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1 erved in all samples with enhanced levels of sitosterol.
2 er selectively for cholesterol compared with sitosterol.
3 trong preference for cholesterol rather than sitosterol.
4 tact cells loaded with either cholesterol or sitosterol.
5 to efficiently esterify both cholesterol and sitosterol.
6 sterol by LCAT was only 15% greater than for sitosterol.
7  an approximately 30-fold increase in plasma sitosterol.
8 heptadecanyl n-octadec-9-enoate (1) and beta-sitosterol (2) on the basis of chromatographic and spect
9 droxyphenethyl alcohol, ocotillone, and beta-sitosterol 3-O-beta-D-glucopyranoside, were also isolate
10  acid, alpha-tocopherol, threonic acid, beta-sitosterol, 4-hydroxybutyric acid, ferulic acid, and tot
11 ne-third nonesterified and consisted of beta-sitosterol (48%), campesterol (27%), and stigmasterol (2
12       The most abundant phytosterol was beta-sitosterol (974-1494 mg/kg) followed by campesterol then
13 , both ACAT1 and ACAT2 were able to esterify sitosterol albeit with lower efficiencies than cholester
14 )-avenasterol, 24-methylene-cholesterol/beta-sitosterol and 24-methylene-cholesterol.
15                                       Plasma sitosterol and campesterol concentrations significantly
16 cholestyramine therapy, the patient's plasma sitosterol and campesterol levels decreased by approxima
17 uence of markedly elevated plasma and tissue sitosterol and campesterol levels, premature atheroscler
18 re analysed, as well as the contents of beta-sitosterol and campesterol oxidation products.
19                            A mixture of beta-sitosterol and campesterol was incorporated into triacyl
20 -keto-, 7-hydroxy- and triol-PS derived from sitosterol and campesterol were 40.0, 34.4, 21.5 and 4.0
21 l (PS) oxidation products (POP) derived from sitosterol and campesterol were measured in 15 foods coo
22 have been: avenasterol, linolenic acid, beta-sitosterol and gadoleico.
23 se inhibitor NB-598 prevented growth in beta-sitosterol and greatly reduced growth in campesterol.
24             Growth of cells cultured in beta-sitosterol and NB-598 was restored by adding small amoun
25 disease characterized by very high levels of sitosterol and other plant sterols and premature atherot
26 bulus terrestris; saw palmetto berries; beta-sitosterol and other related sterols; and wild yams (dio
27 sterol exhibits a stronger ability than beta-sitosterol and stigmasterol to order model membranes.
28 effective for determination of analytes beta-sitosterol and stigmasterol.
29 e most representative phytosterols were beta-sitosterol and stigmasterol.
30 um plant sterols (stigmasterol, avenasterol, sitosterol, and campesterol), cholestanol, and cholester
31 s of the side chain (e.g., campesterol, beta-sitosterol, and desmosterol) supported long-term growth
32                  The antioxidative effect of sitosterol at 1, 2 and 5% levels, in triolein, refined c
33  responsible for the antioxidative effect of sitosterol at frying temperatures.
34 r phytosterols: brassicasterol, campesterol, sitosterol, avenasterol and two phytostanols: sitostanol
35 ase initiates glucan polymerization by using sitosterol-beta-glucoside (SG) as primer.
36 of pure DPPC, pure DOPC, and mixed DOPC-beta-sitosterol bilayers solvated in a vitrification solution
37 a is a bona fide sterol-binding protein with sitosterol-binding properties.
38 ing the levels of sterols, particularly beta-sitosterol, both in grapes and in microvinificates.
39 ]cholesterol and a trace amount of [beta-14C]sitosterol by gavage.
40 elta-tocopherol, beta-carotene, lutein, beta-sitosterol, campesterol and brassicasterol.
41 rs of cholesterol absorption (plant sterols: sitosterol, campesterol) and synthesis (cholesterol prec
42 gosterol), allochthonous (stigmasterol, beta-sitosterol, campesterol, and stigmastanol) and anthropog
43            Cotton fiber membranes synthesize sitosterol-cellodextrins (SCDs) from SG and uridine 5'-d
44 nd plant sterols campesterol/cholesterol and sitosterol/cholesterol (cholesterol absorption markers)
45 was ruled out by documenting a normal plasma sitosterol:cholesterol ratio.
46 e measured in rats treated with cholesterol, sitosterol, cholic acid, deoxycholic acid, ursodeoxychol
47                                              Sitosterol concentrations decreased by 21% (P<0.001) in
48 we show here that macrophages incubated with sitosterol-containing lipoproteins accumulate free stero
49                            Campesterol, beta-sitosterol, Delta(7)-campesterol/Delta(5,24)-stigmastadi
50 isation of pecan nut oils revealed that beta-sitosterol, Delta5-avenasterol, and campesterol were the
51 eased activity 84% (P < .05) and intravenous sitosterol did not change activity.
52 rafficking to the endoplasmic reticulum, and sitosterol-enriched endoplasmic reticulum membranes show
53      The mass ratios of cholesterol ester to sitosterol ester formed by ACAT1 and ACAT2 were 1.6 and
54 rol ferulate, cycloartenol ferulate and beta-sitosterol ferulate.
55 anol ferulate, campesterol ferulate and beta-sitosterol ferulate.
56                The main phytosterol was beta-sitosterol, followed by stigmasterol and campesterol.
57 ethyl to 24-ethyl sterol in the direction of sitosterol formation.
58                             The reduction in sitosterol from baseline was progressive, with further d
59 imulate the transfer of cholesterol and beta-sitosterol from liposomes to heat-treated mitochondria i
60 easing order: campesterol approximately beta-sitosterol&gt;/=stigmasterol>cholesterol.
61            The major plant sterol species is sitosterol; hence the name of the disorder.
62  supplements of zinc, saw palmetto, and beta-sitosterol in relieving BPH symptoms have had mixed resu
63 accumulation of cholestanol, campesterol and sitosterol in serum and stones suggesting their particip
64  whereas PS concentrations (campesterol+beta-sitosterol) increased (P = 0.03) in both groups after PS
65 on of the unfolded protein response and JNK, sitosterol-induced death is caspase-independent and invo
66                                      Second, sitosterol-induced macrophage death does not require ACA
67                          As with FC loading, sitosterol-induced macrophage death requires sterol traf
68            Addition of stigmasterol, but not sitosterol, inhibited SREBP-2 processing and reduced cho
69                         Despite similar beta-sitosterol intakes between the MS and MSF groups, plasma
70               HDL-cholesterol, triglyceride, sitosterol, lathosterol, campesterol, and proprotein con
71 C57BL6J had almost twice the campesterol and sitosterol levels compared with parental CASARk mice, an
72 Ibalpha were strongly correlated with plasma sitosterol levels in samples from human sitosterolemic p
73  significantly more ACAT activity than their sitosterol-loaded counterparts.
74                                           In sitosterol-loaded microsomes, both ACAT1 and ACAT2 were
75 le/surfactant, "Nok" (i.e., SPGS-550-M; beta-sitosterol methoxypolyethyleneglycol succinate), soon to
76 cture of non-polar dimers formed during beta-sitosterol oxidation at 180 degrees C in the presence of
77 edominant non-polar dimer formed during beta-sitosterol oxidative degradation has a configuration of
78                                     The beta-sitosterol oxides (7alpha/7beta-hydroxy, beta/alpha-epox
79 s diminished by about 21% based on low serum sitosterol (P = 0.0269) and campesterol (P = 0.0231) to
80 rhetinic acid, lupeol, ursolic acid and beta-sitosterol showed a strong Th2-inclination and anti-infl
81 H, linoleic acid, saturated fatty acids beta-sitosterol, sn-1 and 3 diglyceride values.
82 g detection (ELSD) was used to quantify beta-sitosterol, stigmasterol, campesterol, and alpha-, delta
83 CAT1-expressing cells esterified 4-fold more sitosterol than the ACAT2 cells.
84 rs differing in polarity, formed during beta-sitosterol thermo-oxidation.
85 des C22-sterol desaturase that converts beta-sitosterol to stigmasterol, was dramatically induced upo
86 s using an abundant plant feedstock and beta-sitosterol, together with succinic anhydride and PEG-550
87 , diosgenin, betulinic acid, escin, and beta-sitosterol treatments significantly inhibited both IL-2
88 e of ripening were Delta(7)-campesterol/beta-sitosterol, uvaol/stigmasterol, clerosterol/Delta(5)-ave
89 1.29%) and lower oxidation susceptibility of sitosterol vs. campesterol.
90           The presence of enhanced levels of sitosterol was found to significantly decrease TG polyme
91 selectively esterified cholesterol even when sitosterol was loaded into the microsomes.
92 en 62.0% and 75.7% of total sterols and beta-sitosterol was the first sterol in the two samples.
93                     Concerning sterols, beta-sitosterol was the main component in seed oils extracted
94                                         beta-Sitosterol was the most abundant component in berry tiss
95                                         beta-Sitosterol was the most important and representative phy
96                  Radiolabeled cholesterol or sitosterol was transferred from donor liposomes to G5- a
97 rols (PS: campesterol, stigmasterol and beta-sitosterol) was evaluated at 180 degrees C for up to 180
98 erentiate cholesterol from the plant sterol, sitosterol, was compared with that of the sterol esterif
99 ermination were 0.25g; 2.5mL; 300s; and beta-sitosterol were 0.25g; 5.4mL; 300s.
100 ta-tocopherol, campesterol, stigmasterol and sitosterol were quantified via GC-MS.
101 lites, sinigrin, quercetin, campesterol, and sitosterol, were confirmed to regulate stomatal closure
102 reduced intestinal uptake of cholesterol and sitosterol, with dramatically reduced plasma phytosterol

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