ライフサイエンスコーパス: skeletal muscle cell

1 he proton-motive force, throughout the mouse skeletal muscle cell.

2 determining the metabolic health of the aged skeletal muscle cell.

3 panded AR causes damage to motor neurons and skeletal muscle cells.

4 n secretion and enhances glucose uptake into skeletal muscle cells.

5 ully dependent on mTOR signalling within the skeletal muscle cells.

6 tatively regulated by PGC-1alpha in cultured skeletal muscle cells.

7 cal for maintenance of metabolic function in skeletal muscle cells.

8 hancer that supports Igf2 gene activation in skeletal muscle cells.

9 esicle translocation and glucose uptake into skeletal muscle cells.

10 orm as the mediator of NO-induced effects in skeletal muscle cells.

11 eta, localized at the Golgi complex in mouse skeletal muscle cells.

12 s a negative modulator of agrin signaling in skeletal muscle cells.

13 sponsive Glut4-storage vesicles from fat and skeletal muscle cells.

14 , insulin resistance, and protein wasting in skeletal muscle cells.

15 creased insulin-stimulated glucose uptake in skeletal muscle cells.

16 ilization, and fatty acid oxidation (FAO) in skeletal muscle cells.

17 lecules that induce PGC-1alpha expression in skeletal muscle cells.

18 -fos expression by junctional epithelial and skeletal muscle cells.

19 in zebrafish embryos, and in vitro in murine skeletal muscle cells.

20 el to study the differentiation of fetal rat skeletal muscle cells.

21 A) to promote inappropriate proliferation of skeletal muscle cells.

22 ved in TNF-alpha-induced MMP-9 production in skeletal muscle cells.

23 I-induced inhibition of insulin signaling in skeletal muscle cells.

24 retain expression of some proteins common to skeletal muscle cells.

25 giform papillae in the tongue, as well as in skeletal muscle cells.

26 blunted SRF-dependent transcription in C2C12 skeletal muscle cells.

27 ain flotillin-1, a marker of lipid rafts, in skeletal muscle cells.

28 y physiological form of Ca(V)1.1 channels in skeletal muscle cells.

29 se did not determine reovirus replication in skeletal muscle cells.

30 K(b) induced ER stress response in C(2)C(12) skeletal muscle cells.

31 without altering the mRNA level in cultured skeletal muscle cells.

32 ed for peptide selection against C2C12 mouse skeletal muscle cells.

33 ed reduction in IRS-1 expression in cultured skeletal muscle cells.

34 by insulin and hyperosmotic stress in L6 rat skeletal muscle cells.

35 human endothelial cells and also apparently skeletal muscle cells.

36 sulin-independent basal glucose uptake in L6 skeletal muscle cells.

37 enchymal stem cells and dedifferentiation in skeletal muscle cells.

38 pstream of genes differentially expressed in skeletal muscle cells.

39 ipant in the agrin-MuSK signaling pathway of skeletal muscle cells.

40 for tissue-specific expression in liver and skeletal muscle cells.

41 (nNOS), which is expressed constitutively in skeletal muscle cells.

42 tive calcium entry (CCE) in Jurkat and in L6 skeletal muscle cells.

43 hat is expressed specifically in cardiac and skeletal muscle cells.

44 ompartment to the plasma membrane in fat and skeletal muscle cells.

45 yte chemoattractant protein 1) production by skeletal muscle cells.

46 abolism and gene expression in primary human skeletal muscle cells.

47 us genome inserted into the genome of murine skeletal muscle cells.

48 is critical for terminal differentiation of skeletal muscle cells.

49 nt potentiation of L-type Ca(2+) channels in skeletal muscle cells.

50 anslocation in both 3T3-L1 adipocytes and L6 skeletal muscle cells.

51 +)](i) in type I astrocytes, neurons, and in skeletal muscle cells.

52 ptional control seen in ordinary cardiac and skeletal muscle cells.

53 t the z-line of the sarcomere of cardiac and skeletal muscle cells.

54 drial morphology and fission protein Drp1 in skeletal muscle cells.

55 s of myositis patients and in cultured human skeletal muscle cells.

56 ultrastructural changes in both cardiac and skeletal muscle cells.

57 ck the differentiation of certain neural and skeletal muscle cells.

58 creased functional voltage sensors in single skeletal muscle cells.

59 its through activation of gene expression in skeletal muscle cells.

60 K by causing Ser(485/491) phosphorylation in skeletal muscle cells.

61 cipitated with PARP from extracts of primary skeletal muscle cells.

62 in synthesis are regulated by PKBalpha in L6 skeletal muscle cells.

63 ntiation of epithelial, glial, neuronal, and skeletal muscle cells.

64 n of desmin-reactive deposits in cardiac and skeletal muscle cells.

65 and insulin signaling is validated in human skeletal muscle cells.

66 nchored protein kinase, as it does in native skeletal muscle cells.

67 le Ca2+ channel as expressed in mouse 129CB3 skeletal muscle cells.

68 ble-homeodomain transcription factor DUX4 in skeletal muscle cells.

69 egulate proliferation and differentiation of skeletal muscle cells.

70 ogical functions of these receptors in mouse skeletal muscle cells.

71 hat SMAD3 suppresses FNDC5 and PGC-1alpha in skeletal muscle cells.

72 ogical functions of these receptors in mouse skeletal muscle cells.

73 ncode proteins of the contractile complex of skeletal muscle cells.

74 or-derived microvesicles induce apoptosis of skeletal muscle cells.

75 gehog (Hh) signaling, and differentiation of skeletal muscle cells.

76 ting the multifunctional role of Staufen1 in skeletal muscle cells.

77 the PGC-1alpha-mediated hypoxic response in skeletal muscle cells.

78 ARP2/3 subunit p41ARC is a PAK1 substrate in skeletal muscle cells.

79 vascular endothelial growth factor (VEGF) in skeletal muscle cells.

80 ose uptake by target tissues such as fat and skeletal muscle cells.

81 ed rates of complete fatty acid oxidation in skeletal muscle cells.

82 that lncRNA AK017368 is highly expressed in skeletal muscle cells.

83 ylcholine receptors (AChRs) to the center of skeletal muscle cells.

84 en species that promote damage in dystrophic skeletal muscle cells.

85 ptional activation during differentiation of skeletal muscle cells.

86 a mechanism that was independent of mTOR in skeletal muscle cells.

87 ically, the inhibition of mTOR signalling in skeletal muscle cells.

88 e molecules that are up-regulated on injured skeletal-muscle cells.

89 uscle cells (96-well-plate format) and human skeletal muscle cells (24-well-plate format).

90 NE and play a role in nuclear positioning in skeletal muscle cells [8-12].

91 -glutamyltransferase (GT) activity in rat L6 skeletal muscle cells (96-well-plate format) and human s

92 Migrating muscle cells, but not all skeletal muscle cells, also expressed MMP-13.

93 m activated macrophages is critical for both skeletal muscle cell and hBD-MSCs death in PIRI-CLI.

94 ffect of mediating insulin on SREBP-1 in L-6 skeletal muscle cells and 3T3 L1 adipocytes, using wortm

95 d Akt and ERK1/2 phosphorylation in cultured skeletal muscle cells and C2C12 myotubes.

96 reduced SRF activity in differentiated C2C12 skeletal muscle cells and cardiac myocytes.

97 otonic decreasing amplitude distributions in skeletal muscle cells and cardiomyocytes, confirming the

98 small heat shock protein (HspB8) in ischemic skeletal muscle cells and enhanced ischemic muscle autop

99 ia-induced apoptosis in both endothelial and skeletal muscle cells and enhanced proliferation in both

100 d Mlx associate with mitochondria in primary skeletal muscle cells and erythroblast K562 cells.

101 interstitial space, thus reducing binding to skeletal muscle cells and glucose uptake.

102 e very low in undifferentiated human primary skeletal muscle cells and myoblasts (HSMM) but increased

103 in the fetal tissue and in undifferentiated skeletal muscle cells and myoblasts.

104 The overly active RyRs in cardiac and skeletal muscle cells and neuronal cells would result in

105 elial cells, HeLa cells, and human embryonic skeletal muscle cells and optic nerve head (ONH) astrocy

106 stricting the expression of foreign genes to skeletal muscle cells and presumably to other cells that

107 and the biological activity of Pip6a-PMO in skeletal muscle cells and primary cardiomyocytes.

108 red the contractile properties of individual skeletal muscle cells and the activation and relaxation

109 se findings indicate that prion infection of skeletal muscle cells and the epithelial layer in the to

110 The role of skeletal muscle cells and their contribution to the immu

111 d is sufficient to convert a fibroblast to a skeletal muscle cell, and, as such, is a model system in

112 in pancreatic beta-cells, cardiac myocytes, skeletal muscle cells, and a cloned KATP composed of two

113 trite was abolished in SIRT3-deficient human skeletal muscle cells, and in SIRT3 knockout mice fed a

114 Ca(V)1 family Ca(2+) channels in cardiac and skeletal muscle cells, and reveal a unique ion channel r

115 dly activated by TNF-alpha in differentiated skeletal muscle cells, and that TNF-alpha/NF-kappaB sign

116 ow fiber-specific gene promoters in cultured skeletal muscle cells, and the calcineurin inhibitor, cy

117 n at S307 in endothelial cells, hepatocytes, skeletal muscle cells, and vascular smooth muscle cells.

118 lbeit at a low frequency, in adult mammalian skeletal muscle cells; and iii) a major contributor to t

119 trate for the first time that normal primary skeletal muscle cells are capable of secreting IL-1beta

120 To determine whether skeletal muscle cells are directly affected by space tra

121 f the C-terminal lobe of troponin C (TnC) in skeletal muscle cells as a step toward elucidating the m

122 INK4A) to promote malignant proliferation of skeletal muscle cells as an early step in ARMS tumorigen

123 muscle (VSM) cells, but also in cardiac and skeletal muscle cells as well as in kidney.

124 factor hypoxia-inducible factor-1 (HIF-1) in skeletal muscle cells, as well as invading myeloid cells

125 e developed a hybrid muscle powered by C2C12 skeletal muscle cells based on the functionalized multi-

126 e relevant cells, specifically primary human skeletal muscle cells because these cells can be convert

127 ulation, a previously-developed model of the skeletal muscle cell bioenergetic system was used to sim

128 hed that mitogens inhibit differentiation of skeletal muscle cells, but the insulin-like growth facto

129 slocation to the cell surface in cardiac and skeletal muscle cells by activating a PI3K dependent pat

130 s indicate that Pip6a-PMO is taken up in the skeletal muscle cells by an energy- and caveolae-mediate

131 nctional sarcoplasmic reticulum in heart and skeletal muscle cells by an undefined mechanism.

132 regulate many genes expressed in cardiac and skeletal muscle cells by binding to myocyte-specific chl

133 odel of insulin-stimulated glucose uptake in skeletal muscle cells by implicating p41ARC as a new com

134 e have shown 'robust' production of lung and skeletal muscle cells by marrow cells in the presence of

135 rin receptor-1 and ferritin-H in hepatic and skeletal muscle cells by promoting the binding of iron r

136 Infection of mammalian skeletal muscle cells by Trichinella spiralis causes hos

137 A co-culture of skeletal muscle cells (C2C12) and cholinergic neurons, g

138 tivated under hypoxic conditions in cultured skeletal muscle cells (C2C12).

139 ure and mouse nerve-muscle ex-vivo) that the skeletal muscle cell constantly senses, through two iden

140 additional defect, namely impaired smooth-to-skeletal muscle cell conversion in the abdominal segment

141 ccur in islet beta cells, adipocytes, and/or skeletal muscle cells correlate with increased susceptib

142 We have developed a primary skeletal muscle cell culture model derived from normal p

143 Exposure of human skeletal muscle cell culture to type 1 interferons produ

144 ndoplasmic reticulum stress and autophagy in skeletal muscle cell death and dysfunction in myositis.

145 e and secrete AGE-albumin, which induced the skeletal muscle cell death and injected hBD-MSCs in PIRI

146 Here, overexpression of XIAP in cultured skeletal muscle cells decreased protein degradation indu

147 pared to adult myoblast cultures, children's skeletal muscle cells demonstrated higher basal and day

148 appearance of Ca(2+) sparks in permeabilized skeletal muscle cells depends on the fibre's oxidative s

149 , both considered critical components of the skeletal muscle cell differentiation program.

150 RK downstream kinase, is a novel mediator of skeletal muscle cell differentiation through its regulat

151 However, Panxs functions in skeletal muscle cell differentiation, and proliferation

152 to the inhibitory role of Notch signaling on skeletal muscle cell differentiation, the Notch pathway

153 Oncogenic Ha-Ras is a potent inhibitor of skeletal muscle cell differentiation, yet the Ras effect

154 t TAK1 is an important upstream regulator of skeletal muscle cell differentiation.

155 these tumor cells allowed the initiation of skeletal muscle cell differentiation.

156 ndocannabinoids and cannabinoid receptors in skeletal muscle cell differentiation.

157 noblotting and kinase assays) in cultured L6 skeletal muscle cells during 30 min of cyclic stretch an

158 xpression in developing smooth, cardiac, and skeletal muscle cells during early embryogenesis.

159 and inhibited protein degradation in L6 rat skeletal muscle cells (EC(50) 4 muM) mediated in part by

160 These results demonstrate that skeletal muscle cells express functionally active NF-AT

161 Endothelial cells, macrophages, and skeletal muscle cells expressed TG throughout the healin

162 In summary, our data suggest that raised skeletal muscle cell expression of GRalpha and 11beta -H

163 ene regulatory network determines cardiac or skeletal muscle cell fates.

164 t the Notch pathway promotes vascular versus skeletal muscle cell fates.

165 nes encoding mitochondrial proteins in human skeletal muscle cells following treatment with 1alpha,25

166 domyosarcoma (aRMS), an aggressive cancer of skeletal muscle cells for which patient outcomes remain

167 derived conditionally immortalized precursor skeletal muscle cells from caveolin-3 transgenic and nul

168 Skeletal muscle cells from DM patients fail to induce cy

169 tly been made toward the production of human skeletal muscle cells from induced pluripotent stem (iPS

170 In summary, skeletal muscle cells from type 2 diabetic patients disp

171 ament arrays, is an essential contributor to skeletal muscle-cell fusion in developing mouse embryos.

172 In fat and skeletal muscle cells, glucose transporter isoform 4 (Gl

173 In extracts of fat and skeletal muscle cells, Glut4 is predominantly found in s

174 These data indicate that skeletal muscle cell grafting gives rise to a subpopulat

175 Skeletal muscle cells grown on vertically aligned CNTs i

176 Given this novel role for PGF2alpha in skeletal muscle cell growth, these studies raise caution

177 anscriptional activities in undifferentiated skeletal muscle cells have not yet been determined.

178 Skeletal muscle cells have served as a paradigm for unde

179 Although previous studies in skeletal muscle cells have shown that HDAC4 lacking seri

180 pletion from mdx mice prevented compensatory skeletal muscle cell hypertrophy, decreased myofiber cen

181 rounded the role of calcineurin in mediating skeletal muscle cell hypertrophy.

182 terization of the underlying SOCE current in skeletal muscle cells (I(SkCRAC)) has not been reported.

183 We show that Bcl-2 expression in skeletal muscle cells identifies an early stage of the m

184 Furthermore, treatment of skeletal muscle cells in culture (C2C12 myotubes) with a

185 Terminal differentiation of skeletal muscle cells in culture is inhibited by a numbe

186 tochondrial network in cardiomyocytes and L6 skeletal muscle cells in culture.

187 gs between identified spinal motoneurons and skeletal muscle cells in larval zebrafish.

188 the mitochondrial morphology of mouse C2C12 skeletal muscle cells in response to heat acclimation an

189 vation and cellular localization of NF-AT in skeletal muscle cells in vitro.

190 rincipal structural component of caveolae in skeletal muscle cells in vivo.

191 re identified in immunoprecipitates of human skeletal muscle cells in vivo.

192 is integrin in commitment of differentiating skeletal muscle cells in vivo.

193 entin expression was up-regulated on injured skeletal-muscle cells in vitro and was expressed in musc

194 e prion protein, PrP(Sc), accumulates within skeletal muscle cells, in addition to axons, in the tong

195 of stem cell myogenesis (transformation into skeletal muscle cells) includes several stages character

196 on of three alpha-dystroglycan glycoforms in skeletal muscle cells, including two minor glycoforms ma

197 Stimulation by IFN-gamma in skeletal muscle cells induces CIITA expression as well a

198 In primary human skeletal muscle cells, inhibition and overexpression str

199 In fat and skeletal muscle cells, insulin-responsive vesicles, or I

200 The differentiation and maturation of skeletal muscle cells into functional fibers is coordina

201 nsulin resistance induced by high glucose in skeletal muscle cells is a consequence of Nox2 activatio

202 nic stem cells (hESCs) to differentiate into skeletal muscle cells is an important criterion in using

203 ortant early event in the differentiation of skeletal muscle cells is exit from the cell cycle, after

204 -like growth factor I (IGF-I) in L6 cultured skeletal muscle cells is inhibited by the glucocorticoid

205 Contraction of skeletal muscle cells is initiated by a well-known signa

206 se during excitation-contraction coupling of skeletal muscle cells is initiated by the functional int

207 The innervation of embryonic skeletal muscle cells is marked by the redistribution of

208 that oncogenic Ras-induced proliferation of skeletal muscle cells is mediated via a unique and novel

209 A hallmark of Nrg-1 signaling in skeletal muscle cells is the activation of extracellular

210 enabled us to demonstrate that mTOR, within skeletal muscle cells, is the rapamycin-sensitive elemen

211 s, namely myelomonocytic cells, osteoblasts, skeletal muscle cells, keratinocytes, and T lymphocytes.

212 glutamine synthetase (GS) is induced in rat skeletal muscle cells (L-6) in response to treatment wit

213 Differentiation of skeletal muscle cells, like most other cell types, requi

214 lly characterized calcium release units in a skeletal muscle cell line (1B5) lacking Ry1R.

215 Overexpression of ZnT7 in a rat skeletal muscle cell line (L6) increased Irs2 mRNA expre

216 In the L6E9 skeletal muscle cell line and in 10T1/2 fibroblasts, a p

217 ork of differentiating cultures of the mouse skeletal muscle cell line C2.

218 There was no benefit for a differentiated skeletal muscle cell line culture (C2C12 cells), suggest

219 An L6 rat skeletal muscle cell line expressing ss-galactosidase (s

220 +/K+-ATPase activation by insulin in the rat skeletal muscle cell line L6.

221 1B5s are a mouse skeletal muscle cell line that carries a null mutation f

222 toskeletal linkage protein dystrophin, and a skeletal muscle cell line, 129 CB3.

223 ature IGF-I were performed in C2C12 cells, a skeletal muscle cell line.

224 tinguish the differentiated state of a mouse skeletal muscle cell line.

225 of a lacZ reporter gene in early cardiac and skeletal muscle cell lineages and in a subset of arteria

226 regulation of gene expression in cardiac and skeletal muscle cell lineages.

227 Skeletal muscle cell lines and subsets of primary cells

228 ase reporter gene analysis using cardiac and skeletal muscle cell lines demonstrated a pattern of dis

229 We treated skeletal muscle cell lines with ryanodine, at concentrat

230 nderstanding the immunologic capabilities of skeletal muscle cells may provide important clues not on

231 4 (Glut4) to the plasma membrane in fat and skeletal muscle cells may represent a primary defect in

232 that is thought to stabilize the cardiac and skeletal muscle cell membranes during contraction.

233 mine whether strain-dependent differences in skeletal muscle cells might account for the differential

234 ertrophy and failure as well as in the C2C12 skeletal muscle cell model of differentiation; (3) the a

235 sults in loss of maternal Igf2 repression in skeletal muscle cells, most strikingly in the tongue, la

236 o-5N loaded into the SR of single, mammalian skeletal muscle cells (murine flexor digitorum brevis my

237 ts the hypothesis that activation of RyR3 in skeletal muscle cells must be indirect and provides the

238 of protein phosphatase-1 (PP-1(G)) in L6 rat skeletal muscle cell myogenesis.

239 In mouse C(2)C(12) cells, similarly to human skeletal muscle cells, myotube formation increased the e

240 te the existence of SOCE in freshly isolated skeletal muscle cells obtained from embryonic days 15 an

241 same panel of genetic changes, altering the skeletal muscle cell of origin led to different tumor mo

242 Skeletal muscle cells of skalpha2(-/-) mice completely l

243 In skeletal muscle cells, oleic acid treatment increased in

244 irect effects of TNF-alpha on differentiated skeletal muscle cells or the signaling mechanisms involv

245 In primary skeletal muscle cells, PGC-1beta induction of endogenous

246 4 (Glut4) to the plasma membrane of fat and skeletal muscle cells plays the key role in postprandial

247 coma by converting less differentiated human skeletal muscle cell precursors (SkMC) and committed hum

248 moted both fetal and postnatal primary human skeletal muscle cell precursors to bypass the senescence

249 o, activation of PPARdelta in adipocytes and skeletal muscle cells promotes fatty acid oxidation and

250 t that SIRT6 depletion in cardiac as well as skeletal muscle cells promotes myostatin (Mstn) expressi

251 icated that the addition of motor neurons to skeletal muscle cells reduced the secretion of GDNF by s

252 lated activation of FGF receptors (FGFRs) in skeletal muscle cells represses terminal myogenic differ

253 GLUT4 recruitment to the plasma membrane of skeletal muscle cells requires F-actin remodeling.

254 human cardiac alpha-actin (HCA) promoter in skeletal muscle cells requires the integrity of DNA bind

255 Taken together, our data show that skeletal muscle cells respond to defective myosin chaper

256 shown that lack of expression of triadins in skeletal muscle cells results in significant increase of

257 mediator of adipose tissue inflammation and skeletal muscle cell (SkMC) insulin sensitivity and to q

258 ls and in committed cell lineages, including skeletal muscle cells (SMC).

259 However, differentiation of hESCs into skeletal muscle cells still remains a challenge, often r

260 ut not Bcl-2, is expressed in cultured human skeletal muscle cells stimulated with proinflammatory cy

261 ls are not grouped into tetrads as in normal skeletal muscle cells suggesting that anchoring to Ry1Rs

262 to be expressed in neuronal, pancreatic, and skeletal muscle cells, suggesting a widespread role in r

263 Skeletal muscle cell survival and differentiation into m

264 tion-competent vesicular carriers in fat and skeletal muscle cells that deliver Glut4 to the plasma m

265 Myoblasts are precursor skeletal muscle cells that differentiate into fused, mul

266 mechanism triggered by mechanical stretch of skeletal muscle cells that leads to an EGR1-dependent tr

267 diffraction of frog (Rana temporaria) single skeletal muscle cells that, although the well-known thin

268 Here we show using differentiated skeletal muscle cells, that tumor necrosis factor (TNF)

269 f the expression of the genes is confined to skeletal muscle cells, the CD8(+) T-cell response is muc

270 ncogenic Ras inhibits the differentiation of skeletal muscle cells through the activation of multiple

271 (MURF) expressed specifically in cardiac and skeletal muscle cells throughout pre- and postnatal mous

272 kappaB activity, but the contribution of the skeletal muscle cell to this process has been unclear.

273 We conclude that commitment of skeletal muscle cells to differentiation is calcium and

274 tein that connects the actin cytoskeleton in skeletal muscle cells to extracellular matrix.

275 the cross talk between human adipocytes and skeletal muscle cells to identify mechanisms linking adi

276 f a functional signaling pathway that allows skeletal muscle cells to sense and react to nutrient ava

277 ntify genome-wide binding of MyoD in several skeletal muscle cell types.

278 In vitro (macrophages, endothelial cells, skeletal muscle cells under normal and hypoxia serum sta

279 Skeletal muscle cells undergo in vitro maturation result

280 erstood, but may be related to the fact that skeletal muscle cells, unlike heart cells, are electrica

281 cell culture-based model of damage to C2C12 skeletal muscle cells using the calcium ionophore, A2318

282 erent in undifferentiated and differentiated skeletal muscle cells (vesicular versus nuclear).

283 ide (NO) induces mitochondrial biogenesis in skeletal muscle cells via upregulation of the peroxisome

284 In skeletal muscle cells, voltage-dependent potentiation of

285 ormin (Met) action on glucose uptake (GU) in skeletal muscle cells was investigated.

286 A subset of skeletal muscle cells was positive with 7E11 (7 of 7 cas

287 tion, Id3 mRNA was detected in proliferating skeletal muscle cells, was further induced by basic fibr

288 Using skeletal muscle cells, we studied effects of (a) inactiv

289 ion demonstrated activation of LacZ when the skeletal muscle cells were implanted into hearts of -MHC

290 -derived neural stem cells in coculture with skeletal muscle cells were induced to become neurons exp

291 in C57BL/6 and BALB/c mice, endothelial and skeletal muscle cells were subjected to hypoxia and nutr

292 ected by space travel, tissue-cultured avian skeletal muscle cells were tissue engineered into bioart

293 ar events that transduce these signals, MM14 skeletal muscle cells were transfected with expression v

294 AAV6, which demonstrate robust infection in skeletal muscle cells, were less effective in crossing t

295 steroid drugs is comparable in rat and human skeletal muscle cells, which emphasizes the potential of

296 hemical and morphological differentiation of skeletal muscle cells while having a minimal effect on p

297 In contrast to brown preadipocytes or skeletal muscle cells, white preadipocytes express Tcf21

298 Acute treatment (30 min) of cultured human skeletal muscle cells with either INH resulted in a dose

299 Transfecting skeletal muscle cells with shRNAs specific for PUM2 up-r

300 During differentiation, skeletal muscle cells withdraw from the cell cycle and f