ライフサイエンスコーパス: skeletal myocyte

1 cose sensing that augments insulin action in skeletal myocytes.

2 tty acid oxidation during differentiation in skeletal myocytes.

3 a marked up-regulation of macroautophagy in skeletal myocytes.

4 s and enhanced basal glucose uptake in human skeletal myocytes.

5 strained calcineurin activity in cardiac and skeletal myocytes.

6 entified incorporated into capillaries among skeletal myocytes.

7 s significantly reduced activity in cultured skeletal myocytes.

8 sfected IGF-1 gene expression in postmitotic skeletal myocytes activates calcineurin-mediated calcium

9 nhancers associate with the DUX4 promoter in skeletal myocytes and activate transcription when epigen

10 we demonstrated that Six4 is TrexBF in mouse skeletal myocytes and embryonic day 10 chick skeletal an

11 nsible for transcription activation in C2C12 skeletal myocytes and H9C2 cardiomyocytes.

12 taining localized VEGF protein expression to skeletal myocytes and infiltrating T cells in the ischem

13 f apoptosis that is expressed in cardiac and skeletal myocytes and neurons.

14 in murine monocyte macrophages, mouse C2C12 skeletal myocytes and rat adrenal pheochromocytoma PC12

15 by conditioned media from cardiac myocytes, skeletal myocytes, and brain astrocytes.

16 T1R-enriched exosomes target cardiomyocytes, skeletal myocytes, and mesenteric resistance vessels and

17 itro by infection of rat cardiomyocytes, rat skeletal myocytes, and mouse fibroblasts with the vector

18 anent transfections in ventricular myocytes, skeletal myocytes, and nonmyocytic cells to map regulato

19 -regulated within differentiated cardiac and skeletal myocytes, and persistently high expression is o

20 ary and duodenal mesenchyme, the cardiac and skeletal myocytes, and the mesenchymal precartilage and

21 he enhancer decreased expression in cultured skeletal myocytes approximately 10- and 2-fold, respecti

22 hysiological properties of human iPS-derived skeletal myocytes are strictly similar to those of their

23 ted expression of VEGF mRNA and protein from skeletal myocytes as well as endothelial cells in the is

24 nd DME2 which activate DUX4-fl expression in skeletal myocytes but not fibroblasts.

25 Inadequate insulin action in skeletal myocytes contributes to hyperglycemia in diabet

26 on in channel regulation was investigated in skeletal myocytes cultured from wild-type mice, mdx mice

27 nd mutants were expressed in primary chicken skeletal myocytes, decreased assembly of Tmod1 mutants w

28 erized as a protein important in cardiac and skeletal myocyte differentiation and is expressed in a d

29 iologic processes such as T-cell activation, skeletal myocyte differentiation, and cardiac hypertroph

30 esses, such as T-cell activation, apoptosis, skeletal myocyte differentiation, and cardiac hypertroph

31 cesses such as T-cell activation, apoptosis, skeletal myocyte differentiation, and cardiac hypertroph

32 DNA-binding activity are upregulated during skeletal myocyte differentiation.

33 UCP3 and that the absence of UCP3 in primary skeletal myocytes exacerbates hypoxia-induced reactive o

34 Skeletal myocytes express the type 2 deiodinase (D2), wh

35 Ca2+ waves previously described in dyspedic skeletal myocytes expressing the cardiac RyR-2.

36 el Na currents from cell-attached patches of skeletal myocytes from mice heterozygous (DMPK(+/-)) and

37 Similar findings were obtained with human skeletal myocytes (HSMs) and freshly isolated rat neonat

38 c effects of insulin-like growth factor-1 on skeletal myocytes in vitro.

39 ng that CrAT overexpression in primary human skeletal myocytes increased glucose uptake and attenuate

40 hat calcineurin-dependent gene regulation in skeletal myocytes is mediated also by MEF2 transcription

41 Expression of TRPC3 channels in skeletal myocytes is up-regulated by neuromuscular activ

42 ansition from rest to contractions in single skeletal myocytes isolated from Xenopus laevis lumbrical

43 We found that mouse C2C12 skeletal myocytes lack tie2, yet dose-dependently adhere

44 ith secretory functions, such as cardiac and skeletal myocytes, Leydig cells, prostatic epithelium, a

45 the chondrocyte, osteoblast, adipocyte, and skeletal myocyte lineages in vitro and in vivo.

46 Unlike skeletal myocytes, mammalian adult cardiomyocytes cannot

47 rstand the net impact of local adipocytes on skeletal myocyte metabolism.

48 present study validates the human iPS-based skeletal myocyte model in comparison with the embryonic

49 mutant PABPN1 which forms aggregates within skeletal myocyte nuclei.

50 PF leads to an approximately 30% increase in skeletal myocyte oxygen consumption.

51 complex-previously found to be important in skeletal myocyte physiology-is now argued to be a molecu

52 e transcription was specified to cardiac and skeletal myocytes, recapitulating precisely the expressi

53 Activation of calcineurin in skeletal myocytes selectively up-regulates slow-fiber-sp

54 In skeletal myocytes serum response factor (SRF) has been s

55 n unexpected mechanism of glucose sensing in skeletal myocytes that contributes to homeostasis and th

56 us nebulin was replaced with mini-nebulin in skeletal myocytes, thin filaments extended beyond the en

57 files in dexamethasone-treated primary human skeletal myocytes using a cDNA microarray, which contain

58 upling machinery of both iPS- and ES-derived skeletal myocytes was functional and specific, but did n

59 Mice lacking PKD1 in skeletal myocytes were resistant to Ang II-induced muscl

60 roliferating fetal brain cells and postnatal skeletal myocytes which exhibit 'catch-up growth', there