ライフサイエンスコーパス: siphon

1 ng siphon are much higher than in the pinned siphon.

2 in the morphogenesis of the tunicate atrial siphon.

3 estive gland, foot, gill, gonad, mantle, and siphon.

4 ulic resistance controlled multiplexed micro-siphoning allowing for the continuous concentration of s

5 on together) compared with focal turbulence (siphon alone).

6 using a surface-modified glass capillary to siphon and eject cells.

7 morphosis disrupts development of the atrial siphon and gill slits, structures which form where invag

8 imary mechanosensory neurons innervating the siphon and having their somata in the left E (LE) cluste

9 actions required for formation of the atrial siphon and highlight the role of atrial ectoderm during

10 to the release of K+ onto blood vessels (K+ siphoning) and to vessel relaxation.

11 hole animals and reduced preparations (tail, siphon, and CNS) was more rapid after longer exposures t

12 In reduced preparations (tail, siphon, and CNS), we show that treatment with 100 microm

13 xity of the peripheral nervous system of the siphon, and the importance of direct tests of the variou

14 chanosensory thresholds in the freely moving siphon are much higher than in the pinned siphon.

15 strostomy tube (A-Tube) and the AspireAssist siphon assembly (Aspire Bariatrics, King of Prussia, PA)

16 the oil did not form an electrospray but was siphoned away from the tip.

17 s they were localized in the oral and atrial siphons, branchial gill slits, endostyle, and gut.

18 onally actuated valving strategies including siphoning, capillary and centrifugo-pneumatic dissolvabl

19 This inhibition was probably due to siphoning catalase into the slow peroxidative reaction.

20 together, these four enzymes are proposed to siphon CoA from primary metabolism to create the side ch

21 Either pinching or pinning the siphon decreases LE cell mechanosensory threshold and en

22 ructures which form where invaginated atrial siphon ectoderm apposes pharyngeal endoderm.

23 Using siphon-elicited gill withdrawal, we demonstrate habituat

24 ed in this preparation, which corresponds to siphon flaring in the intact animal.

25 esent two direct tests of this glial cell-K+ siphoning hypothesis of neurovascular coupling.

26 peripheral cell types and innervation of the siphon in stage 12 juveniles (chosen to allow observatio

27 hesis, the results demonstrate that glial K+ siphoning in the retina does not contribute significantl

28 often fail to activate the LE cells when the siphon is unrestrained.

29 known about corresponding responses when the siphon is unrestrained.

30 mechanical stimulation of the tightly pinned siphon, little is known about corresponding responses wh

31 To investigate the contribution of the LE siphon mechanosensory cells, we recorded from them and g

32 s between identified interneurons (L29s) and siphon MNs (LFSs).

33 and in the abdominal ganglion, in which the siphon MNs are located.

34 A siphon motor neuron and two siphon sensory neurons, both

35 itatory synapse between L29 interneurons and siphon motor neurons (MNs) in Aplysia.

36 ory cells, we recorded from them and gill or siphon motor neurons during the same siphon stimulation

37 tamate-evoked potential (Glu-EP) in isolated siphon motor neurons in cell culture.

38 ctions from LE siphon sensory neurons to LFS siphon motor neurons make a substantial contribution to

39 EPSPs from LE siphon sensory neurons to LFS siphon motor neurons mediate approximately one-third of

40 the effect of injecting botulinum toxin into siphon motor neurons on dishabituation of the siphon-wit

41 Siphon motor neurons were individually dissociated from

42 d in inter- and motor neurons, including LFS siphon motor neurons, and therefore tested whether HCN c

43 rawal reflex (SWR) and the responsiveness of siphon motor neurons.

44 te to activate central neurons and reflexive siphon movements often fail to activate the LE cells whe

45 ier/Olf/EBF (COE), the determinant of atrial siphon muscle (ASM) specification.

46 Here, we show that myogenesis in the atrial siphon muscles (ASMs) and oral siphon muscles (OSMs), wh

47 nalis also generate precursors of the atrial siphon muscles (ASMs).

48 in the atrial siphon muscles (ASMs) and oral siphon muscles (OSMs), which control the exhalant and in

49 expressing motor ganglion neurons and atrial siphon muscles.

50 ant chains, concerns exist that these chains siphon NDDs from the deceased donor wait list and that d

51 en passant extracellular recordings from the siphon nerve in semi-intact preparations.

52 We found that the siphon nerve splits into three major branches, leading u

53 es cells fated to form the atrium and atrial siphon of adult Ciona.

54 The siphon of Aplysia californica has several functions, inc

55 hdrawal reflex to tactile stimulation of the siphon of Aplysia, a mechanism that has emerged as an ex

56 lineage tracer to show that the early atrial siphon of the metamorphic juvenile, including its apertu

57 activation of NSC channels would reduce the siphoning of K+ via the Muller cells.

58 of mRNA and microRNA expression during oral siphon (OS) regeneration in Ciona robusta, and the deriv

59 d reversed by a pressure-initiated capillary siphoning (PICS) phenomenon, which offers improved CE re

60 Likewise, tunicate atrial siphon primordia and posterior (otic, lateral line, and

61 vertebrate otic placodes and tunicate atrial siphon primordia are thought to be homologous based on m

62 al domain subsequently gave rise to the oral siphon primordia in tunicates (with neurosecretory cells

63 Since both siphon primordia in tunicates give rise to sparse popula

64 neural tube, migrate into the body wall and siphon primordia, and subsequently differentiate as pigm

65 al derivatives, including the palps and oral siphon primordium (OSP).

66 We show that the siphon primordium arises within a non-dividing field of

67 s for the development of the tunicate atrial siphon primordium, thought to share homology with the ve

68 Ms), which control the exhalant and inhalant siphons, respectively, also requires Mrf We characterize

69 ined changes in the strength of monosynaptic siphon sensorimotor connections in the abdominal ganglio

70 hat tail nerve shock-induced facilitation of siphon sensorimotor synapses also depends on elevated po

71 that the associative enhancement of Aplysia siphon sensorimotor synapses in a cellular analog of cla

72 revious studies suggested that plasticity at siphon sensory neuron synapses contributes to habituatio

73 found that monosynaptic connections from LE siphon sensory neurons to LFS siphon motor neurons make

74 We estimated that monosynaptic EPSPs from LE siphon sensory neurons to LFS siphon motor neurons media

75 they suggest that other as yet unidentified siphon sensory neurons with lower thresholds and shorter

76 A siphon motor neuron and two siphon sensory neurons, both of which were presynaptical

77 nges are consistent with previous results in siphon sensory neurons.

78 ent to posttetanic potentiation (PTP) at the siphon sensory to motor neuron (SN-MN) synapse.

79 contingent (experimental) animal received a siphon shock each time its gill relaxed below a criterio

80 stimulus (the CS-), delivered to a different siphon site, is unpaired with the US.

81 Surprisingly, in both tail and siphon SN-MN synapses, there was an inverse relationship

82 gill or siphon motor neurons during the same siphon stimulation that has been used in behavioral expe

83 dispensing through independently addressable siphon structures or to relocate solutions against the c

84 greater for LE neurons that fire during the siphon tap and correlates significantly with the enhance

85 classical conditioning of the reflex with a siphon tap conditioned stimulus (CS) and tail shock unco

86 complex PSP elicited in an LFS neuron by the siphon tap, and greater facilitation of the monosynaptic

87 adaptation attenuates the evoked response to siphon taps delivered during water turbulence.

88 d stimulus (CS) delivered to one side of the siphon (the CS+) is paired with a noxious unconditioned

89 positions, a preset fraction of the flow is siphoned to separate the smaller cells from the main flo

90 ore rapid after diffuse turbulence (tail and siphon together) compared with focal turbulence (siphon

91 a newly designed shower drain, disinfecting siphons underneath the sinks, and rimless toilets.

92 neuron by water-movement stimulation of the siphon, which does not cause firing of LE cells.

93 he CS and US produces greater enhancement of siphon withdrawal and evoked firing of LFS neurons, grea

94 elates significantly with the enhancement of siphon withdrawal and evoked firing of the LFS neurons.

95 The threshold required to elicit siphon withdrawal and the duration of siphon withdrawal

96 , identical to that producing habituation in siphon withdrawal in freely moving animals, also produce

97 ubstantial contribution to the reflex in the siphon withdrawal preparation.

98 e recorded evoked firing of LFS neurons, the siphon withdrawal produced by stimulation of an LFS neur

99 ambient environment can regulate the Aplysia siphon withdrawal reflex (SWR) by changing the environme

100 examined the modulation of the tail-induced siphon withdrawal reflex by repeated noxious stimuli app

101 The neural circuit mediating the siphon withdrawal reflex in Aplysia provides a useful ne

102 k-induced sensitization of the tail-elicited siphon withdrawal reflex in Aplysia to examine the role

103 of short- and long-term sensitization of the siphon withdrawal reflex in Aplysia.

104 Using the tail-elicited siphon withdrawal reflex of Aplysia, which is mediated i

105 oped a simplified preparation of the Aplysia siphon withdrawal reflex that allows one to examine beha

106 dies of habituation, such as in the gill and siphon withdrawal reflex to tactile stimulation of the s

107 te to classical conditioning of the gill and siphon withdrawal reflex.

108 ch contribute to dynamic gain control in the siphon withdrawal reflex.

109 ucture of sensory neurons mediating the tail-siphon withdrawal reflex.

110 elicit siphon withdrawal and the duration of siphon withdrawal were not regulated by the circadian cl

111 ex behaviors in Aplysia, tail withdrawal and siphon withdrawal, both elicited by threshold-level tail

112 (<30 min) for sensitization of tail-elicited siphon withdrawal, whereas repeated spaced shocks induce

113 cellular analog of classical conditioning of siphon withdrawal.

114 mplified gill-withdrawal preparation, in the siphon-withdrawal preparation we found no qualitative di

115 Furthermore, in the siphon-withdrawal preparation, all of the various cellul

116 es a stable reduction in the duration of the siphon-withdrawal reflex (SWR) and the responsiveness of

117 ory for long-term sensitization (LTS) of the siphon-withdrawal reflex (SWR) as late as 7 d after trai

118 ral adaptation and sensory adaptation in the siphon-withdrawal reflex (SWR) of Aplysia californica.

119 involved in simple forms of learning of the siphon-withdrawal reflex in a semiintact preparation.

120 ory for long-term sensitization (LTS) of the siphon-withdrawal reflex in the marine snail Aplysia cal

121 ituation and dishabituation of the gill- and siphon-withdrawal reflex in this preparation.

122 Classical conditioning of Aplysia's siphon-withdrawal reflex is thought to be due to a presy

123 new simplified preparation for studying the siphon-withdrawal reflex of Aplysia in which it is relat

124 nts that induce long-term enhancement of the siphon-withdrawal reflex, or long-term synaptic facilita

125 sing a simplified preparation of the Aplysia siphon-withdrawal reflex, we previously found that assoc

126 in a semi-intact preparation of the Aplysia siphon-withdrawal reflex.

127 n in a simplified preparation of the Aplysia siphon-withdrawal reflex.

128 iphon motor neurons on dishabituation of the siphon-withdrawal reflex.

129 s well as the duration of both the gill- and siphon-withdrawal reflexes were measured after either ta

130 roduces behavioral adaptation in the Aplysia siphon-withdrawal response (SWR).