were included and divided into hemorrhagic a

76 aorta en face and the cross-sectional aortic sinus, decreased macrophage number and apoptosis, and pr

77 al: a stem cell hypothesis with a urogenital sinus-derived progeny of all prostatic epithelial cells

78 related sinus, which results in inflammatory sinus disease.

79 mical variations and their relation to known sinus drainage pathways.

80 to look for mucosal disease of the paranasal sinuses, drainage pathways, and presence of anatomical v

81 ent of inflammatory changes in the paranasal sinuses due to different parameters of width (W) and len

82 venous reflux (CVR) in patients with lateral sinus dural arteriovenous fistulas (DAVFs).

83 Our study aimed to evaluate a sinus/edge-corrected (SEC) ZTAC (ZTACSEC), relative to a

84 Afterward, in the pseudo-CT, sinus/edges were automatically estimated as a binary mas

85 Sinus elevation is a reliable and often-used technique.

86 esent study, it was concluded that maxillary sinus elevation with 100% ABB gives predictable results,

87 Renal sinus fat (RSF) is a perivascular fat compartment locate

88 etween fetuin-A, RSF and kidney, human renal sinus fat cells (RSFC) were isolated and cocultured with

89 TA in a patient with dural carotid-cavernous sinus fistula (CCF), which was complicated by increased

90 This study investigates influence of the sinus floor configuration on dimensional stability of gr

91 tal side of each implant were taken, and the sinus floor configuration was classified into concave, a

92 to concave, angle, and flat according to the sinus floor profile at the implant site.

93 ed as the angle between the implant axis and sinus floor, was measured.

94 t to occur when the pneumococci in the upper sinus follow the olfactory nerves and enter the CNS thro

95 f subcutaneous tissues resulting in mass and sinus formation and a discharge that contains grains.

96 Mean initial gain of sinus grafted bone height was 7.0 +/- 1.9 mm, and later

97 y of grafted bone height after the osteotome sinus grafting procedure.

98 grafted bone height was revealed in the flat sinus group compared with the concave group (P <0.001).

99 0 studies, 4 addressed patients with carotid sinus hypersensitivity, and the remaining 6 addressed va

100 count for development of a single urogenital sinus in females exposed to excessive androgen during a

101 an adult male who presented with discharging sinus in the oropharynx.

102 old male presented with pain and discharging sinus in the upper abdomen.

103 implants were placed in 30 grafted maxillary sinuses in 24 patients.

104 tic fever, common colds, rubella and chronic sinus infection, in over 200,000 individuals of European

105 region causes an ideal ground for parasanal sinus infections, by preventing the drainage and ventila

106 illness symptoms (gastrointestinal illness, sinus infections, ear infections, infected wounds).

107 In today's diagnostics of paranasal sinus infections, the role of evaluation of OMC anatomic

108 ly requires S1pr1, whereas movement from the sinus into the parenchyma involves the integrin LFA1 and

109 on in 4 cases and paranasal and/or cavernous sinus invasion in 3 cases.

110 NSD ultrasonography and vTCD of the straight sinus is a promising and easily available technique for

111 Chronic presacral sinus is a significant clinical problem that deserves mo

112 The shape feature, a more pronounced distal sinus, is associated with the colder, drier growing seas

113 issing inflammatory lesions in the paranasal sinuses, it is reasonable to use CT windows dedicated fo

114 r7 (-/-) mice showed reduced aortic arch and sinus lesion areas.

115 rapidly with dramatic increases in coronary sinus levels indicative of myocardial release.

116 Periapical surgeries, implants and maxillary sinus lift are performed on routine basis.

117 o periapical surgery, implant placement, and sinus lifts.

118 In contrast, subcapsular sinus macrophages (SSMs) exposed to lymph-borne HSV-1 we

119 where the perimeter of CD169(+) sub capsular sinus macrophages was disrupted.

120 ons of pathological changes in the paranasal sinuses may be due to selection of unsuitable CT windows

121 as strains, potentially originating from the sinuses, may seed the allograft leading to infections an

122 inflammatory changes in at least one of the sinuses.Measurements of the same inflammatory lesions we

123 and smoking may result in thickening of the sinus membrane.

124 Sphenoid sinus metastasis from hepatocellular carcinoma (HCC) has

125 We present a case of solitary sphenoid sinus metastasis of a 2.7 x 2.3 cm single HCC lesion.

126 dence that UNGD is associated with nasal and sinus, migraine headache, and fatigue symptoms in a gene

127 tional natural gas development and nasal and sinus, migraine headache, and fatigue symptoms in Pennsy

128 The carotid sinus nerve (CSN) conveys electrical signals from the ch

129 Carotid sinus nerve denervation (CSD) has recently been shown to

130 We have previously shown that carotid sinus nerve denervation (CSD) reduces arterial blood pre

131 ttenuated by either resection of the carotid sinus nerve or propranolol.

132 ression of key transcriptional regulators of sinus node and atrial conduction, including Nkx2-5 (NK2

133 RATIONALE: Familial sinus node and atrioventricular conduction dysfunction i

134 have structurally normal atria and preserved sinus node architecture, but expression of key transcrip

135 d in 88% (N=71) of EBW, as opposed to 21% of sinus node breakthrough waves (N=5; P<0.001).

136 EBW were referred to as sinus node breakthrough waves if they were the earliest

137 A total of 218 EBW and 57 sinus node breakthrough waves were observed in 168 patie

138 sights into the complex relationship between sinus node disease and atrial arrhythmias.

139 Although sinus node dysfunction (SND) and atrial arrhythmias freq

140 ration family (n=25) with autosomal dominant sinus node dysfunction (SND) and atrioventricular block

141 uld be a therapeutic target for pathological sinus node dysfunction in veteran athletes.

142 Sinus node dysfunction occurred in 12 patients in the GP

143 eatment for patients with bradycardia due to sinus node dysfunction or atrioventricular block.

144 Ten patients with sinus node dysfunction scheduled for dual-chamber pacema

145 s more major adverse events, major bleeding, sinus node dysfunction, and pacemaker implantation.

146 The sinus node inhibitor ivabradine was approved for patient

147 Further experiments showed that in the sinus node of swim-trained mice, upregulation of miR-423

148 in reaction showed remodeling of miRs in the sinus node of swim-trained mice.

149 orary review summarizes current knowledge on sinus node pathophysiology with the broader goal of yiel

150 Heart rate, sinus node recovery time, Wenckebach cycle length, and a

151 uding slices obtained from the region of the sinus node.

152 stro-esophageal reflux, retinal disease, and sinus-node dysfunction, whereas related heterozygotes ha

153 n an overall proportion of chronic presacral sinus of 9.5%.

154 nstrate that FVIII localizes in the marginal sinus of the spleen of FVIII-deficient mice shortly afte

155 007), without significant differences at the sinus of Valsalva (16.3+/-1.9 versus 16.3+/-1.9 mm/m(2);

156 aorta; and type 3, isolated dilation of the sinus of Valsalva and/or sinotubular junction.

157 ronary artery (AAOCA) from the inappropriate sinus of Valsalva is increasingly recognized by cardiac

158 d more frequently isolated dilatation of the sinus of Valsalva or sinotubular junction (14.2% versus

159 on was defined as isolated dilatation of the sinus of Valsalva or sinotubular junction, isolated dila

160 Macrophage numbers in aortic sinuses of CD11d(-/-) mice were reduced without affectin

161 asonable to use CT windows dedicated for the sinuses or bones.

162 ecretory and basal cells being of urogenital sinus origin.

163 epicardial ablation from within the coronary sinus (P<0.01) and the total length of the MIL (29.3+/-6

164 th larger volume of maxillary sinuses (right sinus: p=0.005; left sinus: p=0.048).

165 axillary sinuses (right sinus: p=0.005; left sinus: p=0.048).

166 eased incidence of atrioventricular block or sinus pause with ozanimod.

167 Significant heart rate slowing and frequent sinus pauses are observed in iNICD mice when compared wi

168 cal changes, resulting in sinus bradycardia, sinus pauses, and a susceptibility to atrial arrhythmias

169 abnormalities including bradycardic events, sinus pauses, atrioventricular block, premature ventricu

170 late, Lamina papyracea, Onodi cell, Sphenoid sinus pneumatization, and (anterior) Ethmoidal artery.

171 ng lectin, helps retain the cells within the sinus, preventing their loss in lymph flow.

172 thought to be assimilated by the urogenital sinus primordial mesenchyme in males during fetal develo

173 G mutation, ranolazine had no effects on the sinus rate or QRS width but shortened the QTc from 509+/

174 hese vessels, running alongside dural venous sinuses, recapitulates the meningeal lymphatic system of

175 without axial planes, through the paranasal sinuses, reconstructed in a sharp algorithm and acquired

176 the number of shocks required to convert to sinus rhythm (2.25+/-1.24 versus 2.41+/-1.22, P=0.31).

177 At 1-year follow-up, 70% remained in sinus rhythm (85% out-of-antiarrhythmic drugs).

178 according to AF duration: PsAF presenting in sinus rhythm (AF induced), PsAF <12 months, and PsAF >12

179 mpared with left-sided heart disease-TR with sinus rhythm (all P<0.05).

180 and posterior wall/septum ablation while in sinus rhythm (group 1), versus same ablation in group 1

181 d with better prognosis only for patients in sinus rhythm (HR: 1.16 per 10 beats/min increase, 95% CI

182 greater all-cause mortality for patients in sinus rhythm (n = 14,166; adjusted HR: 1.11 per 10 beats

183 el omnipolar peak-to-peak voltages (Vmax) in sinus rhythm (SR) and AF.

184 A total of 1262 patients manifested sinus rhythm (SR) at baseline/SR at discharge, 113 SR ba

185 bclinical cardiomyopathy that persists after sinus rhythm (SR) restoration, providing a substrate for

186 results with those obtained in patients with sinus rhythm (SR).

187 ony, which also occurs to some degree during sinus rhythm (SR).

188 dation set AF terminated in 57%, 61% were in sinus rhythm after 4.6 years.

189 in 81% during catheter ablation, 77% were in sinus rhythm after 6 years and multiple ablations.

190 In patients with persistent AF, PVAI in sinus rhythm after direct current cardioversion is assoc

191 F: pulmonary vein antral isolation (PVAI) in sinus rhythm after direct current cardioversion versus P

192 ced ventricular rate by 12 beats/min in both sinus rhythm and AF.

193 l unipolar electrograms were recorded during sinus rhythm and ectopic activation, together with pseud

194 tion from body surface potential maps during sinus rhythm and localizing endocardial and epicardial s

195 Among 4,021 obese individuals with sinus rhythm and no history of atrial fibrillation, 2,00

196 ry vein triggers improves the maintenance of sinus rhythm and reverses disease progression.

197 resence of conduction disorders in BB during sinus rhythm and to study their relation with AF.

198 surface potential maps were recorded during sinus rhythm and ventricular stimulation from 27 endocar

199 Her echocardiography demonstrated normal sinus rhythm at 73 beats per minute.

200 s, left atrium volume >165 mL, absent normal sinus rhythm at admission for EAM, and inducibility of a

201 mbined model of ECG and clinical parameters, sinus rhythm at long-term follow-up could be predicted w

202 Only 76% of patients were in normal sinus rhythm at the beginning of EAM.

203 terone Antagonist (TOPCAT) trial who were in sinus rhythm at the time of echocardiography.

204 llation episodes alternating with periods of sinus rhythm at the time of implantation had a better su

205 tic resonance imaging volumetric data during sinus rhythm for all patients.

206 ial activation mapping were performed during sinus rhythm in 22 patients before pulmonary vein isolat

207 ation (ie, cardioversion) and maintenance of sinus rhythm in patients with atrial fibrillation are re

208 r pharmacological therapy for maintenance of sinus rhythm in patients with both paroxysmal and persis

209 ial benefits of risk factor modification for sinus rhythm maintenance.

210 At 5 years, 73% were in sinus rhythm off antiarrhythmic medications after single

211 ibrillation the first day were reported with sinus rhythm on day 2.

212 ure with reduced ejection fraction in either sinus rhythm or atrial fibrillation (AF).

213 FIRM ablation terminated AF to sinus rhythm or atrial flutter or tachycardia in 59% (PA

214 ith rheumatic mitral valve disease in either sinus rhythm or persistent AF were analyzed using a comb

215 lectrograms were collected using CARTO3v4 in sinus rhythm or ventricular pacing and reviewed for ripp

216 Mortality was lower for patients in sinus rhythm randomized to beta-blockers (HR: 0.73 vs. p

217 Stable sinus rhythm restoration was immediate in 61.5% of patie

218 ular rate control and require restoration of sinus rhythm to improve their quality of life.

219 Absolute timing difference for sinus rhythm was 10+/-5 and 11+/-8 ms respectively, and

220 Stable sinus rhythm was maintained in 32 patients (64%) of the

221 Acute conversion to sinus rhythm was observed in 2 patients after ablation o

222 lectrodes, interelectrode distance: 2 mm) of sinus rhythm was performed in 185 patients during corona

223 culated activation for the in situ hearts in sinus rhythm was similar to patterns recorded in Langend

224 We randomly assigned 1922 patients in sinus rhythm who were scheduled for elective cardiac sur

225 The restoration of sinus rhythm with CA results in significant improvements

226 ythm data, 382 (17%) had AF, 1,602 (70%) had sinus rhythm, and 308 (13%) had "other" rhythm.

227 40%, New York Heart Association class II-IV, sinus rhythm, and heart rate >/=70 beats per minute) and

228 ls, vernakalant-resistant AF was reverted to sinus rhythm, and reinduction of AF by burst pacing (50

229 During sinus rhythm, APD was shorter in LWHs compared to LANG h

230 ssociated with atrial activation compared to sinus rhythm, but has limitations in providing specific

231 Risk in Communities (ARIC) study who were in sinus rhythm, free of valvular disease, and had acceptab

232 uires multiple procedures to maintain stable sinus rhythm.

233 rt failure with reduced ejection fraction in sinus rhythm.

234 atio, 5.43; 95% CI, 3.24-9.12) compared with sinus rhythm.

235 1 hour post procedure for the maintenance of sinus rhythm.

236 scar accurately identify VT channels during sinus rhythm.

237 emaker implantation, whereas the rest are in sinus rhythm.

238 with better prognosis, but only for those in sinus rhythm.

239 found in atrial appendages from patients in sinus rhythm.

240 ricular endocardial mapping was performed in sinus rhythm.

241 litude signals usually occurring late during sinus rhythm.

242 e further intervention to maintain long term sinus rhythm.

243 tients with left-sided heart disease-TR with sinus rhythm.

244 patients with AF compared with patients with sinus rhythm: 10.6+/-5.5 versus 4.7+/-3.5 g, P<0.001.

245 In females, the sinus ridge descends posteriorly to allow the vaginal op

246 reveal a temporal window of development when sinus ridge fate is determined.

247 opulation of mesenchymal cells that regulate sinus ridge morphogenesis.

248 on sexual differentiation of the urogenital sinus ridge, an epithelial thickening that forms where t

249 s with CAH, androgens inhibit descent of the sinus ridge.

250 as connected with larger volume of maxillary sinuses (right sinus: p=0.005; left sinus: p=0.048).

251 evel is the best screening test but petrosal sinus sampling for ACTH may be necessary to distinguish

252 like lymphocytes that survey the subcapsular sinus (SCS) and associated macrophages for pathogen entr

253 mphatics and preferentially bind subcapsular sinus (SCS) CD169(+) macrophages in tumor-draining lymph

254 Aortic sinus sections were stained with Sudan IV for assessment

255 aved-caspase 3 antibody) increased in aortic sinus slices measured as percentage of lesion by 4 mo.

256 th multi-ion channel block, with significant sinus slowing and increased PR, QRS, QT, and QTc interva

257 he optic disc [p=0.881], and bilateral dural sinus stenosis [p=0.837], Mann-Whitney U test).

258 of the optic disc, and bilateral transverse sinus stenosis.

259 The position of the anterior cardinal sinus suggests that it may aid in pressurization equilib

260 not examined the impact of CRS or endoscopic sinus surgery (ESS) upon asthma quality of life (QOL) an

261 a at 3-5 years after a functional endoscopic sinus surgery (FESS) and correlate these data to symptom

262 Although functional endoscopic sinus surgery is an effective means of treating patients

263 ch contribute to a phenotype resembling sick sinus syndrome.

264 nous transcranial Doppler (vTCD) of straight sinus systolic flow velocity (FVsv), and methods derived

265 Peak and mean sinus tachycardia rates were significantly reduced after

266 ed in two patients in the brexanolone group (sinus tachycardia, n=1; somnolence, n=1) and in two pati

267 n promise in the management of inappropriate sinus tachycardia.

268 alysis included 44 CT scans of the paranasal sinuses that were performed in adults.

269 al fluid barrier situated close to the dural sinuses, the site of recently discovered CNS lymphatics.

270 4.46, p=0.01) and isolated superior sagittal sinus thrombosis (HR=0.39, 95% CI=0.14 to 1.04, p=0.05)

271 cephalopathy associated with cerebral venous sinus thrombosis and disseminated primary JCPyV infectio

272 nt on imaging, but transient cerebral venous sinus thrombosis was present.

273 tic presentation of a lesion was presence of sinus tract at buccal or facial abscess of apical portio

274 modality technique, endoscopic/percutaneous sinus tract necrosectomy) were performed.

275 nically, are lesions around implant apex and sinus tract.

276 differential diagnosis of nodular lesions or sinus tracts present in the axillae, groin, perineal, an

277 gland develops from the embryonic urogenital sinus (UGS).

278 Video-assisted ablation of pilonidal sinus (VAAPS) is a new minimally invasive treatment base

279 hat give rise to the left ventricle (LV) and sinus venosus (SV) are still ambiguous.

280 rive from at least two progenitor pools, the sinus venosus and endocardium.

281 ronary endothelium (CoE) originates from the sinus venosus and ventricular endocardium.

282 cardium is activated in multiple cases where sinus venosus angiogenesis is stunted.

283 are able to expand and compensate for faulty sinus venosus development in Apj mutants, leading to nor

284 , we demonstrate that the endocardium of the sinus venosus is a source for the hepatic plexus.

285 ELA)-APJ signaling axis is only required for sinus venosus-derived progenitors.

286 ST-elevation myocardial infarction and sinus venous tract thrombosis occurred as a complication

287 y bilateral concha bullosa affects maxillary sinus volumes.

288 Mean volume of the right maxillary sinus was 17.794 cm(3), while for the left one it was 17

289 distance between PSAA and floor of maxillary sinus was 9.96 mm.

290 In CSA group, the coronary sinus was cannulated via subclavian or femoral venous ap

291 Routine multi-slice CT of the paranasal sinuses was performed to look for mucosal disease of the

292 Patients with chronic nonrecurrent pilonidal sinus were randomized to minimally invasive (76 patients

293 y located in the spleen, lining the marginal sinus where they sense inflammation and capture Ag from

294 ent to both cortical and medullary lymphatic sinuses where the T cells exhibited intense probing beha

295 n compromise drainage pathway of the related sinus, which results in inflammatory sinus disease.

296 ne attenuation differences and misclassified sinuses, which result in patient-dependent performance v

297 ome of the procedure was more predictable in sinuses with a concave floor and small implant-intruding

298 els to coalesce into large flat hyperplastic sinuses with no distinctive hierarchical organization.

299 ormed gradients extending from the lymphatic sinuses, with reduced abundance in the deep LN paracorte

300 e given a high fat diet, CC appear in aortic sinus within 1 week.