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1 ynaptic contact between motor axons and each skeletal muscle fibre.
2 myocyte preparation or a skinned slow-twitch skeletal muscle fibre.
3 lasma membrane of many cell types, including skeletal muscle fibres.
4 of DOX-induced myopathy in both cardiac and skeletal muscle fibres.
5 aintaining the resting membrane potential of skeletal muscle fibres.
6 ochrome c oxidase (COX)-deficient regions in skeletal muscle fibres.
7 molecular remodelling of the triads in adult skeletal muscle fibres.
8 iac myocytes and fast-twitch and slow-twitch skeletal muscle fibres.
9 low and similar between cardiac myocytes and skeletal muscle fibres.
10 forms insoluble aggregates in the nuclei of skeletal muscle fibres.
11 hancement of force production in fast twitch skeletal muscle fibres.
12 r physiological functions in adult mammalian skeletal muscle fibres.
13 genomic actions in isolated intact mammalian skeletal muscle fibres.
14 regarding the role of potassium released by skeletal muscle fibres.
15 PABPN1 forms aggregates within the nuclei of skeletal muscle fibres.
16 ontribute to mitochondrial ROS generation in skeletal muscle fibres.
17 m)) following osmotic challenge in amphibian skeletal muscle fibres.
18 CICR to operate even in such fully polarized skeletal muscle fibres.
19 s, as well as in fast-twitch and slow-twitch skeletal muscle fibres.
20 f sarcomeres in series (sarcomere number) in skeletal muscle fibres.
21 been measured in single, frog (Rana pipiens) skeletal muscle fibres.
22 anism was investigated in permeabilized frog skeletal muscle fibres.
23 y motor neurons regulates gene expression in skeletal muscle fibres.
24 normal expression of laminin alpha2 on their skeletal muscle fibres.
25 ensitivities of contraction in slow and fast skeletal muscle fibres.
27 enes implicated in structure and function of skeletal muscle fibres (ACTG1), neuronal maintenance and
29 generated from AMP that is released from the skeletal muscle fibres and dephosphorylated by ecto 5'nu
30 -mediated ROS production in both cardiac and skeletal muscle fibres and the prevention of DOX-induced
31 ane repair is therefore an active process in skeletal muscle fibres, and dysferlin has an essential r
32 gical functions of glucocorticoids, in adult skeletal muscle fibres, are mediated by a glucocorticoid
33 dy weight and composition, leg lean mass and skeletal muscle fibre area all remained unchanged follow
35 ration of NO in isolated single mature mouse skeletal muscle fibres at rest and following a period of
38 assessment of cellular respiration in intact skeletal muscle fibre bundles obtained from the extensor
45 fully polarized, fluo-3-loaded, intact frog skeletal muscle fibres by exposure to hypertonic Ringer
47 Ia histone deacetylases (HDACs) move between skeletal muscle fibre cytoplasm and nuclei in response t
48 cle fibres, but not for the earlier steps of skeletal muscle fibre differentiation, elongation, fusio
49 )(on)) and cessation (V(o)(2)off)) in single skeletal muscle fibres differing in oxdidative capacity,
50 ctions onset ( ) and cessation ( ) in single skeletal muscle fibres differing in oxidative capacity,
51 the Ca(2+)-fura-2 reaction) from single rat skeletal muscle fibres, either fully dissociated from th
53 may help to explain the weakness observed in skeletal muscle fibres from mdx mice and, possibly, Duch
54 ron Radiation Facility from small bundles of skeletal muscle fibres from Rana esculenta at sarcomere
55 gulation was studied in mechanically skinned skeletal muscle fibres from rat extensor digitorium long
57 cell membrane of many cell types, including skeletal muscle fibres; however, the exact localisation
58 lock of D2 or disruption of the Dio2 gene in skeletal muscle fibres impaired acute exercise-induced P
59 an release dilator concentrations of K+ from skeletal muscle fibres in CH rats as proposed for N rats
61 e NO generation in quiescent and contracting skeletal muscle fibres in real time, although peroxynitr
62 These mutations produce depolarization of skeletal muscle fibres in response to reduced extracellu
63 scle contraction, adenosine is released from skeletal muscle fibres independently of NO and acts dire
65 genomic actions of DHT in isolated mammalian skeletal muscle fibres is to stimulate amino acid uptake
66 pact of ageing on structure and functions of skeletal muscle fibres, likely to be due to a complex in
67 ocal nature of contraction-induced injury to skeletal muscle fibres may arise from heterogeneities in
69 sults suggest that cooperative activation of skeletal muscle fibres occurs primarily through spread o
70 impaired excitation-contraction coupling in skeletal muscle fibres of the mdx mouse, a model of the
72 fication in intact voltage-clamped amphibian skeletal muscle fibres studied in the gluconate-containi
73 in both the ventricular myocardium and slow skeletal muscle fibres such as the masseter and is an im
74 f activation, unloaded shortening of skinned skeletal muscle fibres takes place in two phases: an ini
75 ed to a greater extent in rabbit fast-twitch skeletal muscle fibres than in slow-twitch fibres from b
76 ce that adenosine can stimulate receptors on skeletal muscle fibres that are coupled to ATP-sensitive
77 itochondrial disease and shown in individual skeletal muscle fibres that there exist different patter
78 se data demonstrate in these isolated single skeletal muscle fibres that unchanged peak [Ca(2+)](c) i
79 tosolic ROS balance is compromised in intact skeletal muscle fibres that underwent osmotic shock and
80 both rat slow-twitch and rabbit fast-twitch skeletal muscle fibres the rate of tension redevelopment
81 imation of most of the properties of IKir in skeletal muscle fibres, the model demonstrates that a su
82 analysis for the first time in single human skeletal muscle fibres to measure muscle mechanics, incl
83 bular (t) system of mechanically skinned rat skeletal muscle fibres to measure SOCE during intracellu
84 tional, 12 week aerobic training protocol on skeletal muscle fibre type distribution and satellite ce
86 at such charging phenomena should persist in skeletal muscle fibres unable to release stored Ca(2+).
87 ients in mouse flexor digitorum brevis (FDB) skeletal muscle fibres under voltage clamp, using confoc
89 of tension to [Ca2+] that occurs in skinned skeletal muscle fibres upon stretch also occurs in intac
91 lcium concentration ([Ca2+]i) in intact frog skeletal muscle fibres was determined at two fibre lengt
92 nt protein (HDAC4-GFP) expressed in isolated skeletal muscle fibres, we now show that activation of P
95 ndividual sarcomeres of voltage-clamped frog skeletal muscle fibres were examined by laser scanning c
97 tension and sarcomere length of relaxed frog skeletal muscle fibres were measured in response to impo
98 ar-neighbour RU interactions, in contrast to skeletal muscle fibres where the effect was enhanced.
99 man disease Duchenne muscular dystrophy, has skeletal muscle fibres which display incompletely unders
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