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1 ays, 2 new employees presented with the same skin changes.
2 opathy, monoclonal plasma cell neoplasm, and skin changes.
3 aly, endocrinopathy, monoclonal protein, and skin changes.
4    Initially 16 employees developed pruritic skin changes.
5 rm mass in the left flank with no associated skin changes.
6 eripheral edema, ascites, or effusions), and skin changes.
7 no significant weight loss, diarrhea or GVHD skin changes.
8  facial feature, muscles tone abnormalities, skin changes and breathing difficulties.
9 e relatively rapid and steady improvement of skin changes and knee joint contractures in patients wit
10 ars of Rag1-/- mice, IL-23 initially induced skin changes and levels of Il22 mRNA that were indisting
11 as observed between the intensity of trophic skin changes and the pulsatility index.
12 isual acuity, clinically observable tattooed skin changes, and biopsy results.
13 re performed in most individuals, noticeable skin changes are observed approximately 1 month after a
14 were minimal; there were small or no adverse skin changes at the maximum effective myotoxic doses.
15                                              Skin changes caused by ionizing radiation have been scie
16 isorder (PCD), organomegaly, endocrinopathy, skin changes, edema, sclerotic bone lesions, and thrombo
17             Most of these hyperproliferative skin changes improve when a MEK inhibitor is co-administ
18            To analyze the causative agent of skin changes in employees of a company that produced her
19  administration of benzoyl peroxide produces skin changes in the hairless mouse that qualitatively re
20 ce overexpressing ornithine decarboxylase in skin, changes in tissue polyamine levels, particularly p
21 od monocytes reduces DC accumulation and the skin changes induced either by injecting IL-23 or by app
22 support the hypothesis that inflammation and skin changes involving activin A cause some sensory neur
23 galy, Endocrinopathy, Monoclonal protein and Skin changes) is a paraneoplastic disorder with a 'demye
24 ltiple endocrinopathies, monoclonal protein, skin changes) is a rare disease associated with a plasma
25 s with anti-Th/To antibodies had more subtle skin changes, less severe vascular involvement, and less
26 distinguish those patients with the hallmark skin changes of DM and no clinical evidence of muscle di
27 pus erythematosus, dermatomyositis), and the skin changes of potentially fatal disorders such as graf
28 edis artery decreased with increased trophic skin changes (r=-0.431, P=0.009).
29                                      Because skin changes recurred after discontinuation of imatinib
30 hin weeks of stopping imatinib mesylate, the skin changes recurred in each patient.
31 organomegaly, endocrinopathy, M protein, and skin changes) remains poorly understood.
32                                          The skin changes resolved in all patients, with a faster res
33 ulation by autoantibodies contributes to the skin changes seen in autoimmune thyroid disease.
34 o rejection, manifesting pathologically with skin changes that form the basis of the Banff 2007 class
35 logy from other inflammatory myopathies with skin changes that have prominent perimysial connective t
36                        This review discusses skin changes that may suggest a psychological problem an
37 ciated with only mild side effects including skin changes, transient headache, hyperlipidemia, transa
38 itch signals to the CNS and for the dramatic skin changes triggered by dry-skin-evoked itch and scrat
39 milar to pseudoxanthoma elasticum (PXE), the skin changes were found to be due to amyloid deposition
40 to those already existing in the literature, skin changes were more marked in animals exposed to gado
41 her and aunt, also manifesting with PXE-like skin changes, were heterozygous carriers of a missense m
42 tant mice were protected from psoriasis-like skin changes, which identified a role for Sox13-dependen

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