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1 injection into the artery that perfused the sinus node.
2 different connexin phenotypes in the intact sinus node.
3 nique to study the I(f) pacemaker current in sinus node.
4 uding slices obtained from the region of the sinus node.
5 ntrinsic electrophysiological changes in the sinus node.
6 vivo in mice and in vitro in the denervated sinus node.
7 bundle on the spread of activation from the sinus node.
8 ifferent aspects of autonomic effects on the sinus node.
10 ression of key transcriptional regulators of sinus node and atrial conduction, including Nkx2-5 (NK2
13 represent vagal activity at the level of the sinus node and may not accurately reflect efferent vagal
14 pping for radiofrequency modification of the sinus node and the long-term success rate of the procedu
15 diary sites of impulse generation within the sinus node and/or atrium in patients with inappropriate
18 have structurally normal atria and preserved sinus node architecture, but expression of key transcrip
19 BP-C(t/+) mice have normal ECG intervals and sinus node, atrial, and ventricular conduction and refra
20 ycardia may result from abnormalities of the sinus node, atrioventricular node, or the His-Purkinje s
24 ively attenuate the high discharge rate from sinus node cells, causing inappropriate sinus tachycardi
25 dependent changes in the architecture of the sinus node comprise an increasing ratio between fibrobla
26 ng, responses of the donor sinus node (DSN) (sinus node controlling heart rate) and recipient sinus n
28 ncy of occurrence of this current within the sinus node decrease, as does its contribution to automat
32 th human cardiovascular phenotypes including sinus node disease, atrial fibrillation, ventricular tac
37 minal reflex testing, responses of the donor sinus node (DSN) (sinus node controlling heart rate) and
38 I (hazard ratio, 4.0; P=0.04), and previous sinus node dysfunction (hazard ratio, 8.0; 95% confidenc
42 ration family (n=25) with autosomal dominant sinus node dysfunction (SND) and atrioventricular block
43 ut genetic overlap has not been reported for sinus node dysfunction (SND) and noncompaction cardiomyo
44 ildren experienced more frequent episodes of sinus node dysfunction (SND) compared with older subject
53 cribes an arrhythmia phenotype attributed to sinus node dysfunction and diagnosed by electrocardiogra
56 rmal SAN function and the pathophysiology of sinus node dysfunction and suggest new potential targets
57 more likely to occur in patients with early sinus node dysfunction and those with longer follow-up.
60 ant and persistent atrioventricular block or sinus node dysfunction can occur and indicate a need for
61 iciency in mice may cause the stress-induced sinus node dysfunction found in many aged individuals an
63 stroke in a population of patients paced for sinus node dysfunction in a large prospective clinical t
65 o determine the early and late incidences of sinus node dysfunction in patients systematically and un
66 node function between the 2 stages, 23% had sinus node dysfunction in the early postoperative period
67 r its blood supply is a significant cause of sinus node dysfunction in the orthotopic heart transplan
69 patients, which typically worsens with time, sinus node dysfunction in the transplanted heart usually
72 nus node function between the 2 stages, late sinus node dysfunction is common and more likely to occu
74 tained atrial tachyarrhythmia, implying that sinus node dysfunction is unlikely to be the dominant me
75 ity, spontaneous type I ECG, and presence of sinus node dysfunction might be considered as risk facto
79 nical trials in patients with pacemakers for sinus node dysfunction or atrioventricular block (AVB) a
82 cardiac arrhythmia syndrome associated with sinus node dysfunction that is distinct from long QT syn
83 tment of pacemaker syndrome in patients with sinus node dysfunction treated with ventricular-based (V
84 ain containing 1 (Popdc1) or Popdc2 leads to sinus node dysfunction under stressed conditions in aged
85 , whereas observed survival of patients with sinus node dysfunction was not significantly different f
89 art failure hospitalization in patients with sinus node dysfunction who require pacemaker therapy is
91 iminished P-wave amplitude characteristic of sinus node dysfunction, an AF risk factor in human patie
93 cardiac conduction disorder associated with sinus node dysfunction, arrhythmia, and right and occasi
94 conduction, and human SCN5A mutations cause sinus node dysfunction, atrial fibrillation, conductiona
95 nd at 6 months, decreased R wave amplitudes, sinus node dysfunction, cardiac hypertrophy, interstitia
96 cing (DDDR) and ventricular pacing (VVIR) in sinus node dysfunction, demonstrated no difference in de
98 ficant clinical manifestation of progressive sinus node dysfunction, is the most frequent indication
99 ulmonary connection may increase the risk of sinus node dysfunction, previous studies have not report
101 of atrial fibrillation and in patients with sinus node dysfunction, reduces heart failure symptoms w
102 we studied a family with DCM associated with sinus node dysfunction, supraventricular tachyarrhythmia
115 ing are alternative treatment approaches for sinus-node dysfunction that causes clinically significan
116 domly assigned a total of 2010 patients with sinus-node dysfunction to dual-chamber pacing (1014 pati
117 hamber pacing were observed in patients with sinus-node dysfunction, but not in those with atrioventr
120 stro-esophageal reflux, retinal disease, and sinus-node dysfunction, whereas related heterozygotes ha
123 ts underwent electrophysiological testing of sinus node function a mean of 9 +/- 3 days after surgery
125 ss the effects of reduced Cx40 expression on sinus node function and conduction velocity at different
126 Although most patients (81%) regained normal sinus node function between the 2 stages, 23% had sinus
129 We observed no differences between groups in sinus node function, and ventricular arrhythmias were no
130 -induced chronic atrial fibrillation (AF) on sinus node function, intra-atrial conduction, and atrial
133 e at the SVC-Ao fat pad (a few fibers to the sinus node go directly to the RPV fat pad) before projec
135 developmental requirement for Cx40 in normal sinus node impulse initiation at 15.5 days postcoitus.
139 uggest that sympathetic reinnervation of the sinus node is accompanied by partial restoration of norm
142 effects of PTH and PTHrP on isolated rabbit sinus node, isolated canine Purkinje fibers, and disaggr
144 riate pacing for sinus node dysfunction, the sinus node may recover and permanent pacing may be disco
145 theter- or surgically- based right atrial or sinus node modification may be helpful, but even this is
146 re to determine whether Cx43 is expressed by sinus node myocytes, to characterize the spectrum of con
150 d be considered for patients with underlying sinus node or atrioventricular conduction disturbances,
151 ritory infarcted and whether it includes the sinus node or AV node or important neuroreceptors; wheth
152 pectrum of connexin expression phenotypes in sinus node pacemaker cells, and to define the spatial di
154 rate, associated with a higher incidence of sinus node (pacemaker) disease and electronic pacemaker
155 orary review summarizes current knowledge on sinus node pathophysiology with the broader goal of yiel
156 series who were documented to have a normal sinus node preoperatively, only 1 patient required a per
165 tricular conduction properties and prolonged sinus node recovery time; and (c) inducible ventricular
167 e are summarized, including determination of sinus node recovery times, AV conduction properties, and
168 These results indicate that cells of the sinus node region exhibit a substantial TTX-sensitive cu
171 nK (-/-) hearts is highly restricted, to the sinus-node region, caudal atrial septum, and proximal co
173 e caused by surgical trauma, ischemia to the sinus node, rejection, drug therapy and increasing donor
174 We used laser capture microdissection on a sinus node reporter mouse line to isolate RNA from PCs f
175 s node controlling heart rate) and recipient sinus node (RSN) in the innervated remnant right atrium
180 images showed activation spreading from the sinus node to the rest of the atria, ending at the left
181 tion of the exciting discovery of the AV and sinus nodes, truly landmarks in our understanding of car
182 Potential mechanisms such as effects on the sinus node, ventricular efficiency, or autonomic functio
185 of connexins could create regions within the sinus node with different conduction properties, thereby
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