戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 kin temperature, sweating, and laser-Doppler skin blood flow.
2 and ButOH increase erythema as a function of skin blood flow.
3 ser-Doppler flowmetry to provide an index of skin blood flow.
4 ugh a neurogenic mechanism that also affects skin blood flow.
5 sity, but neither affected histamine-induced skin blood flow.
6 circadian and ultradian (12 h) variations in skin blood flow.
7  function, keratinocyte differentiation, and skin blood flow.
8        These measurements were compared with skin blood flow after various stimuli: heating the skin,
9 ema and PG increased erythema with decreased skin blood flow, all as a function of ADHIB haplotype.
10 pler flowmetry (LDF) was used as an index of skin blood flow and cutaneous vascular conductance (CVC)
11       Previous studies show that the rise in skin blood flow and cutaneous vascular conductance durin
12 attenuated in aged skin resulting in greater skin blood flow and heat loss during cold exposure.
13 t of thermal stimuli or distal scratching on skin blood flow and histamine-induced itch in healthy vo
14 ds histamine iontophoresis was performed and skin blood flow and itch intensity were measured immedia
15 d flow but reduced histamine-induced maximal skin blood flow and itch intensity.
16    Scratching reduced mean histamine-induced skin blood flow and itch intensity.
17 application in healthy and irritated skin on skin blood flow and its relationship to barrier function
18 ormation process, informed by data regarding skin blood flow and reactive hyperemia in response to pr
19                                          Paw skin blood flow, angiographic score, and capillary densi
20 tions of skin sympathetic nerve activity and skin blood flow, arterial pressure, and R-R intervals, o
21 osed workers had significantly lower resting skin blood flow at both 21 degrees C and 4 degrees C tha
22 ing of a limb causes not only an increase in skin blood flow but also in muscle blood flow.
23 ation was found between skin temperature and skin blood flow but not with TEWL.
24            Noxious heat pain increased basal skin blood flow but reduced histamine-induced maximal sk
25  that baroreflexes are capable of modulating skin blood flow, but the effects of baroreceptor loading
26  P did not produce a significant increase in skin blood flow compared to the initial baseline or the
27 ause of enhanced thermoregulatory demand for skin blood flow coupled with dehydration and hyperthermi
28 o and metabolic activity in vivo via altered skin blood flow (Doppler velocimeter) and erythema (refl
29 ing rate (evaporative heat loss) and reduced skin blood flow (dry heat loss) for a given core tempera
30 hetic nerve activity (SSNA) that control the skin blood flow during heat stress in CHF patients.
31               These data suggest the rise in skin blood flow during whole body heating contains an H1
32                             The reduction in skin blood flow during whole-body cooling is impaired in
33 ation as there was no observable increase in skin blood flow following a second administration of sub
34 icroneurography) from the peroneal nerve and skin blood flow (forearm laser Doppler) in 9 patients wi
35                                              Skin blood flow has been imaged during stimulation of fi
36 for matricellular proteins in the regulation skin blood flow has never been proposed.
37  Laser-Doppler flowmetry was used to measure skin blood flow in a total of 18 subjects.
38  and, correspondingly, greater reductions in skin blood flow in HTN.
39 ng the role of substance P in the control of skin blood flow in humans.
40 e more important in regulating microvascular skin blood flow in regions rich in arteriovenous anastom
41               The initial, rapid increase in skin blood flow in response to direct application of hea
42 increase in SNA was accompanied by decreased skin blood flow, increased skin vascular resistance, and
43  prostaglandin production to the increase in skin blood flow induced following the iontophoresis of A
44  we measured skin SNA (microneurography) and skin blood flow (laser Doppler velocimetry) as well as h
45 es, heart rate, beat-by-beat blood pressure, skin blood flow (laser-Doppler flowmetry), local sweat r
46  arm from heart level such that the sites of skin blood flow measurement were 34 +/- 1 cm below the h
47 seline ; P < 0.01) and greater reductions in skin blood flow (NTN: -16 +/- 2%baseline vs. HTN: -28 +/
48 -noxious warming the skin did not affect the skin blood flow or itch intensity.
49                   Nicotine decreased resting skin blood flow (P < 0.05); this response was inhibited
50 cated on sensory nerves, would attenuate the skin blood flow response to local heating in humans.
51                                  The initial skin blood flow response to rapid local heating is an ax
52                                              Skin blood flow responses to graded intradermal microdia
53         The mechanisms involved in the human skin blood flow responses to iontophoretic application o
54 l fluid that would be related with a rise in skin blood flow (SkBF) and temperature sensation.
55 ed with intradermal microdialysis to measure skin blood flow (SkBF) during graded ET-A (BQ-123) and E
56  72-week-old male mice underwent analysis of skin blood flow (SkBF) via laser Doppler in response to
57                                              Skin blood flow (SkBF) was continuously monitored by las
58                                              Skin blood flow (SkBF) was monitored by laser-Doppler fl
59           Nicotine affects the regulation of skin blood flow (SkBF), but the mechanisms involved are
60                      Esophageal temperature, skin blood flow, sweat rate, and perceived thermal sensa
61 -Doppler flowmetry, and CVC was the ratio of skin blood flow to mean arterial pressure.
62                      Mean arterial pressure, skin blood flow via laser-Doppler flowmetry and core tem
63 es C for 20-30 min until a stable plateau in skin blood flow was achieved.
64 in at 4 microl min(-1) in sites 3 and 4, and skin blood flow was allowed to return to baseline (appro
65                   The subsequent increase in skin blood flow was allowed to return to baseline (appro
66                                              Skin blood flow was also monitored over adjacent sites t
67                                      Forearm skin blood flow was measured by laser-Doppler flowmetry,
68                                  An index of skin blood flow was measured directly over each microdia
69                                              Skin blood flow was measured while local skin heating (4
70 al heating to 42 degrees C was performed and skin blood flow was measured with laser Doppler flowmetr
71 1 older (61-77 years) men and women, forearm skin blood flow was monitored at three sites using laser
72 s) and 11 older (62-76 years) men and women, skin blood flow was monitored at two forearm sites with
73                                              Skin blood flow was monitored by laser-Doppler flowmetry
74                                              Skin blood flow was monitored during antidromic stimulat
75 bstance P was then delivered to the skin and skin blood flow was monitored for 45-60 min.
76                                 In addition, skin blood flow was monitored over an area of forearm sk
77               In supine individuals, forearm skin blood flow was monitored via laser-Doppler flowmetr
78               The techniques used to monitor skin blood flow were laser Doppler perfusion imaging and
79                     Baseline measurements of skin blood flow were obtained on the flexor aspect of th
80 ance P produced a dose-dependent increase in skin blood flow with the concentrations of substance P t
81                                IPA increases skin blood flow without erythema and PG increased erythe

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。