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1 kin temperature, sweating, and laser-Doppler skin blood flow.
2 and ButOH increase erythema as a function of skin blood flow.
3 ser-Doppler flowmetry to provide an index of skin blood flow.
4 ugh a neurogenic mechanism that also affects skin blood flow.
5 sity, but neither affected histamine-induced skin blood flow.
6 circadian and ultradian (12 h) variations in skin blood flow.
7 function, keratinocyte differentiation, and skin blood flow.
9 ema and PG increased erythema with decreased skin blood flow, all as a function of ADHIB haplotype.
10 pler flowmetry (LDF) was used as an index of skin blood flow and cutaneous vascular conductance (CVC)
13 t of thermal stimuli or distal scratching on skin blood flow and histamine-induced itch in healthy vo
14 ds histamine iontophoresis was performed and skin blood flow and itch intensity were measured immedia
17 application in healthy and irritated skin on skin blood flow and its relationship to barrier function
18 ormation process, informed by data regarding skin blood flow and reactive hyperemia in response to pr
20 tions of skin sympathetic nerve activity and skin blood flow, arterial pressure, and R-R intervals, o
21 osed workers had significantly lower resting skin blood flow at both 21 degrees C and 4 degrees C tha
25 that baroreflexes are capable of modulating skin blood flow, but the effects of baroreceptor loading
26 P did not produce a significant increase in skin blood flow compared to the initial baseline or the
27 ause of enhanced thermoregulatory demand for skin blood flow coupled with dehydration and hyperthermi
28 o and metabolic activity in vivo via altered skin blood flow (Doppler velocimeter) and erythema (refl
29 ing rate (evaporative heat loss) and reduced skin blood flow (dry heat loss) for a given core tempera
33 ation as there was no observable increase in skin blood flow following a second administration of sub
34 icroneurography) from the peroneal nerve and skin blood flow (forearm laser Doppler) in 9 patients wi
40 e more important in regulating microvascular skin blood flow in regions rich in arteriovenous anastom
42 increase in SNA was accompanied by decreased skin blood flow, increased skin vascular resistance, and
43 prostaglandin production to the increase in skin blood flow induced following the iontophoresis of A
44 we measured skin SNA (microneurography) and skin blood flow (laser Doppler velocimetry) as well as h
45 es, heart rate, beat-by-beat blood pressure, skin blood flow (laser-Doppler flowmetry), local sweat r
46 arm from heart level such that the sites of skin blood flow measurement were 34 +/- 1 cm below the h
47 seline ; P < 0.01) and greater reductions in skin blood flow (NTN: -16 +/- 2%baseline vs. HTN: -28 +/
50 cated on sensory nerves, would attenuate the skin blood flow response to local heating in humans.
55 ed with intradermal microdialysis to measure skin blood flow (SkBF) during graded ET-A (BQ-123) and E
56 72-week-old male mice underwent analysis of skin blood flow (SkBF) via laser Doppler in response to
64 in at 4 microl min(-1) in sites 3 and 4, and skin blood flow was allowed to return to baseline (appro
70 al heating to 42 degrees C was performed and skin blood flow was measured with laser Doppler flowmetr
71 1 older (61-77 years) men and women, forearm skin blood flow was monitored at three sites using laser
72 s) and 11 older (62-76 years) men and women, skin blood flow was monitored at two forearm sites with
80 ance P produced a dose-dependent increase in skin blood flow with the concentrations of substance P t
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