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1 er is paramount in evaluation of cardiac and skeletal myocytes.
2 tty acid oxidation during differentiation in skeletal myocytes.
3  a marked up-regulation of macroautophagy in skeletal myocytes.
4 cose sensing that augments insulin action in skeletal myocytes.
5 strained calcineurin activity in cardiac and skeletal myocytes.
6 entified incorporated into capillaries among skeletal myocytes.
7 s and enhanced basal glucose uptake in human skeletal myocytes.
8 s significantly reduced activity in cultured skeletal myocytes.
9  of in vitro differentiated control and FSHD skeletal myocytes.
10 sfected IGF-1 gene expression in postmitotic skeletal myocytes activates calcineurin-mediated calcium
11 nhancers associate with the DUX4 promoter in skeletal myocytes and activate transcription when epigen
12 we demonstrated that Six4 is TrexBF in mouse skeletal myocytes and embryonic day 10 chick skeletal an
13 nsible for transcription activation in C2C12 skeletal myocytes and H9C2 cardiomyocytes.
14 taining localized VEGF protein expression to skeletal myocytes and infiltrating T cells in the ischem
15 f apoptosis that is expressed in cardiac and skeletal myocytes and neurons.
16  in murine monocyte macrophages, mouse C2C12 skeletal myocytes and rat adrenal pheochromocytoma PC12
17  by conditioned media from cardiac myocytes, skeletal myocytes, and brain astrocytes.
18 T1R-enriched exosomes target cardiomyocytes, skeletal myocytes, and mesenteric resistance vessels and
19 itro by infection of rat cardiomyocytes, rat skeletal myocytes, and mouse fibroblasts with the vector
20 anent transfections in ventricular myocytes, skeletal myocytes, and nonmyocytic cells to map regulato
21 -regulated within differentiated cardiac and skeletal myocytes, and persistently high expression is o
22 ary and duodenal mesenchyme, the cardiac and skeletal myocytes, and the mesenchymal precartilage and
23 he enhancer decreased expression in cultured skeletal myocytes approximately 10- and 2-fold, respecti
24 hysiological properties of human iPS-derived skeletal myocytes are strictly similar to those of their
25 yosin heavy chain]) expressed in cardiac and skeletal myocytes, are a leading cause of hypertrophic c
26 ted expression of VEGF mRNA and protein from skeletal myocytes as well as endothelial cells in the is
27 mitochondrial oxidative energy metabolism in skeletal myocytes both in vitro and in vivo.
28 nd DME2 which activate DUX4-fl expression in skeletal myocytes but not fibroblasts.
29                 Inadequate insulin action in skeletal myocytes contributes to hyperglycemia in diabet
30 on in channel regulation was investigated in skeletal myocytes cultured from wild-type mice, mdx mice
31 nd mutants were expressed in primary chicken skeletal myocytes, decreased assembly of Tmod1 mutants w
32 erized as a protein important in cardiac and skeletal myocyte differentiation and is expressed in a d
33 iologic processes such as T-cell activation, skeletal myocyte differentiation, and cardiac hypertroph
34 esses, such as T-cell activation, apoptosis, skeletal myocyte differentiation, and cardiac hypertroph
35 cesses such as T-cell activation, apoptosis, skeletal myocyte differentiation, and cardiac hypertroph
36  DNA-binding activity are upregulated during skeletal myocyte differentiation.
37 UCP3 and that the absence of UCP3 in primary skeletal myocytes exacerbates hypoxia-induced reactive o
38                                              Skeletal myocytes express the type 2 deiodinase (D2), wh
39  Ca2+ waves previously described in dyspedic skeletal myocytes expressing the cardiac RyR-2.
40 patients with type 2 diabetes have increased skeletal myocyte fat storage: the so-called "athlete's p
41 el Na currents from cell-attached patches of skeletal myocytes from mice heterozygous (DMPK(+/-)) and
42    Similar findings were obtained with human skeletal myocytes (HSMs) and freshly isolated rat neonat
43 c effects of insulin-like growth factor-1 on skeletal myocytes in vitro.
44 ng that CrAT overexpression in primary human skeletal myocytes increased glucose uptake and attenuate
45 eart failure-is accompanied by intracellular skeletal myocyte iron depletion, which can be exacerbate
46 hat calcineurin-dependent gene regulation in skeletal myocytes is mediated also by MEF2 transcription
47              Expression of TRPC3 channels in skeletal myocytes is up-regulated by neuromuscular activ
48 ansition from rest to contractions in single skeletal myocytes isolated from Xenopus laevis lumbrical
49                    We found that mouse C2C12 skeletal myocytes lack tie2, yet dose-dependently adhere
50 ith secretory functions, such as cardiac and skeletal myocytes, Leydig cells, prostatic epithelium, a
51  the chondrocyte, osteoblast, adipocyte, and skeletal myocyte lineages in vitro and in vivo.
52 03065140), we characterised and compared the skeletal myocyte lipid signature of 29 male endurance at
53                                       Unlike skeletal myocytes, mammalian adult cardiomyocytes cannot
54 rstand the net impact of local adipocytes on skeletal myocyte metabolism.
55 milies in intact native murine gastrocnemius skeletal myocytes, minimising artefactual [Ca(2+)](i) pe
56  present study validates the human iPS-based skeletal myocyte model in comparison with the embryonic
57  mutant PABPN1 which forms aggregates within skeletal myocyte nuclei.
58 PF leads to an approximately 30% increase in skeletal myocyte oxygen consumption.
59 to reporter mice specific for progenitors of skeletal myocytes (Pax7(+) and MyoD(+)) and VSMCs (Prrx1
60  complex-previously found to be important in skeletal myocyte physiology-is now argued to be a molecu
61             Furthermore, Sirt1 deficiency in skeletal myocytes promotes insulin resistance.
62 e transcription was specified to cardiac and skeletal myocytes, recapitulating precisely the expressi
63                 Activation of calcineurin in skeletal myocytes selectively up-regulates slow-fiber-sp
64                                           In skeletal myocytes serum response factor (SRF) has been s
65 d pSN spheroids cultured interact with human skeletal myocytes showing the formation of annulospiral
66                              Thus, unlike in skeletal myocytes, Sirt1 deficiency in the endothelium p
67 n unexpected mechanism of glucose sensing in skeletal myocytes that contributes to homeostasis and th
68 ple parallel myofibrils and give cardiac and skeletal myocytes their distinct striated appearance.
69 us nebulin was replaced with mini-nebulin in skeletal myocytes, thin filaments extended beyond the en
70 files in dexamethasone-treated primary human skeletal myocytes using a cDNA microarray, which contain
71 upling machinery of both iPS- and ES-derived skeletal myocytes was functional and specific, but did n
72                         Mice lacking PKD1 in skeletal myocytes were resistant to Ang II-induced muscl
73 roliferating fetal brain cells and postnatal skeletal myocytes which exhibit 'catch-up growth', there