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1  as few as 9 beats per minute, with profound sinus arrhythmia.
2 tion between respiration and the respiratory sinus arrhythmia.
3 ch of it arises independently of respiratory sinus arrhythmia.
4 ardiac vagal tone and pronounced respiratory sinus arrhythmia.
5 herapies, such as biological pacemakers, for sinus arrhythmias.
6                            Vagally modulated sinus arrhythmia and BRS were also eliminated after chro
7 acteristics (cardiac measures of respiratory sinus arrhythmia and pre-ejection period), central nervo
8 sympathetic tone, as measured by respiratory sinus arrhythmia, and sympathetic control of the heart,
9 tion and provide a framework for respiratory sinus arrhythmia as there is an increase in heart rate d
10  Evidence strongly suggests that respiratory sinus arrhythmia both contributes to and buffers respira
11   These results demonstrate that respiratory sinus arrhythmia can actually contribute to respiratory
12 iratory systems, which influence respiratory sinus arrhythmia, do not affect CRPS.
13 pression of these variabilities, respiratory sinus arrhythmia does not always reflect a purely vagal
14                       Therefore, respiratory sinus arrhythmia does not represent simple baroreflex bu
15                         Expiration-triggered sinus arrhythmia (ETA) is a potent and independent post-
16  related to expiration (expiration-triggered sinus arrhythmia [ETA]) from short-term recordings of el
17 f cardiac dysfunction including bradycardia, sinus arrhythmia, idiopathic ventricular fibrillation, c
18 nent in HRV similar to mammalian respiratory sinus arrhythmia in an amphibian, the toad Rhinella schn
19                                  Respiratory sinus arrhythmia is small at rapid, and large at slow br
20 f the principal explanations for respiratory sinus arrhythmia is that it reflects arterial baroreflex
21 te into our model the effects of respiratory sinus arrhythmia, Mayer waves and the important very low
22 A determination by quantifying the amount of sinus arrhythmia related to expiration (expiration-trigg
23 PS and the traditionally studied respiratory sinus arrhythmia represent different aspects of the card
24 as elevated during pup exposure, respiratory sinus arrhythmia (RSA) did not differ between these male
25          We investigated whether respiratory sinus arrhythmia (RSA) in healthy humans originated from
26                             This respiratory sinus arrhythmia (RSA) occurs by modulation of premotor
27 ssessed at multiple time points, respiratory sinus arrhythmia (RSA) was measured in the three face-to
28                                  Respiratory sinus arrhythmia (RSA), a measure of cardiac vagal modul
29  baroreflex function and reduced respiratory sinus arrhythmia (RSA), possibly representing a direct p
30 -amylase (sAA), heart rate (HR), respiratory sinus arrhythmia (RSA), subjective stress perception and
31 phase with respiration to induce respiratory sinus arrhythmia (RSA).
32 increased autonomic functioning (respiratory sinus arrhythmia, RSA) and maternal attachment behavior
33  of this 'respiratory gating' is respiratory sinus arrhythmia, the rhythmic fluctuations of electroca

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