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1 ents developed cancer (excluding nonmelanoma skin cancer).
2 hnique for non-Hodgkin lymphoma and melanoma skin cancer.
3 rmatology clinic underwent biopsy to exclude skin cancer.
4 the sun's UV radiation is a leading cause of skin cancer.
5 ard, kappa, 0.41-0.63), for the diagnosis of skin cancer.
6 to most effectively predict the incidence of skin cancer.
7 for solar ultraviolet (UV) radiation-induced skin cancer.
8 Melanoma is the most aggressive type of skin cancer.
9 rcinoma (cSCC) is the most common metastatic skin cancer.
10 physical appearance and risk of keratinocyte skin cancer.
11 rare and aggressive, yet highly immunogenic skin cancer.
12 lation of mutations and an increased risk of skin cancer.
13 is associated with the risks of cataract and skin cancer.
14 cataract in left eyes and left-sided facial skin cancer.
15 erkel cell carcinoma (MCC), a lethal form of skin cancer.
16 ous history of cancer other than nonmelanoma skin cancer.
17 lieved that people with dark skin cannot get skin cancer.
18 and have a very high incidence of UV-induced skin cancer.
19 splant recipients have a higher incidence of skin cancer.
20 in those considered to be at higher risk for skin cancer.
21 biquitous carcinogen in sunlight that causes skin cancer.
22 ]) wanted to learn more about how to prevent skin cancer.
23 for the chemoprevention and therapy against skin cancer.
24 l proportion can progress into squamous cell skin cancer.
25 cipients (OTRs) are at an increased risk for skin cancer.
26 Melanoma is a lethal form of skin cancer.
27 ded by AICDA) links chronic inflammation and skin cancer.
28 lts aged 18 to 60 years without a history of skin cancer.
29 ous HSCT recipients had no increased risk of skin cancer.
30 ase in incidence of melanoma and nonmelanoma skin cancer.
31 ad to strategies for preventing and treating skin cancer.
32 lts aged 18 to 60 years without a history of skin cancer.
33 hat tomato consumption would protect against skin cancer.
34 NER protects against skin cancer.
35 Cutaneous melanoma (CM) is the most lethal skin cancer.
36 presents the identification of the deadliest skin cancer.
37 are highly expressed in human patients with skin cancer.
38 melanoma (MM) is the most aggressive form of skin cancer.
39 are considered potential early precursors of skin cancer.
40 ase in incidence of melanoma and nonmelanoma skin cancer.
41 g adulthood, increase the risk of developing skin cancer.
42 ntial therapeutic targets in this metastatic skin cancer.
43 and sunburn and thus prevent future cases of skin cancer.
44 233 benign meningiomas, and 1856 nonmelanoma skin cancers.
45 improving the chemotherapeutic efficiency of skin cancers.
46 t malignancies, meningiomas, and nonmelanoma skin cancers.
47 nt lesions will permit us to prevent or cure skin cancers.
48 y of the biodegradable nanogels for treating skin cancers.
49 dependent manner and was detectable in human skin cancers.
50 mination yielded a higher absolute number of skin cancers.
51 ncer, which has the highest mortality of all skin cancers.
52 Malignant melanoma is the deadliest of skin cancers.
53 idence, we estimated 5- and 10-year risks of skin cancers.
54 ant cancers, of which 115 cases (88.5%) were skin cancers.
55 gher risk of all-site, urothelial, lung, and skin cancers.
56 bedrock and tool of choice for the study of skin cancers.
57 s for prevention or treatment of UVR-induced skin cancers.
58 hair development, hair growth disorders, and skin cancers.
59 gy in clinical practice for the diagnosis of skin cancer?
61 otal, 1982 individuals were screened, and 47 skin cancers (2.4%) were histologically confirmed, inclu
62 ] vs 19 [6.1%] answered yes), and history of skin cancer (76 [33.3%] vs 15 [4.8%] answered yes) (all
63 Among those reporting no prior history of skin cancer, a similar model with 10 factors predicted k
64 ll carcinoma (cuSCC) comprises 15-20% of all skin cancers, accounting for over 700,000 cases in USA a
65 ortance: Keratinocyte carcinoma (nonmelanoma skin cancer) accounts for substantial burden in terms of
67 cell polyomavirus (MCV) causes an aggressive skin cancer after prolonged infection and requires an ac
69 of organ transplanted, time to diagnosis of skin cancer after transplantation, and history of condyl
70 nts in the genesis of many cancers including skin cancer and are often implicated in tumor progressio
71 an ultraviolet radiation (UV)-induced human skin cancer and from a mouse model of urethane-induced c
74 lear for all main cancers except nonmelanoma skin cancer and was stronger for cancers of poorer progn
76 .2%; 4 of 27 patients [14.8%] diagnosed with skin cancers and 5 of 11 patients [45.5%] diagnosed with
83 cer (subhazard ratio, 2.1; 95% CI, 1.2-3.7), skin cancer as the index posttransplant cancer (subhazar
85 s the deadliest form of commonly encountered skin cancer because of its rapid progression towards met
87 evelopment of prostate cancer, colon cancer, skin cancer, breast cancer, lung cancer and pancreas can
88 d lung; malignant skin melanoma; nonmelanoma skin cancer; breast; cervical; uterine; ovarian; prostat
89 Surgeon General's Call to Action to Prevent Skin Cancer broadly identified research gaps, but specif
90 nt recipients carry a substantial measurable skin cancer burden at any given time and require frequen
92 Organ transplant recipients with the highest skin cancer burden were Australian born, were fair skinn
94 well established than those for other common skin cancers, but recent evidence has highlighted a pote
95 on may increase the risk of solar UV-induced skin cancer by promoting photochemical damage to the NER
96 yV10) among controls from a population-based skin cancer case-control study (n = 460) conducted in Ne
99 roven melanomas in 134 patients treated in 9 skin cancer centers in Spain, France, Italy, and Austria
103 vioral research addressing all points of the skin cancer control continuum, measuring interventions t
104 eceiving treatment of a condition other than skin cancer (controls) at the dermatology clinics at the
105 the pump-probe signals from melanin in human skin cancers correlate well with clinical concern, but i
106 ystem, we found that brain, lung, colon, and skin cancers could be detected in situ during surgery wi
108 oscopy (RCM) improves diagnostic accuracy in skin cancer detection when combined with dermoscopy; how
112 inflammation-induced AID expression promotes skin cancer development independently of UV damage and s
114 ons: (1) How accurate is teledermatology for skin cancer diagnosis compared with usual care (face-to-
117 s: The influence of the screening program on skin cancer epidemiological findings and the cost per qu
118 The influence of the screening program on skin cancer epidemiological findings and the cost per qu
122 rs with the strongest effects were >20 prior skin cancers excised (odds ratio 8.57, 95% confidence in
123 who at baseline reported no past history of skin cancer excisions and no more than five destructivel
124 ed information on skin, hair, and eye color; skin cancer family history; and sun exposure history, su
125 and 1.71 (95% CI, 0.88-3.33) for nonmelanoma skin cancers for survivors with reference characteristic
127 These same risk factors were associated with skin cancer formation when the analysis was limited to n
130 The number of excisions needed to detect 1 skin cancer from clinical visual skin examinations varie
133 eases in the United States, the incidence of skin cancer has important public health consequences, in
136 oma, and lung, pancreatic, and nonepithelial skin cancers (higher during function intervals), and kid
137 in patients with and without a pretransplant skin cancer history was 31.6% and 7.4%, respectively (P
139 ractices that further increase their risk of skin cancer; however, gaps in the literature exist in yo
140 represent the vast majority of all cases of skin cancer; however, they rarely result in death or sub
141 ransplant skin cancer included pretransplant skin cancer (HR, 4.69; 95% CI, 3.26-6.73), male sex (HR,
145 lid-organ transplant recipients, the risk of skin cancer in hematopoietic stem-cell transplant (HSCT)
147 n the use of sirolimus for the prevention of skin cancer in nonrenal OTRs or those already diagnosed
148 ic and clinical factors and the incidence of skin cancer in nonwhite organ transplant recipients.
151 s research has reported an increased risk of skin cancer in solid organ transplant recipients (OTRs),
152 ecipients with histopathologically confirmed skin cancer in the 3-month baseline period was estimated
155 k of squamous cell carcinoma (SCC) and other skin cancers in organ transplant recipients (OTRs), but
156 keratinocyte cancers (KCs) overall and other skin cancers in relation to azathioprine treatment.
159 significant risk factors for posttransplant skin cancer included pretransplant skin cancer (HR, 4.69
161 evaluate the risk factors for posttransplant skin cancer, including squamous cell carcinoma (SCC), me
165 he risk factors and trends in posttransplant skin cancer is fundamental to targeted screening and pre
174 of sun/UV exposure-related illness, such as skin cancer, is seriously concerning public health autho
175 -induced DNA damage, a major risk factor for skin cancers, is primarily repaired by nucleotide excisi
177 Melanoma, one of the deadliest forms of skin cancer, kills nearly 10,000 people in the United St
178 onship between sunburn and risk of different skin cancers (melanoma, squamous cell carcinoma (SCC), a
180 clinical visual skin examination in reducing skin cancer morbidity and mortality and death from any c
184 s across the United States in the Transplant Skin Cancer Network during 1 of 2 calendar years (either
185 ffects of UV-B radiation against nonmelanoma skin cancer (NMSC) are exerted via signaling mechanisms
186 reviewed the DSCMs for residual nonmelanoma skin cancer (NMSC) before and after a brief training ses
187 atients with a periocular region nonmelanoma skin cancer (NMSC) or a nonperiocular NMSC causing a com
188 or any incident cancer excluding nonmelanoma skin cancer (NMSC) were 1.06 (95% CI, 1.02-1.09), 1.06 (
189 k than HIV-uninfected persons of nonmelanoma skin cancer (NMSC), defined as basal cell carcinoma (BCC
190 ing Mohs micrographic surgery of nonmelanoma skin cancer (NMSC), inflammation in histologic frozen se
191 suppression is a risk factor for nonmelanoma skin cancer (NMSC), particularly squamous cell tumors.
192 billing codes and categorized as nonmelanoma skin cancer (NMSC), viral-linked and "other" cancers.
194 ss all three trials, adjudicated nonmelanoma skin cancer occurred in five patients who received tofac
195 V radiation (UVR)-induced skin pigmentation, skin cancers, ocular surface disease, and, in some patie
196 referrals overall and those for presumptive skin cancer or actinic keratoses, skin biopsies, or PCP
198 in biopsies, and PCP diagnostic accuracy for skin cancer or precancer compared with dermatologist dia
200 t public health consequences, including poor skin cancer outcomes, in part because of late-stage diag
201 Secondary endpoints included the course of skin cancers over 3 periods (first transplantation, retu
203 skin diseases, hyperpigmentation, psoriasis, skin cancer, pachyonychia congenital) caused by aberrant
204 To study temporal trends for the risk of skin cancer, particularly SCC, after organ transplantati
205 liquid biopsies from brain, breast, lung and skin cancer patients were classified in 2.4 cumulative s
208 ence interval = [1.07, 2.43]), reported past skin cancer (prevalence ratio =3.39, 95% confidence inte
209 e behavior that can help reduce the risk for skin cancer, prevent sunburns, mitigate photoaging, and
212 r tanning frequency and behaviors related to skin cancer prevention and to investigate whether these
213 r tanning frequency and behaviors related to skin cancer prevention and to investigate whether these
214 importance of sun protection and facilitate skin cancer prevention and, therefore, decrease the skin
216 lusions and Relevance: Important barriers to skin cancer prevention were lack of knowledge, the belie
220 against loss of bone mass, chronic diseases, skin cancer, prostate cancer, and cardiovascular disease
221 ted with an increased risk of posttransplant skin cancer, PTLD, solid organ cancer, death and graft f
223 en compared with the number of biopsy-proven skin cancers recorded over a similar period before the f
225 l Institute of Health (NIH) grants targeting skin cancer-related behaviors and relevant outcomes.
226 ant applications from 2000 to 2014 targeting skin cancer-related behaviors or testing behavioral inte
227 morbidities and mortalities associated with skin cancers requires sustained research with the goal o
228 s Follow-up Study (1992-2010) to investigate skin cancer risk associated with history of severe sunbu
230 , Spanish, or Haitian Creole assessing their skin cancer risk perception, knowledge, sun protective b
235 ing the approach to the analysis of over 200 skin cancer samples, we demonstrate its utility for disc
237 mplementation of a campaign promoting annual skin cancer screening by FBSE, including training of PCP
238 oma deaths in a region with population-based skin cancer screening compared with no change or slight
239 is critical to emphasize sun protection and skin cancer screening in individuals who tan indoors.
242 the cost-effectiveness of 2 population-based skin cancer screening methods and to assess their budget
243 the cost-effectiveness of 2 population-based skin cancer screening methods and to assess their budget
246 are payer perspective) of 2 population-based skin cancer screening programs in Belgium compared with
247 are payer perspective) of 2 population-based skin cancer screening programs in Belgium compared with
250 Only limited evidence was identified for skin cancer screening, particularly regarding potential
254 2.60; 95% CI, 2.27-2.98), and posttransplant skin cancer (SHR, 2.92; 95% CI, 2.52-3.39), PTLD (SHR, 1
255 the highest and moderate risk of developing skin cancer (skin types I, II, III, and IV) than in skin
256 osttransplant malignancy was classified into skin cancer, solid tumor, and posttransplant lymphoproli
257 nscriptional repressor, is down-modulated in skin cancer stromal cells, and Atf3 knockout mice develo
258 nsplant cancer were history of pretransplant skin cancer (subhazard ratio, 2.1; 95% CI, 1.2-3.7), ski
260 6 outcomes (mean difference of 0.5 or more): skin cancer, surgical complications, cognition, blood pr
265 Although highly expressed in normal skin and skin cancer, the role of the atypical E2Fs, E2F7 and E2F
267 tial as a novel target for the prevention of skin cancer through its role in the regulation of STAT3
268 f video-mosaics of melanoma and non-melanoma skin cancers, to demonstrate potential clinical utility.
269 but little is known about the feasibility of skin cancer training and clinical skin examination (CSE)
270 This pilot study suggests that PCP-based skin cancer training and screening are feasible and have
272 city, Fitzpatrick type, type and location of skin cancer, type of organ transplanted, time to diagnos
273 ed based on age, sex, history of nonmelanoma skin cancer, US geographic region, and population densit
274 igand status is associated with keratinocyte skin cancers using a population-based study of basal cel
281 incidence of spontaneous tumors, especially skin cancer, was observed in adult BMT-rescued DNA-PKcs(
282 nalysis, age, sex, and previous diagnosis of skin cancer were not significantly associated with the p
283 Potential risk factors for posttransplant skin cancer were tested using multivariate Cox regressio
286 ce of all-type cancer (excluding nonmelanoma skin cancers), which was evaluated using Kaplan-Meier su
287 noma constitutes the most aggressive form of skin cancer, which further metastasizes into a deadly fo
289 or head and neck, genitalia, hands, and feet skin cancers, which may represent an additional financia
290 imers (CPDs) are DNA photoproducts linked to skin cancer, whose mutagenicity depends in part on their
291 idence on the effectiveness of screening for skin cancer with a clinical visual skin examination in r
292 e to assess the net benefit of screening for skin cancer with a clinical visual skin examination is l
293 tificial intelligence capable of classifying skin cancer with a level of competence comparable to der
295 Merkel cell carcinoma is a rare, aggressive skin cancer with poor prognosis in patients with advance
296 (MCC) is a highly aggressive neuroendocrine skin cancer with profound but poorly understood resistan
298 rapy, melanoma remains the deadliest form of skin cancer, with a 5-year survival rate of only 15%.
299 e counseled more effectively on the signs of skin cancer, with focused discussion points contingent o
300 er had a lower risk of developing subsequent skin cancer, with no increased risk for overall mortalit
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