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   1 erved in all samples with enhanced levels of sitosterol.                                             
     2 er selectively for cholesterol compared with sitosterol.                                             
     3 trong preference for cholesterol rather than sitosterol.                                             
     4 tact cells loaded with either cholesterol or sitosterol.                                             
     5 to efficiently esterify both cholesterol and sitosterol.                                             
     6 sterol by LCAT was only 15% greater than for sitosterol.                                             
     7  an approximately 30-fold increase in plasma sitosterol.                                             
     8 heptadecanyl n-octadec-9-enoate (1) and beta-sitosterol (2) on the basis of chromatographic and spect
     9 droxyphenethyl alcohol, ocotillone, and beta-sitosterol 3-O-beta-D-glucopyranoside, were also isolate
    10  acid, alpha-tocopherol, threonic acid, beta-sitosterol, 4-hydroxybutyric acid, ferulic acid, and tot
    11 ne-third nonesterified and consisted of beta-sitosterol (48%), campesterol (27%), and stigmasterol (2
  
    13 , both ACAT1 and ACAT2 were able to esterify sitosterol albeit with lower efficiencies than cholester
  
  
    16 cholestyramine therapy, the patient's plasma sitosterol and campesterol levels decreased by approxima
    17 uence of markedly elevated plasma and tissue sitosterol and campesterol levels, premature atheroscler
  
  
    20 -keto-, 7-hydroxy- and triol-PS derived from sitosterol and campesterol were 40.0, 34.4, 21.5 and 4.0
    21 l (PS) oxidation products (POP) derived from sitosterol and campesterol were measured in 15 foods coo
  
    23 se inhibitor NB-598 prevented growth in beta-sitosterol and greatly reduced growth in campesterol.   
  
    25 disease characterized by very high levels of sitosterol and other plant sterols and premature atherot
    26 bulus terrestris; saw palmetto berries; beta-sitosterol and other related sterols; and wild yams (dio
    27 sterol exhibits a stronger ability than beta-sitosterol and stigmasterol to order model membranes.   
  
  
    30 um plant sterols (stigmasterol, avenasterol, sitosterol, and campesterol), cholestanol, and cholester
    31 s of the side chain (e.g., campesterol, beta-sitosterol, and desmosterol) supported long-term growth 
  
  
    34 r phytosterols: brassicasterol, campesterol, sitosterol, avenasterol and two phytostanols: sitostanol
  
    36 of pure DPPC, pure DOPC, and mixed DOPC-beta-sitosterol bilayers solvated in a vitrification solution
  
  
  
  
    41 rs of cholesterol absorption (plant sterols: sitosterol, campesterol) and synthesis (cholesterol prec
    42 gosterol), allochthonous (stigmasterol, beta-sitosterol, campesterol, and stigmastanol) and anthropog
  
    44 nd plant sterols campesterol/cholesterol and sitosterol/cholesterol (cholesterol absorption markers) 
  
    46 e measured in rats treated with cholesterol, sitosterol, cholic acid, deoxycholic acid, ursodeoxychol
  
    48 we show here that macrophages incubated with sitosterol-containing lipoproteins accumulate free stero
  
    50 isation of pecan nut oils revealed that beta-sitosterol, Delta5-avenasterol, and campesterol were the
  
    52 rafficking to the endoplasmic reticulum, and sitosterol-enriched endoplasmic reticulum membranes show
  
  
  
  
  
  
    59 imulate the transfer of cholesterol and beta-sitosterol from liposomes to heat-treated mitochondria i
  
  
    62  supplements of zinc, saw palmetto, and beta-sitosterol in relieving BPH symptoms have had mixed resu
    63 accumulation of cholestanol, campesterol and sitosterol in serum and stones suggesting their particip
    64  whereas PS concentrations (campesterol+beta-sitosterol) increased (P = 0.03) in both groups after PS
    65 on of the unfolded protein response and JNK, sitosterol-induced death is caspase-independent and invo
  
  
  
  
  
    71 C57BL6J had almost twice the campesterol and sitosterol levels compared with parental CASARk mice, an
    72 Ibalpha were strongly correlated with plasma sitosterol levels in samples from human sitosterolemic p
  
  
    75 le/surfactant, "Nok" (i.e., SPGS-550-M; beta-sitosterol methoxypolyethyleneglycol succinate), soon to
    76 cture of non-polar dimers formed during beta-sitosterol oxidation at 180 degrees C in the presence of
    77 edominant non-polar dimer formed during beta-sitosterol oxidative degradation has a configuration of 
  
    79 s diminished by about 21% based on low serum sitosterol (P = 0.0269) and campesterol (P = 0.0231) to 
    80 rhetinic acid, lupeol, ursolic acid and beta-sitosterol showed a strong Th2-inclination and anti-infl
  
    82 g detection (ELSD) was used to quantify beta-sitosterol, stigmasterol, campesterol, and alpha-, delta
  
  
    85 des C22-sterol desaturase that converts beta-sitosterol to stigmasterol, was dramatically induced upo
    86 s using an abundant plant feedstock and beta-sitosterol, together with succinic anhydride and PEG-550
    87 , diosgenin, betulinic acid, escin, and beta-sitosterol treatments significantly inhibited both IL-2 
    88 e of ripening were Delta(7)-campesterol/beta-sitosterol, uvaol/stigmasterol, clerosterol/Delta(5)-ave
  
  
  
  
  
  
  
  
    97 rols (PS: campesterol, stigmasterol and beta-sitosterol) was evaluated at 180 degrees C for up to 180
    98 erentiate cholesterol from the plant sterol, sitosterol, was compared with that of the sterol esterif
  
  
   101 lites, sinigrin, quercetin, campesterol, and sitosterol, were confirmed to regulate stomatal closure 
   102 reduced intestinal uptake of cholesterol and sitosterol, with dramatically reduced plasma phytosterol
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