ライフサイエンスコーパス: skin carcinoma

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1 n form of skin damage and is associated with skin carcinoma.

2 oss of Pten are mutually exclusive events in skin carcinomas.

3 ic mice induces the formation of spontaneous skin carcinomas.

4 omas or p53 deficiency to induce spontaneous skin carcinomas.

5 factor for the increasing incidence of human skin carcinomas.

6 dherin that is aberrantly expressed in human skin carcinomas.

7 er, including highly prevalent UV-associated skin carcinomas.

8 various agents such as retinoic acid, and in skin carcinomas.

9 Similar effects were observed in human skin carcinoma A431 cells expressing mutant p53.

10 non-small cell lung carcinoma, squamous cell skin carcinoma and lymphoma.

11 Because PACE4 is overexpressed in skin carcinomas and in vitro overexpression of PACE4 res

12 n the epidermis inhibited the development of skin carcinomas and metastases in mice.

13 ss reported for the treatment of superficial skin carcinomas by photodynamic therapy with topical app

14 The excision of Rce1 from a Rce1(flox/flox) skin carcinoma cell line also significantly retarded the

15 to the overexpression of COX-2 in the mouse skin carcinoma cell line JWF2, we investigated cis- and

16 e model of UVB-induced skin cancer and human skin carcinoma cells and show that the mutant p53 confor

17 in-associated NKG2d+ gammadelta cells killed skin carcinoma cells by a mechanism that was sensitive t

18 reduced myofibroblast differentiation of E4 skin carcinoma cells in a tumor allograft.

19 also found that adherent TRAIL-R-expressing skin carcinoma cells were TRAIL resistant in vitro but w

20 ted eicosanoid synthesis that is observed in skin carcinomas contributes to the growth and progressio

21 creased susceptibility to chemically induced skin carcinoma formation, whereas the number of papillom

22 gesting that this pathway is dispensable for skin carcinoma formation.

23 Instead of skin carcinomas, however, the chemical treatment protoco

24 The frequency of squamous cell skin carcinoma in organ transplant patients is around 10

25 Chemically induced skin carcinomas in mice are a paradigm for epithelial ne

26 EV-HPVs cause warts and increase the risk of skin carcinomas in otherwise healthy individuals.

27 About 20%-30% of Hras mutant mouse skin carcinomas induced by chemical initiation/promotion

28 Development of UVB-induced skin carcinoma is a multistep and complex process.

29 However, skin carcinoma numbers were very similar, suggesting tha

30 arding the incidence trends of squamous cell skin carcinoma (SCC) and rare skin tumors, including Mer

31 s or nullizygous for p53 develop spontaneous skin carcinomas, which normally are rare in p53-deficien

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