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1 , atrial standstill, conduction disease, and sinus node dysfunction.
2 r (DDDR) versus ventricular (VVIR) pacing in sinus node dysfunction.
3 of dual-chamber versus ventricular pacing in sinus node dysfunction.
4 ich reduced Na+ channel function might cause sinus node dysfunction.
5 vent diseases such as atrial fibrillation or sinus node dysfunction.
6 closely resemble those observed in clinical sinus node dysfunction.
7 d risk of death during pacemaker therapy for sinus node dysfunction.
8 with subsequent stroke in patients paced for sinus node dysfunction.
9 and exhibited ventricular preexcitation and sinus node dysfunction.
10 nts (92%) to determine the late incidence of sinus node dysfunction.
11 4 years after the Fontan operation, 44% had sinus node dysfunction.
12 principally in the subgroup of patients with sinus-node dysfunction.
14 iminished P-wave amplitude characteristic of sinus node dysfunction, an AF risk factor in human patie
19 cribes an arrhythmia phenotype attributed to sinus node dysfunction and diagnosed by electrocardiogra
22 rmal SAN function and the pathophysiology of sinus node dysfunction and suggest new potential targets
23 more likely to occur in patients with early sinus node dysfunction and those with longer follow-up.
27 cardiac conduction disorder associated with sinus node dysfunction, arrhythmia, and right and occasi
28 conduction, and human SCN5A mutations cause sinus node dysfunction, atrial fibrillation, conductiona
29 hamber pacing were observed in patients with sinus-node dysfunction, but not in those with atrioventr
31 ant and persistent atrioventricular block or sinus node dysfunction can occur and indicate a need for
32 nd at 6 months, decreased R wave amplitudes, sinus node dysfunction, cardiac hypertrophy, interstitia
33 cing (DDDR) and ventricular pacing (VVIR) in sinus node dysfunction, demonstrated no difference in de
35 iciency in mice may cause the stress-induced sinus node dysfunction found in many aged individuals an
37 I (hazard ratio, 4.0; P=0.04), and previous sinus node dysfunction (hazard ratio, 8.0; 95% confidenc
40 stroke in a population of patients paced for sinus node dysfunction in a large prospective clinical t
42 o determine the early and late incidences of sinus node dysfunction in patients systematically and un
43 node function between the 2 stages, 23% had sinus node dysfunction in the early postoperative period
44 r its blood supply is a significant cause of sinus node dysfunction in the orthotopic heart transplan
46 patients, which typically worsens with time, sinus node dysfunction in the transplanted heart usually
49 nus node function between the 2 stages, late sinus node dysfunction is common and more likely to occu
51 tained atrial tachyarrhythmia, implying that sinus node dysfunction is unlikely to be the dominant me
52 ficant clinical manifestation of progressive sinus node dysfunction, is the most frequent indication
53 ity, spontaneous type I ECG, and presence of sinus node dysfunction might be considered as risk facto
57 nical trials in patients with pacemakers for sinus node dysfunction or atrioventricular block (AVB) a
60 ulmonary connection may increase the risk of sinus node dysfunction, previous studies have not report
62 of atrial fibrillation and in patients with sinus node dysfunction, reduces heart failure symptoms w
65 ration family (n=25) with autosomal dominant sinus node dysfunction (SND) and atrioventricular block
66 ut genetic overlap has not been reported for sinus node dysfunction (SND) and noncompaction cardiomyo
67 ildren experienced more frequent episodes of sinus node dysfunction (SND) compared with older subject
72 we studied a family with DCM associated with sinus node dysfunction, supraventricular tachyarrhythmia
73 cardiac arrhythmia syndrome associated with sinus node dysfunction that is distinct from long QT syn
74 ing are alternative treatment approaches for sinus-node dysfunction that causes clinically significan
76 domly assigned a total of 2010 patients with sinus-node dysfunction to dual-chamber pacing (1014 pati
77 tment of pacemaker syndrome in patients with sinus node dysfunction treated with ventricular-based (V
78 ain containing 1 (Popdc1) or Popdc2 leads to sinus node dysfunction under stressed conditions in aged
79 , whereas observed survival of patients with sinus node dysfunction was not significantly different f
83 stro-esophageal reflux, retinal disease, and sinus-node dysfunction, whereas related heterozygotes ha
84 art failure hospitalization in patients with sinus node dysfunction who require pacemaker therapy is
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